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J. Field - One of the best experts on this subject based on the ideXlab platform.
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OBJECTIVE IMPROVEMENTS IN ALGODY FOLLOWING REGIONAL INTRAVENOUS GU
2016Co-Authors: J. Field, C. Monk, R M AtkinsAbstract:17 patients with established Algodystrophy following trauma were treated with serial intravenous regional guanethidine blockade. Subjective improvement occurred in all cases. This imp~ovem~~nt could be confirmed quantitatively using objective assessment criteria. The subjective and objective improvements demonstrated in each of the features of Algodystrophy correlate closely (p < ~.OO~). There appears to be no difference in the effectiveness of one block compared to brother in the same patient. Journal qf Hand Surgery (British and European Volume, 1993) I8B: 339-342 Algodystrophy (reflex sympathetic dystrophy or Siideck’s atrophy) is a syndrome affecting the extremit-ies, and occurs most frequently following trauma (Doury et al, 1981). In the upper limb, the condition consists of excessive tenderness of the fingers, joint stiffness, swelling and vasomotor instability (alterations in temperature, colour and sweating). As the condition progresses, atro-phy of the subcutaneous tissues, soft tissue contracture and regional osteoporosis occur. Hannington-Kiff first reported the technique of intra-venous regional guanethidine for treatment of algodys-trophy in 1974. Since then it has been widely accepted as an effective treatment for the condition, although the necessity of using anything apart from the tourniquet has recently been questioned (Glynn et al, personal communication). Hitherto studies have relied mainly on subjective evidence of improvement using this treatmen
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Measurement of finger stiffness in Algodystrophy.
Hand clinics, 2003Co-Authors: J. FieldAbstract:Using a single, simple goniometer measuring the range of joint movement at each joint in the hand, the author has developed a simple method of assessing joint stiffness and Algodystrophy. This not only involves testing all joints in the fingers but also is a simple technique that can be performed in the outpatient clinic. Arthrography has been used to measure resistance of passive motion of joints, finding that stiffness is worse in the morning and improves after physiotherapy and after injection with steroids. The author's method does not actively measure the resistance to passive motion in a joint, as does the arthrograph. As the patient recovers with time, however (see Fig. 2), the range of movement increases and this may be equivalent to the improvement seen in Bramley et al. Goniometry has been found to be a sensitive test for diagnosing Algodystrophy. Together with dolorimetry, however, the specificity of the two tests combined increases significantly while maintaining adequate sensitivity to use as a diagnostic tool. Consequently, it is possible to diagnose Algodystrophy at 1 week following Colles' fracture. This finding has important implications in that it probably means the initial injury is responsible for the disease causation rather than anything constitutional or psychological.
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Algodystrophy and its association with Dupuytren's disease
Journal of hand surgery (Edinburgh Scotland), 1999Co-Authors: J. A. Livingstone, J. FieldAbstract:Seventy-two patients were examined 9 weeks after sustaining a Colles’ fracture of the wrist for evidence of Algodystrophy. They were examined 18 months later for evidence of Dupuytren’s disease to determine the incidence of the association between the two conditions. Forty-one per cent of all patients had evidence of Dupuytren’s disease at 18 months following Colles’ fracture. Sixty-seven per cent of patients with Algodystrophy had evidence of Dupuytren’s disease compared with 19% of patients who showed no features of Algodystrophy.
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Algodystrophy is an early complication of colles fracture what are the implications
Journal of Hand Surgery (European Volume), 1997Co-Authors: J. Field, R M AtkinsAbstract:One hundred patients who had sustained a Colles' fracture were observed for features of Algodystrophy at 1, 5, 9 and 12 weeks following injury. The diagnosis of Algodystrophy was possible as soon as 1 week after fracture. Early diagnosis has important clinical implications: the aetiological factors may become apparent and different treatment modalities be identified; furthermore, early treatment can be started, limiting the morbidity of the condition. It is proposed that patients with features of Algodystrophy require physiotherapy after a Colles' fracture. Those without features may not.
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Psychological Distress Associated with Algodystrophy
Journal of hand surgery (Edinburgh Scotland), 1997Co-Authors: J. Field, F. V. GardnerAbstract:We have carried out a prospective study of the level of psychological distress in patients with Colles' fractures. Those who developed Algodystrophy did not show increased distress before the onset of the condition.
J. A. Kanis - One of the best experts on this subject based on the ideXlab platform.
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British Journal of Rheumatology 1991;30:291-294 BRIEF REPORT THE USE OF NASAL CALCITONIN IN THE TREATMENT OF POST- TRAUMATIC Algodystrophy
2016Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Calcitonin is widely used in the treatment of Algodystrophy but a major disadvantage is the need for its parenteral adminis-tration. For this reason, we evaluated the effect of 400 iu of nasal calcitonin in the treatment of post-traumatic algo-dystrophy in a prospective randomized double-blind study. We found no demonstrable effect on the clinical or skeletal progression of the disorder using sensitive methods of measuring the response to treatment. There was, however, a small but significant hypocalcaemic response in the treatment group despite no change in the other indices of bone turnover. Possible reasons for this lack of clinical and skeletal effect are discussed. KEY WORDS: Colles ' fracture. Reflex sympathetic dystrophy. Bone density. Serum calcium. THE treatment of Algodystrophy (reflex sympathetic dystrophy) includes sympathectomy, either by chemi-cal blockade, local anaesthetic infusion or surgery. These techniques are invasive and not without mor-bidity. Several drugs have been used including calci-tonin [1], corticosteroids [2], beta-blockers [3J and nifedipine [4]. Of these, calcitonin has received interest because of its analgesic action [5,6], effects o
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British Journal of Rheumatology 1994^3:240-248 ORIGINAL CLINICAL PAPERS Algodystrophy: AN UNDER-RECOGNIZED COMPLICATION OF MINOR
2016Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Algodystrophy is a poorly recognized condition of uncertain aetiology which presents with pain and tenderness, vascular instability, swelling and stiffness of an affected limb. It is most commonly seen after trauma. In order to ascertain its inci-dence, natural history and the degree of morbidity induced we studied prospectively 274 patients with Colles ' fracture. Algodystrophy, as judged by the presence of bone pain or tenderness, vasomotor symptoms, swelling and stiffness of the hand was noted in 28 % of patients. There was a significant association between the presence of these features (P<0.0001). The degree of trauma sustained was identified as a predisposing factor. Actuarial analysis showed a gradual resolution of symptoms. Six months after injury, the proportion of patients complaining of pain and swelling had fallen to 20-30%, vas-cular instability and tenderness to 50 % and stiffness to 80%. Failure to improve was associated with a significant loss of hand function (P<0.0001). By 1 yr, pain and tenderness, vascular instability and swelling had decreased still further but stiffness was still apparent in 50%. We believe that Algodystrophy is a neglected disorder and is far more common than formerly appreciated. Although it often resolves spontaneously, it is associated with a significant increase in short-term morbidity in the majority of patients and persistent dysfunction in a minority. KEY WORDS: Algodystrophy, Reflex sympathetic dystrophy, Natural history, Trauma, Colles ' fracture. Algodystrophy, or reflex sympathetic dystrophy, is a clinical syndrome characterized by pain, tenderness
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Algodystrophy: AN UNDER-RECOGNIZED COMPLICATION OF MINOR TRAUMA
British journal of rheumatology, 1994Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Algodystrophy is a poorly recognized condition of uncertain aetiology which presents with pain and tenderness, vascular instability, swelling and stiffness of an affected limb. It is most commonly seen after trauma. In order to ascertain its incidence, natural history and the degree of morbidity induced we studied prospectively 274 patients with Colles' fracture. Algodystrophy, as judged by the presence of bone pain or tenderness, vasomotor symptoms, swelling and stiffness of the hand was noted in 28% of patients. There was a significant association between the presence of these features (P < 0.0001). The degree of trauma sustained was identified as a predisposing factor. Actuarial analysis showed a gradual resolution of symptoms. Six months after injury, the proportion of patients complaining of pain and swelling had fallen to 20-30%, vascular instability and tenderness to 50% and stiffness to 80%. Failure to improve was associated with a significant loss of hand function (P < 0.0001). By 1 yr, pain and tenderness, vascular instability and swelling had decreased still further but stiffness was still apparent in 50%. We believe that Algodystrophy is a neglected disorder and is far more common than formerly appreciated. Although it often resolves spontaneously, it is associated with a significant increase in short-term morbidity in the majority of patients and persistent dysfunction in a minority.
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CHANGES IN CORTICAL AND TRABECULAR BONE IN Algodystrophy
British journal of rheumatology, 1993Co-Authors: D. R. Bickerstaff, D. Charlesworth, J. A. KanisAbstract:We studied the pattern of bone loss in the hand of 77 patients with Colles' fracture using metacarpal morphometry, single photon absorptiometry and a radiographic scoring system. Forty-four patients had post-traumatic Algodystrophy and the remainder served as controls. Both groups were immobilized in the same manner and for the same period of time and both showed loss of bone during immobilization. The loss of bone 7 weeks after fracture was significantly greater in Algodystrophy than in controls both at cortical (P < 0.05) and at trabecular sites (P < 0.001). Recovery of bone occurred in the control patients by 19 weeks after fracture at cortical sites and by 31 weeks in trabecular bone. In contrast, the bone loss seen in patients with Algodystrophy persisted for the 6-month duration of the follow-up, and up to 1 year in all nine patients studied for longer. These findings indicate that post-traumatic Algodystrophy is associated with regional skeletal losses greater than those following uncomplicated fracture and may result in irreversible changes in the structure and thus the strength of the bony architecture.
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THE USE OF NASAL CALCITONIN IN THE TREATMENT OF POST-TRAUMATIC Algodystrophy
British journal of rheumatology, 1991Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Calcitonin is widely used in the treatment of Algodystrophy but a major disadvantage is the need for its parenteral administration. For this reason, we evaluated the effect of 400 iu of nasal calcitonin in the treatment of post-traumatic Algodystrophy in a prospective randomized double-blind study. We found no demonstrable effect on the clinical or skeletal progression of the disorder using sensitive methods of measuring the response to treatment. There was, however, a small but significant hypocalcaemic response in the treatment group despite no change in the other indices of bone turnover. Possible reasons for this lack of clinical and skeletal effect are discussed.
Andrzej Zyluk - One of the best experts on this subject based on the ideXlab platform.
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Algodystrophy in children and adolescents: a review.
Polish orthopedics and traumatology, 2013Co-Authors: Andrzej ZylukAbstract:Algodystrophy occurs in children significantly less frequently than in adults. Symptomatology, course, responsiveness to treatment and prognosis of the pediatric disease is also different from the "adult" form. This paper presents substantial peculiarities of pediatric Algodystrophy: it occurs after relatively minor trauma, involves lower limb more frequently than upper limb and presents with pain, paleness and coldness of the skin in the affected part, as well as with serious functional impairment. Diagnosis of the condition is based on clinical grounds and no imaging is necessary to confirm the diagnosis. Psychological distress is suspected in the development of the disease in children and adolescents, but there is no definitive evidence supporting this view. Treatment of pediatric Algodystrophy should be complex and consist of physical therapy, psychotherapy, pain therapy and, in selected cases, sympathetic and somatic blocks. The literature emphasizes very poor knowledge of Algodystrophy in the pediatric community.
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Prevention of Algodystrophy of the upper limb
Chirurgia narzadow ruchu i ortopedia polska, 2007Co-Authors: Andrzej ZylukAbstract:Treatment of Algodystrophy is a challenge. Early recognition and immediate commencement of effective therapy gives a real chance of recovery, whereas progression into chronic phase is associated with poor prognosis and disability of the limb. Prevention of the condition is a reasonable approach, but little is known about it. Some authors have claimed that careful operative technique, anatomic dissection, avoidance of nerve traction, proper postoperative care and early mobilization of the limb after trauma or operation can reduce risk the frequency of Algodystrophy, but this has not been scientifically confirmed. This paper presents current knowledge about prophylaxis of Algodystrophy, based on literature and author's own experience. It was stated that, as yet, no specific, effective measures are known to prevent Algodystrophy. Administration of 0.5 g vitamin C for 50 days after fracture of the distal radius reduces risk of the condition, but is was reported in only one study. Operation of unstable distal radial fractures, avoiding of tight, uncomfortable plaster casts and painful, forced physiotherapy is believed to reduce risk of Algodystrophy, but it is based on observation rather than scientific evidence. Patients who recovered form Algodystrophy are not more susceptible to recurrence after further trauma or surgery. An early recognition of incipient Algodystrophy and immediate commencement of effective therapy (i.e., with Mannitol and Dexamethasone) was found to be the most important part of prophylaxis of the development of the florid condition.
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Chronic, refractory Algodystrophy
Chirurgia narzadow ruchu i ortopedia polska, 2006Co-Authors: Andrzej ZylukAbstract:Among the patients suffered form Algodystrophy, a subgroup characterized as refractory can be selected. This subgroup consists of exclusively young women, in whom the syndrome develops as a consequence of relatively minor trauma and frequently the course is associated with neurological symptoms and signs. The main problem in these patients is to control a severe, spontaneous pain of the affected limb, while impaired function of the hand is of less importance; in almost all cases the disease leads to almost total disability of the affected limb. The cause of the progressive course of the Algodystrophy and resistance to the treatment in these patients remains obscure. We present a series of 9 women in the mean age of 33 years, with chronic, refractory Algodystrophy of the upper limb lasting mean of 13 months at presentation. The diagnosis of the syndrome was based on clinical grounds, and the treatment included the following methods: mannitol combined with dexamethasone, regional intravenous blocks with methylprednosolone, regional intravenous blocks with phentolamine, phenoxybenzamine, sympathectomy, salmon calcitonin, continuous brachial plexus anaesthesia with bupivacaine, amitryptyline, gabapentin, thaildomid and botulin toxin. Four of the nine patients responded partially to the treatment, one underwent amputation of the affected hand, two are still under control after implantation of the catheter for continuous brachial plexus anaesthesia, and in remaining two patients the long lasting treatments totally failed.
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Shoulder pain and limited mobility in the course of Algodystrophy of the hand
Chirurgia narzadow ruchu i ortopedia polska, 2004Co-Authors: Andrzej Zyluk, Piotr Puchalski, Beata ZylukAbstract:UNLABELLED Algodystrophy typically affects distal parts of upper extremity: the hand and the wrist. Some patients, however, complain of pain and limited mobility in shoulder joint and these symptoms are not related to predisposed injury but appear secondary in the course of the condition. Cause and pathogenesis of this disorder in unknown. The objectives of the study were to investigate incidence of shoulder complains in the patients suffered of Algodystrophy of the hand and to evaluate results of the treatment with local steroid injections of proximal insertion of the tendon of biceps muscle. Based on notes of 78 patients with Algodystrophy within the hand, the incidence of shoulder complains was analysed. Evaluation of the results of the treatment of 18 patients was performed. RESULTS 24 patients (31%) complained of pain and limited mobility in shoulder joint. In 17 of these patients (71%) Algodystrophy was proceeded by fracture of the distal radius. In 16 patients shoulder complains appeared at the same time with Algodystrophy of the hand and in 8 they delayed for 1-3 months. X-rays performed in all patients revealed in 3 only mild osteopenia within proximal part of humerus. All patients had proximal part of bicipital tendons of the biceps muscle tender for palpation suggesting inflammation of this tendon. In 18 patients methylprednisolone and lignocaine was injected 1-3 times locally in the vicinity of the painful tendon. In 15 of these patients (83%) treatment was successful and relief of the pain was achieved within the period from 2 days to 4 weeks. Final assessment of the treatment of Algodystrophy was done mean at 13 months after the treatment was completed. Shoulder complaints disappeared in 17 from 24 patients (71%), and in 7 features persisted but were of milder intensity. These 7 patients had significantly worse final result of the treatment of the Algodystrophy of the hand than rest of the group.
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Early diagnosis of post-traumatic Algodystrophy
Chirurgia narzadow ruchu i ortopedia polska, 2003Co-Authors: Andrzej Zyluk, Piotr PuchalskiAbstract:The results of the retrospective analysis of the chronology and sequence of symptoms and signs occurrence in 64 patients who developed Algodystrophy as a consequence of trauma/operation are presented. The following features were noted: diffuse pain, swelling, reduction of movement of fingers, temperature and colour changes of the limb. Basing on these data, we determined how early after accident a set of features sufficient to establish the diagnosis could be detected. It was found that diagnosis of Algodystrophy could be made at 1 week in 22 patients (34%), at 2 weeks in 37 (58%), at 3 weeks in 46 (72%) and at 4 weeks in 53 patients (83%). Only in 11 patients (17%) Algodystrophy appeared later than 1 month after initial trauma. The syndrome has been eventually diagnosed at 1 month only in 20 patients (31%). A traditional approach to the Algodystrophy as a chronic pain syndrome caused probably considerable delay in the diagnosis, which occurred in almost 70% of our patients. The results of the study show that Algodystrophy can be diagnosed in most cases as early as at 1 month after initial trauma. Early diagnosis prevents of development of florid condition and allow the patients to recover in relatively short time.
D. R. Bickerstaff - One of the best experts on this subject based on the ideXlab platform.
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British Journal of Rheumatology 1991;30:291-294 BRIEF REPORT THE USE OF NASAL CALCITONIN IN THE TREATMENT OF POST- TRAUMATIC Algodystrophy
2016Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Calcitonin is widely used in the treatment of Algodystrophy but a major disadvantage is the need for its parenteral adminis-tration. For this reason, we evaluated the effect of 400 iu of nasal calcitonin in the treatment of post-traumatic algo-dystrophy in a prospective randomized double-blind study. We found no demonstrable effect on the clinical or skeletal progression of the disorder using sensitive methods of measuring the response to treatment. There was, however, a small but significant hypocalcaemic response in the treatment group despite no change in the other indices of bone turnover. Possible reasons for this lack of clinical and skeletal effect are discussed. KEY WORDS: Colles ' fracture. Reflex sympathetic dystrophy. Bone density. Serum calcium. THE treatment of Algodystrophy (reflex sympathetic dystrophy) includes sympathectomy, either by chemi-cal blockade, local anaesthetic infusion or surgery. These techniques are invasive and not without mor-bidity. Several drugs have been used including calci-tonin [1], corticosteroids [2], beta-blockers [3J and nifedipine [4]. Of these, calcitonin has received interest because of its analgesic action [5,6], effects o
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British Journal of Rheumatology 1994^3:240-248 ORIGINAL CLINICAL PAPERS Algodystrophy: AN UNDER-RECOGNIZED COMPLICATION OF MINOR
2016Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Algodystrophy is a poorly recognized condition of uncertain aetiology which presents with pain and tenderness, vascular instability, swelling and stiffness of an affected limb. It is most commonly seen after trauma. In order to ascertain its inci-dence, natural history and the degree of morbidity induced we studied prospectively 274 patients with Colles ' fracture. Algodystrophy, as judged by the presence of bone pain or tenderness, vasomotor symptoms, swelling and stiffness of the hand was noted in 28 % of patients. There was a significant association between the presence of these features (P<0.0001). The degree of trauma sustained was identified as a predisposing factor. Actuarial analysis showed a gradual resolution of symptoms. Six months after injury, the proportion of patients complaining of pain and swelling had fallen to 20-30%, vas-cular instability and tenderness to 50 % and stiffness to 80%. Failure to improve was associated with a significant loss of hand function (P<0.0001). By 1 yr, pain and tenderness, vascular instability and swelling had decreased still further but stiffness was still apparent in 50%. We believe that Algodystrophy is a neglected disorder and is far more common than formerly appreciated. Although it often resolves spontaneously, it is associated with a significant increase in short-term morbidity in the majority of patients and persistent dysfunction in a minority. KEY WORDS: Algodystrophy, Reflex sympathetic dystrophy, Natural history, Trauma, Colles ' fracture. Algodystrophy, or reflex sympathetic dystrophy, is a clinical syndrome characterized by pain, tenderness
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Algodystrophy: AN UNDER-RECOGNIZED COMPLICATION OF MINOR TRAUMA
British journal of rheumatology, 1994Co-Authors: D. R. Bickerstaff, J. A. KanisAbstract:Algodystrophy is a poorly recognized condition of uncertain aetiology which presents with pain and tenderness, vascular instability, swelling and stiffness of an affected limb. It is most commonly seen after trauma. In order to ascertain its incidence, natural history and the degree of morbidity induced we studied prospectively 274 patients with Colles' fracture. Algodystrophy, as judged by the presence of bone pain or tenderness, vasomotor symptoms, swelling and stiffness of the hand was noted in 28% of patients. There was a significant association between the presence of these features (P < 0.0001). The degree of trauma sustained was identified as a predisposing factor. Actuarial analysis showed a gradual resolution of symptoms. Six months after injury, the proportion of patients complaining of pain and swelling had fallen to 20-30%, vascular instability and tenderness to 50% and stiffness to 80%. Failure to improve was associated with a significant loss of hand function (P < 0.0001). By 1 yr, pain and tenderness, vascular instability and swelling had decreased still further but stiffness was still apparent in 50%. We believe that Algodystrophy is a neglected disorder and is far more common than formerly appreciated. Although it often resolves spontaneously, it is associated with a significant increase in short-term morbidity in the majority of patients and persistent dysfunction in a minority.
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CHANGES IN CORTICAL AND TRABECULAR BONE IN Algodystrophy
British journal of rheumatology, 1993Co-Authors: D. R. Bickerstaff, D. Charlesworth, J. A. KanisAbstract:We studied the pattern of bone loss in the hand of 77 patients with Colles' fracture using metacarpal morphometry, single photon absorptiometry and a radiographic scoring system. Forty-four patients had post-traumatic Algodystrophy and the remainder served as controls. Both groups were immobilized in the same manner and for the same period of time and both showed loss of bone during immobilization. The loss of bone 7 weeks after fracture was significantly greater in Algodystrophy than in controls both at cortical (P < 0.05) and at trabecular sites (P < 0.001). Recovery of bone occurred in the control patients by 19 weeks after fracture at cortical sites and by 31 weeks in trabecular bone. In contrast, the bone loss seen in patients with Algodystrophy persisted for the 6-month duration of the follow-up, and up to 1 year in all nine patients studied for longer. These findings indicate that post-traumatic Algodystrophy is associated with regional skeletal losses greater than those following uncomplicated fracture and may result in irreversible changes in the structure and thus the strength of the bony architecture.
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Radiographic changes in Algodystrophy of the hand
Journal of hand surgery (Edinburgh Scotland), 1991Co-Authors: D. R. Bickerstaff, D. P. O’doherty, John A. KanisAbstract:It is not certain to what extent the radiographic features of post-traumatic Algodystrophy can be differentiated from disuse atrophy following trauma. A semi-quantitative scoring system has been devised, grading the dominant features seen on radiography in both conditions following Colles' fracture. The technique had intra- and inter-observer errors of 14% and 17% respectively. When applied prospectively 7 weeks after Colles' fractures, there was a significantly greater score in patients with Algodystrophy (p less than 0.001) than those without. Analysis of the scores was performed to identify a score for the optimal identification of Algodystrophy. At a score of four or greater, 87.5% of patients with Algodystrophy were positively identified with a positive predictive value of 83%. It is suggested that use of this scoring system will aid in the early diagnosis of Algodystrophy.
G. C. Bannister - One of the best experts on this subject based on the ideXlab platform.
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features of Algodystrophy ten years after colles fracture
Journal of Hand Surgery (European Volume), 1992Co-Authors: J. Field, D. Warwick, G. C. BannisterAbstract:Abstract The long-term outcome of Algodystrophy is unknown. Ten years after Cones' fracture, 26% of 55 cases showed features of the syndrome. The finding of poor finger function three months following the fracture correlated significantly with the presence of components of Algodystrophy after ten years.
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Features of Algodystrophy ten years after Colles’ fracture
1992Co-Authors: J. Field, D. Warwick, G. C. BannisterAbstract:The long-term outcome of Algodystrophy is unknown. Ten years after Colles ’ fracture, 26 % of 55 cases showed features of the syndrome. The finding of poor finger function three months following the fracture correlated significantly with the presence of components of Algodystrophy after ten years. Journal of Hand Surgery (British Volume, 1992) 17B: 318-320 Algodystrophy (Siideck’s atrophy or reflex sympathetic dystrophy) is a poorly defined condition consisting of pain and tenderness, vasomotor instability, swelling and joint stiffness. Later, atrophy of the skin and subcutaneous tissues may occur with joint contracture and regional osteoporosis (Doury et al., 1981). In previous series of Colles ’ fracture the reported prevalence of Algodystrophy has been between 0.1% (Bacorn and Kurtzke, 1953) and 10 % (LidstrGm, 1959) of cases. In some series Algodystrophy is not reporte
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long term prognosis of displaced colles fracture a 10 year prospective review
Injury-international Journal of The Care of The Injured, 1992Co-Authors: J. Field, D. Warwick, G. C. Bannister, A G F GibsonAbstract:Abstract In all, 85 per cent of Colles' fractures are clinically satisfactory after 10 years. Clinical results improve little after 3 months. Displaced intraarticular fracture, wrist deformity and Algodystrophy are associated with an unsatisfactory outcome.
