The Experts below are selected from a list of 21 Experts worldwide ranked by ideXlab platform
Lyn Kiers - One of the best experts on this subject based on the ideXlab platform.
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Association of acute inflammatory demyelinating polyneuropathy with acute lymphoblastic leukaemia and HLA-A11
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 1998Co-Authors: Andrew Grigg, Brian D. Tait, Stephen M. Davis, Lyn KiersAbstract:Acute inflammatory demyelinating polyneuropathy (Guillain-Barré syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described. The HLA-A11 Antigen was expressed by all patients, two of whom were Chinese, raising the possibility of a genetically-based immunological predisposition to GBS in this patient group.
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Association of acute inflammatory demyelinating polyneuropathy with acute lymphoblastic leukaemia and HLA-A11
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 1998Co-Authors: Andrew Grigg, Brian D. Tait, Stephen M. Davis, Lyn KiersAbstract:Acute inflammatory demyelinating polyneuropathy (Guillain-Barre syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described. The HLA-A11 Antigen was expressed by all patients, two of whom were Chinese, raising the possibility of a genetically-based immunological predisposition to GBS in this patient group.
Zulay Layrisse - One of the best experts on this subject based on the ideXlab platform.
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Strong association between major histocompatibility complex class I Antigens and immune aberrations among healthy Venezuelans
Human immunology, 1995Co-Authors: Nina J. Makhatadze, Pedro Sanches-llamozas, Maria Teresa Franco, Zulay LayrisseAbstract:Immune reactivity indicators studied among 55 unrelated Venezuelan mestizo subjects included lymphoproliferative response to polyclonal mitogen (PHA, Con A, PwM) stimulation, NK cell activity, and enumeration of peripheral blood mononuclear cells. HLA-A, -B, -C, -DR, and -DQ Antigens were determined by serologic typing. A strong association between impairment of the parameters studied and MHC class I Antigens A11 and A3 was found. Subjects with decreased suboptimal (0.5 micrograms/ml) PHA response as well as decreased optimal (0.5 micrograms/ml) Con A response showed high incidence of HLA-A11 Antigen (RR = 81, p = 0.001, pc = 0.021 and RR = 54, p = 0.0029, respectively). Subjects with decreased suboptimal (0.5 micrograms/ml) PHA response HLA-A11- with only one exception, were either HLA-A1+ or HLA-A3+. These Antigens belong to the same CREG, share public epitopes, and have low incidence in the Venezuelan mestizo population. Six of 10 persons with decreased CD16 subset count (5.17% +/- 0.23% vs 11.69% +/- 0.44%) had HLA-A3 Antigen (RR = 17, p = 0.001, pc = 0.021). The data indicate a possible contribution of HLA-A11,A3, molecules through their private and/or public determinants to immune response aberrations which under certain conditions may result in development of diseases.
Andrew Grigg - One of the best experts on this subject based on the ideXlab platform.
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Association of acute inflammatory demyelinating polyneuropathy with acute lymphoblastic leukaemia and HLA-A11
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 1998Co-Authors: Andrew Grigg, Brian D. Tait, Stephen M. Davis, Lyn KiersAbstract:Acute inflammatory demyelinating polyneuropathy (Guillain-Barré syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described. The HLA-A11 Antigen was expressed by all patients, two of whom were Chinese, raising the possibility of a genetically-based immunological predisposition to GBS in this patient group.
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Association of acute inflammatory demyelinating polyneuropathy with acute lymphoblastic leukaemia and HLA-A11
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 1998Co-Authors: Andrew Grigg, Brian D. Tait, Stephen M. Davis, Lyn KiersAbstract:Acute inflammatory demyelinating polyneuropathy (Guillain-Barre syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described. The HLA-A11 Antigen was expressed by all patients, two of whom were Chinese, raising the possibility of a genetically-based immunological predisposition to GBS in this patient group.
Nina J. Makhatadze - One of the best experts on this subject based on the ideXlab platform.
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Strong association between major histocompatibility complex class I Antigens and immune aberrations among healthy Venezuelans
Human immunology, 1995Co-Authors: Nina J. Makhatadze, Pedro Sanches-llamozas, Maria Teresa Franco, Zulay LayrisseAbstract:Immune reactivity indicators studied among 55 unrelated Venezuelan mestizo subjects included lymphoproliferative response to polyclonal mitogen (PHA, Con A, PwM) stimulation, NK cell activity, and enumeration of peripheral blood mononuclear cells. HLA-A, -B, -C, -DR, and -DQ Antigens were determined by serologic typing. A strong association between impairment of the parameters studied and MHC class I Antigens A11 and A3 was found. Subjects with decreased suboptimal (0.5 micrograms/ml) PHA response as well as decreased optimal (0.5 micrograms/ml) Con A response showed high incidence of HLA-A11 Antigen (RR = 81, p = 0.001, pc = 0.021 and RR = 54, p = 0.0029, respectively). Subjects with decreased suboptimal (0.5 micrograms/ml) PHA response HLA-A11- with only one exception, were either HLA-A1+ or HLA-A3+. These Antigens belong to the same CREG, share public epitopes, and have low incidence in the Venezuelan mestizo population. Six of 10 persons with decreased CD16 subset count (5.17% +/- 0.23% vs 11.69% +/- 0.44%) had HLA-A3 Antigen (RR = 17, p = 0.001, pc = 0.021). The data indicate a possible contribution of HLA-A11,A3, molecules through their private and/or public determinants to immune response aberrations which under certain conditions may result in development of diseases.
Stephen M. Davis - One of the best experts on this subject based on the ideXlab platform.
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Association of acute inflammatory demyelinating polyneuropathy with acute lymphoblastic leukaemia and HLA-A11
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 1998Co-Authors: Andrew Grigg, Brian D. Tait, Stephen M. Davis, Lyn KiersAbstract:Acute inflammatory demyelinating polyneuropathy (Guillain-Barré syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described. The HLA-A11 Antigen was expressed by all patients, two of whom were Chinese, raising the possibility of a genetically-based immunological predisposition to GBS in this patient group.
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Association of acute inflammatory demyelinating polyneuropathy with acute lymphoblastic leukaemia and HLA-A11
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia, 1998Co-Authors: Andrew Grigg, Brian D. Tait, Stephen M. Davis, Lyn KiersAbstract:Acute inflammatory demyelinating polyneuropathy (Guillain-Barre syndrome) developed in three patients receiving chemotherapy for acute lymphoblastic leukaemia or the closely related entity lymphoblastic lymphoma, a relationship that has not been previously described. The HLA-A11 Antigen was expressed by all patients, two of whom were Chinese, raising the possibility of a genetically-based immunological predisposition to GBS in this patient group.