The Experts below are selected from a list of 417 Experts worldwide ranked by ideXlab platform
Suresh Nair - One of the best experts on this subject based on the ideXlab platform.
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symptomatic posterior fossa and supratentorial Subdural Hygromas as a rare complication following foramen magnum decompression for chiari malformation type i
Journal of Neurosurgery, 2011Co-Authors: Biji Bahuleyan, Girish Menon, Easwer Hariharan, Mridul Sharma, Suresh NairAbstract:Symptomatic Subdural Hygroma due to foramen magnum decompression for Chiari malformation Type I is extremely rare. The authors present their experience with 2 patients harboring such lesions and discuss treatment issues. They conclude that the possibility of Subdural Hygromas should be considered in all patients presenting with increased intracranial tension following foramen magnum decompression for Chiari malformation Type I. Immediate neuroimaging and appropriate surgical intervention provides a good outcome.
Stephen Honeybul - One of the best experts on this subject based on the ideXlab platform.
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incidence and risk factors for post traumatic hydrocephalus following decompressive craniectomy for intractable intracranial hypertension and evacuation of mass lesions
Journal of Neurotrauma, 2012Co-Authors: Stephen Honeybul, Kwok M HoAbstract:Abstract There continues to be a considerable interest in decompressive craniectomy in the management of severe traumatic brain injury (TBI). Though technically straightforward, the procedure is not without significant complications. In this study we assessed the incidence and risk factors for the development of Subdural Hygroma and hydrocephalus after decompressive craniectomy. A total of 195 patients who had had a decompressive craniectomy for severe TBI between 2004 and 2010 at the two major trauma centers in Western Australia were considered. Of the 166 patients who survived after the acute hospital stay, 93 (56%; 95% confidence interval [CI] 48,63%) developed Subdural Hygroma; 45 patients (48%) had unilateral and 48 patients (52%) had bilateral Subdural Hygromas. Of the 159 patients who survived more than 6 months after surgery, 72 (45%; 95% CI 38,53%) developed radiological evidence of ventriculomegaly, and 26 of these 72 patients (36%; 95% CI 26,48%) developed clinical evidence of hydrocephalus and...
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incidence and risk factors for post traumatic hydrocephalus following decompressive craniectomy for intractable intracranial hypertension and evacuation of mass lesions
Journal of Neurotrauma, 2012Co-Authors: Stephen HoneybulAbstract:There continues to be a considerable interest in decompressive craniectomy in the management of severe traumatic brain injury (TBI). Though technically straightforward, the procedure is not without significant complications. In this study we assessed the incidence and risk factors for the development of Subdural Hygroma and hydrocephalus after decompressive craniectomy. A total of 195 patients who had had a decompressive craniectomy for severe TBI between 2004 and 2010 at the two major trauma centers in Western Australia were considered. Of the 166 patients who survived after the acute hospital stay, 93 (56%; 95% confidence interval [CI] 48,63%) developed Subdural Hygroma; 45 patients (48%) had unilateral and 48 patients (52%) had bilateral Subdural Hygromas. Of the 159 patients who survived more than 6 months after surgery, 72 (45%; 95% CI 38,53%) developed radiological evidence of ventriculomegaly, and 26 of these 72 patients (36%; 95% CI 26,48%) developed clinical evidence of hydrocephalus and required a ventriculoperitoneal (VP) shunt. Maximum intracranial pressure prior to decompression (p=0.005), Subdural Hygroma (p=0.012), and a lower admission Glasgow Coma Scale score (p=0.009), were significant risk factors for hydrocephalus after decompressive craniectomy. Hydrocephalus requiring a VP shunt was associated with a higher risk of unfavorable neurological outcomes at 18 months (odds ratio 7.46; 95%CI 1.17,47.4; p=0.033), after adjusting for other factors. Our results showed a clear association between injury severity, Subdural Hygroma, and hydrocephalus, suggesting that damage to the cerebrospinal fluid drainage pathways contributes to the primary brain injury rather than the margin of the craniectomy as the factor responsible for these complications.
Jeanpaul Misson - One of the best experts on this subject based on the ideXlab platform.
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spontaneous arachnoid cyst rupture in a previously asymptomatic child a case report
European Journal of Paediatric Neurology, 2004Co-Authors: Annelise Poirrier, Issa Ngossotetanye, M Mouchamps, Jeanpaul MissonAbstract:Abstract Arachnoid cysts are benign congenital cavities arising in the subarachnoid space. Non-traumatic Subdural effusion of cerebrospinal fluid is a rare complication requiring surgical treatment. Case Report: A 15-year-old boy was admitted to the hospital because of symptoms related to acute intracranial hypertension (headache and vomiting). The cerebral CT-scan revealed a Subdural Hygroma adjoining a voluminous Sylvian arachnoid cyst. Two arachnoid cysts were incidentally discovered 11 years before this dramatic complication. Moreover, the patient had suffered a cerebral concussion 2 years earlier, but interestingly did not develop cystic hemorrhage or rupture, contrary to numerous cases previously described in the literature. The location of the cysts and their regular follow-up did not allow foreseeing a cystic rupture. Hygroma evacuation was first performed after which a Subdural peritoneal shunting, using a programmable opening pressure valve, was implanted. Spontaneous rupture into the Subdural space represents an unusual complication of arachnoid cysts. Clinical aspects, radiographic findings, pathogenesis and surgical management are described. It is important to point out that Subdural Hygroma or haematoma should never be excluded in the absence of trauma history, even in the case of small non-progressive cysts regularly supervised.
Biji Bahuleyan - One of the best experts on this subject based on the ideXlab platform.
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symptomatic posterior fossa and supratentorial Subdural Hygromas as a rare complication following foramen magnum decompression for chiari malformation type i
Journal of Neurosurgery, 2011Co-Authors: Biji Bahuleyan, Girish Menon, Easwer Hariharan, Mridul Sharma, Suresh NairAbstract:Symptomatic Subdural Hygroma due to foramen magnum decompression for Chiari malformation Type I is extremely rare. The authors present their experience with 2 patients harboring such lesions and discuss treatment issues. They conclude that the possibility of Subdural Hygromas should be considered in all patients presenting with increased intracranial tension following foramen magnum decompression for Chiari malformation Type I. Immediate neuroimaging and appropriate surgical intervention provides a good outcome.
Steven L. Giannotta - One of the best experts on this subject based on the ideXlab platform.
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arachnoid cyst rupture producing Subdural Hygroma and intracranial hypertension case reports
Neurosurgery, 1997Co-Authors: Felipe C Albuquerque, Steven L. GiannottaAbstract:OBJECTIVE: To analyze the association between arachnoid cysts and Subdural Hygromas. METHODS: We reviewed five cases of arachnoid cysts that ruptured, producing acute Subdural Hygromas. The surgical management and diagnostic methods used are assessed. RESULTS: Five male patients ranging in age from 6 to 25 years sustained the rupture of arachnoid cysts, which produced acute Subdural Hygromas. Four of the patients had incurred blunt head trauma. All patients presented with symptoms referable to intracranial hypertension. The pathognomonic features of a middle fossa arachnoid cyst (MFAC) were noted on the computed tomographic scans and/or magnetic resonance images of each patient. The Hygroma exerted mass effect on the ipsilateral hemisphere and was noted to be under significant pressure at the time of surgical intervention in each case. Two of the five cases are unique in the literature. In one, a coexisting quadrigeminal cyst ruptured, producing a Subdural Hygroma ipsilateral to the MFAC and dilating the basal cisterns. In the other, the MFAC ruptured into the basal cisterns as well as into the Subdural space. The MFAC in each of the remaining three patients ruptured into the Subdural space alone. All patients were treated with drainage of the Subdural space. In the two patients in whom the basal cisterns were involved, both the Hygromas and the MFACs failed to change significantly in size. The Hygromas resolved completely and the MFACs decreased in size considerably in the three patients without cisternal involvement. CONCLUSION: The rupture of an arachnoid cyst can produce a Subdural Hygroma and intracranial hypertension. The latter mandates emergent drainage of the Subdural space. In patients in whom the basal cisterns are not dilated by cyst rupture, both the MFACs and Hygromas resolve after Subdural drainage.
