The Experts below are selected from a list of 360 Experts worldwide ranked by ideXlab platform
Nobuo Yanagisawa - One of the best experts on this subject based on the ideXlab platform.
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persistent Hemiballism with striatal hyperintensity on t1 weighted mri in a diabetic patient a 6 year follow up study
Journal of the Neurological Sciences, 1999Co-Authors: Takao Hashimoto, Hiroyuki Yahikozawa, Norinao Hanyu, Nobuo YanagisawaAbstract:The combination of Hemiballism, hyperglycemia and hyperintensity of the striatum on T1-weighted MRI constitutes a unique syndrome. We report the follow-up of a patient with this disorder whose Hemiballism was sustained for over 5 years. High density on CT of the right striatum turned into normodensity in 4 months, and hyperintensity on T1-weighted MRI and hypointensity on T2-weighted MRI of the lesion were resolved in 18 months. A decreased perfusion of the lesion by SPECT remained 37 months after onset. There was no volume change of the lesion during the course of the illness. The radiological features support the possible pathology of either or both petechial hemorrhage and astrocytosis with high protein concentration after ischemic insult. The Hemiballism may result from selective damage of GABA/enkephalin-containing neurons in the striatum and can persist without the primary histological changes causing the striatal T1-hyperintensity in this disorder.
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Hemiballism with striatal hyperintensity on t1 weighted mri in diabetic patients a unique syndrome
Journal of the Neurological Sciences, 1994Co-Authors: Hiroyuki Yahikozawa, Norinao Hanyu, Kanji Yamamoto, Takao Hashimoto, Koji Shimozono, Shinichi Nakagawa, Nobuo YanagisawaAbstract:We report 3 diabetic patients who developed Hemiballism without involvement of the subthalamic nucleus. Each patient exhibited vigorous, flinging, ballistic involuntary movements in the extremities and slight facial grimacing involving one side of the body. Although diabetes was poorly controlled in all 3, each patient was nonketotic at the onset of Hemiballism. Magnetic resonance imaging (MRI), in these patients showed abnormalities in the striatum contralateral to the Hemiballism that were characterized by an increase in intensity on T1-weighted images and a slight decrease in intensity on T2-weighted images, and these changes persisted for more than 2 months. The striatal lesions are presumed to have developed following mild ischemia in the territory of the lateral striate branches of the middle cerebral artery. This combination of Hemiballism and striatal lesions in diabetic patients may constitute a unique syndrome.
S.-h. Han - One of the best experts on this subject based on the ideXlab platform.
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Subthalamic lesion on MR imaging in a patient with nonketotic hyperglycemia-induced Hemiballism.
AJNR. American journal of neuroradiology, 2008Co-Authors: Hae-rim Kim, Won-jin Moon, I.k. Lee, H.y. Kim, S.-h. HanAbstract:SUMMARY: Hemiballism with corresponding striatal T1 hyperintensity on MR imaging has occasionally been reported in patients with nonketotic hyperglycemia. However, the subthalamic nucleus lesion, which is believed to be pathogenetically related to Hemiballism, is rarely documented in a living patient with nonketotic hyperglycemia. We describe a patient with nonketotic hyperglycemia-induced Hemiballism, whose responsible lesion (ie, the subthalamus) was demonstrated by MR imaging.
Hae-rim Kim - One of the best experts on this subject based on the ideXlab platform.
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Subthalamic lesion on MR imaging in a patient with nonketotic hyperglycemia-induced Hemiballism.
AJNR. American journal of neuroradiology, 2008Co-Authors: Hae-rim Kim, Won-jin Moon, I.k. Lee, H.y. Kim, S.-h. HanAbstract:SUMMARY: Hemiballism with corresponding striatal T1 hyperintensity on MR imaging has occasionally been reported in patients with nonketotic hyperglycemia. However, the subthalamic nucleus lesion, which is believed to be pathogenetically related to Hemiballism, is rarely documented in a living patient with nonketotic hyperglycemia. We describe a patient with nonketotic hyperglycemia-induced Hemiballism, whose responsible lesion (ie, the subthalamus) was demonstrated by MR imaging.
Takao Hashimoto - One of the best experts on this subject based on the ideXlab platform.
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Hemiballism with hyperglycemia and striatal t1 mri hyperintensity an autopsy report
Movement Disorders, 2001Co-Authors: S Ohara, Shinichi Nakagawa, K Tabata, Takao HashimotoAbstract:We report on an autopsy findings of a 92-year-old male with Hemiballism-hemichorea associated with hyperglycemia and striatal hyperintensity on T1-weighed magnetic resonance imaging (MRI), a recently described clinicoradiological syndrome. Histologically, the putamen contralateral to the Hemiballism consisted of multiple foci of recent infarcts associated with reactive astrocytic and interneuronal response. Substrate responsible for the MRI signal changes is still inconclusive. © Movement Disorder Society.
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persistent Hemiballism with striatal hyperintensity on t1 weighted mri in a diabetic patient a 6 year follow up study
Journal of the Neurological Sciences, 1999Co-Authors: Takao Hashimoto, Hiroyuki Yahikozawa, Norinao Hanyu, Nobuo YanagisawaAbstract:The combination of Hemiballism, hyperglycemia and hyperintensity of the striatum on T1-weighted MRI constitutes a unique syndrome. We report the follow-up of a patient with this disorder whose Hemiballism was sustained for over 5 years. High density on CT of the right striatum turned into normodensity in 4 months, and hyperintensity on T1-weighted MRI and hypointensity on T2-weighted MRI of the lesion were resolved in 18 months. A decreased perfusion of the lesion by SPECT remained 37 months after onset. There was no volume change of the lesion during the course of the illness. The radiological features support the possible pathology of either or both petechial hemorrhage and astrocytosis with high protein concentration after ischemic insult. The Hemiballism may result from selective damage of GABA/enkephalin-containing neurons in the striatum and can persist without the primary histological changes causing the striatal T1-hyperintensity in this disorder.
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Hemiballism with striatal hyperintensity on t1 weighted mri in diabetic patients a unique syndrome
Journal of the Neurological Sciences, 1994Co-Authors: Hiroyuki Yahikozawa, Norinao Hanyu, Kanji Yamamoto, Takao Hashimoto, Koji Shimozono, Shinichi Nakagawa, Nobuo YanagisawaAbstract:We report 3 diabetic patients who developed Hemiballism without involvement of the subthalamic nucleus. Each patient exhibited vigorous, flinging, ballistic involuntary movements in the extremities and slight facial grimacing involving one side of the body. Although diabetes was poorly controlled in all 3, each patient was nonketotic at the onset of Hemiballism. Magnetic resonance imaging (MRI), in these patients showed abnormalities in the striatum contralateral to the Hemiballism that were characterized by an increase in intensity on T1-weighted images and a slight decrease in intensity on T2-weighted images, and these changes persisted for more than 2 months. The striatal lesions are presumed to have developed following mild ischemia in the territory of the lateral striate branches of the middle cerebral artery. This combination of Hemiballism and striatal lesions in diabetic patients may constitute a unique syndrome.
Chen Chang - One of the best experts on this subject based on the ideXlab platform.
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presence of activated microglia in a high signal lesion on t1 weighted mr images a biopsy sample re examined
American Journal of Neuroradiology, 2007Co-Authors: Dine Shan, Hungchi Pan, Michael Muhuo Teng, Chen ChangAbstract:To determine the pathologic process responsible for the signal intensity changes of hyperintense putamen on T1-weighted MR images ([Fig 1 A ][1]), we had previously performed stereotactic biopsy in a 72-year-old man 3 months after the onset of stroke and Hemiballism.[1][2] The results revealed a