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Anthony P Farrell - One of the best experts on this subject based on the ideXlab platform.
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locomotory behaviour and post exercise physiology in relation to swimming speed Gait transition and metabolism in free swimming smallmouth bass micropterus dolomieu
The Journal of Experimental Biology, 2004Co-Authors: Stephan J Peake, Anthony P FarrellAbstract:We examined swimming behaviour, Gait recruitment and post-exercise muscle glycogen, muscle lactate, plasma lactate and oxygen consumption in smallmouth bass ( Micropterus dolomieu ; 24–38 cm fork length) that voluntarily ascended a 25 m raceway against water velocities ranging from 40 to 120 cm s–1. Physiological parameters were referenced to additional measurements made following exhaustive exercise in a static tank and aerobic exercise in a swim tunnel. Maximum speeds maintained exclusively using a steady Gait in the raceway ranged from 53.6 to 97.3 cm s–1 and scaled positively with fish length. Minimum swimming speeds maintained exclusively through recruitment of an Unsteady Gait were also positively correlated to fish length and ranged from 81.4 to 122.9 cm s–1. Fish switched between steady and Unsteady swimming at intermediate speeds. Smallmouth bass always maintained a positive ground speed in the raceway; however, those that primarily swam using a steady Gait to overcome low to moderate water velocities (20–50 cm s–1) maintained mean ground speeds of ∼20 cm s–1. By contrast, mean ground speeds of fish that primarily recruited an Unsteady locomotory Gait increased significantly with water velocity, which resulted in an inverse relationship between exercise intensity and duration. We interpret this behaviour as evidence that Unsteady swimming was being fuelled by the limited supply of anaerobic substrates in the white muscle. This hypothesis is supported by the fact that Unsteady swimming fish showed significantly lower muscle glycogen levels, higher lactate concentrations (muscle and plasma) and higher post-exercise oxygen consumption rates compared with fish that used a steady Gait. The reduction in passage time achieved by fish using an Unsteady Gait allowed them to ascend the raceway with relatively minor post-exercise metabolic imbalances, relative to individuals chased to exhaustion.
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locomotory behaviour and post exercise physiology in relation to swimming speed Gait transition and metabolism in free swimming smallmouth bass micropterus dolomieu
The Journal of Experimental Biology, 2004Co-Authors: Stephan J Peake, Anthony P FarrellAbstract:We examined swimming behaviour, Gait recruitment and post-exercise muscle glycogen, muscle lactate, plasma lactate and oxygen consumption in smallmouth bass (Micropterus dolomieu; 24-38 cm fork length) that voluntarily ascended a 25 m raceway against water velocities ranging from 40 to 120 cm s(-1). Physiological parameters were referenced to additional measurements made following exhaustive exercise in a static tank and aerobic exercise in a swim tunnel. Maximum speeds maintained exclusively using a steady Gait in the raceway ranged from 53.6 to 97.3 cm s(-1) and scaled positively with fish length. Minimum swimming speeds maintained exclusively through recruitment of an Unsteady Gait were also positively correlated to fish length and ranged from 81.4 to 122.9 cm s(-1). Fish switched between steady and Unsteady swimming at intermediate speeds. Smallmouth bass always maintained a positive ground speed in the raceway; however, those that primarily swam using a steady Gait to overcome low to moderate water velocities (20-50 cm s(-1)) maintained mean ground speeds of approximately 20 cm s(-1). By contrast, mean ground speeds of fish that primarily recruited an Unsteady locomotory Gait increased significantly with water velocity, which resulted in an inverse relationship between exercise intensity and duration. We interpret this behaviour as evidence that Unsteady swimming was being fuelled by the limited supply of anaerobic substrates in the white muscle. This hypothesis is supported by the fact that Unsteady swimming fish showed significantly lower muscle glycogen levels, higher lactate concentrations (muscle and plasma) and higher post-exercise oxygen consumption rates compared with fish that used a steady Gait. The reduction in passage time achieved by fish using an Unsteady Gait allowed them to ascend the raceway with relatively minor post-exercise metabolic imbalances, relative to individuals chased to exhaustion.
Stephan J Peake - One of the best experts on this subject based on the ideXlab platform.
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locomotory behaviour and post exercise physiology in relation to swimming speed Gait transition and metabolism in free swimming smallmouth bass micropterus dolomieu
The Journal of Experimental Biology, 2004Co-Authors: Stephan J Peake, Anthony P FarrellAbstract:We examined swimming behaviour, Gait recruitment and post-exercise muscle glycogen, muscle lactate, plasma lactate and oxygen consumption in smallmouth bass ( Micropterus dolomieu ; 24–38 cm fork length) that voluntarily ascended a 25 m raceway against water velocities ranging from 40 to 120 cm s–1. Physiological parameters were referenced to additional measurements made following exhaustive exercise in a static tank and aerobic exercise in a swim tunnel. Maximum speeds maintained exclusively using a steady Gait in the raceway ranged from 53.6 to 97.3 cm s–1 and scaled positively with fish length. Minimum swimming speeds maintained exclusively through recruitment of an Unsteady Gait were also positively correlated to fish length and ranged from 81.4 to 122.9 cm s–1. Fish switched between steady and Unsteady swimming at intermediate speeds. Smallmouth bass always maintained a positive ground speed in the raceway; however, those that primarily swam using a steady Gait to overcome low to moderate water velocities (20–50 cm s–1) maintained mean ground speeds of ∼20 cm s–1. By contrast, mean ground speeds of fish that primarily recruited an Unsteady locomotory Gait increased significantly with water velocity, which resulted in an inverse relationship between exercise intensity and duration. We interpret this behaviour as evidence that Unsteady swimming was being fuelled by the limited supply of anaerobic substrates in the white muscle. This hypothesis is supported by the fact that Unsteady swimming fish showed significantly lower muscle glycogen levels, higher lactate concentrations (muscle and plasma) and higher post-exercise oxygen consumption rates compared with fish that used a steady Gait. The reduction in passage time achieved by fish using an Unsteady Gait allowed them to ascend the raceway with relatively minor post-exercise metabolic imbalances, relative to individuals chased to exhaustion.
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locomotory behaviour and post exercise physiology in relation to swimming speed Gait transition and metabolism in free swimming smallmouth bass micropterus dolomieu
The Journal of Experimental Biology, 2004Co-Authors: Stephan J Peake, Anthony P FarrellAbstract:We examined swimming behaviour, Gait recruitment and post-exercise muscle glycogen, muscle lactate, plasma lactate and oxygen consumption in smallmouth bass (Micropterus dolomieu; 24-38 cm fork length) that voluntarily ascended a 25 m raceway against water velocities ranging from 40 to 120 cm s(-1). Physiological parameters were referenced to additional measurements made following exhaustive exercise in a static tank and aerobic exercise in a swim tunnel. Maximum speeds maintained exclusively using a steady Gait in the raceway ranged from 53.6 to 97.3 cm s(-1) and scaled positively with fish length. Minimum swimming speeds maintained exclusively through recruitment of an Unsteady Gait were also positively correlated to fish length and ranged from 81.4 to 122.9 cm s(-1). Fish switched between steady and Unsteady swimming at intermediate speeds. Smallmouth bass always maintained a positive ground speed in the raceway; however, those that primarily swam using a steady Gait to overcome low to moderate water velocities (20-50 cm s(-1)) maintained mean ground speeds of approximately 20 cm s(-1). By contrast, mean ground speeds of fish that primarily recruited an Unsteady locomotory Gait increased significantly with water velocity, which resulted in an inverse relationship between exercise intensity and duration. We interpret this behaviour as evidence that Unsteady swimming was being fuelled by the limited supply of anaerobic substrates in the white muscle. This hypothesis is supported by the fact that Unsteady swimming fish showed significantly lower muscle glycogen levels, higher lactate concentrations (muscle and plasma) and higher post-exercise oxygen consumption rates compared with fish that used a steady Gait. The reduction in passage time achieved by fish using an Unsteady Gait allowed them to ascend the raceway with relatively minor post-exercise metabolic imbalances, relative to individuals chased to exhaustion.
Tao Jiang - One of the best experts on this subject based on the ideXlab platform.
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nitrous oxide induced subacute combined degeneration with longitudinally extensive myelopathy with inverted v sign on spinal mri a case report and literature review
BMC Neurology, 2017Co-Authors: Junliang Yuan, Shuang Kun Wang, Tao JiangAbstract:Nitrous oxide (N2O), a long-standing anesthetic, is also neurotoxic by interfering with the bioavailability of vitamin B12 if abused. A few case studies have reported the neurological and psychiatric complications of N2O. Here, we reported a patient of N2O induced subacute combined degeneration (SCD) with longitudinally extensive myelopathy with inverted V-sign exhibiting progressive limb paresthesia and Unsteady Gait. This case raises the awareness of an important mechanism of neural toxicity of N2O, and clinical physicians should be well recognized this in the field of substance-related disorders.
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Nitrous oxide induced subacute combined degeneration with longitudinally extensive myelopathy with inverted V-sign on spinal MRI: a case report and literature review
BMC, 2017Co-Authors: Junliang Yuan, Shuang Kun Wang, Tao JiangAbstract:Abstract Background Nitrous oxide (N2O), a long-standing anesthetic, is also neurotoxic by interfering with the bioavailability of vitamin B12 if abused. A few case studies have reported the neurological and psychiatric complications of N2O. Case presentation Here, we reported a patient of N2O induced subacute combined degeneration (SCD) with longitudinally extensive myelopathy with inverted V-sign exhibiting progressive limb paresthesia and Unsteady Gait. Conclusions This case raises the awareness of an important mechanism of neural toxicity of N2O, and clinical physicians should be well recognized this in the field of substance-related disorders
Junliang Yuan - One of the best experts on this subject based on the ideXlab platform.
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nitrous oxide induced subacute combined degeneration with longitudinally extensive myelopathy with inverted v sign on spinal mri a case report and literature review
BMC Neurology, 2017Co-Authors: Junliang Yuan, Shuang Kun Wang, Tao JiangAbstract:Nitrous oxide (N2O), a long-standing anesthetic, is also neurotoxic by interfering with the bioavailability of vitamin B12 if abused. A few case studies have reported the neurological and psychiatric complications of N2O. Here, we reported a patient of N2O induced subacute combined degeneration (SCD) with longitudinally extensive myelopathy with inverted V-sign exhibiting progressive limb paresthesia and Unsteady Gait. This case raises the awareness of an important mechanism of neural toxicity of N2O, and clinical physicians should be well recognized this in the field of substance-related disorders.
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Nitrous oxide induced subacute combined degeneration with longitudinally extensive myelopathy with inverted V-sign on spinal MRI: a case report and literature review
BMC, 2017Co-Authors: Junliang Yuan, Shuang Kun Wang, Tao JiangAbstract:Abstract Background Nitrous oxide (N2O), a long-standing anesthetic, is also neurotoxic by interfering with the bioavailability of vitamin B12 if abused. A few case studies have reported the neurological and psychiatric complications of N2O. Case presentation Here, we reported a patient of N2O induced subacute combined degeneration (SCD) with longitudinally extensive myelopathy with inverted V-sign exhibiting progressive limb paresthesia and Unsteady Gait. Conclusions This case raises the awareness of an important mechanism of neural toxicity of N2O, and clinical physicians should be well recognized this in the field of substance-related disorders
Hindi Al Hindi - One of the best experts on this subject based on the ideXlab platform.
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Papillary tumor of the pineal region presenting with Foster Kennedy sign
Hematology Oncology and Stem Cell Therapy, 2015Co-Authors: Joyce N. Mbekeani, Khaled Abdulshafi, Maher I Hassounah, Selwa A. Al Hazzaa, Manzoor Ahmed, Hindi Al HindiAbstract:Foster Kennedy syndrome is defined as unilateral optic nerve atrophy, contralateral disc edema and anosmia. It was originally thought to be pathognomonic of space occupying lesions in the anterior fossa causing direct compression of the optic and olfactory nerves and later intracranial hypertension and contralateral disc edema. Subsequent reports of non-tumor and non-anterior fossa causes, most commonly bilateral sequential anterior ischemic optic neuropathy, have been described. We report a 29 year old man who presented with Unsteady Gait and diminution of vision over 2 months. He displayed findings typical of Parinaud’s syndrome, right disc edema and left optic atrophy. MRI revealed a large pineal tumor associated with hydrocephalus and evidence of increased intracranial pressure. Papillary tumor of the pineal region (PTPR) was diagnosed on histopathology. To our knowledge, this is the first report of this very rare tumor, occurring in association with the Foster Kennedy sign.
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Papillary tumor of the pineal region presenting with Foster Kennedy sign
'Elsevier BV', 2015Co-Authors: Joyce N. Mbekeani, Khaled Abdulshafi, Maher I Hassounah, Selwa A. Al Hazzaa, Manzoor Ahmed, Hindi Al HindiAbstract:Foster Kennedy syndrome is defined as unilateral optic nerve atrophy, contralateral disc edema and anosmia. It was originally thought to be pathognomonic of space occupying lesions in the anterior fossa causing direct compression of the optic and olfactory nerves and later intracranial hypertension and contralateral disc edema. Subsequent reports of non-tumor and non-anterior fossa causes, most commonly bilateral sequential anterior ischemic optic neuropathy, have been described. We report a 29 year old man who presented with Unsteady Gait and diminution of vision over 2 months. He displayed findings typical of Parinaud’s syndrome, right disc edema and left optic atrophy. MRI revealed a large pineal tumor associated with hydrocephalus and evidence of increased intracranial pressure. Papillary tumor of the pineal region (PTPR) was diagnosed on histopathology. To our knowledge, this is the first report of this very rare tumor, occurring in association with the Foster Kennedy sign. Keywords: Foster Kennedy syndrome, Intracranial hypertension, Optic atrophy, Papillary tumor of the pineal region, Papilledem
