The Experts below are selected from a list of 261 Experts worldwide ranked by ideXlab platform

Mustafa Tekin - One of the best experts on this subject based on the ideXlab platform.

  • Autosomal Dominant Inheritance in a recently described zmiz1 related neurodevelopmental disorder case report of siblings and an affected parent
    American Journal of Medical Genetics Part A, 2020
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    : ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

  • Autosomal Dominant Inheritance in a recently described ZMIZ1‐related neurodevelopmental disorder: Case report of siblings and an affected parent
    American journal of medical genetics. Part A, 2019
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

Ingrid Winship - One of the best experts on this subject based on the ideXlab platform.

Kumarie Latchman - One of the best experts on this subject based on the ideXlab platform.

  • Autosomal Dominant Inheritance in a recently described zmiz1 related neurodevelopmental disorder case report of siblings and an affected parent
    American Journal of Medical Genetics Part A, 2020
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    : ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

  • Autosomal Dominant Inheritance in a recently described ZMIZ1‐related neurodevelopmental disorder: Case report of siblings and an affected parent
    American journal of medical genetics. Part A, 2019
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

Jane Juusola - One of the best experts on this subject based on the ideXlab platform.

  • Autosomal Dominant Inheritance in a recently described zmiz1 related neurodevelopmental disorder case report of siblings and an affected parent
    American Journal of Medical Genetics Part A, 2020
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    : ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

  • Autosomal Dominant Inheritance in a recently described ZMIZ1‐related neurodevelopmental disorder: Case report of siblings and an affected parent
    American journal of medical genetics. Part A, 2019
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

Madison Calder - One of the best experts on this subject based on the ideXlab platform.

  • Autosomal Dominant Inheritance in a recently described zmiz1 related neurodevelopmental disorder case report of siblings and an affected parent
    American Journal of Medical Genetics Part A, 2020
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    : ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.

  • Autosomal Dominant Inheritance in a recently described ZMIZ1‐related neurodevelopmental disorder: Case report of siblings and an affected parent
    American journal of medical genetics. Part A, 2019
    Co-Authors: Kumarie Latchman, Madison Calder, Dayna Morel, Lindsay Rhodes, Jane Juusola, Mustafa Tekin
    Abstract:

    ZMIZ1, zinc finger MIZ-domain containing 1, has recently been described in association with syndromic intellectual disability in which the primary phenotypic features include intellectual disability/developmental delay, seizures, hearing loss, behavioral issues, failure to thrive, and various congenital malformations. Most reported cases have been found to result from de novo mutations except for one set of three siblings in which parental testing could not be performed. With informed consent from the family, we report on a father and his two sons demonstrating Autosomal Dominant Inheritance of a novel pathogenic ZMIZ1 variant, c.1310delC (p.Pro437ArgfsX84), causing this recently described neurodevelopmental syndrome. While they all show syndromic findings along with short stature and intellectual disability, only one child had sensorineural hearing loss. Moreover, severity of intellectual disability and eyelid ptosis were variable among the affected members. Our report demonstrates that phenotypic features of ZMIZ1-related neurodevelopmental syndrome are variable even within the same family and that parental testing to identify a mildly affected parent is needed.