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Trevor A. Jackson - One of the best experts on this subject based on the ideXlab platform.

  • Cloning Serratia entomophila Antifeeding Genes—a Putative Defective Prophage Active against the Grass Grub Costelytra zealandica
    Journal of bacteriology, 2004
    Co-Authors: Mark R. H. Hurst, Travis R. Glare, Trevor A. Jackson
    Abstract:

    Serratia entomophila and Serratia proteamaculans (Enterobacteriaceae) cause amber disease in the grass grub Costelytra zealandica (Coleoptera: Scarabaeidae), an important pasture pest in New Zealand. Larval disease symptoms include cessation of feeding, clearance of the gut, amber coloration, and eventual death. A 155-kb plasmid, pADAP, carries the genes sepA, sepB, and sepC, which are essential for production of amber disease symptoms. Transposon insertions in any of the sep genes in pADAP abolish gut clearance but not cessation of feeding, indicating the presence of an Antifeeding gene(s) elsewhere on pADAP. Based on deletion analysis of pADAP and subsequent sequence data, a 47-kb clone was constructed, which when placed in either an Escherichia coli or a Serratia background exerted strong Antifeeding Activity and often led to rapid death of the infected grass grub larvae. Sequence data show that the Antifeeding component is part of a large gene cluster that may form a defective prophage and that six potential members of this prophage are present in Photorhabdus luminescens subsp. laumondii TTO1, a species which also has sep gene homologues.

  • cloning serratia entomophila Antifeeding genes a putative defective prophage active against the grass grub costelytra zealandica
    Journal of Bacteriology, 2004
    Co-Authors: Mark R. H. Hurst, Travis R. Glare, Trevor A. Jackson
    Abstract:

    Serratia entomophila and Serratia proteamaculans (Enterobacteriaceae) cause amber disease in the grass grub Costelytra zealandica (Coleoptera: Scarabaeidae), an important pasture pest in New Zealand. Larval disease symptoms include cessation of feeding, clearance of the gut, amber coloration, and eventual death. A 155-kb plasmid, pADAP, carries the genes sepA, sepB, and sepC, which are essential for production of amber disease symptoms. Transposon insertions in any of the sep genes in pADAP abolish gut clearance but not cessation of feeding, indicating the presence of an Antifeeding gene(s) elsewhere on pADAP. Based on deletion analysis of pADAP and subsequent sequence data, a 47-kb clone was constructed, which when placed in either an Escherichia coli or a Serratia background exerted strong Antifeeding Activity and often led to rapid death of the infected grass grub larvae. Sequence data show that the Antifeeding component is part of a large gene cluster that may form a defective prophage and that six potential members of this prophage are present in Photorhabdus luminescens subsp. laumondii TTO1, a species which also has sep gene homologues.

Mark R. H. Hurst - One of the best experts on this subject based on the ideXlab platform.

  • Cloning Serratia entomophila Antifeeding Genes—a Putative Defective Prophage Active against the Grass Grub Costelytra zealandica
    Journal of bacteriology, 2004
    Co-Authors: Mark R. H. Hurst, Travis R. Glare, Trevor A. Jackson
    Abstract:

    Serratia entomophila and Serratia proteamaculans (Enterobacteriaceae) cause amber disease in the grass grub Costelytra zealandica (Coleoptera: Scarabaeidae), an important pasture pest in New Zealand. Larval disease symptoms include cessation of feeding, clearance of the gut, amber coloration, and eventual death. A 155-kb plasmid, pADAP, carries the genes sepA, sepB, and sepC, which are essential for production of amber disease symptoms. Transposon insertions in any of the sep genes in pADAP abolish gut clearance but not cessation of feeding, indicating the presence of an Antifeeding gene(s) elsewhere on pADAP. Based on deletion analysis of pADAP and subsequent sequence data, a 47-kb clone was constructed, which when placed in either an Escherichia coli or a Serratia background exerted strong Antifeeding Activity and often led to rapid death of the infected grass grub larvae. Sequence data show that the Antifeeding component is part of a large gene cluster that may form a defective prophage and that six potential members of this prophage are present in Photorhabdus luminescens subsp. laumondii TTO1, a species which also has sep gene homologues.

  • cloning serratia entomophila Antifeeding genes a putative defective prophage active against the grass grub costelytra zealandica
    Journal of Bacteriology, 2004
    Co-Authors: Mark R. H. Hurst, Travis R. Glare, Trevor A. Jackson
    Abstract:

    Serratia entomophila and Serratia proteamaculans (Enterobacteriaceae) cause amber disease in the grass grub Costelytra zealandica (Coleoptera: Scarabaeidae), an important pasture pest in New Zealand. Larval disease symptoms include cessation of feeding, clearance of the gut, amber coloration, and eventual death. A 155-kb plasmid, pADAP, carries the genes sepA, sepB, and sepC, which are essential for production of amber disease symptoms. Transposon insertions in any of the sep genes in pADAP abolish gut clearance but not cessation of feeding, indicating the presence of an Antifeeding gene(s) elsewhere on pADAP. Based on deletion analysis of pADAP and subsequent sequence data, a 47-kb clone was constructed, which when placed in either an Escherichia coli or a Serratia background exerted strong Antifeeding Activity and often led to rapid death of the infected grass grub larvae. Sequence data show that the Antifeeding component is part of a large gene cluster that may form a defective prophage and that six potential members of this prophage are present in Photorhabdus luminescens subsp. laumondii TTO1, a species which also has sep gene homologues.

Travis R. Glare - One of the best experts on this subject based on the ideXlab platform.

  • Cloning Serratia entomophila Antifeeding Genes—a Putative Defective Prophage Active against the Grass Grub Costelytra zealandica
    Journal of bacteriology, 2004
    Co-Authors: Mark R. H. Hurst, Travis R. Glare, Trevor A. Jackson
    Abstract:

    Serratia entomophila and Serratia proteamaculans (Enterobacteriaceae) cause amber disease in the grass grub Costelytra zealandica (Coleoptera: Scarabaeidae), an important pasture pest in New Zealand. Larval disease symptoms include cessation of feeding, clearance of the gut, amber coloration, and eventual death. A 155-kb plasmid, pADAP, carries the genes sepA, sepB, and sepC, which are essential for production of amber disease symptoms. Transposon insertions in any of the sep genes in pADAP abolish gut clearance but not cessation of feeding, indicating the presence of an Antifeeding gene(s) elsewhere on pADAP. Based on deletion analysis of pADAP and subsequent sequence data, a 47-kb clone was constructed, which when placed in either an Escherichia coli or a Serratia background exerted strong Antifeeding Activity and often led to rapid death of the infected grass grub larvae. Sequence data show that the Antifeeding component is part of a large gene cluster that may form a defective prophage and that six potential members of this prophage are present in Photorhabdus luminescens subsp. laumondii TTO1, a species which also has sep gene homologues.

  • cloning serratia entomophila Antifeeding genes a putative defective prophage active against the grass grub costelytra zealandica
    Journal of Bacteriology, 2004
    Co-Authors: Mark R. H. Hurst, Travis R. Glare, Trevor A. Jackson
    Abstract:

    Serratia entomophila and Serratia proteamaculans (Enterobacteriaceae) cause amber disease in the grass grub Costelytra zealandica (Coleoptera: Scarabaeidae), an important pasture pest in New Zealand. Larval disease symptoms include cessation of feeding, clearance of the gut, amber coloration, and eventual death. A 155-kb plasmid, pADAP, carries the genes sepA, sepB, and sepC, which are essential for production of amber disease symptoms. Transposon insertions in any of the sep genes in pADAP abolish gut clearance but not cessation of feeding, indicating the presence of an Antifeeding gene(s) elsewhere on pADAP. Based on deletion analysis of pADAP and subsequent sequence data, a 47-kb clone was constructed, which when placed in either an Escherichia coli or a Serratia background exerted strong Antifeeding Activity and often led to rapid death of the infected grass grub larvae. Sequence data show that the Antifeeding component is part of a large gene cluster that may form a defective prophage and that six potential members of this prophage are present in Photorhabdus luminescens subsp. laumondii TTO1, a species which also has sep gene homologues.

Shin-ichi Tebayashi - One of the best experts on this subject based on the ideXlab platform.

  • Antifeedants of Indian barnyard millet, Echinochloa frumentacea link, against brown planthopper, Nilaparvata lugens (Stål).
    Zeitschrift für Naturforschung C, 2008
    Co-Authors: Kabir Md Alamgir, Sachi Matsumoto, Shin-ichi Tebayashi
    Abstract:

    : Eight compounds isolated from Indian barnyard millet have been identified as L-malic acid, trans-aconitic acid, (+)-isocitric acid, 5-O-caffeoylquinic acid, 4-O-caffeoylquinic acid, isocarlinoside, 2"-O-rhamnosylisoorientin, and 7-O-(2"-O-glucuronosyl)glucuronosyltricin, respectively. These compounds showed high Antifeeding Activity against brown planthopper only when they were combined.

  • antifeedants against acusta despesta from the japanese cedar cryptomeria japonica
    Zeitschrift für Naturforschung C, 2001
    Co-Authors: Xiao-hui Chen, Takehiro Kashiwagi, Shin-ichi Tebayashi, Chul-sa Kim, Michio Horiike
    Abstract:

    During our studies on the components in Japanese cedar, Cryptomeria japonica, we found that the crude methanol extract of C. japonica showed intense Antifeeding Activity against one snail species, Acusta despesta, which is well-known as a pest of many vegetables and crops. The active components in the extract were separated into the hexane and ethyl acetate soluble fractions. From the active ethyl acetate soluble fraction, two norlignans, sequirin-C and agatharesinol, were isolated and identified as the active compounds. Both compounds inhibited feeding behavior of A. despesta at 30 microg/cm2 and 40 microg/cm2 concentrations, respectively, when applied by an eggplant leaf or filter paper containing 20 microl of 5% sucrose solution.

  • antifeedants against acusta despesta from the japanese cedar cryptomeria japonica ii
    Bioscience Biotechnology and Biochemistry, 2001
    Co-Authors: Xiao-hui Chen, Takehiro Kashiwagi, Shin-ichi Tebayashi, Michio Horiike
    Abstract:

    The hexane-soluble fr. was found to be as intense in Antifeeding Activity as the crude methnol extract of Cryptomeria japonica against Acusta despesta. This hexane soluble-fraction was used to isolate and identify two sesquiterpenols, (−)-cubebol and (+)-2,7(14),10-bisabolatrien-1-ol-4-one, as the active compounds. Both compounds strongly inhibited the feeding behavior of A. despesta at 120 μg/cm2 and 80 μg/cm2 concentrations, respectively.

Priyo Susatyo - One of the best experts on this subject based on the ideXlab platform.

  • AKTIVITAS ANTI MAKAN EKSTRAK DAUN SIRSAK (ANNONA MURICATA L.) DAN PENGARUHNYA TERHADAP INDEKS NUTRISI SERTA TERHADAP STRUKTUR MEMBRAN PERITROFIK LARVA INSTAR V SPODOPTERA LITURA F.
    Universitas Lampung, 2013
    Co-Authors: Trisnowati B. Ambarningrum, Endang A. Setyowati, Priyo Susatyo
    Abstract:

    Antifeeding Activity of soursop leaf extract    (Annona muricata L.) and its effect on nutrition indices and the microscopic structure of peritrophic membrane of the fifth-instar larvae of Spodoptera litura F. were studied.  Antifeeding test was conducted by choice method. Discs of caisin leaf were dipped into the ether fraction of soursop leaf extract with concentrations of 0; 0.63; 2.50; and 10.00%. Nutrition indices and peritrophic membrane structure were observed by giving fifth-instar larvae  one of the five diets, every group of diet was added to various concentrations of  the ether fraction of soursop leaf extract, containing either 0; 0.63; 1.25; 2.50; and 5.00%. The result showed that  soursop leaf extract had Antifeeding Activity at tested concentration 2.50%. Relative consumption rate (RCR), relative growth rate (RGR), and  efficiency of conversion of  ingested food  (ECI) were significantly lower in the case of treated larvae than that of the controls. However the efficiency of conversion of digested food  (ECD), approximate digestibility (AD), and peritrophic membrane structure treated with  soursop leaf extract were not significantly affected as compared to those in  controls