The Experts below are selected from a list of 63 Experts worldwide ranked by ideXlab platform
Pierre J. G. M. De Wit - One of the best experts on this subject based on the ideXlab platform.
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Cladosporium fulvum (syn. Passalora fulva), a highly specialized plant pathogen as a model for functional studies on plant pathogenic Mycosphaerellaceae
Molecular plant pathology, 2005Co-Authors: Bart P. H. J. Thomma, H. Peter Van Esse, Pedro W. Crous, Pierre J. G. M. De WitAbstract:SUMMARY Taxonomy: Cladosporium fulvum is an asexual fungus for which no sexual stage is currently known. Molecular data, however, support C. fulvum as a member of the Mycosphaerellaceae, clustering with other taxa having Mycosphaerella teleomorphs . C. fulvum has recently been placed in the anamorph genus Passalora as P. fulva . Its taxonomic disposition is supported by its DNA phylogeny, as well as the distinct scars on its conidial hila, which are typical of Passalora , and unlike Cladosporium s.s. , which has teleomorphs that reside in Davidiella , and not Mycosphaerella . Host range and disease symptoms: The presently known sole host of C. fulvum is tomato (members of the genus Lycopersicon ). C. fulvum is mainly a foliar pathogen. Disease symptoms are most obvious on the abaxial side of the leaf and include patches of white mould that turn brown upon sporulation. Due to stomatal clogging, curling of leaves and wilting can occur, leading to defoliation. C. fulvum as a model pathogen: The interaction between C. fulvum and tomato is governed by a Gene-for-Gene Relationship. A total of eight Avr and Ecp genes, and for four of these also the corresponding plant Cf genes, have been cloned. Obtaining conclusive evidence for Gene-for-Gene Relationships is complicated by the poor availability of genetic tools for most Mycosphaerellaceae ‐ plant interactions. Newly developed tools, including Agrobacterium -mediated transformation and RNAi, added to the genome sequence of its host tomato, which will be available within a few years, render C. fulvum attractive as a model species for plant pathogenic Mycosphaerellaceae. Useful websites: http://www.sgn.cornell.edu /help/about / index.html; http://cogeme.ex.ac.uk
Bart P. H. J. Thomma - One of the best experts on this subject based on the ideXlab platform.
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Cladosporium fulvum (syn. Passalora fulva), a highly specialized plant pathogen as a model for functional studies on plant pathogenic Mycosphaerellaceae
Molecular plant pathology, 2005Co-Authors: Bart P. H. J. Thomma, H. Peter Van Esse, Pedro W. Crous, Pierre J. G. M. De WitAbstract:SUMMARY Taxonomy: Cladosporium fulvum is an asexual fungus for which no sexual stage is currently known. Molecular data, however, support C. fulvum as a member of the Mycosphaerellaceae, clustering with other taxa having Mycosphaerella teleomorphs . C. fulvum has recently been placed in the anamorph genus Passalora as P. fulva . Its taxonomic disposition is supported by its DNA phylogeny, as well as the distinct scars on its conidial hila, which are typical of Passalora , and unlike Cladosporium s.s. , which has teleomorphs that reside in Davidiella , and not Mycosphaerella . Host range and disease symptoms: The presently known sole host of C. fulvum is tomato (members of the genus Lycopersicon ). C. fulvum is mainly a foliar pathogen. Disease symptoms are most obvious on the abaxial side of the leaf and include patches of white mould that turn brown upon sporulation. Due to stomatal clogging, curling of leaves and wilting can occur, leading to defoliation. C. fulvum as a model pathogen: The interaction between C. fulvum and tomato is governed by a Gene-for-Gene Relationship. A total of eight Avr and Ecp genes, and for four of these also the corresponding plant Cf genes, have been cloned. Obtaining conclusive evidence for Gene-for-Gene Relationships is complicated by the poor availability of genetic tools for most Mycosphaerellaceae ‐ plant interactions. Newly developed tools, including Agrobacterium -mediated transformation and RNAi, added to the genome sequence of its host tomato, which will be available within a few years, render C. fulvum attractive as a model species for plant pathogenic Mycosphaerellaceae. Useful websites: http://www.sgn.cornell.edu /help/about / index.html; http://cogeme.ex.ac.uk
Sihai Yang - One of the best experts on this subject based on the ideXlab platform.
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rapid evolution of avirulence genes in rice blast fungus magnaporthe oryzae
BMC Genetics, 2014Co-Authors: Ju Huang, Weina Si, Qiming Deng, Ping Li, Sihai YangAbstract:Background Rice blast fungus Magnaporthe oryzae is one of the most devastating pathogens in rice. Avirulence genes in this fungus share a Gene-for-Gene Relationship with the resistance genes in its host rice. Although numerous studies have shown that rice blast R-genes are extremely diverse and evolve rapidly in their host populations, little is known about the evolutionary patterns of the Avr-genes in the pathogens.
Didier Tharreau - One of the best experts on this subject based on the ideXlab platform.
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evidence of a gene for gene Relationship in the oryza sativa magnaporthe grisea pathosystem
Phytopathology, 1992Co-Authors: D Silue, Jeanloup Notteghem, Didier TharreauAbstract:A cross was made between two field isolates of Magnaporthe grisea pathogenic to rice. Full-sib crosses between F 1 ascospores, followed by backcrosses to the female fertile parent GUY 11, produced perithecia fertile enough for tetrad isolation. Genetic analysis of the pathogen revealed that avirulence to the rice cultivar Pi-n o 4 is controlled by one gene (Avr1-Pi-n o 4) unlinked to the gene MATI-1, which is responsible for mating type. Additionally, resistance of Pi-n o 4 was found to be controlled by one gene. Thus, existence of a Gene-for-Gene Relationship was shown for the pathosystem Oryza sativa-M. grisea
Adrienne R. Hardham - One of the best experts on this subject based on the ideXlab platform.
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Inhibition of Rust-induced Hypersensitive Response in Flax Cells by the Microtubule Inhibitor Oryzalin
Functional Plant Biology, 1997Co-Authors: Issei Kobayashi, Yuhko Kobayashi, Adrienne R. HardhamAbstract:Interactions between the flax rust fungus Melampsora lini and flax Linum usitatissimum L. are governed by a Gene-for-Gene Relationship which determines pathogen virulence or avirulence and host resistance or susceptibility. The present study demonstrates differential sensitivity of M. lini and flax to the microtubule depolymerising drug, oryzalin, such that microtubule depolymerisation in flax cells but not in fungal cells could be obtained. Normally, in an incompatible interaction, a rapid hypersensitive response about 24 h after inoculation inhibits fungal development and invasion. However, in an incompatible interaction in the presence of oryzalin, the occurrence of hypersensitive cell death was delayed and its frequency reduced. This allowed a normally avirulent race of M. lini to form haustoria in living host mesophyll cells at a rate and efficiency similar to that achieved by a virulent race in a compatible interaction during the first 36 h after inoculation. After that time, the incidence of hypersensitive cell death increased and further development of the pathogen was arrested. The results indicate that microtubules play a role in effecting rapid and efficient hypersensitive response in the race–cultivar specific interaction between flax and the flax rust fungus.