The Experts below are selected from a list of 918 Experts worldwide ranked by ideXlab platform

Jun-ying Miao - One of the best experts on this subject based on the ideXlab platform.

  • Phosphatidylcholine‐specific phospholipase C and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of cellular biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P 

  • phosphatidylcholine specific phospholipase c and ros were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of Cellular Biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P < 0.01), then it decreased gradually with the cell differentiation, while the Ca2+-depentent PC-PLC was nearly not changed in the whole process. At the same time, ROS level dramatically decreased during the cell differentiation. To understand the role of PC-PLC and how it performs its function in the vascular endothelial differentiation induced by FGF-2, we suppressed PC-PLC activity by its specific inhibitor D609 (tricyclodecan-9-yl potassium xanthate) at 24 h during the cell differentiation. As a result, the cell differentiation could not progress and the intracellular level of ROS was elevated. The data suggested that PC-PLC and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells. PC-PLC was an important factor in the Blastodisc cell survival and differentiation, and it might perform its function associated with ROS. J. Cell. Biochem. 102: 421–428, 2007. © 2007 Wiley-Liss, Inc.

Bin Huang - One of the best experts on this subject based on the ideXlab platform.

  • Phosphatidylcholine‐specific phospholipase C and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of cellular biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P 

  • phosphatidylcholine specific phospholipase c and ros were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of Cellular Biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P < 0.01), then it decreased gradually with the cell differentiation, while the Ca2+-depentent PC-PLC was nearly not changed in the whole process. At the same time, ROS level dramatically decreased during the cell differentiation. To understand the role of PC-PLC and how it performs its function in the vascular endothelial differentiation induced by FGF-2, we suppressed PC-PLC activity by its specific inhibitor D609 (tricyclodecan-9-yl potassium xanthate) at 24 h during the cell differentiation. As a result, the cell differentiation could not progress and the intracellular level of ROS was elevated. The data suggested that PC-PLC and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells. PC-PLC was an important factor in the Blastodisc cell survival and differentiation, and it might perform its function associated with ROS. J. Cell. Biochem. 102: 421–428, 2007. © 2007 Wiley-Liss, Inc.

Shangli Zhang - One of the best experts on this subject based on the ideXlab platform.

  • Phosphatidylcholine‐specific phospholipase C and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of cellular biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P 

  • phosphatidylcholine specific phospholipase c and ros were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of Cellular Biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P < 0.01), then it decreased gradually with the cell differentiation, while the Ca2+-depentent PC-PLC was nearly not changed in the whole process. At the same time, ROS level dramatically decreased during the cell differentiation. To understand the role of PC-PLC and how it performs its function in the vascular endothelial differentiation induced by FGF-2, we suppressed PC-PLC activity by its specific inhibitor D609 (tricyclodecan-9-yl potassium xanthate) at 24 h during the cell differentiation. As a result, the cell differentiation could not progress and the intracellular level of ROS was elevated. The data suggested that PC-PLC and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells. PC-PLC was an important factor in the Blastodisc cell survival and differentiation, and it might perform its function associated with ROS. J. Cell. Biochem. 102: 421–428, 2007. © 2007 Wiley-Liss, Inc.

Weiwei Wang - One of the best experts on this subject based on the ideXlab platform.

  • Phosphatidylcholine‐specific phospholipase C and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of cellular biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P 

  • phosphatidylcholine specific phospholipase c and ros were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of Cellular Biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P < 0.01), then it decreased gradually with the cell differentiation, while the Ca2+-depentent PC-PLC was nearly not changed in the whole process. At the same time, ROS level dramatically decreased during the cell differentiation. To understand the role of PC-PLC and how it performs its function in the vascular endothelial differentiation induced by FGF-2, we suppressed PC-PLC activity by its specific inhibitor D609 (tricyclodecan-9-yl potassium xanthate) at 24 h during the cell differentiation. As a result, the cell differentiation could not progress and the intracellular level of ROS was elevated. The data suggested that PC-PLC and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells. PC-PLC was an important factor in the Blastodisc cell survival and differentiation, and it might perform its function associated with ROS. J. Cell. Biochem. 102: 421–428, 2007. © 2007 Wiley-Liss, Inc.

Jing Zhao - One of the best experts on this subject based on the ideXlab platform.

  • Phosphatidylcholine‐specific phospholipase C and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of cellular biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P 

  • phosphatidylcholine specific phospholipase c and ros were involved in chicken Blastodisc differentiation to vascular endothelial cells
    Journal of Cellular Biochemistry, 2007
    Co-Authors: Jing Zhao, Bao-xiang Zhao, Weiwei Wang, Bin Huang, Shangli Zhang, Jun-ying Miao
    Abstract:

    To find the key factors that were involved in the survival and vascular endothelial differentiation of chick blatodisc induced by fibroblast growth factor 2 (FGF-2), we built a chick vasculogenesis model in vitro. Subsequently, the activities of phosphatidylcholine-specific phospholipase C (PC-PLC), including Ca2+-dependent and -independent PC-PLC, and the level of reactive oxygen species (ROS) were evaluated during the endothelial differentiation of chick Blastodisc. The results showed that Ca2+-indepentent PC-PLC underwent a remarkable increase in 24 h (P < 0.01), then it decreased gradually with the cell differentiation, while the Ca2+-depentent PC-PLC was nearly not changed in the whole process. At the same time, ROS level dramatically decreased during the cell differentiation. To understand the role of PC-PLC and how it performs its function in the vascular endothelial differentiation induced by FGF-2, we suppressed PC-PLC activity by its specific inhibitor D609 (tricyclodecan-9-yl potassium xanthate) at 24 h during the cell differentiation. As a result, the cell differentiation could not progress and the intracellular level of ROS was elevated. The data suggested that PC-PLC and ROS were involved in chicken Blastodisc differentiation to vascular endothelial cells. PC-PLC was an important factor in the Blastodisc cell survival and differentiation, and it might perform its function associated with ROS. J. Cell. Biochem. 102: 421–428, 2007. © 2007 Wiley-Liss, Inc.