Tendinitis

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Andrew Carr - One of the best experts on this subject based on the ideXlab platform.

  • calcific Tendinitis natural history and association with endocrine disorders
    Journal of Shoulder and Elbow Surgery, 2007
    Co-Authors: P Harvie, T C B Pollard, Andrew Carr
    Abstract:

    A retrospective, observational cohort study of 102 consecutive patients (125 shoulders) with calcific Tendinitis is presented. Of the patients, 73 (71.6%) were women and 29 (28.4%) were men. Compared with population prevalences, significant levels of endocrine disorders were found. We compared 66 patients (62 women [93.9%] and 4 men [6.1%]; mean age, 50.3 years) (81 shoulders) with associated endocrine disease with 36 patients (11 women [30.6%] and 25 men [69.4%]); mean age, 52.4 years) (44 shoulders) without endocrine disease. The endocrine cohort was significantly younger than the non-endocrine cohort when symptoms started (mean, 40.9 years and 46.9 years, respectively), had significantly longer natural histories (mean, 79.7 months compared with 47.1 months), and had a significantly higher proportion who underwent operative treatment (46.9% compared with 22.7%). Disorders of thyroid and estrogen metabolism may contribute to calcific Tendinitis etiology. Classifying calcific Tendinitis into type I (idiopathic) and type II (secondary or endocrine-related) aids prognosis and management.

  • calcific Tendinitis natural history and association with endocrine disorders
    Journal of Bone and Joint Surgery-british Volume, 2005
    Co-Authors: P Harvie, T C B Pollard, Andrew Carr
    Abstract:

    The purpose of this study was to investigate the association of endocrine disease with calcific Tendinitis and the effects that such disease has on its natural history. A retrospective observational cohort study of 102 consecutive patients (125 shoulders) with calcific Tendinitis is presented. Seventy-three (71.6 %) female, 29 (28.4 %) male. Compared with population prevalences, significant levels of endocrine disorders were found in our study cohort. Sixty-six patients (81 shoulders, 62 female (93.9 %), 4 male (6.1 %), mean age 50.3 years) with associated endocrine disease were compared with 36 patients (44 shoulders, 11 female (30.6 %), 25 male (69.4 %), mean age 52.4 years) without endocrine disease. The endocrine cohort were significantly younger than the non-endocrine cohort when symptoms started (mean 40.9 years and 46.9 years respectively, p=0.0026), had significantly longer natural histories (mean 79.7 months compared with 47.1 months, p=0.0015) and a significantly higher proportion underwent operative treatment (46.9 % compared with 22.7 %, p=0.0014). Disorders of thyroid and oestrogen metabolism may contribute to calcific Tendinitis aetiology. Classifying calcific Tendinitis into Type I idiopathic and Type II secondary or endocrine-related aids prognosis and management.

M Braier - One of the best experts on this subject based on the ideXlab platform.

  • extracorporeal shock wave treatment for chronic calcific Tendinitis of the shoulder
    The Journal of Rheumatology, 1998
    Co-Authors: A Spindler, A Berman, E Lucero, M Braier
    Abstract:

    Objective. To study the clinical and radiological response of chronic calcific Tendinitis of the shoulder to extracorporeal shock wave treatment (ESWT). Methods. The study included 3 female patients, 42, 48, and 50 years of age, all with calcified Tendinitis of the rotator cuff. All had severe shoulder pain and limitation of motion. ESWT was done in one session with an extracorporeal shock wave lithotripter. Results. After 24 hours, a fragmentation of calcification was achieved, and the patients had no pain and had entirely regained their joint movement. After 2 years of followup they were clinically and radiologically asymptomatic, and there were no adverse effects or other complications. Conclusion. With its good tolerance, safety, and clinical and radiologic response, ESWT should be considered as an alternative therapy in the treatment of chronic calcific Tendinitis of the shoulder refractory to other therapies.

P Harvie - One of the best experts on this subject based on the ideXlab platform.

  • calcific Tendinitis natural history and association with endocrine disorders
    Journal of Shoulder and Elbow Surgery, 2007
    Co-Authors: P Harvie, T C B Pollard, Andrew Carr
    Abstract:

    A retrospective, observational cohort study of 102 consecutive patients (125 shoulders) with calcific Tendinitis is presented. Of the patients, 73 (71.6%) were women and 29 (28.4%) were men. Compared with population prevalences, significant levels of endocrine disorders were found. We compared 66 patients (62 women [93.9%] and 4 men [6.1%]; mean age, 50.3 years) (81 shoulders) with associated endocrine disease with 36 patients (11 women [30.6%] and 25 men [69.4%]); mean age, 52.4 years) (44 shoulders) without endocrine disease. The endocrine cohort was significantly younger than the non-endocrine cohort when symptoms started (mean, 40.9 years and 46.9 years, respectively), had significantly longer natural histories (mean, 79.7 months compared with 47.1 months), and had a significantly higher proportion who underwent operative treatment (46.9% compared with 22.7%). Disorders of thyroid and estrogen metabolism may contribute to calcific Tendinitis etiology. Classifying calcific Tendinitis into type I (idiopathic) and type II (secondary or endocrine-related) aids prognosis and management.

  • calcific Tendinitis natural history and association with endocrine disorders
    Journal of Bone and Joint Surgery-british Volume, 2005
    Co-Authors: P Harvie, T C B Pollard, Andrew Carr
    Abstract:

    The purpose of this study was to investigate the association of endocrine disease with calcific Tendinitis and the effects that such disease has on its natural history. A retrospective observational cohort study of 102 consecutive patients (125 shoulders) with calcific Tendinitis is presented. Seventy-three (71.6 %) female, 29 (28.4 %) male. Compared with population prevalences, significant levels of endocrine disorders were found in our study cohort. Sixty-six patients (81 shoulders, 62 female (93.9 %), 4 male (6.1 %), mean age 50.3 years) with associated endocrine disease were compared with 36 patients (44 shoulders, 11 female (30.6 %), 25 male (69.4 %), mean age 52.4 years) without endocrine disease. The endocrine cohort were significantly younger than the non-endocrine cohort when symptoms started (mean 40.9 years and 46.9 years respectively, p=0.0026), had significantly longer natural histories (mean 79.7 months compared with 47.1 months, p=0.0015) and a significantly higher proportion underwent operative treatment (46.9 % compared with 22.7 %, p=0.0014). Disorders of thyroid and oestrogen metabolism may contribute to calcific Tendinitis aetiology. Classifying calcific Tendinitis into Type I idiopathic and Type II secondary or endocrine-related aids prognosis and management.

Josef Simbrunner - One of the best experts on this subject based on the ideXlab platform.

  • pathways in the diagnosis of prevertebral Tendinitis
    European Journal of Radiology, 2012
    Co-Authors: Robert Vollmann, Georg Philipp Hammer, Josef Simbrunner
    Abstract:

    INTRODUCTION: The prevertebral Tendinitis is an inflammatory process, which affects the cervicothoracic prevertebral muscles. This extremely rare entity was first described by Hartley and Fahlgren in 1964 and until now there are just some case reports dealing with this process. Unfortunately it is quite easy to misdiagnose or mistake the prevertebral Tendinitis as an abscess, because of the imaging features. The aim of this case series is to offer guidelines in the diagnosis of this rare disease to prevent unnecessary surgery. MATERIAL AND METHODS: Six patients with already by imaging or retrospectively after surgery by pathologic report diagnosed prevertebral Tendinitis were included in this study. None of these patients suffered from a chronically inflammatory disease. Three patients just received contrast enhanced computed tomography (CT) and another group of three patients received magnetic resonance imaging (MRI). In two out of three MRI examinations, we additionally performed diffusion weighted images and calculated the apparent diffusion coefficient (ADC) map. The laboratory reports obtained on the day of the computed tomography (CT) or magnetic resonance imaging (MRI) examinations were reviewed for C-reactive protein (CRP) and white blood cell count (WBCC). RESULTS: All patients revealed a prevertebral cervical effusion. Five out of six patients showed amorphous calcifications in the tendon of the prevertebral muscles. In one case calcifications could not be identified at all because of very strong beam hardening artefacts caused by dental prothesis. The CRP values were increased in all patients (mean value 44.9 mg/l; SD ± 28.3). However, WBCC remained normal (mean value 8.4G/l; SD ± 2.7). Only for the two patients who received DWI it was possible to assess the quality of the prevertebral fluid accumulation and to detect the benign prevertebral effusion, which is typical for the retropharyngeal Tendinitis. CONCLUSION: According to the experience with our patients the best imaging feature is MRI with DWI and ADC map to reveal the benign prevertebral effusion and confirm the diagnosis of prevertebral Tendinitis. In some cases MRI might not be available. Here we recommend CT scans to detect typical prevertebral calcifications. Especially a slight elevation of CRP and normal WBCC make the prevertebral Tendinitis more likely.

A Spindler - One of the best experts on this subject based on the ideXlab platform.

  • extracorporeal shock wave treatment for chronic calcific Tendinitis of the shoulder
    The Journal of Rheumatology, 1998
    Co-Authors: A Spindler, A Berman, E Lucero, M Braier
    Abstract:

    Objective. To study the clinical and radiological response of chronic calcific Tendinitis of the shoulder to extracorporeal shock wave treatment (ESWT). Methods. The study included 3 female patients, 42, 48, and 50 years of age, all with calcified Tendinitis of the rotator cuff. All had severe shoulder pain and limitation of motion. ESWT was done in one session with an extracorporeal shock wave lithotripter. Results. After 24 hours, a fragmentation of calcification was achieved, and the patients had no pain and had entirely regained their joint movement. After 2 years of followup they were clinically and radiologically asymptomatic, and there were no adverse effects or other complications. Conclusion. With its good tolerance, safety, and clinical and radiologic response, ESWT should be considered as an alternative therapy in the treatment of chronic calcific Tendinitis of the shoulder refractory to other therapies.