The Experts below are selected from a list of 786 Experts worldwide ranked by ideXlab platform
Steven L Galetta - One of the best experts on this subject based on the ideXlab platform.
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editors note teaching video neuroimages Palsy of conjugate Horizontal Gaze and face due to isolated abducens nuclear infarction
Neurology, 2018Co-Authors: Megan Alcauskas, Steven L GalettaAbstract:In reference to “Teaching Video NeuroImages: Palsy of conjugate Horizontal Gaze and face due to isolated abducens nuclear infarction,” Dr. Miller points out that the title may mislead readers into thinking that both the Horizontal Gaze Palsy and the facial nerve Palsy were caused by the abducens nuclear infarction, rather than the ipsilateral facial Palsy being caused by damage to the facial nerve fascicles bordering the abducens nucleus. Author Kim agrees with Dr. Miller's concerns and has issued a correction to the title, changing it to “Palsy of conjugate Horizontal Gaze and face due to a restricted infarction involving the abducens nucleus.” The correction appears on page 942. In reference to “Teaching Video NeuroImages: Palsy of conjugate Horizontal Gaze and face due to isolated abducens nuclear infarction,” Dr. Miller points out that the title may mislead readers into thinking that both the Horizontal Gaze Palsy and the facial nerve Palsy were caused by the abducens nuclear infarction, rather than the ipsilateral facial Palsy being caused by damage to the facial nerve fascicles bordering the abducens nucleus.
Jeong Min Hwang - One of the best experts on this subject based on the ideXlab platform.
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teaching video neuroimages Horizontal Gaze Palsy with progressive scoliosis
Neurology, 2019Co-Authors: Hee Kyung Yang, Jeong Yoon Choi, Kyung Seok Park, Jeong Min HwangAbstract:A 10-year-old girl presented with abnormal eye movement since birth. She was born from consanguineous parents. Ocular ductions showed complete Horizontal Gaze Palsy, partial limitation of upGaze, and synergistic convergence (figure, A; video 1). Her pupils were round and isocoric. She showed severe thoracolumbar scoliosis. Uncrossed corticospinal and dorsal column–medial lemniscal pathways were demonstrated during intraoperative neuromonitoring of scoliosis surgery. Brain MRI revealed a bifid shape of the pons and medulla oblongata (figure, B–D), which is a typical finding of Horizontal Gaze Palsy with progressive scoliosis.1,2 Bilateral oculomotor, trochlear, and abducens nerves were normally identified (figure, E).
Caroline Tilikete - One of the best experts on this subject based on the ideXlab platform.
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teaching video neuroimages bilateral complete Horizontal Gaze Palsy with preserved convergence the 1 1 syndrome
Neurology, 2016Co-Authors: Damien Biotti, Caroline TiliketeAbstract:Bilateral complete Horizontal Gaze Palsy, the 1 + 1 syndrome, is extremely rare. An otherwise healthy 25-year-old woman shows a recently acquired complete Horizontal saccadic Palsy. Its association with the absence of improvement with oculocephalic maneuvers and the preservation of convergence clinically suggests a bilateral cranial nerve VI nucleus dysfunction1,2 (video at [Neurology.org][1]; figures 1 and 2). Main causes include CNS inflammation, metabolic/toxic disorders, and regional expansive lesions. Mimickers such as myasthenia gravis or Fisher syndrome and anti-GQ1b-mediated disorders have to be meticulously excluded. [1]: http://neurology.org/lookup/doi/10.1212/WNL.0000000000003096
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Teaching Video NeuroImages: Bilateral complete Horizontal Gaze Palsy with preserved convergence: The 1 + 1 syndrome
Neurology, 2016Co-Authors: Damien Biotti, Caroline TiliketeAbstract:Bilateral complete Horizontal Gaze Palsy, the 1 + 1 syndrome, is extremely rare. An otherwise healthy 25-year-old woman shows a recently acquired complete Horizontal saccadic Palsy. Its association with the absence of improvement with oculocephalic maneuvers and the preservation of convergence clinically suggests a bilateral cranial nerve VI nucleus dysfunction1,2 (video at [Neurology.org][1]; figures 1 and 2). Main causes include CNS inflammation, metabolic/toxic disorders, and regional expansive lesions. Mimickers such as myasthenia gravis or Fisher syndrome and anti-GQ1b-mediated disorders have to be meticulously excluded. [1]: http://neurology.org/lookup/doi/10.1212/WNL.0000000000003096
Neil R Miller - One of the best experts on this subject based on the ideXlab platform.
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reader response teaching video neuroimages Palsy of conjugate Horizontal Gaze and face due to isolated abducens nuclear infarction
Neurology, 2018Co-Authors: Neil R MillerAbstract:I read and viewed with interest the Teaching Video NeuroImage by Kim et al.1 Clearly, the reported patient did not have an “isolated” abducens nuclear infarction, as that would have caused a truly isolated ipsilateral Horizontal Gaze Palsy. The ipsilateral facial Palsy was caused by damage to the facial nerve fascicles adjacent to the abducens nucleus. Although the authors indicate this in the body of the text,1 the title of the article suggests that both the Horizontal Gaze Palsy and the facial Palsy were caused by the abducens nuclear infarction. I would hate for those who simply read the title of the otherwise excellent vignette to think that facial nerve fibers are somehow located within the abducens nerve nucleus. It is confusing enough for physicians to remember that the nucleus contains both axons destined for the ipsilateral lateral rectus and axons of the medial longitudinal fasciculus destined for the contralateral medial rectus so that abducens nuclear lesions cause an ipsilateral Horizontal Gaze Palsy rather than a sixth nerve Palsy.
Kenji Ohtsuka - One of the best experts on this subject based on the ideXlab platform.
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Vertical-torsional oscillations and dissociated bilateral Horizontal Gaze Palsy in a patient with a pontine cavernous angioma
Journal of Neurology Neurosurgery and Psychiatry, 2005Co-Authors: Noriaki Washio, Yasuo Suzuki, T Yamaki, M Kase, Kenji OhtsukaAbstract:We report the case of a 16 year old girl with vertical-torsional oscillations. She had a 4 year history of bilateral Horizontal Gaze Palsy caused by a cavernous angioma in the medial part of the dorsal pons. She presented with vertical oscillopsia that had worsened during the past 3 months. Unilateral three dimensional eye movements and bilateral Horizontal eye movements were recorded using a magnetic search coil method and direct current electro-oculography, respectively. She had vertical-torsional oscillations (average frequency: 3.0 Hz) leaving vertical saccades and pursuits intact. The average amplitudes of the vertical and torsional components were 2.0° and 0.6°, respectively. Her Horizontal rapid eye movements were severely impaired; however, her Horizontal pursuits and slow phases of vestibulo-ocular reflex were only partially impaired (gain
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vertical torsional oscillations and dissociated bilateral Horizontal Gaze Palsy in a patient with a pontine cavernous angioma
Journal of Neurology Neurosurgery and Psychiatry, 2005Co-Authors: Noriaki Washio, Yasuo Suzuki, T Yamaki, M Kase, Kenji OhtsukaAbstract:We report the case of a 16 year old girl with vertical-torsional oscillations. She had a 4 year history of bilateral Horizontal Gaze Palsy caused by a cavernous angioma in the medial part of the dorsal pons. She presented with vertical oscillopsia that had worsened during the past 3 months. Unilateral three dimensional eye movements and bilateral Horizontal eye movements were recorded using a magnetic search coil method and direct current electro-oculography, respectively. She had vertical-torsional oscillations (average frequency: 3.0 Hz) leaving vertical saccades and pursuits intact. The average amplitudes of the vertical and torsional components were 2.0° and 0.6°, respectively. Her Horizontal rapid eye movements were severely impaired; however, her Horizontal pursuits and slow phases of vestibulo-ocular reflex were only partially impaired (gain<0.3, oculomotor range<±9°). Convergence and divergence were intact. Lesions involving the medial part of the dorsal pons and bilateral paramedian pontine reticular formation can induce vertical and torsional oscillations without disruption of vertical rapid eye movements.