The Experts below are selected from a list of 45126 Experts worldwide ranked by ideXlab platform
John M Boyce - One of the best experts on this subject based on the ideXlab platform.
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nosocomial antibiotic associated Diarrhea associated with enterotoxin producing strains of methicillin resistant staphylococcus aureus
The American Journal of Gastroenterology, 2005Co-Authors: John M Boyce, Nancy L HavillAbstract:Nosocomial Antibiotic-Associated Diarrhea Associated with Enterotoxin-Producing Strains of Methicillin-Resistant Staphylococcus Aureus
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Nosocomial antibiotic—associated Diarrhea caused by enterotoxin-producing methicillin-resistant staphylococcus aureus, an unrecognized disease
The American Journal of Gastroenterology, 2003Co-Authors: Mehrnaz Hojjati, Nancy L Havill, Mehdi M Kebria, Ronald J. Vender, John M BoyceAbstract:Nosocomial antibiotic—associated Diarrhea caused by enterotoxin-producing methicillin-resistant staphylococcus aureus, an unrecognized disease
Nancy L Havill - One of the best experts on this subject based on the ideXlab platform.
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nosocomial antibiotic associated Diarrhea associated with enterotoxin producing strains of methicillin resistant staphylococcus aureus
The American Journal of Gastroenterology, 2005Co-Authors: John M Boyce, Nancy L HavillAbstract:Nosocomial Antibiotic-Associated Diarrhea Associated with Enterotoxin-Producing Strains of Methicillin-Resistant Staphylococcus Aureus
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Nosocomial antibiotic—associated Diarrhea caused by enterotoxin-producing methicillin-resistant staphylococcus aureus, an unrecognized disease
The American Journal of Gastroenterology, 2003Co-Authors: Mehrnaz Hojjati, Nancy L Havill, Mehdi M Kebria, Ronald J. Vender, John M BoyceAbstract:Nosocomial antibiotic—associated Diarrhea caused by enterotoxin-producing methicillin-resistant staphylococcus aureus, an unrecognized disease
Per Brobech Mortensen - One of the best experts on this subject based on the ideXlab platform.
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colonic fermentation to short chain fatty acids is decreased in antibiotic associated Diarrhea
Gastroenterology, 1991Co-Authors: Mette Rye Clausen, Helen Bonnen, Michael Tvede, Per Brobech MortensenAbstract:Abstract To elucidate the pathogenesis of Antibiotic-Associated Diarrhea, colonic carbohydrate fermentation was investigated in three groups of subjects—a group of controls, a group of patients with Antibiotic-Associated Diarrhea, and a group of patients receiving antibiotic therapy without Diarrhea. Compared with controls, the colonic fermentation was markedly impaired in patients with Antibiotic-Associated Diarrhea reflected by both very low concentrations (22.1 vs. 59.5 mmol/L; P P
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Colonic fermentation to short-chain fatty acids is decreased in Antibiotic-Associated Diarrhea
Gastroenterology, 1991Co-Authors: Mette Rye Clausen, Helen Bonnen, Michael Tvede, Per Brobech MortensenAbstract:To elucidate the pathogenesis of Antibiotic-Associated Diarrhea, colonic carbohydrate fermentation was investigated in three groups of subjects--a group of controls, a group of patients with Antibiotic-Associated Diarrhea, and a group of patients receiving antibiotic therapy without Diarrhea. Compared with controls, the colonic fermentation was markedly impaired in patients with Antibiotic-Associated Diarrhea reflected by both very low concentrations (22.1 vs. 59.5 mmol/L; P less than 0.01) and production rates of short-chain fatty acids. In the group of patients without Diarrhea, the effect on the colonic fermentation was dependent on the antibiotic administered. Penicillin and pivampicillin PO did not reduce the concentrations (69.9 and 66.7 mmol/L, respectively) or production rates. Dicloxacillin, erythromycin, and combined IV treatment with ampicillin, netilmicin, and metronidazole reduced both concentrations (27.1, 38.2, and 18.8 mmol/L; P less than 0.01) and production rates of short-chain fatty acids to levels seen in patients with Diarrhea. L-Lactate and D-lactate concentrations were normal in all patients (less than 5 mmol/L), but lactate production was reduced in the patients who had reduced production of short-chain fatty acids, including patients with Diarrhea. Thus, Antibiotic-Associated Diarrhea was always related to reduced fecal concentrations and production rates of short-chain fatty acids and production rates of lactate. These results suggest that the Antibiotic-Associated Diarrhea might be secondary to impaired colonic fermentation in otherwise disposed subjects, resulting in accumulation of luminal carbohydrate and/or decreased short-chain fatty acid-stimulated sodium and water absorption.
Antonio Gasbarrini - One of the best experts on this subject based on the ideXlab platform.
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saccharomyces boulardii and antibiotic associated Diarrhea effectiveness of prophylactic use
The American Journal of Gastroenterology, 2012Co-Authors: Maria Assunta Zocco, Matteo Garcovich, Antonio GasbarriniAbstract:To the Editor: We read with great interest the article by Pozzoni et al. (1) about the effectiveness of Saccharomyces boulardii (S. boulardii) in the prevention of Antibiotic-Associated Diarrhea (AAD) in adults. This is a carefully designed clinical trial, which was conducted with good methodology and low risk of bias. We agree with the authors that AAD prevention is an important topic, especially in hospitalized patients, since it increases costs and hospital length of stay. However, the results of the trial should be interpreted within the context of some limitations.
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Saccharomyces boulardii and Antibiotic-Associated Diarrhea: effectiveness of prophylactic use.
The American journal of gastroenterology, 2012Co-Authors: Maria Assunta Zocco, Matteo Garcovich, Antonio GasbarriniAbstract:To the Editor: We read with great interest the article by Pozzoni et al. (1) about the effectiveness of Saccharomyces boulardii (S. boulardii) in the prevention of Antibiotic-Associated Diarrhea (AAD) in adults
Mette Rye Clausen - One of the best experts on this subject based on the ideXlab platform.
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colonic fermentation to short chain fatty acids is decreased in antibiotic associated Diarrhea
Gastroenterology, 1991Co-Authors: Mette Rye Clausen, Helen Bonnen, Michael Tvede, Per Brobech MortensenAbstract:Abstract To elucidate the pathogenesis of Antibiotic-Associated Diarrhea, colonic carbohydrate fermentation was investigated in three groups of subjects—a group of controls, a group of patients with Antibiotic-Associated Diarrhea, and a group of patients receiving antibiotic therapy without Diarrhea. Compared with controls, the colonic fermentation was markedly impaired in patients with Antibiotic-Associated Diarrhea reflected by both very low concentrations (22.1 vs. 59.5 mmol/L; P P
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Colonic fermentation to short-chain fatty acids is decreased in Antibiotic-Associated Diarrhea
Gastroenterology, 1991Co-Authors: Mette Rye Clausen, Helen Bonnen, Michael Tvede, Per Brobech MortensenAbstract:To elucidate the pathogenesis of Antibiotic-Associated Diarrhea, colonic carbohydrate fermentation was investigated in three groups of subjects--a group of controls, a group of patients with Antibiotic-Associated Diarrhea, and a group of patients receiving antibiotic therapy without Diarrhea. Compared with controls, the colonic fermentation was markedly impaired in patients with Antibiotic-Associated Diarrhea reflected by both very low concentrations (22.1 vs. 59.5 mmol/L; P less than 0.01) and production rates of short-chain fatty acids. In the group of patients without Diarrhea, the effect on the colonic fermentation was dependent on the antibiotic administered. Penicillin and pivampicillin PO did not reduce the concentrations (69.9 and 66.7 mmol/L, respectively) or production rates. Dicloxacillin, erythromycin, and combined IV treatment with ampicillin, netilmicin, and metronidazole reduced both concentrations (27.1, 38.2, and 18.8 mmol/L; P less than 0.01) and production rates of short-chain fatty acids to levels seen in patients with Diarrhea. L-Lactate and D-lactate concentrations were normal in all patients (less than 5 mmol/L), but lactate production was reduced in the patients who had reduced production of short-chain fatty acids, including patients with Diarrhea. Thus, Antibiotic-Associated Diarrhea was always related to reduced fecal concentrations and production rates of short-chain fatty acids and production rates of lactate. These results suggest that the Antibiotic-Associated Diarrhea might be secondary to impaired colonic fermentation in otherwise disposed subjects, resulting in accumulation of luminal carbohydrate and/or decreased short-chain fatty acid-stimulated sodium and water absorption.