The Experts below are selected from a list of 198 Experts worldwide ranked by ideXlab platform
Wolfgang Auffermann - One of the best experts on this subject based on the ideXlab platform.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging
American Heart Journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, Charles B. HigginsAbstract:Abstract The current study used cine magnetic resonance imaging to determine the effect of increasing severity of vaivular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral ( n = 22) or aortic ( n = 17) regurgitation with ( n = 13) and without ( n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 ± 3% in normal volunteers, 64 ± 3% in patients with regurgitation, and 25 ± 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease. Cine MRI can be used to quantitate LV function in volume overload states and to characterize Myocardial Disease.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging.
American heart journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, C B HigginsAbstract:The current study used cine magnetic resonance imaging to determine the effect of increasing severity of valvular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral (n = 22) or aortic (n = 17) regurgitation with (n = 13) and without (n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 +/- 3% in normal volunteers, 64 +/- 3% in patients with regurgitation, and 25 +/- 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease.(ABSTRACT TRUNCATED AT 250 WORDS)
Charles B. Higgins - One of the best experts on this subject based on the ideXlab platform.
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Delayed enhancement magnetic resonance imaging in nonischemic Myocardial Disease.
Journal of thoracic imaging, 2013Co-Authors: Gustavo L. Moraes, Charles B. Higgins, Karen G. OrdovasAbstract:This review highlights the role of delayed enhancement magnetic resonance imaging for the diagnosis of patients with nonischemic Myocardial Disease. The authors discuss the use of delayed enhancement for differentiation between ischemic and nonischemic Myocardial Disease and for narrowing the differential diagnosis when nonischemic etiologies are suspected. In addition, special focus is given to the prognostic applications of delayed enhancement magnetic resonance imaging.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging
American Heart Journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, Charles B. HigginsAbstract:Abstract The current study used cine magnetic resonance imaging to determine the effect of increasing severity of vaivular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral ( n = 22) or aortic ( n = 17) regurgitation with ( n = 13) and without ( n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 ± 3% in normal volunteers, 64 ± 3% in patients with regurgitation, and 25 ± 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease. Cine MRI can be used to quantitate LV function in volume overload states and to characterize Myocardial Disease.
C B Higgins - One of the best experts on this subject based on the ideXlab platform.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging.
American heart journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, C B HigginsAbstract:The current study used cine magnetic resonance imaging to determine the effect of increasing severity of valvular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral (n = 22) or aortic (n = 17) regurgitation with (n = 13) and without (n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 +/- 3% in normal volunteers, 64 +/- 3% in patients with regurgitation, and 25 +/- 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease.(ABSTRACT TRUNCATED AT 250 WORDS)
Nelson B. Schiller - One of the best experts on this subject based on the ideXlab platform.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging
American Heart Journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, Charles B. HigginsAbstract:Abstract The current study used cine magnetic resonance imaging to determine the effect of increasing severity of vaivular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral ( n = 22) or aortic ( n = 17) regurgitation with ( n = 13) and without ( n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 ± 3% in normal volunteers, 64 ± 3% in patients with regurgitation, and 25 ± 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease. Cine MRI can be used to quantitate LV function in volume overload states and to characterize Myocardial Disease.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging.
American heart journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, C B HigginsAbstract:The current study used cine magnetic resonance imaging to determine the effect of increasing severity of valvular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral (n = 22) or aortic (n = 17) regurgitation with (n = 13) and without (n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 +/- 3% in normal volunteers, 64 +/- 3% in patients with regurgitation, and 25 +/- 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease.(ABSTRACT TRUNCATED AT 250 WORDS)
Tae Hwan Lim - One of the best experts on this subject based on the ideXlab platform.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging
American Heart Journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, Charles B. HigginsAbstract:Abstract The current study used cine magnetic resonance imaging to determine the effect of increasing severity of vaivular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral ( n = 22) or aortic ( n = 17) regurgitation with ( n = 13) and without ( n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 ± 3% in normal volunteers, 64 ± 3% in patients with regurgitation, and 25 ± 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease. Cine MRI can be used to quantitate LV function in volume overload states and to characterize Myocardial Disease.
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Noninvasive determination of left ventricular output and wall stress in volume overload and in Myocardial Disease by cine magnetic resonance imaging.
American heart journal, 1991Co-Authors: Wolfgang Auffermann, Stefan Wagner, William W. Holt, Peter T. Buser, Barbara Kircher, Nelson B. Schiller, Tae Hwan Lim, Christopher L. Wolfe, C B HigginsAbstract:The current study used cine magnetic resonance imaging to determine the effect of increasing severity of valvular regurgitation on systolic wall stress and to demonstrate that wall stress was disproportionately increased in relation to the severity of regurgitation in patients with Myocardial Disease. A total of 39 patients with predominantly mitral (n = 22) or aortic (n = 17) regurgitation with (n = 13) and without (n = 26) Myocardial Disease and 10 normal volunteers were examined with cine magnetic resonance imaging (MRI) at 1.5 T. Left ventricular (LV) cardiac output (CO) and peak systolic (PS) wall stress (WS) and end-systolic (ES) WS were calculated from blood pressure recordings, carotid pulse tracings, and wall thickness (h) and diameter (D) measurements obtained from cine MRI. Patients were classified into three degrees of severity according to their LV regurgitant volume (RV). Myocardial Disease was defined by an ejection fraction (EF) of less than 40%. Mean LV EF was 61 +/- 3% in normal volunteers, 64 +/- 3% in patients with regurgitation, and 25 +/- 2% in patients with Myocardial Disease. LV CO was directly related to RV in patients without Myocardial Disease, whereas it was disproportionately low in relation to RV in patients with Myocardial Disease. PS WS was significantly higher in severe mitral and/or aortic regurgitation compared with moderate, mild, and no mitral and/or aortic regurgitation. Compared with the degree of regurgitation, PS WS was disproportionately higher in patients with Myocardial Disease. Thus LV CO and WS rise progressively with increasing severity of regurgitation. Disproportionately high systolic WS relative to RV indicates the presence of Myocardial Disease.(ABSTRACT TRUNCATED AT 250 WORDS)
