The Experts below are selected from a list of 207 Experts worldwide ranked by ideXlab platform
Octavio M. Pontes-neto - One of the best experts on this subject based on the ideXlab platform.
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Conjugate upward Gaze Paralysis with unilateral ptosis caused by a unilateral midbrain infarction
Journal of neurology neurosurgery and psychiatry, 2013Co-Authors: Bruno Lopes Dos Santos, Gustavo Novelino Simão, Octavio M. Pontes-netoAbstract:A 73-year-old woman with atrial fibrillation presented with a sudden right hemiparesis, with diplopia and left ptosis, and was admitted at an Emergency Unit. The neurological examination found fluctuations on consciousness level, predominant crural right hemiparesis and right central facial Paralysis without sensitive abnormalities. The first ophthalmological evaluation showed normal pupillary reflexes, total left ptosis and paresis of adduction of the left eye, with conjugated horizontal palsy for right Gaze and conjugated vertical palsy for upward and downward Gaze on saccadic and smooth pursuit eye movements. The convergence showed paresis of left eye, with reactive pupils, and oculocephalic test was normal. A head CT had no acute ischaemic signs, and after 4 days, she was discharged. The brain magnetic resonance (MR) performed 15 days after the ictus showed a clearly defined left paramedian tegmental mesencephalic infarct (figure 1). Two months after the stroke, the patient had a remarkable improvement of ocular motility, presenting paresis of levator palpebrae, medial and inferior …
Shirley H. Wray - One of the best experts on this subject based on the ideXlab platform.
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Eye Movement Disorders in Clinical Practice - Vertical Gaze and Syndromes of the Midbrain
Eye Movement Disorders in Clinical Practice, 2014Co-Authors: Shirley H. WrayAbstract:reviews the architecture of the midbrain and extensively discusses the linkage of specific diagnostic signs to areas of the midbrain. Paralysis of up- and downGaze, pretectal pupils, eyelid and vergence disorders are among the signs discussed. Here again, case studies and video displays allow detailed presentations of patient history, analysis, diagnosis, and treatment of a range of disorders characterized by supranuclear vertical Gaze Paralysis: the pretectal syndrome, the Sylvian aqueduct syndrome, syndromes of the top of the basilar artery, and a rare case of supranuclear Paralysis of downGaze with clinicopathological correlation. A short section on vergence disorders covers Paralysis of convergence, the third “Parinaud” sign, convergence retraction nystagmus, pretectal pseudobobbing, and oculogyric crises.
Veerle Visser-vandewalle - One of the best experts on this subject based on the ideXlab platform.
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Vertical Gaze palsy after thalamic stimulation for Tourette syndrome: case report.
Neurosurgery, 2007Co-Authors: Linda Ackermans, Yasin Temel, Noël J.c. Bauer, Veerle Visser-vandewalleAbstract:OBJECTIVE: We describe a patient who developed a vertical Gaze Paralysis after deep brain stimulation performed for intractable Tourette syndrome due to a small deep bleeding in the upper mesencephalon. CLINICAL PRESENTATION: A 39-year-old man underwent thalamic deep brain stimulation for intractable Tourette syndrome. Immediately postoperatively, he had diplopia and dizziness. The neurological examination revealed vertical Gaze palsy with preserved vertical oculocephalic movements. A postoperative computed tomography scan revealed a discrete high-density lesion across the midline at the distal end of the left electrode. This area corresponds with the pretectal area, including the rostral interstitial nucleus of the medial longitudinal fasciculus, with sparing of the oculomotor and rubral nuclei. INTERVENTION: Six months postoperatively, maximal upward and downward smooth pursuit eye movements were achieved. Upward saccadic velocities were still reduced by 20 to 25 degrees. CONCLUSION: This case report describes a complication that might demand special attention during the planning of thalamic deep brain stimulation for the treatment of Tourette syndrome. Examination of both horizontal and vertical eye movements during deep brain stimulation surgery is recommended.
Bruno Lopes Dos Santos - One of the best experts on this subject based on the ideXlab platform.
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Conjugate upward Gaze Paralysis with unilateral ptosis caused by a unilateral midbrain infarction
Journal of neurology neurosurgery and psychiatry, 2013Co-Authors: Bruno Lopes Dos Santos, Gustavo Novelino Simão, Octavio M. Pontes-netoAbstract:A 73-year-old woman with atrial fibrillation presented with a sudden right hemiparesis, with diplopia and left ptosis, and was admitted at an Emergency Unit. The neurological examination found fluctuations on consciousness level, predominant crural right hemiparesis and right central facial Paralysis without sensitive abnormalities. The first ophthalmological evaluation showed normal pupillary reflexes, total left ptosis and paresis of adduction of the left eye, with conjugated horizontal palsy for right Gaze and conjugated vertical palsy for upward and downward Gaze on saccadic and smooth pursuit eye movements. The convergence showed paresis of left eye, with reactive pupils, and oculocephalic test was normal. A head CT had no acute ischaemic signs, and after 4 days, she was discharged. The brain magnetic resonance (MR) performed 15 days after the ictus showed a clearly defined left paramedian tegmental mesencephalic infarct (figure 1). Two months after the stroke, the patient had a remarkable improvement of ocular motility, presenting paresis of levator palpebrae, medial and inferior …
C Pierrot-deseilligny - One of the best experts on this subject based on the ideXlab platform.
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Complete bilateral horizontal Gaze Paralysis disclosing multiple sclerosis
Journal of neurology neurosurgery and psychiatry, 2001Co-Authors: D Milea, M Napolitano, H Dechy, P Le Hoang, Jean Yves Delattre, C Pierrot-deseillignyAbstract:Two women presented with bilateral internuclear ophthalmoplegia evolving in a few days to complete bilateral horizontal Gaze Paralysis. Convergence and vertical eye movements were normal. Cerebral MRI showed a few small white matter lesions in the lateral ventricle regions, and, at the brainstem level, a single, small, bilateral lesion affecting the posterior part of the medial pontine tegmentum and responsible for the clinical syndrome. The condition gradually improved in both patients, following a similar progression as at the onset: improvement first involved the adduction movements in both eyes, whereas bilateral abduction paresis still persisted for a few weeks, before complete recovery of eye movements. Bilateral damage to the medial longitudinal fasciculus and subsequent lateral extent of damage to the region of the two abducens emerging fibres may explain the clinical findings. In both cases, the cause was probably multiple sclerosis.
