Subdural Effusion

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Nobuhiko Aoki - One of the best experts on this subject based on the ideXlab platform.

  • infected Subdural Effusion associated with resolving Subdural hematoma case report
    Neurologia Medico-chirurgica, 1997
    Co-Authors: Nobuhiko Aoki, Tatsuo Sakai, Akihiro Oikawa, Touichiro Takizawa, Tsuneo Shishido
    Abstract:

    A 70-year-old male presented with rapid neurological deterioration and fever 3 months after suffering a closed head trauma. He underwent craniotomy for possible Subdural empyema based on computed tomography and clinical findings. Dural incision revealed an outer membrane typical of chronic Subdural hematoma which covered a clear, yellowish fluid containing Campylobacter fetus. Histological examination confirmed the capsule of the hematoma, with a necrotic focus infiltrated by neutrophils. Administration of intravenous imipenem and topical tobramycin and cefalothin achieved total resolution of his neurological deficits. Development of the infected Subdural Effusion was probably secondary to bacterial infection in the pre-existing chronic Subdural hematoma in the resolving stage. The presence of the hematoma capsule always carries the risk of development of an infectious focus.

  • extracerebral fluid collections in infancy role of magnetic resonance imaging in differentiation between Subdural Effusion and subarachnoid space enlargement
    Journal of Neurosurgery, 1994
    Co-Authors: Nobuhiko Aoki
    Abstract:

    The pathological process of extracerebral fluid collections in infancy includes Subdural Effusion and enlargement of the subarachnoid spaces. Both conditions have traditionally been investigated as a single clinical entity, because of difficulty in differentiating between them. The prognosis of Subdural Effusion is not as benign as that of enlargement of subarachnoid spaces, requiring differential diagnosis between these disorders. The present study was conducted to elucidate whether this differentiation could be made on magnetic resonance (MR) images. The series consisted of 16 infants aged 10 months or younger, including eight with verified Subdural Effusion and eight in whom a diagnosis of enlargement of the subarachnoid spaces was achieved by neuroimaging studies other than MR imaging. In all eight patients with Subdural Effusion, the intensity of the fluid was greater than that of cerebrospinal fluid (CSF) in at least one of the sequences using T1-weighted, proton-density, and T2-weighted MR images. The flow-void sign, indicating vessels in the fluid spaces, was not seen in any of these eight patients. On the other hand, in all eight patients with enlargement of the subarachnoid spaces, the fluid was isointense in relation to CSF, and vascular flow-void areas were seen in at least one of the MR imaging sequences. Based on these observations, it is concluded that differentiation between Subdural Effusion and enlargement of the subarachnoid spaces can be established by focusing on two aspects of MR imaging findings: 1) the intensity of the fluid, which is either iso- or hyperintense relative to CSF, and 2) the presence or absence of vascular flow-void areas in the fluid spaces.

  • evolution of chronic Subdural hematoma after burr hole exploration for Subdural Effusion case report
    Neurologia Medico-chirurgica, 1994
    Co-Authors: Nobuhiko Aoki, Tatsuo Sakai, Akihiro Oikawa
    Abstract:

    A 57-year-old male developed Subdural Effusion after head trauma, which remained asymptomatic and unchanged in volume during a follow-up period of 3 months. A typical chronic Subdural hematoma (CSDH) developed 6 weeks after burr-hole exploration in spite of the absence of hematoma capsule or blood components in the Effusion. The CSDH was successfully treated by irrigation and drainage. This case suggests that the presence of blood in Subdural Effusion may be the trigger for evolution of CSDH. We recommend that asymptomatic Subdural Effusion should be followed up without surgical intervention.

Volker Seifert - One of the best experts on this subject based on the ideXlab platform.

  • cerebral venous sinus thrombosis manifesting as bilateral Subdural Effusion
    Acta Neurologica Scandinavica, 2003
    Co-Authors: G Marquardt, Stefan Weidauer, Heinrich Lanfermann, Volker Seifert
    Abstract:

    Three patients with bilateral Subdural Effusion, an exclusive manifestation of cerebral venous sinus thrombosis (CVST), are presented. A possible explanation of this rare occurrence is provided, and the differential therapeutic strategies are discussed. We propose to consider CVST in cases of Subdural Effusions of obscure origin. Appropriate imaging studies should not be delayed if there is suspicion of sinus thrombosis to enable adequate therapy to be started as soon as possible.

Pochou Liliang - One of the best experts on this subject based on the ideXlab platform.

  • contralateral Subdural Effusion related to decompressive craniectomy performed in patients with severe traumatic brain injury
    Injury-international Journal of The Care of The Injured, 2012
    Co-Authors: Haokuang Wang, Chengloong Liang, Yuduan Tsai, Kuowei Wang, Pochou Liliang
    Abstract:

    Abstract Background Contralateral Subdural Effusion caused by decompressive craniectomy (DC) is not uncommon. However, it has rarely been reported. Method From 2004 to 2008, 123 severe traumatic brain injury (TBI) patients were identified as having undergone DC for increased intracranial pressure (IICP) with or without removal of a blood clot or contused brain. Of these 123 patients, nine developed delayed contralateral Subdural Effusion. Demographics, clinical presentations, treatment and outcome were reported. Results The overall incidence of contralateral Subdural Effusion was 7.3%. On average, this complication was found 23 days after DC. Of the nine patients, six had neurological deterioration and received drainage through a burr hole. One patient needed a subsequent subduro-peritoneal shunting because of recurrent Subdural Effusion. Conclusion Contralateral Subdural Effusions may be not uncommon and need more aggressive treatment because of their tendency to cause midline shift. Surgical intervention may be warranted if the patients develop deteriorating clinical manifestations or if the Subdural Effusion has an apparent mass effect.

Alfred Witzmann - One of the best experts on this subject based on the ideXlab platform.

  • bilateral Subdural Effusion and cerebral displacement associated with spontaneous intracranial hypotension diagnostic and management strategies report of two cases
    Journal of Neurosurgery, 2002
    Co-Authors: Nedal Hejazi, Muder Alwitry, Alfred Witzmann
    Abstract:

    The authors describe two patients with bilateral Subdural Effusion and cerebral displacement associated with spontaneous intracranial hypotension (SIH) and discuss the possible pathophysiological origins of these abnormalities. The signs seen on magnetic resonance imaging in both cases, such as tonsillar descent, Subdural Effusion, meningeal enhancement, downward displacement of the optic chiasm, and crowding of this structure and the hypothalamus between the pituitary gland and brain, can help to establish the diagnosis of SIH. Therapy with a lumbar epidural blood patch resulted in the rapid resolution of all symptoms and most morphological abnormalities. The authors propose diagnostic and management strategies based on their own experiences and the reported cases of SIH in the medical literature.

Yuichiro Nonaka - One of the best experts on this subject based on the ideXlab platform.

  • traumatic subacute Subdural Effusion in the posterior fossa associated with secondary acute hydrocephalus in a neonate case report
    Journal of Neurosurgery, 2007
    Co-Authors: Hiroshi Mori, Hiroki Ohashi, Yuichiro Nonaka
    Abstract:

    The authors report on a neonatal patient with traumatic subacute Subdural Effusion in the posterior fossa associated with secondary acute hydrocephalus. The infant fell from his mother's hand onto the floor, injuring his left parietal region. Computed tomography (CT) scans of the patient's head revealed a linear fracture of the left parietal bone, a small contusion in the right temporal lobe, and a small Subdural hematoma in the right posterior fossa with thin Subdural Effusion. Serial CT scans revealed a progressive increase in Subdural Effusion bilaterally in the posterior fossa. On Day 7 the anterior fontanelle was tense and CT scans revealed marked hydrocephalus associated with thick Subdural Effusion in the posterior fossa. External drainage of both the Subdural Effusion and dilated lateral ventricles improved the patient's condition, and no reaccumulation of Subdural Effusion has been observed. The origin and treatment of this rare clinical entity is discussed.