The Experts below are selected from a list of 30 Experts worldwide ranked by ideXlab platform
Shigehiko Suzuki - One of the best experts on this subject based on the ideXlab platform.
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the Lesser Palatine Nerve innervates the levator veli palatini muscle
Plastic and reconstructive surgery. Global open, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Katsuya Kawai, Yoshitaka Matsuura, Shigehiko SuzukiAbstract:The levator veli palatini (LVP) muscle is responsible for the elevation of the soft palate and is critically involved in speech and deglutition. Therefore, repair of the muscle during palatoplasty for cleft palate is essential. However, the innervation of the LVP remains controversial.1–7 The conventional view is the innervation via the pharyngeal plexus, whereas the conflicting view is the double innervation via the pharyngeal plexus and the facial Nerve.
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three dimensional imaging of palatal muscles in the human embryo and fetus development of levator veli palatini and clinical importance of the Lesser Palatine Nerve
Developmental Dynamics, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Toru Kanahashi, Akio Yoneyama, Hirohiko Imai, Tetsuya Matsuda, Tohoru Takeda, Katsuya Kawai, Shigehiko SuzukiAbstract:Background: After palatoplasty, incomplete velopharyngeal closure in speech articulation sometimes persists, despite restoration of deglutition function. The levator veli palatini (LVP) is believed to be significantly involved with velopharyngeal function in articulation; however, the development and innervation of LVP remain obscure. The development of LVP in human embryos and fetuses has not been systematically analyzed using the Carnegie stage (CS) to standardize documentation of development. Results: The anlage of LVP starts to develop at CS 21 beneath the aperture of the auditory tube (AT) to the pharynx. At CS 23, LVP runs along AT over its full length, as evidenced by three-dimensional image reconstruction. In the fetal stage, the Lesser Palatine Nerve (LPN) is in contact with LVP. Conclusions: The positional relationship between LVP and AT three-dimensionally, suggesting that LVP might be derived from the second branchial arch. Based on histological evidence, we hypothesize that motor components from the facial Nerve may run along LPN, believed to be purely sensory. The multiple innervation of LVP by LPN and pharyngeal plexus may explain the postpalatoplasty discrepancy between the partial impairment in articulation vs. the functional restoration of deglutition. That is, the contribution of LPN is greater in articulation than in deglutition. Developmental Dynamics 245:123–131, 2016. © 2015 Wiley Periodicals, Inc.
Hideaki Kishimoto - One of the best experts on this subject based on the ideXlab platform.
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the Lesser Palatine Nerve innervates the levator veli palatini muscle
Plastic and reconstructive surgery. Global open, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Katsuya Kawai, Yoshitaka Matsuura, Shigehiko SuzukiAbstract:The levator veli palatini (LVP) muscle is responsible for the elevation of the soft palate and is critically involved in speech and deglutition. Therefore, repair of the muscle during palatoplasty for cleft palate is essential. However, the innervation of the LVP remains controversial.1–7 The conventional view is the innervation via the pharyngeal plexus, whereas the conflicting view is the double innervation via the pharyngeal plexus and the facial Nerve.
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three dimensional imaging of palatal muscles in the human embryo and fetus development of levator veli palatini and clinical importance of the Lesser Palatine Nerve
Developmental Dynamics, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Toru Kanahashi, Akio Yoneyama, Hirohiko Imai, Tetsuya Matsuda, Tohoru Takeda, Katsuya Kawai, Shigehiko SuzukiAbstract:Background: After palatoplasty, incomplete velopharyngeal closure in speech articulation sometimes persists, despite restoration of deglutition function. The levator veli palatini (LVP) is believed to be significantly involved with velopharyngeal function in articulation; however, the development and innervation of LVP remain obscure. The development of LVP in human embryos and fetuses has not been systematically analyzed using the Carnegie stage (CS) to standardize documentation of development. Results: The anlage of LVP starts to develop at CS 21 beneath the aperture of the auditory tube (AT) to the pharynx. At CS 23, LVP runs along AT over its full length, as evidenced by three-dimensional image reconstruction. In the fetal stage, the Lesser Palatine Nerve (LPN) is in contact with LVP. Conclusions: The positional relationship between LVP and AT three-dimensionally, suggesting that LVP might be derived from the second branchial arch. Based on histological evidence, we hypothesize that motor components from the facial Nerve may run along LPN, believed to be purely sensory. The multiple innervation of LVP by LPN and pharyngeal plexus may explain the postpalatoplasty discrepancy between the partial impairment in articulation vs. the functional restoration of deglutition. That is, the contribution of LPN is greater in articulation than in deglutition. Developmental Dynamics 245:123–131, 2016. © 2015 Wiley Periodicals, Inc.
Sun Ok Song - One of the best experts on this subject based on the ideXlab platform.
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the effect of Lesser Palatine Nerve block on pain after elective pediatric tonsillectomy
Korean Journal of Anesthesiology, 2003Co-Authors: Sun Ok SongAbstract:Background: This prospective double-blind randomized study was performed to evaluate the an- algesic effect of Lesser Palatine Nerve block for postoperative pain control after a pediatric tonsillectomy and to compare the analgesic effects of pre-emptive versus postoperative blocks. Methods: Forty-eight ASA class 1 children scheduled for an elective tonsillectomy were randomized into three groups. Patients received Lesser Palatine Nerve blocks using divided doses of 0.05 ml/kg of 0.2% ropivacaine 5 min prior to the beginning of tonsillectomy (Pre-block group or immediately after surgery (Post-block group. Patients allocated into the Control group did not receive any Nerve block. Postoperative pain was measured immediately after surgery 3 6 12 and 24 hours following operation using a 0 to 4 points pain scale based on a facial expression of pain scale ruler. Side effects and the number of analgesic injections were observed for 24 hours postoperatively. Results: No significant differences in the pain scores were observed immediately after surgery and at 3 6 12 and 24 hours after operation in the three groups (P > 0.05. The number of analgesic injections was similar in the groups. Conclusions: T he results of this study reveal that the Lesser Palatine ner ve block was not effective for postoperative pain control following pediatric tonsillectomy and that the pre-emptive block offered no pain control benefit over the postoperative block. Therefore we do not recommend Lesser Palatine Nerve blocks for the management of postoperative pain after pediatric tonsillectomy. (Korean J An- esthesiol 2003; 44: 820~827
Katsuya Kawai - One of the best experts on this subject based on the ideXlab platform.
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the Lesser Palatine Nerve innervates the levator veli palatini muscle
Plastic and reconstructive surgery. Global open, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Katsuya Kawai, Yoshitaka Matsuura, Shigehiko SuzukiAbstract:The levator veli palatini (LVP) muscle is responsible for the elevation of the soft palate and is critically involved in speech and deglutition. Therefore, repair of the muscle during palatoplasty for cleft palate is essential. However, the innervation of the LVP remains controversial.1–7 The conventional view is the innervation via the pharyngeal plexus, whereas the conflicting view is the double innervation via the pharyngeal plexus and the facial Nerve.
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three dimensional imaging of palatal muscles in the human embryo and fetus development of levator veli palatini and clinical importance of the Lesser Palatine Nerve
Developmental Dynamics, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Toru Kanahashi, Akio Yoneyama, Hirohiko Imai, Tetsuya Matsuda, Tohoru Takeda, Katsuya Kawai, Shigehiko SuzukiAbstract:Background: After palatoplasty, incomplete velopharyngeal closure in speech articulation sometimes persists, despite restoration of deglutition function. The levator veli palatini (LVP) is believed to be significantly involved with velopharyngeal function in articulation; however, the development and innervation of LVP remain obscure. The development of LVP in human embryos and fetuses has not been systematically analyzed using the Carnegie stage (CS) to standardize documentation of development. Results: The anlage of LVP starts to develop at CS 21 beneath the aperture of the auditory tube (AT) to the pharynx. At CS 23, LVP runs along AT over its full length, as evidenced by three-dimensional image reconstruction. In the fetal stage, the Lesser Palatine Nerve (LPN) is in contact with LVP. Conclusions: The positional relationship between LVP and AT three-dimensionally, suggesting that LVP might be derived from the second branchial arch. Based on histological evidence, we hypothesize that motor components from the facial Nerve may run along LPN, believed to be purely sensory. The multiple innervation of LVP by LPN and pharyngeal plexus may explain the postpalatoplasty discrepancy between the partial impairment in articulation vs. the functional restoration of deglutition. That is, the contribution of LPN is greater in articulation than in deglutition. Developmental Dynamics 245:123–131, 2016. © 2015 Wiley Periodicals, Inc.
Shigehito Yamada - One of the best experts on this subject based on the ideXlab platform.
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the Lesser Palatine Nerve innervates the levator veli palatini muscle
Plastic and reconstructive surgery. Global open, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Katsuya Kawai, Yoshitaka Matsuura, Shigehiko SuzukiAbstract:The levator veli palatini (LVP) muscle is responsible for the elevation of the soft palate and is critically involved in speech and deglutition. Therefore, repair of the muscle during palatoplasty for cleft palate is essential. However, the innervation of the LVP remains controversial.1–7 The conventional view is the innervation via the pharyngeal plexus, whereas the conflicting view is the double innervation via the pharyngeal plexus and the facial Nerve.
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three dimensional imaging of palatal muscles in the human embryo and fetus development of levator veli palatini and clinical importance of the Lesser Palatine Nerve
Developmental Dynamics, 2016Co-Authors: Hideaki Kishimoto, Shigehito Yamada, Toru Kanahashi, Akio Yoneyama, Hirohiko Imai, Tetsuya Matsuda, Tohoru Takeda, Katsuya Kawai, Shigehiko SuzukiAbstract:Background: After palatoplasty, incomplete velopharyngeal closure in speech articulation sometimes persists, despite restoration of deglutition function. The levator veli palatini (LVP) is believed to be significantly involved with velopharyngeal function in articulation; however, the development and innervation of LVP remain obscure. The development of LVP in human embryos and fetuses has not been systematically analyzed using the Carnegie stage (CS) to standardize documentation of development. Results: The anlage of LVP starts to develop at CS 21 beneath the aperture of the auditory tube (AT) to the pharynx. At CS 23, LVP runs along AT over its full length, as evidenced by three-dimensional image reconstruction. In the fetal stage, the Lesser Palatine Nerve (LPN) is in contact with LVP. Conclusions: The positional relationship between LVP and AT three-dimensionally, suggesting that LVP might be derived from the second branchial arch. Based on histological evidence, we hypothesize that motor components from the facial Nerve may run along LPN, believed to be purely sensory. The multiple innervation of LVP by LPN and pharyngeal plexus may explain the postpalatoplasty discrepancy between the partial impairment in articulation vs. the functional restoration of deglutition. That is, the contribution of LPN is greater in articulation than in deglutition. Developmental Dynamics 245:123–131, 2016. © 2015 Wiley Periodicals, Inc.