The Experts below are selected from a list of 894 Experts worldwide ranked by ideXlab platform

Peter Kierulf - One of the best experts on this subject based on the ideXlab platform.

  • three new cases of an inborn qualitative Fibrinogen Defect Fibrinogen oslo ii
    Scandinavian Journal of Haematology, 2009
    Co-Authors: H C Godal, Frank Brosstad, Peter Kierulf
    Abstract:

    An autosomally inherited, qualitative Fibrinogen Defect is presented. It is associated with prolonged thrombin clotting time, low plasma Fibrinogen when assayed by a fibrin polymerization test and large amounts of Fibrinogen antigen determinants in the supernatant after clotting. The plasma Fibrinogen level was normal when assayed by an immunological technique or by quantitation of insoluble fibrin under conditions in which fibrin polymerization in enhanced. As judged from N-terminal amino acid analyses, fibrinopeptides were split off at normal speed, and the subunit chains of the Fibrinogen appeared normal when examined on polyacrylamide gels. The abnormality was not associated with bleeding tendency, and other routine coagulation tests gave normal results. The findings are in accordance with the concept of Defective fibrin polymerization.

H C Godal - One of the best experts on this subject based on the ideXlab platform.

  • three new cases of an inborn qualitative Fibrinogen Defect Fibrinogen oslo ii
    Scandinavian Journal of Haematology, 2009
    Co-Authors: H C Godal, Frank Brosstad, Peter Kierulf
    Abstract:

    An autosomally inherited, qualitative Fibrinogen Defect is presented. It is associated with prolonged thrombin clotting time, low plasma Fibrinogen when assayed by a fibrin polymerization test and large amounts of Fibrinogen antigen determinants in the supernatant after clotting. The plasma Fibrinogen level was normal when assayed by an immunological technique or by quantitation of insoluble fibrin under conditions in which fibrin polymerization in enhanced. As judged from N-terminal amino acid analyses, fibrinopeptides were split off at normal speed, and the subunit chains of the Fibrinogen appeared normal when examined on polyacrylamide gels. The abnormality was not associated with bleeding tendency, and other routine coagulation tests gave normal results. The findings are in accordance with the concept of Defective fibrin polymerization.

Frank Brosstad - One of the best experts on this subject based on the ideXlab platform.

  • three new cases of an inborn qualitative Fibrinogen Defect Fibrinogen oslo ii
    Scandinavian Journal of Haematology, 2009
    Co-Authors: H C Godal, Frank Brosstad, Peter Kierulf
    Abstract:

    An autosomally inherited, qualitative Fibrinogen Defect is presented. It is associated with prolonged thrombin clotting time, low plasma Fibrinogen when assayed by a fibrin polymerization test and large amounts of Fibrinogen antigen determinants in the supernatant after clotting. The plasma Fibrinogen level was normal when assayed by an immunological technique or by quantitation of insoluble fibrin under conditions in which fibrin polymerization in enhanced. As judged from N-terminal amino acid analyses, fibrinopeptides were split off at normal speed, and the subunit chains of the Fibrinogen appeared normal when examined on polyacrylamide gels. The abnormality was not associated with bleeding tendency, and other routine coagulation tests gave normal results. The findings are in accordance with the concept of Defective fibrin polymerization.