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Joey S W Kwong - One of the best experts on this subject based on the ideXlab platform.
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sleep duration and risk of All Cause Mortality a flexible non linear meta regression of 40 prospective cohort studies
Sleep Medicine Reviews, 2017Co-Authors: Chang Xu, Matteo Rota, Chao Zhang, Ruixia Yuan, Hong Weng, Xiangyu Meng, Joey S W KwongAbstract:Summary Approximately 27–37% of the general population experience prolonged sleep duration and 12–16% report shortened sleep duration. However, prolonged or shortened sleep duration may be associated with serious health problems. A comprehensive, flexible, non-linear meta-regression with restricted cubic spline (RCS) was used to investigate the dose–response relationship between sleep duration and All-Cause Mortality in adults. Medline (Ovid), Embase, EBSCOhost—PsycINFO, and EBSCOhost—CINAHL Plus databases, reference lists of relevant review articles, and included studies were searched up to Nov. 29, 2015. Prospective cohort studies investigating the association between sleep duration and All-Cause Mortality in adults with at least three categories of sleep duration were eligible for inclusion. We eventuAlly included in our study 40 cohort studies enrolling 2,200,425 participants with 271,507 deaths. A J-shaped association between sleep duration and All-Cause Mortality was present: compared with 7 h of sleep (reference for 24-h sleep duration), both shortened and prolonged sleep durations were associated with increased risk of All-Cause Mortality (4 h: relative risk [RR] = 1.05; 95% confidence interval [CI] = 1.02–1.07; 5 h: RR = 1.06; 95% CI = 1.03–1.09; 6 h: RR = 1.04; 95% CI = 1.03–1.06; 8 h: RR = 1.03; 95% CI = 1.02–1.05; 9 h: RR = 1.13; 95% CI = 1.10–1.16; 10 h: RR = 1.25; 95% CI = 1.22–1.28; 11 h: RR = 1.38; 95% CI = 1.33–1.44; n = 29; P n = 13; P The current evidence suggests that insufficient or prolonged sleep may increase All-Cause Mortality. Women may be more susceptible to short sleep duration on All-Cause Mortality.
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sleep duration and risk of All Cause Mortality a flexible non linear meta regression of 40 prospective cohort studies
Sleep Medicine Reviews, 2017Co-Authors: Tongzu Liu, Matteo Rota, Chao Zhang, Ruixia Yuan, Hong Weng, Xiangyu Meng, Joey S W Kwong, Hui Cai, Mingjun ShiAbstract:Approximately 27-37% of the general population experience prolonged sleep duration and 12-16% report shortened sleep duration. However, prolonged or shortened sleep duration may be associated with serious health problems. A comprehensive, flexible, non-linear meta-regression with restricted cubic spline (RCS) was used to investigate the dose-response relationship between sleep duration and All-Cause Mortality in adults. Medline (Ovid), Embase, EBSCOhost-PsycINFO, and EBSCOhost-CINAHL Plus databases, reference lists of relevant review articles, and included studies were searched up to Nov. 29, 2015. Prospective cohort studies investigating the association between sleep duration and All-Cause Mortality in adults with at least three categories of sleep duration were eligible for inclusion. We eventuAlly included in our study 40 cohort studies enrolling 2,200,425 participants with 271,507 deaths. A J-shaped association between sleep duration and All-Cause Mortality was present: compared with 7 h of sleep (reference for 24-h sleep duration), both shortened and prolonged sleep durations were associated with increased risk of All-Cause Mortality (4 h: relative risk [RR] = 1.05; 95% confidence interval [CI] = 1.02-1.07; 5 h: RR = 1.06; 95% CI = 1.03-1.09; 6 h: RR = 1.04; 95% CI = 1.03-1.06; 8 h: RR = 1.03; 95% CI = 1.02-1.05; 9 h: RR = 1.13; 95% CI = 1.10-1.16; 10 h: RR = 1.25; 95% CI = 1.22-1.28; 11 h: RR = 1.38; 95% CI = 1.33-1.44; n = 29; P < 0.01 for non-linear test). With regard to the night-sleep duration, prolonged night-sleep duration was associated with increased All-Cause Mortality (8 h: RR = 1.01; 95% CI = 0.99-1.02; 9 h: RR = 1.08; 95% CI = 1.05-1.11; 10 h: RR = 1.24; 95% CI = 1.21-1.28; n = 13; P < 0.01 for non-linear test). Subgroup analysis showed females with short sleep duration a day (<7 h) were at high risk of All-Cause Mortality (4 h: RR = 1.07; 95% CI = 1.02-1.13; 5 h: RR = 1.08; 95% CI = 1.03-1.14; 6 h: RR = 1.05; 95% CI = 1.02-1.09), but males were not (4 h: RR = 1.01; 95% CI = 0.96-1.06; 5 h: RR = 1.02; 95% CI = 0.97-1.08; 6 h: RR = 1.02; 95% CI = 0.98-1.06). The current evidence suggests that insufficient or prolonged sleep may increase All-Cause Mortality. Women may be more susceptible to short sleep duration on All-Cause Mortality.
Franz H. Messerli - One of the best experts on this subject based on the ideXlab platform.
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sodium intake life expectancy and All Cause Mortality
European Heart Journal, 2021Co-Authors: Franz H. Messerli, Louis Hofstetter, Lamprini Syrogiannouli, Emrush Rexhaj, George C.m. Siontis, Christian Seiler, Sripal BangaloreAbstract:AIMS Since dietary sodium intake has been identified as a risk factor for cardiovascular disease and premature death, a high sodium intake can be expected to curtail life span. We tested this hypothesis by analysing the relationship between sodium intake and life expectancy as well as survival in 181 countries worldwide. METHODS AND RESULTS We correlated age-standardized estimates of country-specific average sodium consumption with healthy life expectancy at birth and at age of 60 years, death due to non-communicable diseases and All-Cause Mortality for the year of 2010, after adjusting for potential confounders such as gross domestic product per capita and body mass index. We considered global health estimates as provided by World Health Organization. Among the 181 countries included in this analysis, we found a positive correlation between sodium intake and healthy life expectancy at birth (β = 2.6 years/g of daily sodium intake, R2 = 0.66, P < 0.001), as well as healthy life expectancy at age 60 (β = 0.3 years/g of daily sodium intake, R2 = 0.60, P = 0.048) but not for death due to non-communicable diseases (β = 17 events/g of daily sodium intake, R2 = 0.43, P = 0.100). Conversely, All-Cause Mortality correlated inversely with sodium intake (β = -131 events/g of daily sodium intake, R2 = 0.60, P < 0.001). In a sensitivity analysis restricted to 46 countries in the highest income class, sodium intake continued to correlate positively with healthy life expectancy at birth (β = 3.4 years/g of daily sodium intake, R2 = 0.53, P < 0.001) and inversely with All-Cause Mortality (β = -168 events/g of daily sodium intake, R2 = 0.50, P < 0.001). CONCLUSION Our observation of sodium intake correlating positively with life expectancy and inversely with All-Cause Mortality worldwide and in high-income countries argues against dietary sodium intake being a culprit of curtailing life span or a risk factor for premature death. These data are observational and should not be used as a base for nutritional interventions.
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Sodium intake, life expectancy, and All-Cause Mortality.
European heart journal, 2020Co-Authors: Franz H. Messerli, Louis Hofstetter, Lamprini Syrogiannouli, Emrush Rexhaj, George C.m. Siontis, Christian Seiler, Sripal BangaloreAbstract:AIMS Since dietary sodium intake has been identified as a risk factor for cardiovascular disease and premature death, a high sodium intake can be expected to curtail life span. We tested this hypothesis by analysing the relationship between sodium intake and life expectancy as well as survival in 181 countries worldwide. METHODS AND RESULTS We correlated age-standardized estimates of country-specific average sodium consumption with healthy life expectancy at birth and at age of 60 years, death due to non-communicable diseases and All-Cause Mortality for the year of 2010, after adjusting for potential confounders such as gross domestic product per capita and body mass index. We considered global health estimates as provided by World Health Organization. Among the 181 countries included in this analysis, we found a positive correlation between sodium intake and healthy life expectancy at birth (β = 2.6 years/g of daily sodium intake, R2 = 0.66, P
Sripal Bangalore - One of the best experts on this subject based on the ideXlab platform.
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sodium intake life expectancy and All Cause Mortality
European Heart Journal, 2021Co-Authors: Franz H. Messerli, Louis Hofstetter, Lamprini Syrogiannouli, Emrush Rexhaj, George C.m. Siontis, Christian Seiler, Sripal BangaloreAbstract:AIMS Since dietary sodium intake has been identified as a risk factor for cardiovascular disease and premature death, a high sodium intake can be expected to curtail life span. We tested this hypothesis by analysing the relationship between sodium intake and life expectancy as well as survival in 181 countries worldwide. METHODS AND RESULTS We correlated age-standardized estimates of country-specific average sodium consumption with healthy life expectancy at birth and at age of 60 years, death due to non-communicable diseases and All-Cause Mortality for the year of 2010, after adjusting for potential confounders such as gross domestic product per capita and body mass index. We considered global health estimates as provided by World Health Organization. Among the 181 countries included in this analysis, we found a positive correlation between sodium intake and healthy life expectancy at birth (β = 2.6 years/g of daily sodium intake, R2 = 0.66, P < 0.001), as well as healthy life expectancy at age 60 (β = 0.3 years/g of daily sodium intake, R2 = 0.60, P = 0.048) but not for death due to non-communicable diseases (β = 17 events/g of daily sodium intake, R2 = 0.43, P = 0.100). Conversely, All-Cause Mortality correlated inversely with sodium intake (β = -131 events/g of daily sodium intake, R2 = 0.60, P < 0.001). In a sensitivity analysis restricted to 46 countries in the highest income class, sodium intake continued to correlate positively with healthy life expectancy at birth (β = 3.4 years/g of daily sodium intake, R2 = 0.53, P < 0.001) and inversely with All-Cause Mortality (β = -168 events/g of daily sodium intake, R2 = 0.50, P < 0.001). CONCLUSION Our observation of sodium intake correlating positively with life expectancy and inversely with All-Cause Mortality worldwide and in high-income countries argues against dietary sodium intake being a culprit of curtailing life span or a risk factor for premature death. These data are observational and should not be used as a base for nutritional interventions.
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Sodium intake, life expectancy, and All-Cause Mortality.
European heart journal, 2020Co-Authors: Franz H. Messerli, Louis Hofstetter, Lamprini Syrogiannouli, Emrush Rexhaj, George C.m. Siontis, Christian Seiler, Sripal BangaloreAbstract:AIMS Since dietary sodium intake has been identified as a risk factor for cardiovascular disease and premature death, a high sodium intake can be expected to curtail life span. We tested this hypothesis by analysing the relationship between sodium intake and life expectancy as well as survival in 181 countries worldwide. METHODS AND RESULTS We correlated age-standardized estimates of country-specific average sodium consumption with healthy life expectancy at birth and at age of 60 years, death due to non-communicable diseases and All-Cause Mortality for the year of 2010, after adjusting for potential confounders such as gross domestic product per capita and body mass index. We considered global health estimates as provided by World Health Organization. Among the 181 countries included in this analysis, we found a positive correlation between sodium intake and healthy life expectancy at birth (β = 2.6 years/g of daily sodium intake, R2 = 0.66, P
Wei Bao - One of the best experts on this subject based on the ideXlab platform.
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association of skipping breakfast with cardiovascular and All Cause Mortality
Journal of the American College of Cardiology, 2019Co-Authors: Shuang Rong, Linda Snetselaar, Yangbo Sun, Buyun Liu, Robert B Wallace, Wei BaoAbstract:Abstract Background Skipping breakfast is common among U.S. adults. Limited evidence suggests that skipping breakfast is associated with atherosclerosis and cardiovascular disease. Objectives The authors sought to examine the association of skipping breakfast with cardiovascular and All-Cause Mortality. Methods This is a prospective cohort study of a nationAlly representative sample of 6,550 adults 40 to 75 years of age who participated in the National Health and Nutrition Examination Survey III 1988 to 1994. Frequency of breakfast eating was reported during an in-house interview. Death and underlying Causes of death were ascertained by linkage to death records through December 31, 2011. The associations between breakfast consumption frequency and cardiovascular and All-Cause Mortality were investigated by using weighted Cox proportional hazards regression models. Results Among the 6,550 participants (mean age 53.2 years; 48.0% male) in this study, 5.1% never consumed breakfast, 10.9% rarely consumed breakfast, 25.0% consumed breakfast some days, and 59.0% consumed breakfast every day. During 112,148 person-years of follow-up, 2,318 deaths occurred including 619 deaths from cardiovascular disease. After adjustment for age, sex, race/ethnicity, socioeconomic status, dietary and lifestyle factors, body mass index, and cardiovascular risk factors, participants who never consumed breakfast compared with those consuming breakfast everyday had hazard ratios of 1.87 (95% confidence interval: 1.14 to 3.04) for cardiovascular Mortality and 1.19 (95% confidence interval: 0.99 to 1.42) for All-Cause Mortality. Conclusions In a nationAlly representative cohort with 17 to 23 years of follow-up, skipping breakfast was associated with a significantly increased risk of Mortality from cardiovascular disease. Our study supports the benefits of eating breakfast in promoting cardiovascular health.
Ply Chichareon - One of the best experts on this subject based on the ideXlab platform.
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predicting 2 year All Cause Mortality after contemporary pci updating the logistic clinical syntax score
Catheterization and Cardiovascular Interventions, 2021Co-Authors: David Van Klaveren, Kuniaki Takahashi, Ply Chichareon, Rodrigo Modolo, Norihiro Kogame, Chun Chin Chang, Mariusz TomaniakAbstract:Aims We aimed to update the logistic clinical SYNTAX score to predict 2 year All-Cause Mortality after contemporary percutaneous coronary intervention (PCI). Methods and results We analyzed 15,883 patients in the GLOBAL LEADERS study who underwent PCI. The logistic clinical SYNTAX model was updated after imputing missing values by refitting the original model (refitted original model) and fitting an extended new model (new model, with, selection based on the Akaike Information Criterion). External validation was performed in 10,100 patients having PCI at Fu Wai hospital. Chronic obstructive pulmonary disease, prior stroke, current smoker, hemoglobin level, and white blood cell count were identified as additional independent predictors of 2 year All-Cause Mortality and included into the new model. The c-indexes of the original, refitted original and the new model in the derivation cohort were 0.74 (95% CI 0.72-0.76), 0.75 (95% CI 0.73-0.77), and 0.78 (95% CI 0.76-0.80), respectively. The c-index of the new model was lower in the validation cohort than in the derivation cohort, but still showed improved discriminative ability of the newly developed model (0.72; 95% CI 0.67-0.77) compared to the refitted original model (0.69; 95% CI 0.64-0.74). The models overestimated the observed 2 year All-Cause Mortality of 1.11% in the Chinese external validation cohort by 0.54 percentage points, indicating the need for calibration of the model to the Chinese patient population. Conclusions The new model of the logistic clinical SYNTAX score better predicts 2 year All-Cause Mortality after PCI than the original model. The new model could guide clinical decision making by risk stratifying patients undergoing PCI.
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validation of the updated logistic clinical syntax score for All Cause Mortality in the global leaders trial
Eurointervention, 2019Co-Authors: Ply Chichareon, David Van Klaveren, Yoshinobu Onuma, Taku Asano, Kuniaki Takahashi, Rodrigo Modolo, Norihiro Kogame, Chun Chin Chang, Mariusz Tomaniak, Yuki KatagiriAbstract:Aims: The aim of this study was the external validation of the updated logistic clinical SYNTAX score for two-year All-Cause Mortality after PCI in the GLOBAL LEADERS trial. Methods and results: The GLOBAL LEADERS trial was an investigator-initiated, prospective randomised, multicentre, open-label trial comparing two strategies of antiplatelet therapy in 15,991 patients undergoing PCI. As a predefined analysis, we studied the first 4,006 consecutive patients enrolled between July 2013 and April 2014 for whom the anatomic SYNTAX scores were calculated by an independent core lab. The updated logistic clinical SYNTAX score was available in 3,271 patients. Patients were divided into quintiles according to the score. The C-statistic of the updated logistic clinical SYNTAX score for twoyear All-Cause Mortality was 0.71 (95% confidence interval [CI]: 0.64-0.77). The updated logistic clinical SYNTAX score identified patients at very high risk for two-year All-Cause Mortality after PCI. Although it systematicAlly overestimated two-year All-Cause Mortality, predicted and observed two-year All-Cause Mortality in the majority of the patients (four out of five quintiles) were in agreement. Conclusions: OverAll discrimination for two-year All-Cause Mortality of the updated logistic clinical SYNTAX score is either borderline acceptable or possibly helpful. Calibration in the majority of patients is appropriate. The score is potentiAlly useful in selecting enriched high-risk populations.