Pulmonic Stenosis

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William G Williams - One of the best experts on this subject based on the ideXlab platform.

  • management of neonatal critical Pulmonic Stenosis in the balloon valvotomy era
    The Annals of Thoracic Surgery, 1990
    Co-Authors: J Caspi, John G Coles, Lee N Benson, Robert M Freedom, Patricia E Burrows, Jeffrey F Smallhorn, George A Trusler, William G Williams
    Abstract:

    Abstract We evaluated our recent experience with management of neonatal critical Pulmonic Stenosis and intact ventricular septum between 1982 and 1988. Thirty-nine patients (aged less than 3 months) were treated initially by operation (group A, n=19) or with balloon pulmonary valvotomy (group B, n=20). Patients in group A were younger (5 ± 1.3 versus 18 ± 4 days in group B) (mean ± standard error of the mean) and had a greater degree of hypoxia (oxygen tension, 55 ± 4 versus 80 ± 6 mm Hg) ( p

William C. Roberts - One of the best experts on this subject based on the ideXlab platform.

  • full development of consequences of congenital Pulmonic Stenosis in eighty four years
    American Journal of Cardiology, 2017
    Co-Authors: William C. Roberts, Paul A Grayburn, Joseph M Guileyardo, Robert C Stoler
    Abstract:

    Described herein is an 84-year-old woman, the oldest reported, with severe Pulmonic Stenosis who underwent a highly successful Pulmonic valvotomy at age 77 and highly unsuccessfully attempted percutaneous Pulmonic valve implantation at age 84. During the 84 years she developed nearly all clinical and morphologic consequences of Pulmonic Stenosis, including heavy calcification of the Pulmonic valve, heavy calcification of the tricuspid valve annulus, severe right ventricular wall thickening without ventricular cavity dilation, aneurysm of the pulmonary truck, multiple focal ventricular wall scars without narrowing of the epicardial coronary arteries, wall thickening and luminal narrowing of the intramural coronary arteries, and extremely low 12-lead QRS electrocardiographic voltage.

  • sudden death right ventricular infarction and abnormal right ventricular intramural coronary arteries in isolated congenital valvular Pulmonic Stenosis
    American Journal of Cardiology, 1993
    Co-Authors: Jamshid Shirani, Abarmard Maziar Zafari, William C. Roberts
    Abstract:

    Abstract Isolated congenital valvular pulmonk Stenosis is a relatively common anomaly of the heart. Fontana and Edwards' found Pulmonic Stenosis in 8 of 357 patients (2%) with congenital cardiac disease studied at necropsy. In unoperated patients with severe forms of this congenital valvular Pulmonic Stenosis, only 2 (aged 19 and 26 years, both men) had died suddenly.* Only 1 of 186 sudden deaths in patients with congenital cardiovascular disease (age range 1 to 21 years) was caused by Pulmonic Stenosis? This report describes a patient with isolated pulmonk Stenosis who died suddenly, and at necropsy also was found to have right ventricular infarction and abnormal intramural coronary arteries.

Wenpin Lien - One of the best experts on this subject based on the ideXlab platform.

  • infundibular Pulmonic Stenosis with intact ventricular septum a report of 15 surgically corrected patients
    International Journal of Cardiology, 1993
    Co-Authors: Kougi Shyu, Chuenden Tseng, Ingsh Chiu, C R Hung, Yungzu Tseng, Wenpin Lien
    Abstract:

    Abstract Infundibular Pulmonic Stenosis with intact ventricular septum of primary origin is an uncommon condition. We report 15 such patients (nine males and six females, aged 7–36 years) who had undergone surgical correction for the anomaly during the period between 1975 and 1992. The occurrence of this clinical setting represents 0.19% (15/7826) of all cardiac operations and 0.46% (15/3222) of congenital heart diseases undergoing surgical correction during that period of time. The lesion was of discrete fibromuscular hypertrophy of the infundibulum in all 15 patients. The presenting symptoms of most patients were exertional dyspnea and syncope; however, five patients with severe obstruction were asymptomatic. The peak systolic pressure gradient across the infundibulum ranged from 71 to 230 mmHg. There was only one operative death; the remainder had remained well following the surgery over a mean follow-up period of 35 months. Surgical correction for infundibular Pulmonic Stenosis is rewarding in the absence of heart failure.

James W Buchanan - One of the best experts on this subject based on the ideXlab platform.

  • pathogenesis of single right coronary artery and Pulmonic Stenosis in english bulldogs
    Journal of Veterinary Internal Medicine, 2001
    Co-Authors: James W Buchanan
    Abstract:

    : English Bulldogs are the most common breed to have Pulmonic Stenosis. Previous studies showed that this congenital heart abnormality in Bulldogs frequently is caused by a circumpulmonary left coronary artery originating from a single right coronary artery. Fetal anasarca also occurs often in Bulldogs and might represent congestive heart failure, but the cause is unknown. To determine if fetal anasarca is associated with a coronary anomaly and Pulmonic Stenosis, major coronary arteries were studied in 6 bulldog puppies with fetal anasarca. Five of the puppies had normal coronary arteries, and this led to the conclusion that fetal anasarca usually is not associated with major coronary abnormalities or Pulmonic Stenosis. The 6th puppy had single right coronary artery with circumpulmonary left coronary artery and moderate subvalvular Pulmonic Stenosis. Serial section histology suggests that the underlying cause of this syndrome is malformation of the left aortic sinus (of Valsalva) and inversion of the proximal segment of the left main coronary artery.

  • Pulmonic Stenosis caused by single coronary artery in dogs four cases 1965 1984
    Javma-journal of The American Veterinary Medical Association, 1990
    Co-Authors: James W Buchanan
    Abstract:

    : Single right coronary artery (CA) associated with Pulmonic Stenosis was found in 3 English Bulldogs and a Boxer, suggesting a genetic predisposition for the associated anomalies. The left main coronary branch arose from the single right CA, encircled the Pulmonic root over hypoplastic Pulmonic valves at the level of the obstruction, and appeared to be the primary cause of underlying Pulmonic Stenosis. Patch-graft surgery to relieve Pulmonic Stenosis caused death in 1 dog when the unrecognized anomalous CA was served during the procedure. The anomalous left CA was detectable by use of angiocardiography in all 4 dogs and was recognized before surgery in 2 of them. In 1 dog, a right ventricle-to-pulmonary trunk-bridging conduit was implanted to improve outflow from the right side of the heart.

J Caspi - One of the best experts on this subject based on the ideXlab platform.

  • management of neonatal critical Pulmonic Stenosis in the balloon valvotomy era
    The Annals of Thoracic Surgery, 1990
    Co-Authors: J Caspi, John G Coles, Lee N Benson, Robert M Freedom, Patricia E Burrows, Jeffrey F Smallhorn, George A Trusler, William G Williams
    Abstract:

    Abstract We evaluated our recent experience with management of neonatal critical Pulmonic Stenosis and intact ventricular septum between 1982 and 1988. Thirty-nine patients (aged less than 3 months) were treated initially by operation (group A, n=19) or with balloon pulmonary valvotomy (group B, n=20). Patients in group A were younger (5 ± 1.3 versus 18 ± 4 days in group B) (mean ± standard error of the mean) and had a greater degree of hypoxia (oxygen tension, 55 ± 4 versus 80 ± 6 mm Hg) ( p