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Andres Soto - One of the best experts on this subject based on the ideXlab platform.
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Peripheral Vertigo Classification. Consensus Document. Otoneurology Committee of the Spanish Otorhinolaryngology Society (2003–2006)
Acta Otorrinolaringologica, 2008Co-Authors: Constantino Morera, Herminio Perez, Nicolas Perez, Andres SotoAbstract:There are many different Vertigo classifications and different denominations are frequently used for the same clinical processes. The Otoneurology Committee of the Spanish Society for Otorhinolaryngology and Head and Neck Pathology proposes an eminently practical classification of peripheral Vertigo to facilitate a common terminology that can be easily used by general ENT practitioners. The methodology used has been by consensus within our society and especially among the most outstanding work groups in the area of otoneurology in Spain. Initially Vertigo is divided into single-episode Vertigo and recurring attacks of Vertigo, and these are then sub-divided into 2 groups, depending on whether or not hearing loss is present. Acute Vertigo without hearing loss corresponds to vestibular neuritis and if it is associated with hearing loss, it is due to labyrinthitis of different aetiologies and cochleovestibular neuritis. Recurrent Vertigos without hearing loss are classified as induced, either by posture (BPPV) or pressure (perilymphatic fistula), or as spontaneous, including Migraine-Associated Vertigo, metabolic Vertigo, childhood paroxysmal Vertigo, and Vertigo of vascular causes (TIAs, vertebrobasilar insufficiency). Finally, recurrent Vertigo with hearing loss includes Meniere's disease and others such as Vertigomigraine (with hearing loss), autoimmune pathology of the inner ear, syphilitic infection, and perilymphatic fistula (with hearing loss).
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peripheral Vertigo classification consensus document otoneurology committee of the spanish otorhinolaryngology society 2003 2006
Acta Otorrinolaringologica, 2008Co-Authors: Constantino Morera, Herminio Perez, Nicolas Perez, Andres SotoAbstract:There are many different Vertigo classifications and different denominations are frequently used for the same clinical processes. The Otoneurology Committee of the Spanish Society for Otorhinolaryngology and Head and Neck Pathology proposes an eminently practical classification of peripheral Vertigo to facilitate a common terminology that can be easily used by general ENT practitioners. The methodology used has been by consensus within our society and especially among the most outstanding work groups in the area of otoneurology in Spain. Initially Vertigo is divided into single-episode Vertigo and recurring attacks of Vertigo, and these are then sub-divided into 2 groups, depending on whether or not hearing loss is present. Acute Vertigo without hearing loss corresponds to vestibular neuritis and if it is associated with hearing loss, it is due to labyrinthitis of different aetiologies and cochleovestibular neuritis. Recurrent Vertigos without hearing loss are classified as induced, either by posture (BPPV) or pressure (perilymphatic fistula), or as spontaneous, including Migraine-Associated Vertigo, metabolic Vertigo, childhood paroxysmal Vertigo, and Vertigo of vascular causes (TIAs, vertebrobasilar insufficiency). Finally, recurrent Vertigo with hearing loss includes Meniere's disease and others such as Vertigomigraine (with hearing loss), autoimmune pathology of the inner ear, syphilitic infection, and perilymphatic fistula (with hearing loss).
Toshihisa Murofushi - One of the best experts on this subject based on the ideXlab platform.
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Otolithic Vertigo in children: Report of 3 cases
Acta Oto-Laryngologica Case Reports, 2016Co-Authors: Toshihisa Murofushi, Sakurako Komiyama, Ryota Suizu, Eriko YoshimuraAbstract:AbstractTilting and/or pulling sensation without a sensation of rotation might be caused by otolithic disorders and may be called otolithic Vertigo. We report 3 children (a 5-year-old boy, a 15-year-old girl, and an 8-year-old boy) who presented with episodic tilting or pulling sensation in the roll plane. Common findings of these 3 patients were unilateral abnormal ocular vestibular evoked myogenic potential responses (oVEMP). They did not show any other abnormal findings, and were diagnosed as idiopathic otolithic Vertigo (IOV). On the basis of their medical histories, their episodes might be caused by mechanism similar to Migraine-Associated Vertigo.
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does migraine associated Vertigo share a common pathophysiology with meniere s disease study with vestibular evoked myogenic potential
Cephalalgia, 2009Co-Authors: Toshihisa Murofushi, H Ozeki, A Inoue, A SakataAbstract:To clarify if Migraine-Associated Vertigo (MAV) and Meniere's disease (MD) share a common pathophysiology, vestibular-evoked myogenic potentials (VEMP) were measured in 11 patients with MAV, 11 with unilateral MD and eight healthy subjects. As acoustic stimuli, tone bursts (TB; 250, 500, 1000 and 2000 Hz) were presented. In healthy subjects, 500-Hz TB evoked the largest amplitude. To quantify this tendency, 500-1000 VEMP slope was calculated, and 500-1000 VEMP slope was the smallest on the affected side of MD patients. Among the 11 MD patients, five had significantly decreased 500-1000 VEMP asymmetry (shift of the tuning to 1000 Hz). Three of the 11 MAV patients also showed a significantly decreased 500-1000 VEMP slope. This finding suggests that MAV might share a common pathophysiology with MD. In addition to this finding, four of the other eight MAV patients showed prolonged p13 latencies. This suggests that MAV could consist of patients with different lesion sites.
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Does migraine‐associated Vertigo share a common pathophysiology with Meniere's disease? Study with vestibular‐evoked myogenic potential
Cephalalgia : an international journal of headache, 2009Co-Authors: Toshihisa Murofushi, H Ozeki, A Inoue, A SakataAbstract:To clarify if Migraine-Associated Vertigo (MAV) and Meniere's disease (MD) share a common pathophysiology, vestibular-evoked myogenic potentials (VEMP) were measured in 11 patients with MAV, 11 with unilateral MD and eight healthy subjects. As acoustic stimuli, tone bursts (TB; 250, 500, 1000 and 2000 Hz) were presented. In healthy subjects, 500-Hz TB evoked the largest amplitude. To quantify this tendency, 500-1000 VEMP slope was calculated, and 500-1000 VEMP slope was the smallest on the affected side of MD patients. Among the 11 MD patients, five had significantly decreased 500-1000 VEMP asymmetry (shift of the tuning to 1000 Hz). Three of the 11 MAV patients also showed a significantly decreased 500-1000 VEMP slope. This finding suggests that MAV might share a common pathophysiology with MD. In addition to this finding, four of the other eight MAV patients showed prolonged p13 latencies. This suggests that MAV could consist of patients with different lesion sites.
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Neuro-otological findings at the acute stage of Vertigo in migraine associated Vertigo: a report of 2 cases.
Equilibrium Research, 2009Co-Authors: Aki Sakata, Hidenori Ozeki, Haruka Nakahara, Toshihisa MurofushiAbstract:Although many patients with both episodic Vertigo and migraine have been reported, we have few opportunities to observe pathological nystagmus during an attack in these patients. We report herein on two cases with migraine associated Vertigo (MAV) in which neuro-otological findings could be observed at the acute stage of Vertigo. The first patient was a 46-year-old woman, with migraine without aura. She visited our clinic complaining of an episodic dizzy feeling with a migrainous headache and phonophobia. On the first examination, she did not show nystagmus or disequilibrium. Four months later, she had further acute Vertigo attacks, during which she showed right beating spontaneous nystagmus lasting for 3 hours. Her Vertigo attack and headache are currently well-controlled with lomerizine. The second case was a 47-year-old woman who visited our clinic complaining of Vertigo accompanied by a migrainous headache and left tinnitus. On the first examination, there were no remarkable findings except for 35% canal paresis of the left ear on caloric testing. Later, she showed direction fixed spontaneous nystagmus during a Vertigo attack lasting for a few days. This patient's Vertigo attack and headache have also been well-controlled with lomerizine. These cases showed that patients with MAV could have acute asymmetrical disorders of the vestibular system. The lesion site was considered to be in the peripheral vestibular system at least in the second case. However, the actual pathophysiology of MAV remains unclear.
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Migraine-Associated Vertigo: clinical characteristics of Japanese patients and effect of lomerizine, a calcium channel antagonist
Acta oto-laryngologica. Supplementum, 2007Co-Authors: Shinichi Iwasaki, Munetaka Ushio, Yasuhiro Chihara, Ken Ito, Keiko Sugasawa, Toshihisa MurofushiAbstract:Conclusion. Lomerizine, a calcium channel blocker, may be effective as a treatment for Migraine-Associated Vertigo (MAV). Objective. To determine the clinical characteristics of patients with MAV in Japan and the effectiveness of lomerizine. Patients and methods. This was a retrospective chart review carried out in a university hospital of 33 patients who fulfilled the diagnostic criteria for MAV. All patients were initially treated with dietary manipulation. If this therapy was unsuccessful, oral medications, mainly lomerizine, were administered. Medical records were reviewed to find clinical characteristics of patients with MAV and to evaluate the effects of the therapy on Vertigo/dizziness symptoms. Results. A marked female predominance was found (23 women, 10 men). The frequency and the duration of Vertigo varied across patients. About 60% of the patients had cochlear symptoms during an attack, among which bilateral aural fullness was most frequent. Oto-neurological examination showed abnormalities in...
A Sakata - One of the best experts on this subject based on the ideXlab platform.
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Does migraine‐associated Vertigo share a common pathophysiology with Meniere's disease? Study with vestibular‐evoked myogenic potential
Cephalalgia : an international journal of headache, 2009Co-Authors: Toshihisa Murofushi, H Ozeki, A Inoue, A SakataAbstract:To clarify if Migraine-Associated Vertigo (MAV) and Meniere's disease (MD) share a common pathophysiology, vestibular-evoked myogenic potentials (VEMP) were measured in 11 patients with MAV, 11 with unilateral MD and eight healthy subjects. As acoustic stimuli, tone bursts (TB; 250, 500, 1000 and 2000 Hz) were presented. In healthy subjects, 500-Hz TB evoked the largest amplitude. To quantify this tendency, 500-1000 VEMP slope was calculated, and 500-1000 VEMP slope was the smallest on the affected side of MD patients. Among the 11 MD patients, five had significantly decreased 500-1000 VEMP asymmetry (shift of the tuning to 1000 Hz). Three of the 11 MAV patients also showed a significantly decreased 500-1000 VEMP slope. This finding suggests that MAV might share a common pathophysiology with MD. In addition to this finding, four of the other eight MAV patients showed prolonged p13 latencies. This suggests that MAV could consist of patients with different lesion sites.
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does migraine associated Vertigo share a common pathophysiology with meniere s disease study with vestibular evoked myogenic potential
Cephalalgia, 2009Co-Authors: Toshihisa Murofushi, H Ozeki, A Inoue, A SakataAbstract:To clarify if Migraine-Associated Vertigo (MAV) and Meniere's disease (MD) share a common pathophysiology, vestibular-evoked myogenic potentials (VEMP) were measured in 11 patients with MAV, 11 with unilateral MD and eight healthy subjects. As acoustic stimuli, tone bursts (TB; 250, 500, 1000 and 2000 Hz) were presented. In healthy subjects, 500-Hz TB evoked the largest amplitude. To quantify this tendency, 500-1000 VEMP slope was calculated, and 500-1000 VEMP slope was the smallest on the affected side of MD patients. Among the 11 MD patients, five had significantly decreased 500-1000 VEMP asymmetry (shift of the tuning to 1000 Hz). Three of the 11 MAV patients also showed a significantly decreased 500-1000 VEMP slope. This finding suggests that MAV might share a common pathophysiology with MD. In addition to this finding, four of the other eight MAV patients showed prolonged p13 latencies. This suggests that MAV could consist of patients with different lesion sites.
Robert W Baloh - One of the best experts on this subject based on the ideXlab platform.
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Vestibular Migraine I: Mechanisms, Diagnosis, and Clinical Features.
Seminars in Neurology, 2020Co-Authors: Robert W BalohAbstract:Vestibular migraine (VM), also known as migrainous Vertigo or Migraine-Associated Vertigo, is characterized by recurrent vestibular attacks often accompanied by migraine headaches and other migraine symptoms. It is one of the most common presenting complaints to physicians in primary care, otolaryngology, and neurology. Epidemiologic data suggest that VM may affect 1 to 3% of the general population and 10 to 30% of patients seeking treatment for dizziness. Attacks typically last minutes to hours and range from spontaneous and positional Vertigo to extreme sensitivity to self and surround motion. As with headaches, nausea, and vomiting, phonophobia and photophobia are common accompanying symptoms. The clinical spectrum of VM and its underlying pathophysiological mechanisms are just being identified, with much debate about the causal relationship of vestibular symptoms and headache, no evidence-based guidelines for clinical management, limited characterization of its disease burden, and little information about its negative impact on health-related quality of life.
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Phenotypic and genetic analysis of a large family with Migraine-Associated Vertigo
Headache, 2007Co-Authors: Hane Lee, Yoon-hee Cha, Joanna C. Jen, Stanley F. Nelson, Robert W BalohAbstract:Objectives.— To describe a large multigenerational family with Migraine-Associated Vertigo (MAV) combining a detailed phenotypic and genetic analysis. Background.— Migraine-Associated Vertigo is said to be highly prevalent in the general population and, like other migraine syndromes, its etiology is felt to have a strong genetic component. However, so far, there have been no reports of large families with MAV. Methods.— Detailed clinical study was conducted on a large multigenerational family with MAV. Genetic study using identical-by-descent analysis with dense single nucleotide polymorphism (SNP) arrays was performed to examine consistent inheritance pattern among the affecteds. Results.— Clinical features of MAV were variable although most had other migraine symptoms with at least some of their attacks. We did not find a region of the genome shared by all eight subjects with MAV indicating a polygenetic inheritance for MAV even in this single large family. Conclusions.— A region on 11q shared by most affected females may contain a susceptibility allele for MAV that is expressed exclusively or predominantly by women.
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Migraine associated Vertigo.
Journal of Clinical Neurology, 2007Co-Authors: Robert W BalohAbstract:The interrelations of migraine and Vertigo are complex, eluding a simple localization either centrally or peripherally. Spontaneous episodic Vertigo, benign paroxysmal positional Vertigo, and Meniere's disease all occur more frequently in patients with migraine than in those without. Family studies support a hereditary predisposition to migraine associated Vertigo. In this review, we discuss definitions, epidemiology, associated syndromes, neurootological abnormalities, genetics and treatment for patients with migraine and Vertigo.
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Association of progesterone receptor with Migraine-Associated Vertigo
Neurogenetics, 2007Co-Authors: Hane Lee, Robert W Baloh, Yoon-hee Cha, Joanna C. Jen, Lauren Sininger, Stanley F. NelsonAbstract:While migraine has been demonstrated to be familial and have genetic contributions, genome-wide linkage analyses and candidate gene studies have highlighted that migraine is genetically complex. Despite substantial efforts, no consistent replication of linkage or association has been reported for common migraine syndromes. Among the candidate genes tested for association with migraine by several groups were female sex hormone genes based on the observation of a much higher incidence of migraine in females. Migraine-Associated Vertigo (MAV) is a migraine syndrome also much more common in females than males. Because MAV is less common in the general population than migraine or migraine with aura, it may be a better migraine syndrome to detect susceptibility alleles. In this study, we tested the association of two female hormonal genes, progesterone receptor ( PGR ) and estrogen receptor ( ESR1 ), which were previously reported to be associated with migraine in women. We typed 150 MAV subjects and 145 genomic matched control subjects. One SNP (rs1042838) within PGR , which is in high linkage disequilibrium with the functional PROGINS variant, was significantly associated with MAV ( p = 0.0007). Two SNPs (rs2228480 and rs1801132) within ESR1 demonstrated no significant association. No synergistic effect between ESR1 variants and PGR variants was identified.
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migraine associated Vertigo
Acta Oto-laryngologica, 2005Co-Authors: Krister Brantberg, Natalie Trees, Robert W BalohAbstract:Conclusions It is probably not wise to demand a temporal relationship between migraine symptoms and Vertigo for the definition of migrainous Vertigo. When recurrent Vertigo attacks begin at an early age in a patient with normal hearing and migraine, there are few diagnoses other than migraine that need to be considered. Objective The clinical association between migraine and vestibular symptoms, such as dizziness, motion intolerance and spontaneous attacks of Vertigo, is well documented. Recently, investigators have attempted to develop diagnostic criteria for this association. We hypothesized that there are multiple Migraine-Associated vestibular syndromes and studied a more homogenous subset of them (benign recurrent Vertigo). Material and methods A structured interview was conducted over the telephone with 40 patients who presented to our neurotology clinic with benign recurrent Vertigo and met the International Headache Society criteria for migraine. The structured interview was also conducted with 40...
R Giovanni - One of the best experts on this subject based on the ideXlab platform.
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EHMTI-0114. Vestibular migraine, prevalence in vestibular and headache centre and aids for diagnosis
Journal of Headache and Pain, 2014Co-Authors: C Mostardini, G Nola, R GiovanniAbstract:In population-based studies, lifetime prevalence of migraine and Vertigo in the general population of Western industrial nations is approximately 16% and 7%, respectively. A lot terms are used to describe combination of migraine and vestibular symptoms (migranous Vertigo, Migraine-Associated-Vertigo, vertiginous migraine), recently the International Headache Society and the Barany Society (International Society for NeuroOtology) created a consensus document with diagnostic criteria for Vestibular Migraine (VM) to clearly diagnose and compare patient populations in a standardized manner. Evaluate the prevalence of VM is particularly complex for its overlap between otolaryngology and headache experts. Our experience teaches us that, if main symptom of VM is headache, probably patient will be more easily diagnosed migraine but not VM, and if the main symptom is dizziness may not be interviewed on association with headache, failing the diagnosis. Aim of the study is to start a collaboration with the otolaringology to evaluate the utility of self-administered tests to facilitate diagnosis of VM in patients who access to vestibular or headaches clinics.