The Experts below are selected from a list of 240 Experts worldwide ranked by ideXlab platform
P Coriat - One of the best experts on this subject based on the ideXlab platform.
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circulating cardiac troponin t in Myocardial Contusion
Chest, 1997Co-Authors: M Ferjani, Gabriella Droc, Sophie Dreux, M Arthaud, J P Goarin, B Riou, P CoriatAbstract:Study objective Myocardial Contusion may induce life-threatening complications, but its diagnosis is difficult. Circulating cardiac troponin T is considered a highly sensitive and specific marker of Myocardial cell injury. We investigate the value of cardiac troponin T measurement in the diagnosis of Myocardial Contusion. Design Prospective study. Setting Level 1 trauma center Methods We prospectively measured circulating cardiac troponin T and performed echocardiography and continuous Holter monitoring in patients who had suffered blunt trauma. Myocardial Contusion was diagnosed in patients who fulfilled one of the following criteria: (1) an abnormal echocardiography compatible with Myocardial Contusion; (2) severe cardiac rhythm abnormalities; (3) severe cardiac conduction abnormalities; and (4) hemopericardium. Measurements and results One hundred twenty-eight patients were included and Myocardial Contusion was diagnosed in 29 patients. Patients with Myocardial Contusion had more severe trauma, experienced more frequently associated thoracic lesions, and had a lower left ventricular ejection fraction area (48±15 vs 61±10%; p (Aroc), which was significantly different from 0.50 ( Aroc =0.69; 95% confidence interval, 0.56 to 0.80). However, this improvement was not clinically acceptable (sensitivity, 0.31; specificity, 0.91). Conclusions Circulating cardiac troponin T measurement had a slightly greater diagnostic value than usual biological parameters (CK-MB, CK-MB/CK) in Myocardial Contusion. Nevertheless, it was concluded that an elevated circulating cardiac troponin T concentration has no important clinical value in the diagnosis of Myocardial Contusion.
M Ferjani - One of the best experts on this subject based on the ideXlab platform.
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circulating cardiac troponin t in Myocardial Contusion
Chest, 1997Co-Authors: M Ferjani, Gabriella Droc, Sophie Dreux, M Arthaud, J P Goarin, B Riou, P CoriatAbstract:Study objective Myocardial Contusion may induce life-threatening complications, but its diagnosis is difficult. Circulating cardiac troponin T is considered a highly sensitive and specific marker of Myocardial cell injury. We investigate the value of cardiac troponin T measurement in the diagnosis of Myocardial Contusion. Design Prospective study. Setting Level 1 trauma center Methods We prospectively measured circulating cardiac troponin T and performed echocardiography and continuous Holter monitoring in patients who had suffered blunt trauma. Myocardial Contusion was diagnosed in patients who fulfilled one of the following criteria: (1) an abnormal echocardiography compatible with Myocardial Contusion; (2) severe cardiac rhythm abnormalities; (3) severe cardiac conduction abnormalities; and (4) hemopericardium. Measurements and results One hundred twenty-eight patients were included and Myocardial Contusion was diagnosed in 29 patients. Patients with Myocardial Contusion had more severe trauma, experienced more frequently associated thoracic lesions, and had a lower left ventricular ejection fraction area (48±15 vs 61±10%; p (Aroc), which was significantly different from 0.50 ( Aroc =0.69; 95% confidence interval, 0.56 to 0.80). However, this improvement was not clinically acceptable (sensitivity, 0.31; specificity, 0.91). Conclusions Circulating cardiac troponin T measurement had a slightly greater diagnostic value than usual biological parameters (CK-MB, CK-MB/CK) in Myocardial Contusion. Nevertheless, it was concluded that an elevated circulating cardiac troponin T concentration has no important clinical value in the diagnosis of Myocardial Contusion.
Y Watanabe - One of the best experts on this subject based on the ideXlab platform.
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Myocardial Contusion caused by a baseball
Clinical Cardiology, 1996Co-Authors: Masaru Mirukawa, Kunihiko Hirose, Tatsuhiko Mori, Jyunya Kusukawa, Nobuyoshi Tomioka, Y WatanabeAbstract:Myocardial Contusion is a rare type of sports injury. We report a case of Myocardial Contusion caused by a baseball. In this patient, arrhythmias were induced by an exercise test 1 week after injury. That patients with Myocardial Contusion but without arrhythmias at rest need to be treated carefully is emphasized.
Basil M Rudusky - One of the best experts on this subject based on the ideXlab platform.
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Myocardial Contusion culminating in a ruptured pseudoaneurysm of the left ventricle a case report
Angiology, 2003Co-Authors: Basil M RuduskyAbstract:Blunt chest trauma continues to be one of the most common injuries at all ages. Trauma in general is the leading cause of death in the young to middle-aged segment of our population. Blunt chest injury is said to occur in more than one third of all motor vehicle accidents. Myocardial Contusion is the most frequent cardiac injury resulting from blunt chest trauma. Autopsy studies indicate that cardiac trauma was directly associated with death in approxi mately 10% of cases suffering blunt chest injury. Aneurysm formation as a sequela of blunt cardiac trauma is a rare entity and pseudoaneurysm formation is considerably more rare. A case of Myocardial Contusion resulting in Myocardial necrosis, rupture of the ventricle, and pseudoaneurysm formation with subsequent rupture and sudden death is presented.
A Edouard - One of the best experts on this subject based on the ideXlab platform.
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cardiac troponin i and Myocardial Contusion in the rabbit
Anesthesiology, 2000Co-Authors: Naomitsu Okubo, Cecile Hombrouck, Paul Fornes, Claudine Cosson, Kamran Samii, Jean Xavier Mazoit, A EdouardAbstract:Background: Patients with cardiac Contusion have a high risk of cardiac complications during emergency anesthesia. Despite the progress in cardiac imaging, a biologic marker of Myocardial damage such as cardiac troponin I remains useful and has been proposed in clinical practice. The relationship among histologic injury, left-ventricular function, and release of cardiac enzymes and cardiac troponin I has been investigated after a controlled Myocardial Contusion in a rabbit model. Methods: A global trauma (two levels of energy: 250 and 350 mJ) was produced on an isolated preparation of rabbit's heart, of which the temperature, perfusion flow, beating rate, and left-ventricular volume were kept constant. Left-ventricular pressure and its first derivative as a function of time were measured during a 60-min period after the blow; a timed collection of the effluent was made to assess creatine kinase, lactate dehydrogenase, and cardiac troponin I. At the end of the period, an anatomic score of the Contusion was calculated by histologic examination of the hearts. Results: Compared with a control group, the two levels of cardiac trauma resulted in a proportional anatomic injury significantly correlated with left-ventricular dysfunction (Δ%dP/dt max = -16 ± 12 and -36 ± 20% at 3 min, mean ± SD). Transient releases in cardiac markers after the lesser amount of trauma contrasted with a prolonged and biphasic release of cardiac troponin I after the greater amount. Peak cardiac troponin I level was correlated with anatomic injury (p = 0.596, P = 0.001) and negatively correlated with left-ventricular dysfunction (r = -0.375, P = 0.04). Conclusion: Cardiac troponin I is a marker of anatomic and functional consequences of experimental cardiac trauma and may be a predictive indicator of early posttraumatic cardiac complications during the postoperative period.
