The Experts below are selected from a list of 2958 Experts worldwide ranked by ideXlab platform
Martin H. Markowitz - One of the best experts on this subject based on the ideXlab platform.
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women
Annals of Internal Medicine, 2020Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Abstract Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and th...
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women: A Distinct Syndrome
Annals of internal medicine, 1990Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and thrombocytosis. The Nodes displayed typical morphologic changes of the plasma cell variant of multicentric angiofollicular Lymph Node Hyperplasia. The pathologic changes are morphologically distinct from angioimmunoblastic Lymphadenopathy with dysproteinemia although clinical similarities do exist. In these four cases, the Lymphadenopathy was usually bulky and multicentric. There was frequent splenic involvement. The neuropathies were severe and disabling. Clinical courses have been variable with some responses to therapy with steroids and alkylating agents. No neoplastic transformations have occurred. Multicentric angiofollicular Lymph Node Hyperplasia may represent a reactive lesion in which the antigenic stimulus is unknown but results in follicular Hyperplasia, angiogenesis, and the systemic manifestations of hyperimmune stimulation. We believe this clinical syndrome may represent a distinct variant of multicentric angiofollicular Lymph Node Hyperplasia, and it requires close observation for neoplastic transformation and other complications of its multisystem nature.
John M. Feigert - One of the best experts on this subject based on the ideXlab platform.
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women
Annals of Internal Medicine, 2020Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Abstract Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and th...
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women: A Distinct Syndrome
Annals of internal medicine, 1990Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and thrombocytosis. The Nodes displayed typical morphologic changes of the plasma cell variant of multicentric angiofollicular Lymph Node Hyperplasia. The pathologic changes are morphologically distinct from angioimmunoblastic Lymphadenopathy with dysproteinemia although clinical similarities do exist. In these four cases, the Lymphadenopathy was usually bulky and multicentric. There was frequent splenic involvement. The neuropathies were severe and disabling. Clinical courses have been variable with some responses to therapy with steroids and alkylating agents. No neoplastic transformations have occurred. Multicentric angiofollicular Lymph Node Hyperplasia may represent a reactive lesion in which the antigenic stimulus is unknown but results in follicular Hyperplasia, angiogenesis, and the systemic manifestations of hyperimmune stimulation. We believe this clinical syndrome may represent a distinct variant of multicentric angiofollicular Lymph Node Hyperplasia, and it requires close observation for neoplastic transformation and other complications of its multisystem nature.
Morton Coleman - One of the best experts on this subject based on the ideXlab platform.
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women
Annals of Internal Medicine, 2020Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Abstract Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and th...
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women: A Distinct Syndrome
Annals of internal medicine, 1990Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and thrombocytosis. The Nodes displayed typical morphologic changes of the plasma cell variant of multicentric angiofollicular Lymph Node Hyperplasia. The pathologic changes are morphologically distinct from angioimmunoblastic Lymphadenopathy with dysproteinemia although clinical similarities do exist. In these four cases, the Lymphadenopathy was usually bulky and multicentric. There was frequent splenic involvement. The neuropathies were severe and disabling. Clinical courses have been variable with some responses to therapy with steroids and alkylating agents. No neoplastic transformations have occurred. Multicentric angiofollicular Lymph Node Hyperplasia may represent a reactive lesion in which the antigenic stimulus is unknown but results in follicular Hyperplasia, angiogenesis, and the systemic manifestations of hyperimmune stimulation. We believe this clinical syndrome may represent a distinct variant of multicentric angiofollicular Lymph Node Hyperplasia, and it requires close observation for neoplastic transformation and other complications of its multisystem nature.
Julius Schulman - One of the best experts on this subject based on the ideXlab platform.
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women
Annals of Internal Medicine, 2020Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Abstract Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and th...
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women: A Distinct Syndrome
Annals of internal medicine, 1990Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and thrombocytosis. The Nodes displayed typical morphologic changes of the plasma cell variant of multicentric angiofollicular Lymph Node Hyperplasia. The pathologic changes are morphologically distinct from angioimmunoblastic Lymphadenopathy with dysproteinemia although clinical similarities do exist. In these four cases, the Lymphadenopathy was usually bulky and multicentric. There was frequent splenic involvement. The neuropathies were severe and disabling. Clinical courses have been variable with some responses to therapy with steroids and alkylating agents. No neoplastic transformations have occurred. Multicentric angiofollicular Lymph Node Hyperplasia may represent a reactive lesion in which the antigenic stimulus is unknown but results in follicular Hyperplasia, angiogenesis, and the systemic manifestations of hyperimmune stimulation. We believe this clinical syndrome may represent a distinct variant of multicentric angiofollicular Lymph Node Hyperplasia, and it requires close observation for neoplastic transformation and other complications of its multisystem nature.
Donald L. Sweet - One of the best experts on this subject based on the ideXlab platform.
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women
Annals of Internal Medicine, 2020Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Abstract Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and th...
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Multicentric Angiofollicular Lymph Node Hyperplasia with Peripheral Neuropathy, Pseudotumor Cerebri, IgA Dysproteinemia, and Thrombocytosis in Women: A Distinct Syndrome
Annals of internal medicine, 1990Co-Authors: John M. Feigert, Donald L. Sweet, Morton Coleman, Diana Variakojis, Nathaniel Wisch, Julius Schulman, Martin H. MarkowitzAbstract:Four women with multicentric angiofollicular Lymph Node Hyperplasia had a distinct clinical syndrome characterized by peripheral neuropathy, pseudotumor cerebri, IgA dysproteinemia, and thrombocytosis. The Nodes displayed typical morphologic changes of the plasma cell variant of multicentric angiofollicular Lymph Node Hyperplasia. The pathologic changes are morphologically distinct from angioimmunoblastic Lymphadenopathy with dysproteinemia although clinical similarities do exist. In these four cases, the Lymphadenopathy was usually bulky and multicentric. There was frequent splenic involvement. The neuropathies were severe and disabling. Clinical courses have been variable with some responses to therapy with steroids and alkylating agents. No neoplastic transformations have occurred. Multicentric angiofollicular Lymph Node Hyperplasia may represent a reactive lesion in which the antigenic stimulus is unknown but results in follicular Hyperplasia, angiogenesis, and the systemic manifestations of hyperimmune stimulation. We believe this clinical syndrome may represent a distinct variant of multicentric angiofollicular Lymph Node Hyperplasia, and it requires close observation for neoplastic transformation and other complications of its multisystem nature.
