The Experts below are selected from a list of 30 Experts worldwide ranked by ideXlab platform

Shelley D. Smith - One of the best experts on this subject based on the ideXlab platform.

  • Gene × Environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

  • gene environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and Psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

Lauren M. Mcgrath - One of the best experts on this subject based on the ideXlab platform.

  • Gene × Environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

  • gene environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and Psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

Erik G. Willcutt - One of the best experts on this subject based on the ideXlab platform.

  • Gene × Environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

  • gene environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and Psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

Lawrence D. Shriberg - One of the best experts on this subject based on the ideXlab platform.

  • Gene × Environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

  • gene environment interactions in Speech sound disorder predict language and preliteracy outcomes
    Development and Psychopathology, 2007
    Co-Authors: Lauren M. Mcgrath, Erik G. Willcutt, Lawrence D. Shriberg, Shelley D. Smith
    Abstract:

    Few studies have investigated the role of gene x environment interactions (G x E) in Speech, language, and literacy disorders. Currently, there are two theoretical models, the diathesis-stress model and the bioecological model, that make opposite predictions about the expected direction of G x E, because environmental risk factors may either strengthen or weaken the effect of genes on phenotypes. The purpose of the current study was to test for G x E at two Speech sound disorder and reading disability linkage peaks using a sib-pair linkage design and continuous measures of socioeconomic status, home language/literacy environment, and number of ear infections. The interactions were tested using Composite Speech, language, and preliteracy phenotypes and previously identified linkage peaks on 6p22 and 15q21. Results showed five G x E at both the 6p22 and 15q21 locations across several phenotypes and environmental measures. Four of the five interactions were consistent with the bioecological model of G x E. Each of these four interactions involved environmental measures of the home language/literacy environment. The only interaction that was consistent with the diathesis-stress model was one involving the number of ear infections as the environmental risk variable. The direction of these interactions and possible interpretations are explored in the discussion.

Andrew J. Tilley - One of the best experts on this subject based on the ideXlab platform.

  • The association between auditory memory span and Speech rate in children from kindergarten to sixth grade.
    Journal of experimental child psychology, 2002
    Co-Authors: Angela N. Ferguson, Judith A. Bowey, Andrew J. Tilley
    Abstract:

    This study reexamined the association between Speech rate and memory span in children from kindergarten to sixth grade (N = 152) in order to potentially account for the inconsistencies within the published literature on this topic. Some of the inconsistencies in past research may reflect the different methods adopted in assessing Speech rate. In particular, repeating word triples may itself involve memory demands, contaminating the correlation between Speech rate and memory span in younger children. Analyses using Composite Speech rate and memory span measures showed that Speech rate for word triples shared variance with memory span that was independent of Speech rate for single words. Moreover, Speech rate for word triples was largely redundant with age in explaining additional variation in memory span once the effects of Speech rate for single words were controlled.