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René Dom – One of the best experts on this subject based on the ideXlab platform.

  • Cognitive changes in patients with
    Huntington’s disease (HD) and
    asymptomatic carriers of the HD mutation
    Journal of Neurology, 2004
    Co-Authors: Jurgen Lemiere, Marleen Decruyenaere, Gery Evers-kiebooms, Erik Vandenbussche, René Dom

    Abstract:

    Objective Objective information about the onset and progression of cognitive impairment in Huntington’s disease (HD) is very important in the light of appropriate outcome measures when conducting clinical trials. Therefore, we evaluated the progression of cognitive functions in HD patients and asymptomatic carriers of the HD mutation (AC) over a 2.5–year period.We also sought to detect the earliest markers of cognitive impairment in AC. Methods A prospective study comparing HD patients, clinically asymptomatic HD mutation–carriers (AC) and non–carriers (NC). These groups were examined three times during a period of 2.5 years. At baseline the study sample consisted of 49 subjects. Forty–two subjects (19 HD patients, 12 AC and 11 NC) completed three assessments. A battery of neuropsychological Tests measuring intelligence, attention, memory, language, visuospatial perception, and executive functions was performed. Results The performance of HD patients deteriorated on the following cognitive Tests: Symbol Digit Modalities Test (SDMT), Stroop Colour and Word, Boston Naming Test (BNT), Object and Space Perception and Trail Making Test–B. Longitudinal comparison of AC and NC revealed that performances on SDMT, Block Span, Digit Span Backwards, Hopkins Verbal Learning Test (Learning and delayed recall) and Conditional Associative Learning Test are impaired in AC. Conclusions Tasks measuring mainly attention, object and space perception and executive functions adequately assess the progression of HD disease. Other cognitive functions do not significantly deteriorate. Furthermore, problems in attention, working memory, verbal Learning, verbal long–term memory and Learning of random associations are the earliest cognitive manifestations in AC.

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  • Cognitive changes in patients with Huntington’s disease (HD) and asymptomatic carriers of the HD mutation: A longitudinal follow-up study
    Journal of Neurology, 2004
    Co-Authors: Jurgen Lemiere, Marleen Decruyenaere, Gery Evers-kiebooms, Erik Vandenbussche, René Dom

    Abstract:

    Objective information
    about the onset and progression
    of cognitive impairment
    in Huntington’s disease (HD) is
    very important in the light of appropriate
    outcome measures when

    conducting clinical trials. Therefore,
    we evaluated the progression
    of cognitive functions in HD patients
    and asymptomatic carriers
    of the HD mutation (AC) over a
    2.5–year period.We also sought to
    detect the earliest markers of cognitive
    impairment in AC. A prospective study comparing HD
    patients, clinically asymptomatic
    HD mutation–carriers (AC) and
    non–carriers (NC). These groups
    were examined three times during
    a period of 2.5 years. At baseline
    the study sample consisted of 49
    subjects. Forty–two subjects (19 HD
    patients, 12 AC and 11 NC) completed
    three assessments. A battery
    of neuropsychological Tests measuring
    intelligence, attention, memory,
    language, visuospatial perception,
    and executive functions was
    performed. The performance
    of HD patients deteriorated
    on the following cognitive Tests:
    Symbol Digit Modalities Test
    (SDMT), Stroop Colour and Word,
    Boston Naming Test (BNT), Object
    and Space Perception and Trail
    Making Test–B. Longitudinal comparison
    of AC and NC revealed that
    performances on SDMT, Block
    Span, Digit Span Backwards, Hopkins
    Verbal Learning Test (Learning
    and delayed recall) and Conditional
    Associative Learning Test are
    impaired in AC. Tasks
    measuring mainly attention, object
    and space perception and executive
    functions adequately assess the
    progression of HD disease. Other
    cognitive functions do not significantly
    deteriorate. Furthermore,
    problems in attention, working
    memory, verbal Learning, verbal
    long–term memory and Learning of
    random associations are the earliest
    cognitive manifestations in AC.

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Jurgen Lemiere – One of the best experts on this subject based on the ideXlab platform.

  • Cognitive changes in patients with
    Huntington’s disease (HD) and
    asymptomatic carriers of the HD mutation
    Journal of Neurology, 2004
    Co-Authors: Jurgen Lemiere, Marleen Decruyenaere, Gery Evers-kiebooms, Erik Vandenbussche, René Dom

    Abstract:

    Objective Objective information about the onset and progression of cognitive impairment in Huntington’s disease (HD) is very important in the light of appropriate outcome measures when conducting clinical trials. Therefore, we evaluated the progression of cognitive functions in HD patients and asymptomatic carriers of the HD mutation (AC) over a 2.5–year period.We also sought to detect the earliest markers of cognitive impairment in AC. Methods A prospective study comparing HD patients, clinically asymptomatic HD mutation–carriers (AC) and non–carriers (NC). These groups were examined three times during a period of 2.5 years. At baseline the study sample consisted of 49 subjects. Forty–two subjects (19 HD patients, 12 AC and 11 NC) completed three assessments. A battery of neuropsychological Tests measuring intelligence, attention, memory, language, visuospatial perception, and executive functions was performed. Results The performance of HD patients deteriorated on the following cognitive Tests: Symbol Digit Modalities Test (SDMT), Stroop Colour and Word, Boston Naming Test (BNT), Object and Space Perception and Trail Making Test–B. Longitudinal comparison of AC and NC revealed that performances on SDMT, Block Span, Digit Span Backwards, Hopkins Verbal Learning Test (Learning and delayed recall) and Conditional Associative Learning Test are impaired in AC. Conclusions Tasks measuring mainly attention, object and space perception and executive functions adequately assess the progression of HD disease. Other cognitive functions do not significantly deteriorate. Furthermore, problems in attention, working memory, verbal Learning, verbal long–term memory and Learning of random associations are the earliest cognitive manifestations in AC.

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  • Cognitive changes in patients with Huntington’s disease (HD) and asymptomatic carriers of the HD mutation: A longitudinal follow-up study
    Journal of Neurology, 2004
    Co-Authors: Jurgen Lemiere, Marleen Decruyenaere, Gery Evers-kiebooms, Erik Vandenbussche, René Dom

    Abstract:

    Objective information
    about the onset and progression
    of cognitive impairment
    in Huntington’s disease (HD) is
    very important in the light of appropriate
    outcome measures when

    conducting clinical trials. Therefore,
    we evaluated the progression
    of cognitive functions in HD patients
    and asymptomatic carriers
    of the HD mutation (AC) over a
    2.5–year period.We also sought to
    detect the earliest markers of cognitive
    impairment in AC. A prospective study comparing HD
    patients, clinically asymptomatic
    HD mutation–carriers (AC) and
    non–carriers (NC). These groups
    were examined three times during
    a period of 2.5 years. At baseline
    the study sample consisted of 49
    subjects. Forty–two subjects (19 HD
    patients, 12 AC and 11 NC) completed
    three assessments. A battery
    of neuropsychological Tests measuring
    intelligence, attention, memory,
    language, visuospatial perception,
    and executive functions was
    performed. The performance
    of HD patients deteriorated
    on the following cognitive Tests:
    Symbol Digit Modalities Test
    (SDMT), Stroop Colour and Word,
    Boston Naming Test (BNT), Object
    and Space Perception and Trail
    Making Test–B. Longitudinal comparison
    of AC and NC revealed that
    performances on SDMT, Block
    Span, Digit Span Backwards, Hopkins
    Verbal Learning Test (Learning
    and delayed recall) and Conditional
    Associative Learning Test are
    impaired in AC. Tasks
    measuring mainly attention, object
    and space perception and executive
    functions adequately assess the
    progression of HD disease. Other
    cognitive functions do not significantly
    deteriorate. Furthermore,
    problems in attention, working
    memory, verbal Learning, verbal
    long–term memory and Learning of
    random associations are the earliest
    cognitive manifestations in AC.

    Free Register to Access Article

Gery Evers-kiebooms – One of the best experts on this subject based on the ideXlab platform.

  • Cognitive changes in patients with
    Huntington’s disease (HD) and
    asymptomatic carriers of the HD mutation
    Journal of Neurology, 2004
    Co-Authors: Jurgen Lemiere, Marleen Decruyenaere, Gery Evers-kiebooms, Erik Vandenbussche, René Dom

    Abstract:

    Objective Objective information about the onset and progression of cognitive impairment in Huntington’s disease (HD) is very important in the light of appropriate outcome measures when conducting clinical trials. Therefore, we evaluated the progression of cognitive functions in HD patients and asymptomatic carriers of the HD mutation (AC) over a 2.5–year period.We also sought to detect the earliest markers of cognitive impairment in AC. Methods A prospective study comparing HD patients, clinically asymptomatic HD mutation–carriers (AC) and non–carriers (NC). These groups were examined three times during a period of 2.5 years. At baseline the study sample consisted of 49 subjects. Forty–two subjects (19 HD patients, 12 AC and 11 NC) completed three assessments. A battery of neuropsychological Tests measuring intelligence, attention, memory, language, visuospatial perception, and executive functions was performed. Results The performance of HD patients deteriorated on the following cognitive Tests: Symbol Digit Modalities Test (SDMT), Stroop Colour and Word, Boston Naming Test (BNT), Object and Space Perception and Trail Making Test–B. Longitudinal comparison of AC and NC revealed that performances on SDMT, Block Span, Digit Span Backwards, Hopkins Verbal Learning Test (Learning and delayed recall) and Conditional Associative Learning Test are impaired in AC. Conclusions Tasks measuring mainly attention, object and space perception and executive functions adequately assess the progression of HD disease. Other cognitive functions do not significantly deteriorate. Furthermore, problems in attention, working memory, verbal Learning, verbal long–term memory and Learning of random associations are the earliest cognitive manifestations in AC.

    Free Register to Access Article

  • Cognitive changes in patients with Huntington’s disease (HD) and asymptomatic carriers of the HD mutation: A longitudinal follow-up study
    Journal of Neurology, 2004
    Co-Authors: Jurgen Lemiere, Marleen Decruyenaere, Gery Evers-kiebooms, Erik Vandenbussche, René Dom

    Abstract:

    Objective information
    about the onset and progression
    of cognitive impairment
    in Huntington’s disease (HD) is
    very important in the light of appropriate
    outcome measures when

    conducting clinical trials. Therefore,
    we evaluated the progression
    of cognitive functions in HD patients
    and asymptomatic carriers
    of the HD mutation (AC) over a
    2.5–year period.We also sought to
    detect the earliest markers of cognitive
    impairment in AC. A prospective study comparing HD
    patients, clinically asymptomatic
    HD mutation–carriers (AC) and
    non–carriers (NC). These groups
    were examined three times during
    a period of 2.5 years. At baseline
    the study sample consisted of 49
    subjects. Forty–two subjects (19 HD
    patients, 12 AC and 11 NC) completed
    three assessments. A battery
    of neuropsychological Tests measuring
    intelligence, attention, memory,
    language, visuospatial perception,
    and executive functions was
    performed. The performance
    of HD patients deteriorated
    on the following cognitive Tests:
    Symbol Digit Modalities Test
    (SDMT), Stroop Colour and Word,
    Boston Naming Test (BNT), Object
    and Space Perception and Trail
    Making Test–B. Longitudinal comparison
    of AC and NC revealed that
    performances on SDMT, Block
    Span, Digit Span Backwards, Hopkins
    Verbal Learning Test (Learning
    and delayed recall) and Conditional
    Associative Learning Test are
    impaired in AC. Tasks
    measuring mainly attention, object
    and space perception and executive
    functions adequately assess the
    progression of HD disease. Other
    cognitive functions do not significantly
    deteriorate. Furthermore,
    problems in attention, working
    memory, verbal Learning, verbal
    long–term memory and Learning of
    random associations are the earliest
    cognitive manifestations in AC.

    Free Register to Access Article