Straight Sinus

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Philippe Hantson - One of the best experts on this subject based on the ideXlab platform.

  • Bilateral Thalamic Infarction
    The western journal of emergency medicine, 2012
    Co-Authors: Paul Geukens, Thierry Duprez, Philippe Hantson
    Abstract:

    A 35-year-old woman, without previous medical history except oral contraception, presented with sudden onset of stupor and clonic perseveration in the upper limbs. She was aphasic, but communicated by vertical movements of the head. Unenhanced brain computed tomography (CT) demonstrated hyperintensity of the Straight Sinus and hypo-intense areas within both thalami (Figure 1A) leading to a diagnosis of cerebral venous thrombosis (CVT). Despite anticoagulation therapy, the patient deteriorated with a Glasgow Coma Score at 7. A brain magnetic resonance imaging (MRI) obtained two days later confirmed extensive acute venous thrombosis involving the Straight Sinus, the right tentorial Sinus, the left proximal tentorial Sinus and the vein of Galan. A bilateral thalamic venous infarction was confirmed (Figure 1B). Local thrombectomy followed by thrombolysis within the Straight Sinus was unsuccessful. A mild hemorrhagic transformation of the bithalamic infarction was observed on follow-up MRI. Neurological status slowly improved over time. At 3-month follow-up, she had no cognitive deficit and was able to walk spite of residual spasticity. Figure 1. A. Unenhanced brain computed tomography. Strong hyperintensity within Straight Sinus due to the presence of an acute clot (arrow) together with subtle hypo-intensity within both thalamomesencephalic junctions. B. Magnetic resonance imaging with Fluid ... Bilateral infarctions of the thalami may be observed from either arterial or venous origin. They account for only 0.6% of all cerebral infarctions. Occlusion of the top of the basilar artery or of the so-called artery of Percheron – a developmental variant replacing the perforating medial thalamic arteries – is responsible for arterial ischemia. On venous side, the posterior group of thalamic veins drains into the Straight Sinus. A thrombosis of this vessel may also lead to a bilateral partial venous infarct due to the upstream overpressure. The diagnosis of CVT is often challenging, as clinical symptoms are highly variable and unspecific.1 Good clinical recovery may be observed, even after severe and prolonged deficits at early acute stage of the disease course. Common risk factors for CVT, when identified, include pregnancy, early post-partum, oral contraception, hypercoagulability and infection.

A. U. Yildirim - One of the best experts on this subject based on the ideXlab platform.

Paul Geukens - One of the best experts on this subject based on the ideXlab platform.

  • Bilateral Thalamic Infarction
    The western journal of emergency medicine, 2012
    Co-Authors: Paul Geukens, Thierry Duprez, Philippe Hantson
    Abstract:

    A 35-year-old woman, without previous medical history except oral contraception, presented with sudden onset of stupor and clonic perseveration in the upper limbs. She was aphasic, but communicated by vertical movements of the head. Unenhanced brain computed tomography (CT) demonstrated hyperintensity of the Straight Sinus and hypo-intense areas within both thalami (Figure 1A) leading to a diagnosis of cerebral venous thrombosis (CVT). Despite anticoagulation therapy, the patient deteriorated with a Glasgow Coma Score at 7. A brain magnetic resonance imaging (MRI) obtained two days later confirmed extensive acute venous thrombosis involving the Straight Sinus, the right tentorial Sinus, the left proximal tentorial Sinus and the vein of Galan. A bilateral thalamic venous infarction was confirmed (Figure 1B). Local thrombectomy followed by thrombolysis within the Straight Sinus was unsuccessful. A mild hemorrhagic transformation of the bithalamic infarction was observed on follow-up MRI. Neurological status slowly improved over time. At 3-month follow-up, she had no cognitive deficit and was able to walk spite of residual spasticity. Figure 1. A. Unenhanced brain computed tomography. Strong hyperintensity within Straight Sinus due to the presence of an acute clot (arrow) together with subtle hypo-intensity within both thalamomesencephalic junctions. B. Magnetic resonance imaging with Fluid ... Bilateral infarctions of the thalami may be observed from either arterial or venous origin. They account for only 0.6% of all cerebral infarctions. Occlusion of the top of the basilar artery or of the so-called artery of Percheron – a developmental variant replacing the perforating medial thalamic arteries – is responsible for arterial ischemia. On venous side, the posterior group of thalamic veins drains into the Straight Sinus. A thrombosis of this vessel may also lead to a bilateral partial venous infarct due to the upstream overpressure. The diagnosis of CVT is often challenging, as clinical symptoms are highly variable and unspecific.1 Good clinical recovery may be observed, even after severe and prolonged deficits at early acute stage of the disease course. Common risk factors for CVT, when identified, include pregnancy, early post-partum, oral contraception, hypercoagulability and infection.

R. Shane Tubbs - One of the best experts on this subject based on the ideXlab platform.

  • Case report of a vertical Straight Sinus with hydrocephalus and Chiari I malformation.
    Anatomy & cell biology, 2020
    Co-Authors: Dany Hage, Joe Iwanaga, Aaron S. Dumont, R. Shane Tubbs
    Abstract:

    The Straight Sinus is a division of the dural venous Sinuses, found beneath the splenium of the corpus callosum. At the internal occipital protuberance, it comes together with the superior sagittal Sinus and transverse Sinus to form the torcular Herophili. It functions as a major site of venous drainage for the cerebellum, inferior sagittal Sinus, and vein of Galen. Many morphological variations have been reported involving the angulation, positioning, and number of Straight Sinuses present. Patients with Chiari II and III malformations have been observed to have a high incidence of anatomical variation with their dural venous Sinuses, including vertically oriented Straight Sinuses. Additionally, there is a high rate of hydrocephalus in this patient population. Herein, we report a vertically oriented Straight Sinus in a child.

  • The Straight Sinus
    Anatomy Imaging and Surgery of the Intracranial Dural Venous Sinuses, 2020
    Co-Authors: Matthew Protas, R. Shane Tubbs
    Abstract:

    Abstract The Straight Sinus travels posteriorly and inferiorly beneath the splenium of the corpus callosum toward the internal occipital protuberance between the areas where the falx cerebri meets the midline of the tentorium cerebelli. The Straight Sinus receives its major drainage from the great cerebral vein of Galen and is thus very important in drainage of deep brain veins. This chapter details the anatomy, variants, and clinical implications of this intradural venous Sinus.

Yuichiro Tanaka - One of the best experts on this subject based on the ideXlab platform.

  • Total resection of torcular herophili hemangiopericytoma with radial artery graft: case report.
    Neurosurgery, 1995
    Co-Authors: Hisashi Nagashima, Shigeaki Kobayashi, Toshiki Takemae, Yuichiro Tanaka
    Abstract:

    A case of recurrent hemangiopericytoma involving the torcular herophili is reported, with special reference to the reconstruction of the Straight Sinus with a radial artery graft. In preoperative investigations, the tumor was found invading the Straight and transverse Sinuses. The involved occipital superior sagittal Sinus had been removed in the previous surgery. A collateral bypass was observed from the superior sagittal Sinus to the Straight Sinus, and the veins of Rosenthal were not opacified, suggesting poor venous drainage from the deep venous system to the cavernous Sinus. We considered it necessary to reconstruct the Straight and right transverse Sinuses to achieve total removal of the tumor. The affected Sinuses were removed and were successfully reconstructed with a radial artery interposition graft between the Straight and right transverse Sinus. The postoperative course was good, and the bypass was patent on the postoperative angiogram. The details of the operative technique are described.