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L Xie - One of the best experts on this subject based on the ideXlab platform.

  • change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Chinese Journal of Industrial Hygiene and Occupational Diseases, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P<0.05) , with the increase in urinary Mercury concentration. Conclusion Patients with occupational Mercury Poisoning have a high rate of abnormal neural electromyographic findings, which can be used as an important method for early identification of chronic peripheral nerve injuries induced by Mercury Poisoning. The degree of peripheral nerve injuries increases with the increasing time of Mercury exposure and urinary Mercury concentration. Key words: Mercury; Poisoning; Nerve EMG; Peripheral nerves

  • Change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational di, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P

Paul I. Dantzig - One of the best experts on this subject based on the ideXlab platform.

  • persistent palmar plaques another possible cutaneous sign of Mercury Poisoning
    Journal of Toxicology-cutaneous and Ocular Toxicology, 2005
    Co-Authors: Paul I. Dantzig
    Abstract:

    Mercury Poisoning is becoming a health concern due to the extensive pollution of water and fish and the increasing consumption of fish in the human diet. Mercury is extremely toxic to the body, especially the central nervous system, but diagnosis is difficult due to the lack of specific signs. We report four patients with persistent palmar plaques that correlated with blood Mercury levels and cleared upon the elimination of Mercury from the body.

  • Persistent Palmar Plaques—Another Possible Cutaneous Sign of Mercury Poisoning
    Journal of Toxicology: Cutaneous and Ocular Toxicology, 2004
    Co-Authors: Paul I. Dantzig
    Abstract:

    Mercury Poisoning is becoming a health concern due to the extensive pollution of water and fish and the increasing consumption of fish in the human diet. Mercury is extremely toxic to the body, especially the central nervous system, but diagnosis is difficult due to the lack of specific signs. We report four patients with persistent palmar plaques that correlated with blood Mercury levels and cleared upon the elimination of Mercury from the body.

  • A new cutaneous sign of Mercury Poisoning
    Journal of the American Academy of Dermatology, 2003
    Co-Authors: Paul I. Dantzig
    Abstract:

    Chronic Mercury Poisoning is becoming a health concern because of extensive pollution of water and fish, and the increasing consumption of fish in the human diet. Mercury is extremely toxic to the body, especially the central nervous system, but diagnosis is difficult because of the lack of specific signs. A total of 11 patients were observed to have a nonpruritic or mildly pruritic discreet papular and papulovesicular eruption that correlated with high blood Mercury levels. The Mercury evidently came from increased seafood consumption. All of the patients improved when they were placed on either a seafood-free diet or chelation therapy. Physicians should suspect Mercury Poisoning in patients who eat a high-seafood diet who present with an asymptomatic or mildly pruritic papular or papulovesicular eruption.

B Jiang - One of the best experts on this subject based on the ideXlab platform.

  • change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Chinese Journal of Industrial Hygiene and Occupational Diseases, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P<0.05) , with the increase in urinary Mercury concentration. Conclusion Patients with occupational Mercury Poisoning have a high rate of abnormal neural electromyographic findings, which can be used as an important method for early identification of chronic peripheral nerve injuries induced by Mercury Poisoning. The degree of peripheral nerve injuries increases with the increasing time of Mercury exposure and urinary Mercury concentration. Key words: Mercury; Poisoning; Nerve EMG; Peripheral nerves

  • Change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational di, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P

Z X Yang - One of the best experts on this subject based on the ideXlab platform.

  • change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Chinese Journal of Industrial Hygiene and Occupational Diseases, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P<0.05) , with the increase in urinary Mercury concentration. Conclusion Patients with occupational Mercury Poisoning have a high rate of abnormal neural electromyographic findings, which can be used as an important method for early identification of chronic peripheral nerve injuries induced by Mercury Poisoning. The degree of peripheral nerve injuries increases with the increasing time of Mercury exposure and urinary Mercury concentration. Key words: Mercury; Poisoning; Nerve EMG; Peripheral nerves

  • Change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational di, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P

W R Dai - One of the best experts on this subject based on the ideXlab platform.

  • change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Chinese Journal of Industrial Hygiene and Occupational Diseases, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P<0.05) , with the increase in urinary Mercury concentration. Conclusion Patients with occupational Mercury Poisoning have a high rate of abnormal neural electromyographic findings, which can be used as an important method for early identification of chronic peripheral nerve injuries induced by Mercury Poisoning. The degree of peripheral nerve injuries increases with the increasing time of Mercury exposure and urinary Mercury concentration. Key words: Mercury; Poisoning; Nerve EMG; Peripheral nerves

  • Change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors
    Zhonghua lao dong wei sheng zhi ye bing za zhi = Zhonghua laodong weisheng zhiyebing zazhi = Chinese journal of industrial hygiene and occupational di, 2017
    Co-Authors: B Jiang, J Lai, W R Dai, W F Liu, Z X Yang, L Xie
    Abstract:

    Objective To investigate the change in peripheral nervous conduction velocity in patients with occupational chronic Mercury Poisoning and related influencing factors. Methods From February 2011 to December 2014, urinary Mercury examination and neural electromyography were performed for 104 patients with occupational chronic Mercury Poisoning. The data on age, type of work, working years of Mercury exposure, and past medical history were collected, and peripheral nervous conduction velocity and its correlation with age, working years of Mercury exposure, and urinary Mercury concentration were analyzed. Results All the 104 patients with occupational chronic Mercury Poisoning had a mean of 45.37±9.82 years, median (P25, P75) working years of 7 (3, 11) , and a median (P25, P75) urinary Mercury concentration of 88.50 (56.25, 163.03) μg/g Cr. The major clinical manifestations of peripheral nerve injuries were numbness of extremities (20.2%) , hypopselaphesia/hypalgesia or hyperpselaphesia/hyperalgesia (9.6%) , and bone/muscle pain in the extremities (6.7%) . Neural electromyography showed an increase in denervation potential (fibrillation potential or positive sharp wave) and a detection rate of abnormal peripheral nervous conduction velocity as high as 65.4%. The patients with an older age and more working years had a higher incidence rate of abnormal sensory conduction velocity of the ulnar nerve. There were significant reductions in motor and sensory conduction velocities of the median nerve, motor and sensory conduction velocities of the ulnar nerve, motor conduction velocity of the common peroneal nerve, and the sensory conduction velocity of the superficial peroneal nerve (P