The Experts below are selected from a list of 318 Experts worldwide ranked by ideXlab platform

Katherine D. Heidenreich - One of the best experts on this subject based on the ideXlab platform.

  • Nystagmus Discordance with 2-Dimensional Videonystagmography in Posterior Semicircular Canal Benign Paroxysmal Positional Vertigo
    Otolaryngology–Head and Neck Surgery, 2015
    Co-Authors: Crystal M. Vanderheyden, Wendy J. Carender, Katherine D. Heidenreich
    Abstract:

    ObjectiveThe Dix-Hallpike test is a standard component of the videonystagmography test battery and can diagnose posterior semicircular canal benign paroxysmal Positional Vertigo. The purpose of this study is to determine the prevalence of discordant, equivocal, and concordant nystagmus tracings in active posterior semicircular canal benign paroxysmal Positional Vertigo when compared directly with the eye video.Study DesignCase series with chart review of patients diagnosed with posterior semicircular canal benign paroxysmal Positional Vertigo by 2-dimensional videonystagmography from August 1, 2007, to August 1, 2012.SettingA tertiary vestibular test laboratory.Subjects and MethodsNinety-six adults (4 had bilateral involvement) with posterior semicircular canal benign paroxysmal Positional Vertigo were included. A total of 100 videos with accompanying videonystagmography tracings were reviewed to determine nystagmus trajectory as well as globe position. Descriptive statistics were used to describe prevale...

Crystal M. Vanderheyden - One of the best experts on this subject based on the ideXlab platform.

  • Nystagmus Discordance with 2-Dimensional Videonystagmography in Posterior Semicircular Canal Benign Paroxysmal Positional Vertigo
    Otolaryngology–Head and Neck Surgery, 2015
    Co-Authors: Crystal M. Vanderheyden, Wendy J. Carender, Katherine D. Heidenreich
    Abstract:

    ObjectiveThe Dix-Hallpike test is a standard component of the videonystagmography test battery and can diagnose posterior semicircular canal benign paroxysmal Positional Vertigo. The purpose of this study is to determine the prevalence of discordant, equivocal, and concordant nystagmus tracings in active posterior semicircular canal benign paroxysmal Positional Vertigo when compared directly with the eye video.Study DesignCase series with chart review of patients diagnosed with posterior semicircular canal benign paroxysmal Positional Vertigo by 2-dimensional videonystagmography from August 1, 2007, to August 1, 2012.SettingA tertiary vestibular test laboratory.Subjects and MethodsNinety-six adults (4 had bilateral involvement) with posterior semicircular canal benign paroxysmal Positional Vertigo were included. A total of 100 videos with accompanying videonystagmography tracings were reviewed to determine nystagmus trajectory as well as globe position. Descriptive statistics were used to describe prevale...

Douglas E Mattox - One of the best experts on this subject based on the ideXlab platform.

  • single treatment approaches to benign paroxysmal Positional Vertigo
    Archives of Otolaryngology-head & Neck Surgery, 1993
    Co-Authors: Susan J Herdman, Ronald J Tusa, Leonard R Proctor, Douglas E Mattox
    Abstract:

    • Objective. —To determine the effectiveness of two different physical therapy approaches for benign paroxysmal Positional Vertigo. Design. —Randomized study. Setting. —Outpatient clinic. Patients. —Consecutive sample of 60 patients with benign paroxysmal Positional Vertigo. Intervention. —Patients received either a single treatment based on the hypothesis that the Vertigo and nystagmus of benign paroxysmal Positional Vertigo are due to debris adhering to the cupula of the posterior semicircular canal (cupulolithiasis) or a single treatment based on the hypothesis that the debris is free floating in the long arm of the posterior canal (canalithiasis). Patients were reevaluated 1 to 2 weeks after the treatment and again 4 to 6 months later. Outcome. —Treatment outcome was classified as either asymptomatic, more than 70% improved as rated by the patient, or no change. Results. —The treatment designed for cupulolithiasis resulted in remission of Vertigo and nystagmus in 70% of the patients and in improvement of the symptoms in another 20%. The treatment designed for canalithiasis resulted in remission of Vertigo and nystagmus in 57% of the patients and in improvement in another 33%. There was no statistically significant difference between treatments. Conclusions. —These single-treatment approaches are equally effective treatments for benign paroxysmal Positional Vertigo. Further studies are needed to look at the long-term effectiveness of these treatments. ( Arch Otolaryngol Head Neck Surg. 1993;119:450-454)

John J. Zappia - One of the best experts on this subject based on the ideXlab platform.

  • causes of persistent Positional Vertigo following posterior semicircular canal occlusion for benign paroxysmal Positional Vertigo
    Otology & Neurotology, 2018
    Co-Authors: Alexander L Luryi, John J. Zappia, Christopher A Schutt, Dennis I Bojrab, Michael J Larouere, Eric W Sargent, Seilesh Babu
    Abstract:

    OBJECTIVE To report causes of persistent recalcitrant Positional Vertigo (PRPV) after posterior semicircular canal occlusion (PSCO) for benign paroxysmal Positional Vertigo (BPPV). STUDY DESIGN Retrospective chart review. SETTING Single high-volume otology practice. PATIENTS Patients diagnosed with BPPV from 2007 to 2017. INTERVENTION PSCO and follow-up care including diagnostic and particle repositioning maneuvers for recurrent BPPV. MAIN OUTCOME MEASURES PRPV, defined as recalcitrant Positional Vertigo for any reason following PSCO. RESULTS Twenty seven PSCO operations were performed in 26 patients. Twenty five patients (96.2%) had resolution of the Dix-Hallpike test in the operated ear. Eleven patients (42.3%) developed BPPV postoperatively, three (11.5%) in the operated ear and eight (30.8%) in the contralateral ear. Five of eight patients (62.5%) who developed contralateral BPPV had unilateral BPPV preoperatively. Eight patients (30.8%) developed BPPV at least twice after surgery or did not resolve, qualifying as PRPV, and all but one of these events occurred in the nonsurgical ear. No instances of cerebrospinal fluid leak, postoperative infection, facial palsy, clinically significant hearing loss, or death occurred. CONCLUSIONS PSCO is a safe and effective option for recalcitrant BPPV. However, 30.8% of patients, including patients with initially unilateral BPPV, had recalcitrant Positional Vertigo postoperatively, usually due to contralateral BPPV. Patients considering PSCO should be counseled regarding this risk to ensure realistic expectations.

  • Posterior semicircular canal occlusion for benign paroxysmal Positional Vertigo.
    The American journal of otology, 1996
    Co-Authors: John J. Zappia
    Abstract:

    For the small group of patients with benign paroxysmal Positional Vertigo that does not resolve with time or conservative treatment measures, posterior semicircular canal occlusion is considered. In the past 2 years, eight patients have undergone posterior semicircular canal occlusion at the Chicago Otology Group. All patients noted resolution of their Positional Vertigo. No complications were observed, particularly no sensorineural hearing loss was noted. A several-week period of disequilibrium is typical and appears to be the most bothersome aspect of the surgery to the patient.

Fuyuki Tateno - One of the best experts on this subject based on the ideXlab platform.

  • Visual Suppression is Impaired in Spinocerebellar Ataxia Type 6 but Preserved in Benign Paroxysmal Positional Vertigo
    Diagnostics (Basel Switzerland), 2012
    Co-Authors: Masahiko Kishi, Ryuji Sakakibara, Tomoe Yoshida, Masahiko Yamamoto, Mitsuya Suzuki, Manabu Kataoka, Yohei Tsuyusaki, Akihiko Tateno, Fuyuki Tateno
    Abstract:

    Positional Vertigo is a common neurologic emergency and mostly the etiology is peripheral. However, central diseases may mimic peripheral Positional Vertigo at their initial presentation. We here describe the results of a visual suppression test in six patients with spinocerebellar ataxia type 6 (SCA6), a central Positional Vertigo, and nine patients with benign paroxysmal Positional Vertigo (BPPV), the major peripheral Positional Vertigo. As a result, the visual suppression value of both diseases differed significantly; e.g., 22.5% in SCA6 and 64.3% in BPPV (p < 0.001). There was a positive correlation between the visual suppression value and disease duration, cerebellar atrophy, and CAG repeat length of SCA6 but they were not statistically significant. In conclusion, the present study showed for the first time that visual suppression is impaired in SCA6, a central Positional Vertigo, but preserved in BPPV, the major peripheral Positional Vertigo, by directly comparing both groups. The abnormality in the SCA6 group presumably reflects dysfunction in the central visual fixation pathway at the cerebellar flocculus and nodulus. This simple test might aid differential diagnosis of peripheral and central Positional Vertigo at the earlier stage of disease.

  • Visual Suppression is Impaired in Spinocerebellar Ataxia Type 6 but Preserved in Benign Paroxysmal Positional Vertigo
    MDPI AG, 2012
    Co-Authors: Masahiko Kishi, Ryuji Sakakibara, Tomoe Yoshida, Masahiko Yamamoto, Mitsuya Suzuki, Manabu Kataoka, Yohei Tsuyusaki, Akihiko Tateno, Fuyuki Tateno
    Abstract:

    Positional Vertigo is a common neurologic emergency and mostly the etiology is peripheral. However, central diseases may mimic peripheral Positional Vertigo at their initial presentation. We here describe the results of a visual suppression test in six patients with spinocerebellar ataxia type 6 (SCA6), a central Positional Vertigo, and nine patients with benign paroxysmal Positional Vertigo (BPPV), the major peripheral Positional Vertigo. As a result, the visual suppression value of both diseases differed significantly; e.g., 22.5% in SCA6 and 64.3% in BPPV (p < 0.001). There was a positive correlation between the visual suppression value and disease duration, cerebellar atrophy, and CAG repeat length of SCA6 but they were not statistically significant. In conclusion, the present study showed for the first time that visual suppression is impaired in SCA6, a central Positional Vertigo, but preserved in BPPV, the major peripheral Positional Vertigo, by directly comparing both groups. The abnormality in the SCA6 group presumably reflects dysfunction in the central visual fixation pathway at the cerebellar flocculus and nodulus. This simple test might aid differential diagnosis of peripheral and central Positional Vertigo at the earlier stage of disease.disease