The Experts below are selected from a list of 51 Experts worldwide ranked by ideXlab platform

Wei-dong Zhang - One of the best experts on this subject based on the ideXlab platform.

  • Case Report Idiopathic tension mediastinal emphysema cured by video-assisted thoracic surgery: a case report
    2020
    Co-Authors: Xiao-ping Li, Liang Zhang, Lei Li, Bo Yang, Wei-dong Zhang
    Abstract:

    Mediastinal emphysema is a status of gas retention in mediastinum, usually caused by airway system injury or Esophagus Rupture. Healthy people without trauma and basic disease emerging unexplained mediastinal emphysema are called idiopathic mediastinal emphysema. While tension pneumomediastinum is an exceptional and potentially lethal condition, the increased intramediastinal pressure with a severe oppression of heart, vena cava and pulmonary vein leads to impaired central venous return through the cavae system, restricted right heart diastolic filling, and collapse of the cardiac chambers, resulting in reduced stroke volume, cardiac output and dysae- mia. This young people-prone disease was initially reported by Hamman in 1939 and usually has a good prognosis so that no special treatments were needed. In this study, however, we presented a young boy with idiopathic tension mediastinal emphysema combined with dysaemia and respiratory failure. Fortunately, he was finally successful treated by VATS after heteropathy and incision on suprasternal fossae.

  • Idiopathic tension mediastinal emphysema cured by video-assisted thoracic surgery: a case report
    International Journal of Clinical and Experimental Medicine, 2015
    Co-Authors: Xiao-ping Li, Liang Zhang, Lei Li, Bo Yang, Wei-dong Zhang
    Abstract:

    Mediastinal emphysema is a status of gas retention in mediastinum, usually caused by airway system injury or Esophagus Rupture. Healthy people without trauma and basic disease emerging unexplained mediastinal emphysema are called idiopathic mediastinal emphysema. While tension pneumomediastinum is an exceptional and potentially lethal condition, the increased intramediastinal pressure with a severe oppression of heart, vena cava and pulmonary vein leads to impaired central venous return through the cavae system, restricted right heart diastolic filling, and collapse of the cardiac chambers, resulting in reduced stroke volume, cardiac output and dysaemia. This young people-prone disease was initially reported by Hamman in 1939 and usually has a good prognosis so that no special treatments were needed. In this study, however, we presented a young boy with idiopathic tension mediastinal emphysema combined with dysaemia and respiratory failure. Fortunately, he was finally successful treated by VATS after heteropathy and incision on suprasternal fossae.

Hiroshi Saeki - One of the best experts on this subject based on the ideXlab platform.

  • Pathophysiology of Vomiting and Esophageal Perforation in Boerhaave’s Syndrome
    Digestive Diseases and Sciences, 2020
    Co-Authors: Hiroyuki Kuwano, Takehiko Yokobori, Yuji Kumakura, Kyoichi Ogata, Tomonori Yoshida, Kengo Kuriyama, Makoto Sakai, Makoto Sohda, Tatsuya Miyazaki, Hiroshi Saeki
    Abstract:

    Background and Aims Boerhaave’s syndrome, involving Esophagus Rupture, is considered a pathological response to vomiting that may occur just before perforation. However, the mechanism of vomiting and occurrence of this disease have not been clearly demonstrated. Methods We identified patients with esophageal perforation between 1995 and 2017 and reviewed endoscopic findings at retching during upper gastrointestinal endoscopy. Finally, we proposed a theory for the underlying pathological mechanism. Results We retrospectively investigated 10 patients with esophageal perforation between 1995 and 2017. All patients presented after vomiting associated with large volumes of food and alcohol intake. Nine were treated by primary closure of the perforation and drainage of the thoracic cavity, and one was conservatively treated. In all cases, the perforations were longitudinal tears (1–4 cm) and located in the left of the Esophagus, just above the gastric cardia. Conclusions We hypothesize that vomiting occurred by retrograde propagation of gastrointestinal motor contraction from the jejunum to the gastric antrum, followed by prolapse of the gastric fornix mucosal into the Esophagus. Subsequent esophageal perforation probably resulted from excessive prolapse due to strong contraction and destruction of the muscularis mucosa of the left side of abdominal Esophagus, with longitudinal stretching of the whole left esophageal wall due to traction. We also propose that Boerhaave’s syndrome is defined as “post-emetic esophageal perforation” to ensure broader recognition and more expedient diagnosis and treatment. Remaining conditions without any definite causes may be labeled “idiopathic” or “spontaneous” Rupture of the Esophagus.

  • Pathophysiology of Vomiting and Esophageal Perforation in Boerhaave's Syndrome.
    Digestive Diseases and Sciences, 2020
    Co-Authors: Hiroyuki Kuwano, Takehiko Yokobori, Yuji Kumakura, Kyoichi Ogata, Tomonori Yoshida, Kengo Kuriyama, Makoto Sakai, Makoto Sohda, Tatsuya Miyazaki, Hiroshi Saeki
    Abstract:

    BACKGROUND AND AIMS: Boerhaave's syndrome, involving Esophagus Rupture, is considered a pathological response to vomiting that may occur just before perforation. However, the mechanism of vomiting and occurrence of this disease have not been clearly demonstrated. METHODS: We identified patients with esophageal perforation between 1995 and 2017 and reviewed endoscopic findings at retching during upper gastrointestinal endoscopy. Finally, we proposed a theory for the underlying pathological mechanism. RESULTS: We retrospectively investigated 10 patients with esophageal perforation between 1995 and 2017. All patients presented after vomiting associated with large volumes of food and alcohol intake. Nine were treated by primary closure of the perforation and drainage of the thoracic cavity, and one was conservatively treated. In all cases, the perforations were longitudinal tears (1-4 cm) and located in the left of the Esophagus, just above the gastric cardia. CONCLUSIONS: We hypothesize that vomiting occurred by retrograde propagation of gastrointestinal motor contraction from the jejunum to the gastric antrum, followed by prolapse of the gastric fornix mucosal into the Esophagus. Subsequent esophageal perforation probably resulted from excessive prolapse due to strong contraction and destruction of the muscularis mucosa of the left side of abdominal Esophagus, with longitudinal stretching of the whole left esophageal wall due to traction. We also propose that Boerhaave's syndrome is defined as "post-emetic esophageal perforation" to ensure broader recognition and more expedient diagnosis and treatment. Remaining conditions without any definite causes may be labeled "idiopathic" or "spontaneous" Rupture of the Esophagus.

Xiao-ping Li - One of the best experts on this subject based on the ideXlab platform.

  • Case Report Idiopathic tension mediastinal emphysema cured by video-assisted thoracic surgery: a case report
    2020
    Co-Authors: Xiao-ping Li, Liang Zhang, Lei Li, Bo Yang, Wei-dong Zhang
    Abstract:

    Mediastinal emphysema is a status of gas retention in mediastinum, usually caused by airway system injury or Esophagus Rupture. Healthy people without trauma and basic disease emerging unexplained mediastinal emphysema are called idiopathic mediastinal emphysema. While tension pneumomediastinum is an exceptional and potentially lethal condition, the increased intramediastinal pressure with a severe oppression of heart, vena cava and pulmonary vein leads to impaired central venous return through the cavae system, restricted right heart diastolic filling, and collapse of the cardiac chambers, resulting in reduced stroke volume, cardiac output and dysae- mia. This young people-prone disease was initially reported by Hamman in 1939 and usually has a good prognosis so that no special treatments were needed. In this study, however, we presented a young boy with idiopathic tension mediastinal emphysema combined with dysaemia and respiratory failure. Fortunately, he was finally successful treated by VATS after heteropathy and incision on suprasternal fossae.

  • Idiopathic tension mediastinal emphysema cured by video-assisted thoracic surgery: a case report
    International Journal of Clinical and Experimental Medicine, 2015
    Co-Authors: Xiao-ping Li, Liang Zhang, Lei Li, Bo Yang, Wei-dong Zhang
    Abstract:

    Mediastinal emphysema is a status of gas retention in mediastinum, usually caused by airway system injury or Esophagus Rupture. Healthy people without trauma and basic disease emerging unexplained mediastinal emphysema are called idiopathic mediastinal emphysema. While tension pneumomediastinum is an exceptional and potentially lethal condition, the increased intramediastinal pressure with a severe oppression of heart, vena cava and pulmonary vein leads to impaired central venous return through the cavae system, restricted right heart diastolic filling, and collapse of the cardiac chambers, resulting in reduced stroke volume, cardiac output and dysaemia. This young people-prone disease was initially reported by Hamman in 1939 and usually has a good prognosis so that no special treatments were needed. In this study, however, we presented a young boy with idiopathic tension mediastinal emphysema combined with dysaemia and respiratory failure. Fortunately, he was finally successful treated by VATS after heteropathy and incision on suprasternal fossae.

Hiroyuki Kuwano - One of the best experts on this subject based on the ideXlab platform.

  • Pathophysiology of Vomiting and Esophageal Perforation in Boerhaave’s Syndrome
    Digestive Diseases and Sciences, 2020
    Co-Authors: Hiroyuki Kuwano, Takehiko Yokobori, Yuji Kumakura, Kyoichi Ogata, Tomonori Yoshida, Kengo Kuriyama, Makoto Sakai, Makoto Sohda, Tatsuya Miyazaki, Hiroshi Saeki
    Abstract:

    Background and Aims Boerhaave’s syndrome, involving Esophagus Rupture, is considered a pathological response to vomiting that may occur just before perforation. However, the mechanism of vomiting and occurrence of this disease have not been clearly demonstrated. Methods We identified patients with esophageal perforation between 1995 and 2017 and reviewed endoscopic findings at retching during upper gastrointestinal endoscopy. Finally, we proposed a theory for the underlying pathological mechanism. Results We retrospectively investigated 10 patients with esophageal perforation between 1995 and 2017. All patients presented after vomiting associated with large volumes of food and alcohol intake. Nine were treated by primary closure of the perforation and drainage of the thoracic cavity, and one was conservatively treated. In all cases, the perforations were longitudinal tears (1–4 cm) and located in the left of the Esophagus, just above the gastric cardia. Conclusions We hypothesize that vomiting occurred by retrograde propagation of gastrointestinal motor contraction from the jejunum to the gastric antrum, followed by prolapse of the gastric fornix mucosal into the Esophagus. Subsequent esophageal perforation probably resulted from excessive prolapse due to strong contraction and destruction of the muscularis mucosa of the left side of abdominal Esophagus, with longitudinal stretching of the whole left esophageal wall due to traction. We also propose that Boerhaave’s syndrome is defined as “post-emetic esophageal perforation” to ensure broader recognition and more expedient diagnosis and treatment. Remaining conditions without any definite causes may be labeled “idiopathic” or “spontaneous” Rupture of the Esophagus.

  • Pathophysiology of Vomiting and Esophageal Perforation in Boerhaave's Syndrome.
    Digestive Diseases and Sciences, 2020
    Co-Authors: Hiroyuki Kuwano, Takehiko Yokobori, Yuji Kumakura, Kyoichi Ogata, Tomonori Yoshida, Kengo Kuriyama, Makoto Sakai, Makoto Sohda, Tatsuya Miyazaki, Hiroshi Saeki
    Abstract:

    BACKGROUND AND AIMS: Boerhaave's syndrome, involving Esophagus Rupture, is considered a pathological response to vomiting that may occur just before perforation. However, the mechanism of vomiting and occurrence of this disease have not been clearly demonstrated. METHODS: We identified patients with esophageal perforation between 1995 and 2017 and reviewed endoscopic findings at retching during upper gastrointestinal endoscopy. Finally, we proposed a theory for the underlying pathological mechanism. RESULTS: We retrospectively investigated 10 patients with esophageal perforation between 1995 and 2017. All patients presented after vomiting associated with large volumes of food and alcohol intake. Nine were treated by primary closure of the perforation and drainage of the thoracic cavity, and one was conservatively treated. In all cases, the perforations were longitudinal tears (1-4 cm) and located in the left of the Esophagus, just above the gastric cardia. CONCLUSIONS: We hypothesize that vomiting occurred by retrograde propagation of gastrointestinal motor contraction from the jejunum to the gastric antrum, followed by prolapse of the gastric fornix mucosal into the Esophagus. Subsequent esophageal perforation probably resulted from excessive prolapse due to strong contraction and destruction of the muscularis mucosa of the left side of abdominal Esophagus, with longitudinal stretching of the whole left esophageal wall due to traction. We also propose that Boerhaave's syndrome is defined as "post-emetic esophageal perforation" to ensure broader recognition and more expedient diagnosis and treatment. Remaining conditions without any definite causes may be labeled "idiopathic" or "spontaneous" Rupture of the Esophagus.

Zhang Heming - One of the best experts on this subject based on the ideXlab platform.

  • diagnosis and treatment of 14 cases of chronic spontaneous Rupture of Esophagus sre
    Journal of Hainan Medical University, 2011
    Co-Authors: Zhang Heming
    Abstract:

    Objective: To discuss the diagnosis and treatment of spontaneous Rupture of Esophagus in order to avoid misdiagnosis and mistreatment.Methods: Retrospective analyses on 14 cases of spontaneous Esophagus Rupture patients that were treated in our hospital during 1995 to 2009 was conducted.Results: Ten of the 14 patients underwent internal repair surgery within 24 hours(9 caes),or within 72 hours(1 case) after onset,and all achieved stage I recovery.One of the 2 patients that underwent esophagectomy + cervical esophagogastrostomy 72 hours after onset achieved stage I recovery,while the other had fistula and recovered after cervical drainage and supportive therapy.One of the 2 patients that were treated with closed thoracic drainag plus jejunostomy died,the other recovered after esophagectomy + cervical esophagogastrostomy.Conclusions: Early diagnosis,peroper treatment and comprehensive nutrients support are key factors to the successful treatment of spontaneous Rupture of Esophagus