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Maria Alice Rostom De Mello - One of the best experts on this subject based on the ideXlab platform.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity.
Diabetology & metabolic syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Alice Rostom De MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity.
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Metabolic responses to acute physical exercise in young rats recovered from Fetal Protein malnutrition with a fructose-rich diet
Lipids in health and disease, 2011Co-Authors: Lucieli Teresa Cambri, Gustavo G. Araujo, Ana Carolina Ghezzi, José Diego Botezelli, Maria Alice Rostom De MelloAbstract:Background: Malnutrition in utero can “program” the Fetal tissues, making them more vulnerable to metabolic disturbances. Also there is association between excessive consumption of fructose and the development of metabolic syndrome. However, there is little information regarding the acute effect of physical exercise on subjects recovered from malnutrition and/or fed with a fructose-rich diet. The objective of this study was to evaluate the metabolic aspects and the response to acute physical exercise in rats recovered from Fetal Protein malnutrition with a fructose-rich diet. Methods: Pregnant Wistar rats were fed with a balanced (B) diet or a low-Protein (L) diet. After birth and until 60 days of age, the offspring were distributed into four groups according to the diet received: B: B diet during the whole experiment; balanced/fructose (BF): B diet until birth and fructose-rich (F) diet afterwards; low Protein/ balanced (LB): L diet until birth and B diet afterwards; low Protein/fructose (LF): L diet until birth and F diet afterwards. Results: The excess fructose intake reduced the body weight gain, especially in the BF group. Furthermore, the serum total cholesterol and the LDL cholesterol were elevated in this group. In the LF group, the serum total cholesterol and the muscle glycogen increased. Acute physical exercise increased the serum concentrations of glucose, triglycerides, HDL cholesterol and liver lipids and reduced the concentrations of muscle glycogen in all groups. Conclusion: An excess fructose intake induced some signs of metabolic syndrome. However, Protein malnutrition appeared to protect against the short term effects of fructose. In other hand, most responses to acute physical exercise were not influenced by early malnutrition and/or by the fructose overload.
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Recovery of rat growth and lipid profiles in adult rats subjected to Fetal Protein malnutrition with a fructose-rich diet.
Nutrition research (New York N.Y.), 2010Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:There is evidence suggesting an association between fructose consumption and the development of metabolic syndrome. In turn, Protein malnutrition in utero is proposed to “program” the Fetal tissues, making them more susceptible to nutritional associated disorders. To test this hypothesis, the present study was designed to analyze body growth and metabolic aspects of rats subjected to Fetal Protein malnutrition and subsequently fed a fructose-rich diet. Wistar rats were distributed into 4 groups: balanced (B) diet—B diet offered the entire experimental period; balanced diet/fructose—B diet until birth and fructose-rich diet (F-60% fructose) until adulthood; low-Protein (L) diet/balanced—L diet until birth and B diet until adulthood; low-Protein diet/fructose (F)—L diet until birth and F diet until adulthood. After nutritional recovery, there was a restoration of serum glucose, total Protein, and albumin concentrations, which were reduced by Fetal malnutrition, and a restoration of the liver glycogen and lipids contents, which were increased by Fetal malnutrition. This restoration was independent of the diet adopted after birth. It was verified that the high fructose diet arrested body growth of the rats independently of the nutritional state during Fetal life and was associated with weight reduction and decrease of the adipose in some regions of the body (P b .05). Moreover, the serum concentrations of triglycerides and total cholesterol, which are indicators of metabolic syndrome, rose in the rats that ingested the fructose-rich diet (P b .05). In summary, high consumption of fructose impairs body growth and alters the circulating lipids independently of the Protein nutrition in utero. © 2010 Elsevier Inc. All rights reserved.
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Aerobic capacity of rats recovered from Fetal malnutrition with a fructose-rich diet.
Applied physiology nutrition and metabolism = Physiologie appliquee nutrition et metabolisme, 2010Co-Authors: Lucieli Teresa Cambri, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:The objective of this study was to analyze the aerobic capacity, through the maximal lactate steady-state (MLSS) protocol, of rats subjected to Fetal Protein malnutrition and recovered with a fruct...
Lucieli Teresa Cambri - One of the best experts on this subject based on the ideXlab platform.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity.
Diabetology & metabolic syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Alice Rostom De MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity
Diabetology & Metabolic Syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Ar MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity. Methods The male offspring born of mothers fed on a balanced or low-Protein diet were split in four groups until 60 days: Balanced (B): balanced diet during the whole period; Balanced/Fructose (BF): balanced diet in utero and fructose-rich diet after birth; Low Protein/Balanced (LB): low-Protein diet in utero and balanced diet after birth; Low Protein/Fructose (LF): low Protein diet in utero and fructose-rich diet after birth. Results Acute physical exercise reduced the muscle glycogen concentrations in all groups, although the LF group showed higher concentrations at rest. There was no difference among the groups in the glucose uptake and oxidation rates in the isolated soleus muscle neither at rest nor after acute exercise. However, glycogen synthesis was higher in the LF muscle than in the others at rest. Acute physical exercise increased glycogen synthesis in all groups, and the LF group showed the highest values. Conclusion The fructose-rich diet administered in rats after Fetal Protein malnutrition alters muscle glycogen concentrations and glycogen synthesis in the rest and after acute exercise at maximal lactate steady-state intensity.
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Metabolic responses to acute physical exercise in young rats recovered from Fetal Protein malnutrition with a fructose-rich diet
Lipids in health and disease, 2011Co-Authors: Lucieli Teresa Cambri, Gustavo G. Araujo, Ana Carolina Ghezzi, José Diego Botezelli, Maria Alice Rostom De MelloAbstract:Background: Malnutrition in utero can “program” the Fetal tissues, making them more vulnerable to metabolic disturbances. Also there is association between excessive consumption of fructose and the development of metabolic syndrome. However, there is little information regarding the acute effect of physical exercise on subjects recovered from malnutrition and/or fed with a fructose-rich diet. The objective of this study was to evaluate the metabolic aspects and the response to acute physical exercise in rats recovered from Fetal Protein malnutrition with a fructose-rich diet. Methods: Pregnant Wistar rats were fed with a balanced (B) diet or a low-Protein (L) diet. After birth and until 60 days of age, the offspring were distributed into four groups according to the diet received: B: B diet during the whole experiment; balanced/fructose (BF): B diet until birth and fructose-rich (F) diet afterwards; low Protein/ balanced (LB): L diet until birth and B diet afterwards; low Protein/fructose (LF): L diet until birth and F diet afterwards. Results: The excess fructose intake reduced the body weight gain, especially in the BF group. Furthermore, the serum total cholesterol and the LDL cholesterol were elevated in this group. In the LF group, the serum total cholesterol and the muscle glycogen increased. Acute physical exercise increased the serum concentrations of glucose, triglycerides, HDL cholesterol and liver lipids and reduced the concentrations of muscle glycogen in all groups. Conclusion: An excess fructose intake induced some signs of metabolic syndrome. However, Protein malnutrition appeared to protect against the short term effects of fructose. In other hand, most responses to acute physical exercise were not influenced by early malnutrition and/or by the fructose overload.
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Recovery of rat growth and lipid profiles in adult rats subjected to Fetal Protein malnutrition with a fructose-rich diet.
Nutrition research (New York N.Y.), 2010Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:There is evidence suggesting an association between fructose consumption and the development of metabolic syndrome. In turn, Protein malnutrition in utero is proposed to “program” the Fetal tissues, making them more susceptible to nutritional associated disorders. To test this hypothesis, the present study was designed to analyze body growth and metabolic aspects of rats subjected to Fetal Protein malnutrition and subsequently fed a fructose-rich diet. Wistar rats were distributed into 4 groups: balanced (B) diet—B diet offered the entire experimental period; balanced diet/fructose—B diet until birth and fructose-rich diet (F-60% fructose) until adulthood; low-Protein (L) diet/balanced—L diet until birth and B diet until adulthood; low-Protein diet/fructose (F)—L diet until birth and F diet until adulthood. After nutritional recovery, there was a restoration of serum glucose, total Protein, and albumin concentrations, which were reduced by Fetal malnutrition, and a restoration of the liver glycogen and lipids contents, which were increased by Fetal malnutrition. This restoration was independent of the diet adopted after birth. It was verified that the high fructose diet arrested body growth of the rats independently of the nutritional state during Fetal life and was associated with weight reduction and decrease of the adipose in some regions of the body (P b .05). Moreover, the serum concentrations of triglycerides and total cholesterol, which are indicators of metabolic syndrome, rose in the rats that ingested the fructose-rich diet (P b .05). In summary, high consumption of fructose impairs body growth and alters the circulating lipids independently of the Protein nutrition in utero. © 2010 Elsevier Inc. All rights reserved.
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Aerobic capacity of rats recovered from Fetal malnutrition with a fructose-rich diet.
Applied physiology nutrition and metabolism = Physiologie appliquee nutrition et metabolisme, 2010Co-Authors: Lucieli Teresa Cambri, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:The objective of this study was to analyze the aerobic capacity, through the maximal lactate steady-state (MLSS) protocol, of rats subjected to Fetal Protein malnutrition and recovered with a fruct...
Ana Carolina Ghezzi - One of the best experts on this subject based on the ideXlab platform.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity.
Diabetology & metabolic syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Alice Rostom De MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity
Diabetology & Metabolic Syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Ar MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity. Methods The male offspring born of mothers fed on a balanced or low-Protein diet were split in four groups until 60 days: Balanced (B): balanced diet during the whole period; Balanced/Fructose (BF): balanced diet in utero and fructose-rich diet after birth; Low Protein/Balanced (LB): low-Protein diet in utero and balanced diet after birth; Low Protein/Fructose (LF): low Protein diet in utero and fructose-rich diet after birth. Results Acute physical exercise reduced the muscle glycogen concentrations in all groups, although the LF group showed higher concentrations at rest. There was no difference among the groups in the glucose uptake and oxidation rates in the isolated soleus muscle neither at rest nor after acute exercise. However, glycogen synthesis was higher in the LF muscle than in the others at rest. Acute physical exercise increased glycogen synthesis in all groups, and the LF group showed the highest values. Conclusion The fructose-rich diet administered in rats after Fetal Protein malnutrition alters muscle glycogen concentrations and glycogen synthesis in the rest and after acute exercise at maximal lactate steady-state intensity.
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Metabolic responses to acute physical exercise in young rats recovered from Fetal Protein malnutrition with a fructose-rich diet
Lipids in health and disease, 2011Co-Authors: Lucieli Teresa Cambri, Gustavo G. Araujo, Ana Carolina Ghezzi, José Diego Botezelli, Maria Alice Rostom De MelloAbstract:Background: Malnutrition in utero can “program” the Fetal tissues, making them more vulnerable to metabolic disturbances. Also there is association between excessive consumption of fructose and the development of metabolic syndrome. However, there is little information regarding the acute effect of physical exercise on subjects recovered from malnutrition and/or fed with a fructose-rich diet. The objective of this study was to evaluate the metabolic aspects and the response to acute physical exercise in rats recovered from Fetal Protein malnutrition with a fructose-rich diet. Methods: Pregnant Wistar rats were fed with a balanced (B) diet or a low-Protein (L) diet. After birth and until 60 days of age, the offspring were distributed into four groups according to the diet received: B: B diet during the whole experiment; balanced/fructose (BF): B diet until birth and fructose-rich (F) diet afterwards; low Protein/ balanced (LB): L diet until birth and B diet afterwards; low Protein/fructose (LF): L diet until birth and F diet afterwards. Results: The excess fructose intake reduced the body weight gain, especially in the BF group. Furthermore, the serum total cholesterol and the LDL cholesterol were elevated in this group. In the LF group, the serum total cholesterol and the muscle glycogen increased. Acute physical exercise increased the serum concentrations of glucose, triglycerides, HDL cholesterol and liver lipids and reduced the concentrations of muscle glycogen in all groups. Conclusion: An excess fructose intake induced some signs of metabolic syndrome. However, Protein malnutrition appeared to protect against the short term effects of fructose. In other hand, most responses to acute physical exercise were not influenced by early malnutrition and/or by the fructose overload.
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Recovery of rat growth and lipid profiles in adult rats subjected to Fetal Protein malnutrition with a fructose-rich diet.
Nutrition research (New York N.Y.), 2010Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:There is evidence suggesting an association between fructose consumption and the development of metabolic syndrome. In turn, Protein malnutrition in utero is proposed to “program” the Fetal tissues, making them more susceptible to nutritional associated disorders. To test this hypothesis, the present study was designed to analyze body growth and metabolic aspects of rats subjected to Fetal Protein malnutrition and subsequently fed a fructose-rich diet. Wistar rats were distributed into 4 groups: balanced (B) diet—B diet offered the entire experimental period; balanced diet/fructose—B diet until birth and fructose-rich diet (F-60% fructose) until adulthood; low-Protein (L) diet/balanced—L diet until birth and B diet until adulthood; low-Protein diet/fructose (F)—L diet until birth and F diet until adulthood. After nutritional recovery, there was a restoration of serum glucose, total Protein, and albumin concentrations, which were reduced by Fetal malnutrition, and a restoration of the liver glycogen and lipids contents, which were increased by Fetal malnutrition. This restoration was independent of the diet adopted after birth. It was verified that the high fructose diet arrested body growth of the rats independently of the nutritional state during Fetal life and was associated with weight reduction and decrease of the adipose in some regions of the body (P b .05). Moreover, the serum concentrations of triglycerides and total cholesterol, which are indicators of metabolic syndrome, rose in the rats that ingested the fructose-rich diet (P b .05). In summary, high consumption of fructose impairs body growth and alters the circulating lipids independently of the Protein nutrition in utero. © 2010 Elsevier Inc. All rights reserved.
José Diego Botezelli - One of the best experts on this subject based on the ideXlab platform.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity
Diabetology & Metabolic Syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Ar MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity. Methods The male offspring born of mothers fed on a balanced or low-Protein diet were split in four groups until 60 days: Balanced (B): balanced diet during the whole period; Balanced/Fructose (BF): balanced diet in utero and fructose-rich diet after birth; Low Protein/Balanced (LB): low-Protein diet in utero and balanced diet after birth; Low Protein/Fructose (LF): low Protein diet in utero and fructose-rich diet after birth. Results Acute physical exercise reduced the muscle glycogen concentrations in all groups, although the LF group showed higher concentrations at rest. There was no difference among the groups in the glucose uptake and oxidation rates in the isolated soleus muscle neither at rest nor after acute exercise. However, glycogen synthesis was higher in the LF muscle than in the others at rest. Acute physical exercise increased glycogen synthesis in all groups, and the LF group showed the highest values. Conclusion The fructose-rich diet administered in rats after Fetal Protein malnutrition alters muscle glycogen concentrations and glycogen synthesis in the rest and after acute exercise at maximal lactate steady-state intensity.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity.
Diabetology & metabolic syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Alice Rostom De MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity.
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Metabolic responses to acute physical exercise in young rats recovered from Fetal Protein malnutrition with a fructose-rich diet
Lipids in health and disease, 2011Co-Authors: Lucieli Teresa Cambri, Gustavo G. Araujo, Ana Carolina Ghezzi, José Diego Botezelli, Maria Alice Rostom De MelloAbstract:Background: Malnutrition in utero can “program” the Fetal tissues, making them more vulnerable to metabolic disturbances. Also there is association between excessive consumption of fructose and the development of metabolic syndrome. However, there is little information regarding the acute effect of physical exercise on subjects recovered from malnutrition and/or fed with a fructose-rich diet. The objective of this study was to evaluate the metabolic aspects and the response to acute physical exercise in rats recovered from Fetal Protein malnutrition with a fructose-rich diet. Methods: Pregnant Wistar rats were fed with a balanced (B) diet or a low-Protein (L) diet. After birth and until 60 days of age, the offspring were distributed into four groups according to the diet received: B: B diet during the whole experiment; balanced/fructose (BF): B diet until birth and fructose-rich (F) diet afterwards; low Protein/ balanced (LB): L diet until birth and B diet afterwards; low Protein/fructose (LF): L diet until birth and F diet afterwards. Results: The excess fructose intake reduced the body weight gain, especially in the BF group. Furthermore, the serum total cholesterol and the LDL cholesterol were elevated in this group. In the LF group, the serum total cholesterol and the muscle glycogen increased. Acute physical exercise increased the serum concentrations of glucose, triglycerides, HDL cholesterol and liver lipids and reduced the concentrations of muscle glycogen in all groups. Conclusion: An excess fructose intake induced some signs of metabolic syndrome. However, Protein malnutrition appeared to protect against the short term effects of fructose. In other hand, most responses to acute physical exercise were not influenced by early malnutrition and/or by the fructose overload.
Carla Ribeiro - One of the best experts on this subject based on the ideXlab platform.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity.
Diabetology & metabolic syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Alice Rostom De MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity.
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Muscle glycogen metabolism changes in rats fed early postnatal a fructose-rich diet after maternal Protein malnutrition: effects of acute physical exercise at the maximal lactate steady-state intensity
Diabetology & Metabolic Syndrome, 2014Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, José Diego Botezelli, Carla Ribeiro, Maria Ar MelloAbstract:Background The objective was to evaluate the muscle glucose metabolism in rats fed a fructose-rich diet after Fetal Protein malnutrition, at rest and after acute physical exercise at maximal lactate steady-state intensity. Methods The male offspring born of mothers fed on a balanced or low-Protein diet were split in four groups until 60 days: Balanced (B): balanced diet during the whole period; Balanced/Fructose (BF): balanced diet in utero and fructose-rich diet after birth; Low Protein/Balanced (LB): low-Protein diet in utero and balanced diet after birth; Low Protein/Fructose (LF): low Protein diet in utero and fructose-rich diet after birth. Results Acute physical exercise reduced the muscle glycogen concentrations in all groups, although the LF group showed higher concentrations at rest. There was no difference among the groups in the glucose uptake and oxidation rates in the isolated soleus muscle neither at rest nor after acute exercise. However, glycogen synthesis was higher in the LF muscle than in the others at rest. Acute physical exercise increased glycogen synthesis in all groups, and the LF group showed the highest values. Conclusion The fructose-rich diet administered in rats after Fetal Protein malnutrition alters muscle glycogen concentrations and glycogen synthesis in the rest and after acute exercise at maximal lactate steady-state intensity.
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Recovery of rat growth and lipid profiles in adult rats subjected to Fetal Protein malnutrition with a fructose-rich diet.
Nutrition research (New York N.Y.), 2010Co-Authors: Lucieli Teresa Cambri, Ana Carolina Ghezzi, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:There is evidence suggesting an association between fructose consumption and the development of metabolic syndrome. In turn, Protein malnutrition in utero is proposed to “program” the Fetal tissues, making them more susceptible to nutritional associated disorders. To test this hypothesis, the present study was designed to analyze body growth and metabolic aspects of rats subjected to Fetal Protein malnutrition and subsequently fed a fructose-rich diet. Wistar rats were distributed into 4 groups: balanced (B) diet—B diet offered the entire experimental period; balanced diet/fructose—B diet until birth and fructose-rich diet (F-60% fructose) until adulthood; low-Protein (L) diet/balanced—L diet until birth and B diet until adulthood; low-Protein diet/fructose (F)—L diet until birth and F diet until adulthood. After nutritional recovery, there was a restoration of serum glucose, total Protein, and albumin concentrations, which were reduced by Fetal malnutrition, and a restoration of the liver glycogen and lipids contents, which were increased by Fetal malnutrition. This restoration was independent of the diet adopted after birth. It was verified that the high fructose diet arrested body growth of the rats independently of the nutritional state during Fetal life and was associated with weight reduction and decrease of the adipose in some regions of the body (P b .05). Moreover, the serum concentrations of triglycerides and total cholesterol, which are indicators of metabolic syndrome, rose in the rats that ingested the fructose-rich diet (P b .05). In summary, high consumption of fructose impairs body growth and alters the circulating lipids independently of the Protein nutrition in utero. © 2010 Elsevier Inc. All rights reserved.
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Aerobic capacity of rats recovered from Fetal malnutrition with a fructose-rich diet.
Applied physiology nutrition and metabolism = Physiologie appliquee nutrition et metabolisme, 2010Co-Authors: Lucieli Teresa Cambri, Carla Ribeiro, Rodrigo Augusto Dalia, Maria Alice Rostom De MelloAbstract:The objective of this study was to analyze the aerobic capacity, through the maximal lactate steady-state (MLSS) protocol, of rats subjected to Fetal Protein malnutrition and recovered with a fruct...