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N. Gurses - One of the best experts on this subject based on the ideXlab platform.
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Hypersensitivity vasculitis induced by cefoperazone/sulbactam
Annals of Clinical Microbiology and Antimicrobials, 2003Co-Authors: I Islek, Ali O Katranci, Ender Ariturk, Sancar Baris, N. GursesAbstract:Background Cefoperazone has not been reported to cause vasculitic complications before. Here, we report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam. Case presentation A 13-year-old girl with appendicitis developed Hypersensitivity vasculitis on the fifth day of cefoperazone/sulbactam therapy. Hypersensitivity vasculitis resolved gradually after removal of the agent on the seventh day and did not recur. Although Hypersensitivity vasculitis has multiple causes, coexistence of Hypersensitivity vasculitis and cefoperazone treatment, and the quite resolution of the disease after removal of the drug, strongly favours a causative relationship. Conclusion To our knowledge, this is the first report of a Hypersensitivity vasculitis associated with cefoperazone.
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Hypersensitivity vasculitis induced by cefoperazone/sulbactam.
Annals of clinical microbiology and antimicrobials, 2003Co-Authors: I Islek, Ali O Katranci, Ender Ariturk, Sancar Baris, N. GursesAbstract:Cefoperazone has not been reported to cause vasculitic complications before. Here, we report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam. A 13-year-old girl with appendicitis developed Hypersensitivity vasculitis on the fifth day of cefoperazone/sulbactam therapy. Hypersensitivity vasculitis resolved gradually after removal of the agent on the seventh day and did not recur. Although Hypersensitivity vasculitis has multiple causes, coexistence of Hypersensitivity vasculitis and cefoperazone treatment, and the quite resolution of the disease after removal of the drug, strongly favours a causative relationship. To our knowledge, this is the first report of a Hypersensitivity vasculitis associated with cefoperazone.
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Hypersensitivity vasculitis induced by cefoperazone/sulbactam
2003Co-Authors: I Islek, Ali O Katranci, Ender Ariturk, Sancar Baris, N. GursesAbstract:© 2003 Islek et al; licensee BioMed Central Ltd. This is an Open Access article: verbatim copying and redistribution of this article are permitted in all media for any purpose, provided this notice is preserved along with the article's original URL. Hypersensitivity vasculitiscefoperazone Background: Cefoperazone has not been reported to cause vasculitic complications before. Here, we report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam. Case presentation: A 13-year-old girl with appendicitis developed Hypersensitivity vasculitis on the fifth day of cefoperazone/sulbactam therapy. Hypersensitivity vasculitis resolved gradually after removal of the agent on the seventh day and did not recur. Although Hypersensitivity vasculitis has multiple causes, coexistence of Hypersensitivity vasculitis and cefoperazone treatment, and the quite resolution of the disease after removal of the drug, strongly favours a causative relationship. Conclusion: To our knowledge, this is the first report of a Hypersensitivity vasculitis associated with cefoperazone. Background Cefoperazone is a common antibiotic drug used mainly to treat serious infections. Reported adverse effects of this drug include urticaria, maculopapular and erythematous skin rash and rarely anaphylaxis and bronchospasm [1,2]. Cefoperazone has not been reported to cause vasculitic complications before. We report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam
M. K. Selgrade - One of the best experts on this subject based on the ideXlab platform.
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Hypersensitivity Reactions in the Respiratory Tract
Comprehensive Toxicology, 2010Co-Authors: Jean F. Regal, M. K. SelgradeAbstract:Chemicals and proteins can cause Hypersensitivity reactions in the respiratory tract via multiple mechanisms. Hypersensitivity responses require a latent period and develop in two stages: An induction or sensitization phase and an elicitation or effector phase with appearance of symptoms. Often, the Hypersensitivity reaction to a given allergen involves a combination of both humoral (Types I–III) and cell mediated (Type IV) components leading to disease. This chapter will focus on Hypersensitivity reactions in the respiratory tract that are most often encountered by toxicologists including allergic rhinitis, allergic asthma, anaphylaxis, and Hypersensitivity pneumonitis. In addition, chronic beryllium disease will be presented as an example of a Th1-mediated Hypersensitivity reaction. Current knowledge of mechanisms will be presented as well as animal models used in research in Hypersensitivity reactions in the respiratory tract.
V.j. Johnson - One of the best experts on this subject based on the ideXlab platform.
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Hypersensitivity Reactions in the Respiratory Tract
Comprehensive Toxicology, 2018Co-Authors: Stefanie C.m. Burleson, V.j. JohnsonAbstract:Chemicals and proteins can cause Hypersensitivity reactions in the respiratory tract via multiple mechanisms. Hypersensitivity responses require a latency period and develop in two stages: An induction or sensitization phase, during which the immune system learns to recognize the allergen, and an elicitation or effector phase with appearance of symptoms. Often, the Hypersensitivity reaction to a given allergen involves a combination of both humoral (Types I–III) and cell-mediated (Type IV) components leading to disease. This article focuses on Hypersensitivity reactions in the respiratory tract that are most often encountered by toxicologists including allergic rhinitis, allergic asthma, anaphylaxis, and Hypersensitivity pneumonitis. In addition, chronic beryllium disease will be presented as an example of a Th1-mediated Hypersensitivity reaction. Current knowledge of mechanisms will be presented as well as animal models used in research in Hypersensitivity reactions in the respiratory tract.
Mika J. Mäkelä - One of the best experts on this subject based on the ideXlab platform.
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The prognosis of wheat Hypersensitivity in children.
Pediatric Allergy and Immunology, 2009Co-Authors: Anne Kotaniemi-syrjänen, Kati Palosuo, Tuomas Jartti, Mikael Kuitunen, Anna S. Pelkonen, Mika J. MäkeläAbstract:Kotaniemi-Syrjanen A, Palosuo K, Jartti T, Kuitunen M, Pelkonen AS, Makela MJ. The prognosis of wheat Hypersensitivity in children. Pediatr Allergy Immunol 2010: 21: e421–e428. © 2009 John Wiley & Sons A/S The study was aimed to determine the natural history of wheat Hypersensitivity, to define risk factors for persistent wheat Hypersensitivity, and to evaluate the development of respiratory allergy in children with wheat Hypersensitivity. The development and subsequent disappearance of wheat Hypersensitivity, clinical findings, skin prick test (SPT) reactivity, and the development of allergic rhinoconjunctivitis and asthma were charted retrospectively in 28 children with wheat Hypersensitivity proven by the open oral challenge at the median age of 21 months (range 6 to 75 months). Appearance of skin symptoms during the diagnostic wheat challenge was related to SPT-positive wheat Hypersensitivity, while the appearance of gastrointestinal symptoms alone was associated with SPT-negative wheat Hypersensitivity (p = 0.002). Wheat was tolerated by 59%, 69%, 84%, and 96%, by age 4, 6, 10, and 16, respectively. Sensitization to gliadin with a SPT wheal of ≥5 mm at the time of the diagnostic challenge was associated with a slower course of recovery from wheat Hypersensitivity (p = 0.019), and a SPT wheal of ≥3 mm to gliadin at any time was associated with the development of asthma (p = 0.022). SPT reactivity to wheat was associated with later SPT reactivity to birch pollen (p = 0.001), and the development of allergic rhinoconjunctivitis (p = 0.001). In conclusion, almost all children with wheat Hypersensitivity can tolerate wheat by adolescence. Sensitization to gliadin is associated with a slower achievement of tolerance and an increased risk of asthma.
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The prognosis of wheat Hypersensitivity in children.
Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology, 2009Co-Authors: Anne Kotaniemi-syrjänen, Kati Palosuo, Tuomas Jartti, Mikael Kuitunen, Anna S. Pelkonen, Mika J. MäkeläAbstract:The study was aimed to determine the natural history of wheat Hypersensitivity, to define risk factors for persistent wheat Hypersensitivity, and to evaluate the development of respiratory allergy in children with wheat Hypersensitivity. The development and subsequent disappearance of wheat Hypersensitivity, clinical findings, skin prick test (SPT) reactivity, and the development of allergic rhinoconjunctivitis and asthma were charted retrospectively in 28 children with wheat Hypersensitivity proven by the open oral challenge at the median age of 21 months (range 6 to 75 months). Appearance of skin symptoms during the diagnostic wheat challenge was related to SPT-positive wheat Hypersensitivity, while the appearance of gastrointestinal symptoms alone was associated with SPT-negative wheat Hypersensitivity (p=0.002). Wheat was tolerated by 59%, 69%, 84%, and 96%, by age 4, 6, 10, and 16, respectively. Sensitization to gliadin with a SPT wheal of >or=5 mm at the time of the diagnostic challenge was associated with a slower course of recovery from wheat Hypersensitivity (p=0.019), and a SPT wheal of >or=3 mm to gliadin at any time was associated with the development of asthma (p=0.022). SPT reactivity to wheat was associated with later SPT reactivity to birch pollen (p=0.001), and the development of allergic rhinoconjunctivitis (p=0.001). In conclusion, almost all children with wheat Hypersensitivity can tolerate wheat by adolescence. Sensitization to gliadin is associated with a slower achievement of tolerance and an increased risk of asthma.
I Islek - One of the best experts on this subject based on the ideXlab platform.
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Hypersensitivity vasculitis induced by cefoperazone/sulbactam
Annals of Clinical Microbiology and Antimicrobials, 2003Co-Authors: I Islek, Ali O Katranci, Ender Ariturk, Sancar Baris, N. GursesAbstract:Background Cefoperazone has not been reported to cause vasculitic complications before. Here, we report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam. Case presentation A 13-year-old girl with appendicitis developed Hypersensitivity vasculitis on the fifth day of cefoperazone/sulbactam therapy. Hypersensitivity vasculitis resolved gradually after removal of the agent on the seventh day and did not recur. Although Hypersensitivity vasculitis has multiple causes, coexistence of Hypersensitivity vasculitis and cefoperazone treatment, and the quite resolution of the disease after removal of the drug, strongly favours a causative relationship. Conclusion To our knowledge, this is the first report of a Hypersensitivity vasculitis associated with cefoperazone.
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Hypersensitivity vasculitis induced by cefoperazone/sulbactam.
Annals of clinical microbiology and antimicrobials, 2003Co-Authors: I Islek, Ali O Katranci, Ender Ariturk, Sancar Baris, N. GursesAbstract:Cefoperazone has not been reported to cause vasculitic complications before. Here, we report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam. A 13-year-old girl with appendicitis developed Hypersensitivity vasculitis on the fifth day of cefoperazone/sulbactam therapy. Hypersensitivity vasculitis resolved gradually after removal of the agent on the seventh day and did not recur. Although Hypersensitivity vasculitis has multiple causes, coexistence of Hypersensitivity vasculitis and cefoperazone treatment, and the quite resolution of the disease after removal of the drug, strongly favours a causative relationship. To our knowledge, this is the first report of a Hypersensitivity vasculitis associated with cefoperazone.
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Hypersensitivity vasculitis induced by cefoperazone/sulbactam
2003Co-Authors: I Islek, Ali O Katranci, Ender Ariturk, Sancar Baris, N. GursesAbstract:© 2003 Islek et al; licensee BioMed Central Ltd. This is an Open Access article: verbatim copying and redistribution of this article are permitted in all media for any purpose, provided this notice is preserved along with the article's original URL. Hypersensitivity vasculitiscefoperazone Background: Cefoperazone has not been reported to cause vasculitic complications before. Here, we report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam. Case presentation: A 13-year-old girl with appendicitis developed Hypersensitivity vasculitis on the fifth day of cefoperazone/sulbactam therapy. Hypersensitivity vasculitis resolved gradually after removal of the agent on the seventh day and did not recur. Although Hypersensitivity vasculitis has multiple causes, coexistence of Hypersensitivity vasculitis and cefoperazone treatment, and the quite resolution of the disease after removal of the drug, strongly favours a causative relationship. Conclusion: To our knowledge, this is the first report of a Hypersensitivity vasculitis associated with cefoperazone. Background Cefoperazone is a common antibiotic drug used mainly to treat serious infections. Reported adverse effects of this drug include urticaria, maculopapular and erythematous skin rash and rarely anaphylaxis and bronchospasm [1,2]. Cefoperazone has not been reported to cause vasculitic complications before. We report a case of Hypersensitivity vasculitis associated with cefoperazone/sulbactam
