The Experts below are selected from a list of 60 Experts worldwide ranked by ideXlab platform
Yoshihiro Shimada - One of the best experts on this subject based on the ideXlab platform.
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A Case of Pachydermoperiostosis with Watery Diarrhea, Giant Gastri Rugae, and Endocrine Disorder
Digestive Endoscopy, 1991Co-Authors: Keiko Kishida, Shiro Fukumoto, Makoto Watanabe, Kyoichi Adachi, Kazutoshi Amano, Miyako Ohtani, Noriyuki Arima, Satoru Ikeda, Yoshihiro ShimadaAbstract:: A 31-year-old man, cook, who had had persistent watery diarrhea for about a month visited our hospital. He had already been diagnosed as having pachydermoperiostosis. An examination of the upper gastrointestinal tract revealed that he had giant Gastric Rugae. The histology was compatible with hypertrophic gastritis accompanied with marked hyperplasia of the fundic gland and foveolar epithelia. The output of Gastric juice was high in volume, although its acidity normal. The barium transit time of the small intestine was reduced to 15 minutes. His diarrhea was, thus, considered to be induced by gastrointestinal hyperfunction. The patient's glucose tolerance was abnormal and basal Cortisol secretion level was high. Pachydermoperiostosis has been the focus of attention for skin and bone changes, and the frequency of this disease with endocrine disorders seems rather high, but accompanying gastrointestinal disorders have so far not often been reported in Japan. The results of our investigation strongly suggest that pachydermoperiostosis is a systemic disease.
Martin Riegler - One of the best experts on this subject based on the ideXlab platform.
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Videoendoscopy and histopathology of the esophagoGastric junction in patients with gastroesophageal reflux disease
Wiener klinische Wochenschrift, 2007Co-Authors: Claudia Ringhofer, Johannes Lenglinger, Margit Eisler, Fritz Wrba, Roland Sedivy, Johannes Zacherl, Enrico P Cosentini, Gerhard Prager, Elena Devyatko, Martin RieglerAbstract:BACKGROUND AND AIMS: During endoscopy the stomach is considered to rise at the level of the 'Gastric' folds; however, anatomical studies have demonstrated that the proximal Gastric folds may in fact be esophageal. This prospective study was designed to assess the histopathology of endoscopically visible proximal Gastric folds in patients with gastroesophageal reflux disease. METHODS: 35 consecutive patients (20 males) with gastroesophageal reflux disease underwent video endoscopy, including biopsy sampling from the endoscopically visible esophagoGastric junction (0 cm, 0.5 cm and 1.0 cm distal to the rise of Gastric folds and 0.5 cm and 1.0 cm proximal to it). Endoscopy was digitally recorded and reviewed for assignment of biopsy level. Columnar-lined esophagus and esophagitis were cataloged according to the Paull-Chandrasoma histopathologic classification and the Los Angeles endoscopic classification. RESULTS: Endoscopy: Normal endoscopic esophagoGastric junction was seen in 11 (31%) patients and visible columnar-lined esophagus ≤ 0.5 cm in 24 (69%). Histology: Columnar-lined esophagus extended 1.0 cm in 22.8% of patients and 0.5 cm in 51.4%, distal to the rise of the Gastric folds. In all patients columnar-lined esophagus was interposed between squamous epithelium and Gastric oxyntic mucosa. Thus, so-called Gastric folds contained mucosa of esophageal origin in all patients. Intestinal metaplasia (Barrett esophagus) was detected in eight (22.9%) patients. CONCLUSIONS: Endoscopy cannot exclude histopathologic columnar-lined esophagus within Gastric Rugae. Thus, visible 'Gastric' folds should not be used for definition of the esophagoGastric junction but as a reference landmark for biopsy sampling during endoscopy. HINTERGRUND: In der Endoskopie beginnt der Magen mit den sogenannten gastralen Schleimhautfalten. Anatomische Studien lassen jedoch vermuten, dass diese Falten zum Ösophagus gehören. Deshalb hat diese Studie die Histologie des Beginns der endoskopisch sichtbaren Magenfalten bei Patienten mit gastroösophagealer Refluxkrankheit untersucht. METHODIK: Bei 35 Patienten (20 Männern und 15 Frauen) mit gastroösophagealer Refluxkrankheit wurden Videoendoskopie und Biopsien aus dem ösophagogastralen Übergang durchgeführt. Endoskopie und Biopsien wurden digital aufgezeichnet. Zylinderepithelösophagus und Ösophagitis wurden entsprechend der Paull-Chandrasoma bzw. Los Angeles Klassifikation beurteilt. ERGEBNISSE: Endoskopie: Ein endoskopisch normaler ösophagogastraler Übergang und ein endoskopisch sichtbarer Zylinderepithelösophagus < 0,5 cm fanden sich bei 11 (31%) bzw. 24 (69%) Patienten. Histologie: Bei 22,8% und 51,4% der Patienten war der Zylinderepithelösophagus in Biopsien 1,0 cm bzw. 0,5 cm distal des Beginns der endoskopischen Magenfalten nachzuweisen. Bei 8 Patienten (22,9%) fand sich eine intestinale Metaplasie (= Barrett Ösophagus). ZUSAMMENFASSUNG: Endoskopisch kann ein histologischer Zylinderepithelösophagus in den so genannten Magenfalten nicht ausgeschlossen werden. Der Beginn der Magenfalten dient als Referenz-Ebene für die Biopsielokalisationen.
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Videoendoscopy and histopathology of the esophagoGastric junction in patients with gastroesophageal reflux disease.
Wiener klinische Wochenschrift, 2007Co-Authors: Claudia Ringhofer, Johannes Lenglinger, Margit Eisler, Fritz Wrba, Roland Sedivy, Johannes Zacherl, Enrico P Cosentini, Gerhard Prager, Elena Devyatko, Martin RieglerAbstract:During endoscopy the stomach is considered to rise at the level of the 'Gastric' folds; however, anatomical studies have demonstrated that the proximal Gastric folds may in fact be esophageal. This prospective study was designed to assess the histopathology of endoscopically visible proximal Gastric folds in patients with gastroesophageal reflux disease. 35 consecutive patients (20 males) with gastroesophageal reflux disease underwent video endoscopy, including biopsy sampling from the endoscopically visible esophagoGastric junction (0 cm, 0.5 cm and 1.0 cm distal to the rise of Gastric folds and 0.5 cm and 1.0 cm proximal to it). Endoscopy was digitally recorded and reviewed for assignment of biopsy level. Columnar-lined esophagus and esophagitis were cataloged according to the Paull-Chandrasoma histopathologic classification and the Los Angeles endoscopic classification. Endoscopy: Normal endoscopic esophagoGastric junction was seen in 11 (31%) patients and visible columnar-lined esophagus < or = 0.5 cm in 24 (69%). Columnar-lined esophagus extended 1.0 cm in 22.8% of patients and 0.5 cm in 51.4%, distal to the rise of the Gastric folds. In all patients columnar-lined esophagus was interposed between squamous epithelium and Gastric oxyntic mucosa. Thus, so-called Gastric folds contained mucosa of esophageal origin in all patients. Intestinal metaplasia (Barrett esophagus) was detected in eight (22.9%) patients. Endoscopy cannot exclude histopathologic columnar-lined esophagus within Gastric Rugae. Thus, visible 'Gastric' folds should not be used for definition of the esophagoGastric junction but as a reference landmark for biopsy sampling during endoscopy.
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Videoendoscopy and histopathology of the esophagoGastric junction in patients with gastroesophageal reflux disease.
Wiener Klinische Wochenschrift, 2007Co-Authors: Claudia Ringhofer, Johannes Lenglinger, Margit Eisler, Fritz Wrba, Roland Sedivy, Johannes Zacherl, Enrico P Cosentini, Gerhard Prager, Elena Devyatko, Martin RieglerAbstract:BACKGROUND AND AIMS: During endoscopy the stomach is considered to rise at the level of the 'Gastric' folds; however, anatomical studies have demonstrated that the proximal Gastric folds may in fact be esophageal. This prospective study was designed to assess the histopathology of endoscopically visible proximal Gastric folds in patients with gastroesophageal reflux disease. METHODS: 35 consecutive patients (20 males) with gastroesophageal reflux disease underwent video endoscopy, including biopsy sampling from the endoscopically visible esophagoGastric junction (0 cm, 0.5 cm and 1.0 cm distal to the rise of Gastric folds and 0.5 cm and 1.0 cm proximal to it). Endoscopy was digitally recorded and reviewed for assignment of biopsy level. Columnar-lined esophagus and esophagitis were cataloged according to the Paull-Chandrasoma histopathologic classification and the Los Angeles endoscopic classification. RESULTS: Endoscopy: Normal endoscopic esophagoGastric junction was seen in 11 (31%) patients and visible columnar-lined esophagus ≤ 0.5 cm in 24 (69%). Histology: Columnar-lined esophagus extended 1.0 cm in 22.8% of patients and 0.5 cm in 51.4%, distal to the rise of the Gastric folds. In all patients columnar-lined esophagus was interposed between squamous epithelium and Gastric oxyntic mucosa. Thus, so-called Gastric folds contained mucosa of esophageal origin in all patients. Intestinal metaplasia (Barrett esophagus) was detected in eight (22.9%) patients. CONCLUSIONS: Endoscopy cannot exclude histopathologic columnar-lined esophagus within Gastric Rugae. Thus, visible 'Gastric' folds should not be used for definition of the esophagoGastric junction but as a reference landmark for biopsy sampling during endoscopy.
Keiko Kishida - One of the best experts on this subject based on the ideXlab platform.
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A Case of Pachydermoperiostosis with Watery Diarrhea, Giant Gastri Rugae, and Endocrine Disorder
Digestive Endoscopy, 1991Co-Authors: Keiko Kishida, Shiro Fukumoto, Makoto Watanabe, Kyoichi Adachi, Kazutoshi Amano, Miyako Ohtani, Noriyuki Arima, Satoru Ikeda, Yoshihiro ShimadaAbstract:: A 31-year-old man, cook, who had had persistent watery diarrhea for about a month visited our hospital. He had already been diagnosed as having pachydermoperiostosis. An examination of the upper gastrointestinal tract revealed that he had giant Gastric Rugae. The histology was compatible with hypertrophic gastritis accompanied with marked hyperplasia of the fundic gland and foveolar epithelia. The output of Gastric juice was high in volume, although its acidity normal. The barium transit time of the small intestine was reduced to 15 minutes. His diarrhea was, thus, considered to be induced by gastrointestinal hyperfunction. The patient's glucose tolerance was abnormal and basal Cortisol secretion level was high. Pachydermoperiostosis has been the focus of attention for skin and bone changes, and the frequency of this disease with endocrine disorders seems rather high, but accompanying gastrointestinal disorders have so far not often been reported in Japan. The results of our investigation strongly suggest that pachydermoperiostosis is a systemic disease.
Claudia Ringhofer - One of the best experts on this subject based on the ideXlab platform.
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Videoendoscopy and histopathology of the esophagoGastric junction in patients with gastroesophageal reflux disease
Wiener klinische Wochenschrift, 2007Co-Authors: Claudia Ringhofer, Johannes Lenglinger, Margit Eisler, Fritz Wrba, Roland Sedivy, Johannes Zacherl, Enrico P Cosentini, Gerhard Prager, Elena Devyatko, Martin RieglerAbstract:BACKGROUND AND AIMS: During endoscopy the stomach is considered to rise at the level of the 'Gastric' folds; however, anatomical studies have demonstrated that the proximal Gastric folds may in fact be esophageal. This prospective study was designed to assess the histopathology of endoscopically visible proximal Gastric folds in patients with gastroesophageal reflux disease. METHODS: 35 consecutive patients (20 males) with gastroesophageal reflux disease underwent video endoscopy, including biopsy sampling from the endoscopically visible esophagoGastric junction (0 cm, 0.5 cm and 1.0 cm distal to the rise of Gastric folds and 0.5 cm and 1.0 cm proximal to it). Endoscopy was digitally recorded and reviewed for assignment of biopsy level. Columnar-lined esophagus and esophagitis were cataloged according to the Paull-Chandrasoma histopathologic classification and the Los Angeles endoscopic classification. RESULTS: Endoscopy: Normal endoscopic esophagoGastric junction was seen in 11 (31%) patients and visible columnar-lined esophagus ≤ 0.5 cm in 24 (69%). Histology: Columnar-lined esophagus extended 1.0 cm in 22.8% of patients and 0.5 cm in 51.4%, distal to the rise of the Gastric folds. In all patients columnar-lined esophagus was interposed between squamous epithelium and Gastric oxyntic mucosa. Thus, so-called Gastric folds contained mucosa of esophageal origin in all patients. Intestinal metaplasia (Barrett esophagus) was detected in eight (22.9%) patients. CONCLUSIONS: Endoscopy cannot exclude histopathologic columnar-lined esophagus within Gastric Rugae. Thus, visible 'Gastric' folds should not be used for definition of the esophagoGastric junction but as a reference landmark for biopsy sampling during endoscopy. HINTERGRUND: In der Endoskopie beginnt der Magen mit den sogenannten gastralen Schleimhautfalten. Anatomische Studien lassen jedoch vermuten, dass diese Falten zum Ösophagus gehören. Deshalb hat diese Studie die Histologie des Beginns der endoskopisch sichtbaren Magenfalten bei Patienten mit gastroösophagealer Refluxkrankheit untersucht. METHODIK: Bei 35 Patienten (20 Männern und 15 Frauen) mit gastroösophagealer Refluxkrankheit wurden Videoendoskopie und Biopsien aus dem ösophagogastralen Übergang durchgeführt. Endoskopie und Biopsien wurden digital aufgezeichnet. Zylinderepithelösophagus und Ösophagitis wurden entsprechend der Paull-Chandrasoma bzw. Los Angeles Klassifikation beurteilt. ERGEBNISSE: Endoskopie: Ein endoskopisch normaler ösophagogastraler Übergang und ein endoskopisch sichtbarer Zylinderepithelösophagus < 0,5 cm fanden sich bei 11 (31%) bzw. 24 (69%) Patienten. Histologie: Bei 22,8% und 51,4% der Patienten war der Zylinderepithelösophagus in Biopsien 1,0 cm bzw. 0,5 cm distal des Beginns der endoskopischen Magenfalten nachzuweisen. Bei 8 Patienten (22,9%) fand sich eine intestinale Metaplasie (= Barrett Ösophagus). ZUSAMMENFASSUNG: Endoskopisch kann ein histologischer Zylinderepithelösophagus in den so genannten Magenfalten nicht ausgeschlossen werden. Der Beginn der Magenfalten dient als Referenz-Ebene für die Biopsielokalisationen.
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Videoendoscopy and histopathology of the esophagoGastric junction in patients with gastroesophageal reflux disease.
Wiener klinische Wochenschrift, 2007Co-Authors: Claudia Ringhofer, Johannes Lenglinger, Margit Eisler, Fritz Wrba, Roland Sedivy, Johannes Zacherl, Enrico P Cosentini, Gerhard Prager, Elena Devyatko, Martin RieglerAbstract:During endoscopy the stomach is considered to rise at the level of the 'Gastric' folds; however, anatomical studies have demonstrated that the proximal Gastric folds may in fact be esophageal. This prospective study was designed to assess the histopathology of endoscopically visible proximal Gastric folds in patients with gastroesophageal reflux disease. 35 consecutive patients (20 males) with gastroesophageal reflux disease underwent video endoscopy, including biopsy sampling from the endoscopically visible esophagoGastric junction (0 cm, 0.5 cm and 1.0 cm distal to the rise of Gastric folds and 0.5 cm and 1.0 cm proximal to it). Endoscopy was digitally recorded and reviewed for assignment of biopsy level. Columnar-lined esophagus and esophagitis were cataloged according to the Paull-Chandrasoma histopathologic classification and the Los Angeles endoscopic classification. Endoscopy: Normal endoscopic esophagoGastric junction was seen in 11 (31%) patients and visible columnar-lined esophagus < or = 0.5 cm in 24 (69%). Columnar-lined esophagus extended 1.0 cm in 22.8% of patients and 0.5 cm in 51.4%, distal to the rise of the Gastric folds. In all patients columnar-lined esophagus was interposed between squamous epithelium and Gastric oxyntic mucosa. Thus, so-called Gastric folds contained mucosa of esophageal origin in all patients. Intestinal metaplasia (Barrett esophagus) was detected in eight (22.9%) patients. Endoscopy cannot exclude histopathologic columnar-lined esophagus within Gastric Rugae. Thus, visible 'Gastric' folds should not be used for definition of the esophagoGastric junction but as a reference landmark for biopsy sampling during endoscopy.
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Videoendoscopy and histopathology of the esophagoGastric junction in patients with gastroesophageal reflux disease.
Wiener Klinische Wochenschrift, 2007Co-Authors: Claudia Ringhofer, Johannes Lenglinger, Margit Eisler, Fritz Wrba, Roland Sedivy, Johannes Zacherl, Enrico P Cosentini, Gerhard Prager, Elena Devyatko, Martin RieglerAbstract:BACKGROUND AND AIMS: During endoscopy the stomach is considered to rise at the level of the 'Gastric' folds; however, anatomical studies have demonstrated that the proximal Gastric folds may in fact be esophageal. This prospective study was designed to assess the histopathology of endoscopically visible proximal Gastric folds in patients with gastroesophageal reflux disease. METHODS: 35 consecutive patients (20 males) with gastroesophageal reflux disease underwent video endoscopy, including biopsy sampling from the endoscopically visible esophagoGastric junction (0 cm, 0.5 cm and 1.0 cm distal to the rise of Gastric folds and 0.5 cm and 1.0 cm proximal to it). Endoscopy was digitally recorded and reviewed for assignment of biopsy level. Columnar-lined esophagus and esophagitis were cataloged according to the Paull-Chandrasoma histopathologic classification and the Los Angeles endoscopic classification. RESULTS: Endoscopy: Normal endoscopic esophagoGastric junction was seen in 11 (31%) patients and visible columnar-lined esophagus ≤ 0.5 cm in 24 (69%). Histology: Columnar-lined esophagus extended 1.0 cm in 22.8% of patients and 0.5 cm in 51.4%, distal to the rise of the Gastric folds. In all patients columnar-lined esophagus was interposed between squamous epithelium and Gastric oxyntic mucosa. Thus, so-called Gastric folds contained mucosa of esophageal origin in all patients. Intestinal metaplasia (Barrett esophagus) was detected in eight (22.9%) patients. CONCLUSIONS: Endoscopy cannot exclude histopathologic columnar-lined esophagus within Gastric Rugae. Thus, visible 'Gastric' folds should not be used for definition of the esophagoGastric junction but as a reference landmark for biopsy sampling during endoscopy.
Shiro Fukumoto - One of the best experts on this subject based on the ideXlab platform.
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A Case of Pachydermoperiostosis with Watery Diarrhea, Giant Gastri Rugae, and Endocrine Disorder
Digestive Endoscopy, 1991Co-Authors: Keiko Kishida, Shiro Fukumoto, Makoto Watanabe, Kyoichi Adachi, Kazutoshi Amano, Miyako Ohtani, Noriyuki Arima, Satoru Ikeda, Yoshihiro ShimadaAbstract:: A 31-year-old man, cook, who had had persistent watery diarrhea for about a month visited our hospital. He had already been diagnosed as having pachydermoperiostosis. An examination of the upper gastrointestinal tract revealed that he had giant Gastric Rugae. The histology was compatible with hypertrophic gastritis accompanied with marked hyperplasia of the fundic gland and foveolar epithelia. The output of Gastric juice was high in volume, although its acidity normal. The barium transit time of the small intestine was reduced to 15 minutes. His diarrhea was, thus, considered to be induced by gastrointestinal hyperfunction. The patient's glucose tolerance was abnormal and basal Cortisol secretion level was high. Pachydermoperiostosis has been the focus of attention for skin and bone changes, and the frequency of this disease with endocrine disorders seems rather high, but accompanying gastrointestinal disorders have so far not often been reported in Japan. The results of our investigation strongly suggest that pachydermoperiostosis is a systemic disease.