The Experts below are selected from a list of 303 Experts worldwide ranked by ideXlab platform
Elena Kokoliou - One of the best experts on this subject based on the ideXlab platform.
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Technology & Behavior
internal, 2020Co-Authors: Elena KokoliouAbstract:the key physiological attributes/arousal indicators (i.e. heart rate variability) and behavioral patterns (i.e. shaving strokes, facial expressions) when
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AI Knowledge base
internal, 2017Co-Authors: Elena KokoliouAbstract:physiological responses and behavioral patterns related to shaving experience and shaving performance. Identify the key physiological attributes/arousal indicators (i.e … – humanoid Robot system. Picard, R. W., Fedor, S. & Ayzenberg, Y. (2015). Multiple Arousal Theory and Daily Life Electrodermal Activity Asymmetry. Emotion
Loukas Martinou - One of the best experts on this subject based on the ideXlab platform.
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Shaving Biometrics
internal, 2017Co-Authors: Loukas MartinouAbstract:of sweat generated by the pores on skin. GSR has long been used to measure physiological arousal. Blood Volume Pulse (BVP). Measures cardiovascular dynamics
David M Rapoport - One of the best experts on this subject based on the ideXlab platform.
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Non-Invasive detection of respiratory effort-related Arousals (REras) by a nasal cannula/pressure transducer system.
Sleep, 2000Co-Authors: Indu Ayappa, Ana C Krieger, Alison Rosen, Rebecca L. O'malley, Robert G. Norman, David M RapoportAbstract:STUDY OBJECTIVES: The published AASM guidelines approve use of a nasal cannula/pressure transducer to detect apneas/hypopneas, but require esophageal manometry for Respiratory Effort-Related Arousals (RERAs). However, esophageal manometry may be poorly tolerated by many subjects. We have shown that the shape of the inspiratory flow signal from a nasal cannula identifies flow limitation and elevated upper-airway resistance. This study tests the hypothesis that detection of flow limitation events using the nasal cannula provides a non-invasive means to identify RERAs. DESIGN: N/A. SETTING: N/A. PATIENTS: 10 UARS/OSAS and 5 normal subjects INTERVENTIONS: N/A. MEASUREMENTS AND RESULTS: All subjects underwent full NPSG. Two scorers identified events from the nasal cannula signal as apneas, hypopneas, and flow limitation events. Two additional scorers identified events from esophageal manometry. Arousals were scored in a separate pass. Interscorer reliability and intersignal agreement were assessed both without and with regard to Arousal. The total number of respiratory events identified by the two scorers of the nasal cannula was similar with an Intraclass Correlation (ICC) =0.96, and was essentially identical to the agreement for the two scorers of esophageal manometry (ICC=0.96). There was good agreement between the number of events detected by the two techniques with a slight bias towards the nasal cannula (4.5 events/hr). There was no statistically significant difference (bias 0.9/hr, 95%CI -0.3-2.0) between the number of nasal cannula flow limitation events terminated by Arousal and manometry events terminated by Arousal (RERAs). CONCLUSION: The nasal cannula/pressure transducer provides a non-invasive reproducible detector of all events in sleep disordered breathing; in particular, it detects the same events as esophageal manometry (RERAs).
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non invasive detection of respiratory effort related Arousals reras by a nasal cannula pressure transducer system
Sleep, 2000Co-Authors: Indu Ayappa, Ana C Krieger, Alison Rosen, Rebecca L Omalley, Robert G. Norman, David M RapoportAbstract:STUDY OBJECTIVES: The published AASM guidelines approve use of a nasal cannula/pressure transducer to detect apneas/hypopneas, but require esophageal manometry for Respiratory Effort-Related Arousals (RERAs). However, esophageal manometry may be poorly tolerated by many subjects. We have shown that the shape of the inspiratory flow signal from a nasal cannula identifies flow limitation and elevated upper-airway resistance. This study tests the hypothesis that detection of flow limitation events using the nasal cannula provides a non-invasive means to identify RERAs. DESIGN: N/A. SETTING: N/A. PATIENTS: 10 UARS/OSAS and 5 normal subjects INTERVENTIONS: N/A. MEASUREMENTS AND RESULTS: All subjects underwent full NPSG. Two scorers identified events from the nasal cannula signal as apneas, hypopneas, and flow limitation events. Two additional scorers identified events from esophageal manometry. Arousals were scored in a separate pass. Interscorer reliability and intersignal agreement were assessed both without and with regard to Arousal. The total number of respiratory events identified by the two scorers of the nasal cannula was similar with an Intraclass Correlation (ICC) =0.96, and was essentially identical to the agreement for the two scorers of esophageal manometry (ICC=0.96). There was good agreement between the number of events detected by the two techniques with a slight bias towards the nasal cannula (4.5 events/hr). There was no statistically significant difference (bias 0.9/hr, 95%CI -0.3-2.0) between the number of nasal cannula flow limitation events terminated by Arousal and manometry events terminated by Arousal (RERAs). CONCLUSION: The nasal cannula/pressure transducer provides a non-invasive reproducible detector of all events in sleep disordered breathing; in particular, it detects the same events as esophageal manometry (RERAs).
Michael I Polkey - One of the best experts on this subject based on the ideXlab platform.
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Neural Respiratory Drive and Arousal in Patients with Obstructive Sleep Apnea Hypopnea.
Sleep, 2015Co-Authors: Sichang Xiao, Baiting He, Joerg Steier, John Moxham, Michael I PolkeyAbstract:STUDY OBJECTIVES: It has been hypothesized that Arousals after apnea and hypopnea events in patients with obstructive sleep apnea are triggered when neural respiratory drive exceeds a certain level, but this hypothesis is based on esophageal pressure data, which are dependent on flow and lung volume. We aimed to determine whether a fixed threshold of respiratory drive is responsible for Arousal at the termination of apnea and hypopnea using a flow independent technique (esophageal diaphragm electromyography, EMGdi) in patients with obstructive sleep apnea. SETTING: Sleep center of state Key Laboratory of Respiratory Disease. PATIENTS: Seventeen subjects (two women, mean age 53 ± 11 years) with obstructive sleep apnea/hypopnea syndrome were studied. METHODS: We recorded esophageal pressure and EMGdi simultaneously during overnight full polysomnography in all the subjects. MEASUREMENTS AND RESULTS: A total of 709 hypopnea events and 986 apnea events were analyzed. There was wide variation in both esophageal pressure and EMGdi at the end of both apnea and hypopnea events within a subject and stage 2 sleep. The EMGdi at the end of events that terminated with Arousal was similar to those which terminated without Arousal for both hypopnea events (27.6% ± 13.9%max vs 29.9% ± 15.9%max, P = ns) and apnea events (22.9% ± 11.5%max vs 22.1% ± 12.6%max, P = ns). The Pes at the end of respiratory events terminated with Arousal was also similar to those terminated without Arousal. There was a small but significant difference in EMGdi at the end of respiratory events between hypopnea and apnea (25.3% ± 14.2%max vs 21.7% ± 13.2%max, P < 0.05]. CONCLUSIONS: Our data do not support the concept that there is threshold of neural respiratory drive that is responsible for Arousal in patients with obstructive sleep apnea.
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Neural respiratory drive and Arousal in patients with obstructive sleep apnea–hypopnea
European Respiratory Journal, 2014Co-Authors: Sichang Xiao, Ying-xin Wu, Baiting He, Joerg Steier, John Moxham, Michael I PolkeyAbstract:Background: Repetitive Arousals after apnea and hypopnea events in patients with obstructive sleep apnea have been hypothesized to be triggered by respiratory effort. Objectives: The purpose of the study is to determine whether the threshold of respiratory drive responsible for Arousal for apnea is the same as that for hypopnea. Methods: Seventeen subjects (two females and fifteen males, mean age 53±11 years) with obstructive sleep apnea and hypopnea were studied by recording diaphragm EMG and esophageal pressure during overnight full polysomnography. Results: A total of 709 hyponea events and 986 apnea events were analyzed. There was wide variation in Pes at the end of both apnea and hypopnea events and the maximal Pes could be four times higher than the minimal Pes at the end of apnea within a subject. 15.5%±14.2% of hypopnea events and 9.5%±11.6% of apnea events during stage 2 were terminated without Arousal. There was significant difference in EMGdi at the end of respiratory events between hypopnea and apnea [(25.3±14.2) % EMGmax. vs (21.7 ±13.2)%EMGmax, p 0.05)] and apnea events [(22.9±11.5) % EMGmax vs (22.1±12.6) % EMGmax, p>0.05]. Conclusions: our data do not support the concept that there is threshold of respiratory drive which responsible for Arousal to resume airflow.
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neural respiratory drive and Arousal in patients with obstructive sleep apnea hypopnea
European Respiratory Journal, 2014Co-Authors: Sichang Xiao, Joerg Steier, John Moxham, Michael I Polkey, Yuanming LuoAbstract:Background: Repetitive Arousals after apnea and hypopnea events in patients with obstructive sleep apnea have been hypothesized to be triggered by respiratory effort. Objectives: The purpose of the study is to determine whether the threshold of respiratory drive responsible for Arousal for apnea is the same as that for hypopnea. Methods: Seventeen subjects (two females and fifteen males, mean age 53±11 years) with obstructive sleep apnea and hypopnea were studied by recording diaphragm EMG and esophageal pressure during overnight full polysomnography. Results: A total of 709 hyponea events and 986 apnea events were analyzed. There was wide variation in Pes at the end of both apnea and hypopnea events and the maximal Pes could be four times higher than the minimal Pes at the end of apnea within a subject. 15.5%±14.2% of hypopnea events and 9.5%±11.6% of apnea events during stage 2 were terminated without Arousal. There was significant difference in EMGdi at the end of respiratory events between hypopnea and apnea [(25.3±14.2) % EMGmax. vs (21.7 ±13.2)%EMGmax, p 0.05)] and apnea events [(22.9±11.5) % EMGmax vs (22.1±12.6) % EMGmax, p>0.05]. Conclusions: our data do not support the concept that there is threshold of respiratory drive which responsible for Arousal to resume airflow.
Cindy M. Meston - One of the best experts on this subject based on the ideXlab platform.
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Understanding sexual Arousal and subjective–genital Arousal desynchrony in women
Nature Reviews Urology, 2019Co-Authors: Cindy M. Meston, Amelia M. StantonAbstract:Sexual Arousal in women comprises two components: genital Arousal and subjective Arousal. Genital Arousal is characterized by genital vasocongestion and other physiological changes that occur in response to sexual stimuli, whereas subjective Arousal refers to mental engagement during sexual activity. For some women, genital Arousal enhances subjective Arousal; for others, the two types of Arousal are desynchronous. However, the relationship between genital and subjective Arousal might not be relevant to the diagnosis and treatment of sexual Arousal dysfunction. Studies have shown that not all women who report sexual Arousal problems have decreased genital Arousal, and only some women with decreased genital Arousal have low subjective Arousal. To develop efficacious treatments for female sexual Arousal dysfunction, researchers need to differentiate the women for whom genital sensations have a critical role in their subjective Arousal from those who are not mentally aroused by genital cues. The mechanisms by which women become aroused and the inputs into Arousal have considerable implications for treatment outcomes. Sexual Arousal in women comprises two components: genital Arousal and subjective Arousal. For some women, genital Arousal enhances subjective Arousal; for others, the two types of Arousal are desynchronous. In this Review, Meston and Stanton describe the mechanisms and the relationship between genital and subjective Arousal and consider how they assist in diagnosis and treatment of sexual Arousal dysfunction and development of treatments for female sexual Arousal dysfunction.
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Understanding sexual Arousal and subjective–genital Arousal desynchrony in women
Nature reviews. Urology, 2019Co-Authors: Cindy M. Meston, Amelia M. StantonAbstract:Sexual Arousal in women comprises two components: genital Arousal and subjective Arousal. Genital Arousal is characterized by genital vasocongestion and other physiological changes that occur in response to sexual stimuli, whereas subjective Arousal refers to mental engagement during sexual activity. For some women, genital Arousal enhances subjective Arousal; for others, the two types of Arousal are desynchronous. However, the relationship between genital and subjective Arousal might not be relevant to the diagnosis and treatment of sexual Arousal dysfunction. Studies have shown that not all women who report sexual Arousal problems have decreased genital Arousal, and only some women with decreased genital Arousal have low subjective Arousal. To develop efficacious treatments for female sexual Arousal dysfunction, researchers need to differentiate the women for whom genital sensations have a critical role in their subjective Arousal from those who are not mentally aroused by genital cues. The mechanisms by which women become aroused and the inputs into Arousal have considerable implications for treatment outcomes.
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The sensitivity of continuous laboratory measures of physiological and subjective sexual Arousal for diagnosing women with sexual Arousal disorder.
The journal of sexual medicine, 2010Co-Authors: Cindy M. Meston, Alessandra H. Rellini, Katie MccallAbstract:Past findings on the diagnostic sensitivity of vaginal photoplethysmography are limited by testing among women with heterogeneous sexual dysfunctions and by the use of statistical techniques that are unable to assess how changes in subjective Arousal are associated with changes in physiological Arousal. The aims of this study were to: (i) test the sensitivity of vaginal photoplethysmography and continuous measures of subjective sexual Arousal in differentiating between women with and without sexual Arousal or orgasm dysfunction; and (ii) examine the diagnostic utility of measuring the synchrony between genital and subjective sexual responses. Sexual Arousal was assessed in sexually healthy women (n = 12), women with orgasm disorder (OD; n = 12), and 38 women who met the criteria for the three subcategories of sexual Arousal dysfunction described by Basson et al. (i.e., genital sexual Arousal disorder [GAD; n = 9], subjective sexual Arousal disorder [SAD; n = 13], and combined genital and subjective Arousal disorder [CAD; n = 16]). Physiological sexual Arousal was assessed using vaginal photoplethysmography, and subjective sexual Arousal was measured continuously and using a Likert-scale in response to sexual videos. Women with GAD showed the lowest and women with CAD showed the highest levels of vaginal pulse amplitude response to erotic stimuli. Women with sexual Arousal disorder showed significantly lower levels of subjective sexual Arousal to erotic stimuli than did sexually healthy women. Relations between subjective and physiological measures of sexual Arousal were significantly weaker among women with sexual Arousal disorder than sexually healthy women or women with OD. Preliminary support was provided for the diagnostic utility of measuring the synchrony between subjective and genital Arousal in women with sexual Arousal disorder. Findings do not support the sensitivity of using vaginal photoplethysmography, or continuous or Likert-scale measures of subjective Arousal for differentiating between subtypes of women with sexual Arousal disorder.
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The Sensitivity of Continuous Laboratory Measures of Physiological and Subjective Sexual Arousal for Diagnosing Women with Sexual Arousal Disorder
The Journal of Sexual Medicine, 2010Co-Authors: Cindy M. Meston, Alessandra H. Rellini, Katie MccallAbstract:Introduction. Past findings on the diagnostic sensitivity of vaginal photoplethysmography are limited by testing among women with heterogeneous sexual dysfunctions and by the use of statistical techniques that are unable to assess how changes in subjective Arousal are associated with changes in physiological Arousal. Aims. The aims of this study were to: (i) test the sensitivity of vaginal photoplethysmography and continuous measures of subjective sexual Arousal in differentiating between women with and without sexual Arousal or orgasm dysfunction; and (ii) examine the diagnostic utility of measuring the synchrony between genital and subjective sexual responses. Methods. Sexual Arousal was assessed in sexually healthy women (n = 12), women with orgasm disorder (OD; n = 12), and 38 women who met the criteria for the three subcategories of sexual Arousal dysfunction described by Basson et al. (i.e., genital sexual Arousal disorder (GAD; n = 9), subjective sexual Arousal disorder (SAD; n = 13), and combined genital and subjective Arousal disorder (CAD; n = 16)). Main Outcome Measures. Physiological sexual Arousal was assessed using vaginal photoplethysmography, and subjective sexual Arousal was measured continuously and using a Likert-scale in response to sexual videos. Results. Women with GAD showed the lowest and women with CAD showed the highest levels of vaginal pulse amplitude response to erotic stimuli. Women with sexual Arousal disorder showed significantly lower levels of subjective sexual Arousal to erotic stimuli than did sexually healthy women. Relations between subjective and physiological measures of sexual Arousal were significantly weaker among women with sexual Arousal disorder than sexually healthy women or women with OD. Conclusion. Preliminary support was provided for the diagnostic utility of measuring the synchrony between subjective and genital Arousal in women with sexual Arousal disorder. Findings do not support the sensitivity of using vaginal photoplethysmography, or continuous or Likert-scale measures of subjective Arousal for differentiating between subtypes of women with sexual Arousal disorder. Meston CM, Rellini AH, and McCall K. The sensitivity of continuous laboratory measures of physiological and subjective sexual Arousal for diagnosing women with sexual Arousal disorder. J Sex Med 2010;7:938-950.
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The Effects of False Positive and False Negative Physiological Feedback on Sexual Arousal: A Comparison of Women with or without Sexual Arousal Disorder
Archives of Sexual Behavior, 2007Co-Authors: Katie M. Mccall, Cindy M. MestonAbstract:The effects of false positive and false negative physiological feedback (vaginal photoplethymograph response print-out) on women's sexual Arousal were examined. Participants included women without sexual dysfunction ( n =16) and women with Sexual Arousal Disorder (SAD; n =15). Measures of subjective sexual Arousal, physiological sexual Arousal (vaginal pulse amplitude), expectancies, affect, and anxiety were obtained in response to viewing an erotic film. Results indicated that false positive feedback significantly increased subjective levels of sexual Arousal, whereas false negative feedback significantly decreased subjective levels of sexual Arousal in both groups. Sexually functional women had overall higher expectancies for sexual Arousal than women with SAD. Unexpectedly, false positive feedback did not significantly impact physiological sexual Arousal in sexually functional women; however, it resulted in significantly decreased responses in physiological sexual Arousal in women with SAD. False negative feedback had no significant effect on physiological sexual response in sexually functional women or women with SAD.