The Experts below are selected from a list of 1266 Experts worldwide ranked by ideXlab platform
Jaime Godoy - One of the best experts on this subject based on the ideXlab platform.
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basal temporal language area
Brain, 1991Co-Authors: Hans Luders, Dudley S Dinner, Elaine Wyllie, Ronald P Lesser, Joseph F Hahn, Harold H Morris, Jaime GodoyAbstract:Language interference was elicited by electrical stimulation of the dominant basal temporal region in 8 out of 22 cases and in none of 7 cases with subdural electrodes implanted over the nondominant temporal lobe. Language interference was elicited by stimulation of electrodes placed over the fusiform gyrus 3-7 cm from the tip of the temporal lobe. Electrical stimulation of the basal temporal language area produced a global receptive and Expressive Aphasia with speech arrest at high stimulus intensities. Other higher cortical function, for example copying complex designs or memory of nonverbal information was intact, in spite of the total inability to process verbal information. At lower stimulus intensities partial Aphasias with a predominant receptive component occurred. Surgical resection of the basal temporal language area produces no lasting language deficit.
Dudley S Dinner - One of the best experts on this subject based on the ideXlab platform.
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Aphasia after hemispherectomy in an adult with early onset epilepsy and hemiplegia
Journal of Neurology Neurosurgery and Psychiatry, 2004Co-Authors: Tobias Loddenkemper, Dudley S Dinner, Cynthia S Kubu, Richard A Prayson, William Bingaman, A Dagirmanjian, Elaine WyllieAbstract:A 55 year old left handed man with left hemisphere subcortical encephalomalacia, seizures, language impairment, and right hemiparesis from a motor vehicle accident at age five was evaluated for epilepsy surgery. The patient continued to speak and followed commands during a left intracarotid amobarbital test (IAT). Left functional hemispherectomy resulted in Expressive Aphasia. Based on postoperative outcome, language was bilateral. The injury after primary development of language function, the predominantly subcortical lesion, and the late timing of surgical intervention well past development and plasticity may have been factors in the emergence of postoperative Aphasia.
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basal temporal language area
Brain, 1991Co-Authors: Hans Luders, Dudley S Dinner, Elaine Wyllie, Ronald P Lesser, Joseph F Hahn, Harold H Morris, Jaime GodoyAbstract:Language interference was elicited by electrical stimulation of the dominant basal temporal region in 8 out of 22 cases and in none of 7 cases with subdural electrodes implanted over the nondominant temporal lobe. Language interference was elicited by stimulation of electrodes placed over the fusiform gyrus 3-7 cm from the tip of the temporal lobe. Electrical stimulation of the basal temporal language area produced a global receptive and Expressive Aphasia with speech arrest at high stimulus intensities. Other higher cortical function, for example copying complex designs or memory of nonverbal information was intact, in spite of the total inability to process verbal information. At lower stimulus intensities partial Aphasias with a predominant receptive component occurred. Surgical resection of the basal temporal language area produces no lasting language deficit.
Elaine Wyllie - One of the best experts on this subject based on the ideXlab platform.
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Aphasia after hemispherectomy in an adult with early onset epilepsy and hemiplegia
Journal of Neurology Neurosurgery and Psychiatry, 2004Co-Authors: Tobias Loddenkemper, Dudley S Dinner, Cynthia S Kubu, Richard A Prayson, William Bingaman, A Dagirmanjian, Elaine WyllieAbstract:A 55 year old left handed man with left hemisphere subcortical encephalomalacia, seizures, language impairment, and right hemiparesis from a motor vehicle accident at age five was evaluated for epilepsy surgery. The patient continued to speak and followed commands during a left intracarotid amobarbital test (IAT). Left functional hemispherectomy resulted in Expressive Aphasia. Based on postoperative outcome, language was bilateral. The injury after primary development of language function, the predominantly subcortical lesion, and the late timing of surgical intervention well past development and plasticity may have been factors in the emergence of postoperative Aphasia.
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basal temporal language area
Brain, 1991Co-Authors: Hans Luders, Dudley S Dinner, Elaine Wyllie, Ronald P Lesser, Joseph F Hahn, Harold H Morris, Jaime GodoyAbstract:Language interference was elicited by electrical stimulation of the dominant basal temporal region in 8 out of 22 cases and in none of 7 cases with subdural electrodes implanted over the nondominant temporal lobe. Language interference was elicited by stimulation of electrodes placed over the fusiform gyrus 3-7 cm from the tip of the temporal lobe. Electrical stimulation of the basal temporal language area produced a global receptive and Expressive Aphasia with speech arrest at high stimulus intensities. Other higher cortical function, for example copying complex designs or memory of nonverbal information was intact, in spite of the total inability to process verbal information. At lower stimulus intensities partial Aphasias with a predominant receptive component occurred. Surgical resection of the basal temporal language area produces no lasting language deficit.
Hans Luders - One of the best experts on this subject based on the ideXlab platform.
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basal temporal language area
Brain, 1991Co-Authors: Hans Luders, Dudley S Dinner, Elaine Wyllie, Ronald P Lesser, Joseph F Hahn, Harold H Morris, Jaime GodoyAbstract:Language interference was elicited by electrical stimulation of the dominant basal temporal region in 8 out of 22 cases and in none of 7 cases with subdural electrodes implanted over the nondominant temporal lobe. Language interference was elicited by stimulation of electrodes placed over the fusiform gyrus 3-7 cm from the tip of the temporal lobe. Electrical stimulation of the basal temporal language area produced a global receptive and Expressive Aphasia with speech arrest at high stimulus intensities. Other higher cortical function, for example copying complex designs or memory of nonverbal information was intact, in spite of the total inability to process verbal information. At lower stimulus intensities partial Aphasias with a predominant receptive component occurred. Surgical resection of the basal temporal language area produces no lasting language deficit.
James J. Riviello - One of the best experts on this subject based on the ideXlab platform.
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Original Article Episodic Epileptic Verbal Auditory Agnosia in Landau Kleffner Syndrome TreatedWith Combination Diazepam and
2016Co-Authors: Orrin Devinsky, Rina Goldberg, Aviva Bojko, Daniel Miles, James J. RivielloAbstract:We report 2 pediatric patients who presented initially with seizures followed by subacute language regression characterized by a verbal auditory agnosia. These previously normal children had no evidence of Expressive Aphasia during their symptomatic periods. Further, in both cases, auditory agnosia was associated with sleep-activated electroencephalographic (EEG) epileptiform activity, consistent with Landau-Kleffner syndrome. However, both cases are unique since the episodic auditory agnosia and sleep-activated EEG epileptiform activity rapidly responded to combination therapy with pulse benzodiazepine and corticosteroids. Fur-ther, in each case, recurrences were characterized by similar symptoms, EEG findings, and beneficial responses to the pulse ben-zodiazepine and corticosteroid therapy. These observations suggest that pulse combination high-dose corticosteroid and benzodiazepine therapy may be especially effective in Landau-Kleffner syndrome
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Episodic epileptic verbal auditory agnosia in Landau Kleffner syndrome treated with combination diazepam and corticosteroids.
Journal of child neurology, 2014Co-Authors: Orrin Devinsky, Rina Goldberg, Daniel K. Miles, Aviva Bojko, James J. RivielloAbstract:We report 2 pediatric patients who presented initially with seizures followed by subacute language regression characterized by a verbal auditory agnosia. These previously normal children had no evidence of Expressive Aphasia during their symptomatic periods. Further, in both cases, auditory agnosia was associated with sleep-activated electroencephalographic (EEG) epileptiform activity, consistent with Landau-Kleffner syndrome. However, both cases are unique since the episodic auditory agnosia and sleep-activated EEG epileptiform activity rapidly responded to combination therapy with pulse benzodiazepine and corticosteroids. Further, in each case, recurrences were characterized by similar symptoms, EEG findings, and beneficial responses to the pulse benzodiazepine and corticosteroid therapy. These observations suggest that pulse combination high-dose corticosteroid and benzodiazepine therapy may be especially effective in Landau-Kleffner syndrome.
