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Silvia Franco Andrade - One of the best experts on this subject based on the ideXlab platform.

  • Effects of experimental amitraz intoxication in cats [Efeitos da intoxicação experimental por amitraz em gatos]
    2015
    Co-Authors: Silvia Franco Andrade, Cecilia Braga Laposy, S F Valente, V M Bettanim, L T Rodrigues, J Marcicano, Michiko Sakate, Presidente Prudente
    Abstract:

    This work studied the effects of experimental amitraz intoxication in cats. Sixteen cats were randomly divided equally into two groups: amitraz group- animals received 1.5 % amitraz at 1mg/kg IV; and the control group- animals without amitraz. Physiological parameters from blood, cardiorespiratory system, and sedation indicators were quantified over time up to 360 minutes. Blood profile, urea, creatinine, alananine aminotransferase and aspartate aminotransferase were not affected by amitraz. Sedation, loss of reflexes, hypothermia, bradycardia, bradyarrhythmia, hypotension, Bradypnea, mydriasis, besides transitory hyperglycemia, hypoinsulinemia and decrease of cortisol levels were observed in cats experimentally exposed to amitraz. The D2-adrenergic effects induced by amitraz intoxication in cats are very similar to the same effects reported in others species, contributing with more information about this type of intoxication to veterinary toxicology

  • Estudo comparativo da intoxicação experimental por amitraz entre cães e gatos
    Universidade de São Paulo, 2008
    Co-Authors: Silvia Franco Andrade, Cecilia Braga Laposy, L T Rodrigues, J Marcicano, Carlos Vianna De Andrade, Thiago Luiz Appel
    Abstract:

    The objective of the present work was to study the characteristics in common and the differences observed in the intoxication by amitraz 1 mg/kg, IV, in dogs and cats. The main clinical signs observed in common were sedation, hypothermia, bradycardia, bradyarrhytmia, hypotension, Bradypnea, mydriasis and transitory hyperglycemia, however the intensity of these signs was different among the species. The hypothermia was more accentuated in cats than in dogs. The dogs were more sensitive to the cardiorespiratory alterations presenting more significant decrease in the heart and breathing frequency, besides occurrence of larger bradyarrhytmia number. The cats presented more prolong mydriasis than the dogs. It was observed hyperglycemia and transitory hypoinsulinemia and transitory decrease of the plasmatic levels of cortisol, however the cats presented a hyperglycemia pick more accentuated and more precocious than the dogs, and regarding the plasmatic levels of cortisol, the dogs presented a decrease more accentuated than the cats. In the medium time of return of the sedation smaller in cat. These results demonstrated that the amitraz intoxication between dogs and cats is very similar, however the cats showed more sensibility to the hypothermia and hyperglycemia induction, besides mydriasis and the medium time of return of sedation more prolong, while the dogs presented a decrease more accentuated in the cardiorespiratory parameters and of the plasmatic levels of cortisol than the cats

  • effects of experimental amitraz intoxication in cats
    Arquivo Brasileiro De Medicina Veterinaria E Zootecnia, 2007
    Co-Authors: Silvia Franco Andrade, Cecilia Braga Laposy, S F Valente, V M Bettanim, L T Rodrigues, Michiko Sakate, J Marcicano
    Abstract:

    This work studied the effects of experimental amitraz intoxication in cats. Sixteen cats were randomly divided equally into two groups: amitraz group - animals received 1.5% amitraz at 1mg/kg IV; and the control group - animals without amitraz. Physiological parameters from blood, cardiorespiratory system, and sedation indicators were quantified over time up to 360 minutes. Blood profile, urea, creatinine, alananine aminotransferase and aspartate aminotransferase were not affected by amitraz. Sedation, loss of reflexes, hypothermia, bradycardia, bradyarrhythmia, hypotension, Bradypnea, mydriasis, besides transitory hyperglycemia, hypoinsulinemia and decrease of cortisol levels were observed in cats experimentally exposed to amitraz. The a2-adrenergic effects induced by amitraz intoxication in cats are very similar to the same effects reported in others species, contributing with more information about this type of intoxication to veterinary toxicology.

  • yohimbine and atipamezole on the treatment of experimentally induced amitraz intoxication in cats
    2006
    Co-Authors: Silvia Franco Andrade, Fabiola Sangiorgio
    Abstract:

    Experimental amitraz intoxication and its reversal by atipamezole and yohimbine were studied in cats. Twenty four cats were randomly divided equally into 3 groups: Group A (amitraz); Group AY (amitraz/yohimbine) and Group AA (amitraz/atipamezole). Sedation, loss of reflexes, hypothermia, bradycardia, bradyarrhythmia, hypotension, Bradypnea, mydriasis, and initial transitory hyperglycemia occurred in Group A. Median intervals for sedation return, in minutes, were significantly lower in amitraz-intoxicated cats treated with either yohimbine or atipamezole. Yohimbine and atipamezole were very effective in the treatment of amitraz intoxication, although atipamezole was more effective in reversal of arrhythmias, mydriasis, and sedation induced by this acaricide in cats.

Josemir W Sander - One of the best experts on this subject based on the ideXlab platform.

  • sudden unexpected death in epilepsy risk factors and potential pathomechanisms
    Nature Reviews Neurology, 2009
    Co-Authors: Rainer Surges, Roland D Thijs, Hanno L Tan, Josemir W Sander
    Abstract:

    sudden unexpected death in epilepsy (UDeP) is the most common cause of death directly related to epilepsy, and most frequently occurs in people with chronic epilepsy. The main risk factors for sUDeP are associated with poorly controlled seizures, suggesting that most cases of sUDeP are seizure- related events. Dysregulation in cardiac and respiratory physiology, dysfunction in systemic and cerebral circulation physiology, and seizure-induced hormonal and metabolic changes might all contribute to sUDeP. Cardiac factors include bradyarrhythmias and asystole, as well as tachyarrhythmias and alterations to cardiac repolarization. Altered electrolytes and blood pH, as well as the release of catecholamines, modulate cardiac excitability and might facilitate arrhythmias. respiratory symptoms are not uncommon during seizures and comprise central apnea or Bradypnea, and, less frequently, obstruction of the airways and neurogenic pulmonary edema. Alterations to autonomic function, such as a reduction in heart rate variability or disturbed baroreflex sensitivity, can impair the body's capacity to cope with challenging situations of elevated stress, such as seizures. Here, we summarize data on the incidence of and risk factors for sUDeP, and consider the pathophysiological aspects of chronic epilepsy that might lead to sudden death. we suggest that sUDeP is caused by the fatal coexistence of several predisposing and triggering factors.

Craig S Henriquez - One of the best experts on this subject based on the ideXlab platform.

J Marcicano - One of the best experts on this subject based on the ideXlab platform.

  • Effects of experimental amitraz intoxication in cats [Efeitos da intoxicação experimental por amitraz em gatos]
    2015
    Co-Authors: Silvia Franco Andrade, Cecilia Braga Laposy, S F Valente, V M Bettanim, L T Rodrigues, J Marcicano, Michiko Sakate, Presidente Prudente
    Abstract:

    This work studied the effects of experimental amitraz intoxication in cats. Sixteen cats were randomly divided equally into two groups: amitraz group- animals received 1.5 % amitraz at 1mg/kg IV; and the control group- animals without amitraz. Physiological parameters from blood, cardiorespiratory system, and sedation indicators were quantified over time up to 360 minutes. Blood profile, urea, creatinine, alananine aminotransferase and aspartate aminotransferase were not affected by amitraz. Sedation, loss of reflexes, hypothermia, bradycardia, bradyarrhythmia, hypotension, Bradypnea, mydriasis, besides transitory hyperglycemia, hypoinsulinemia and decrease of cortisol levels were observed in cats experimentally exposed to amitraz. The D2-adrenergic effects induced by amitraz intoxication in cats are very similar to the same effects reported in others species, contributing with more information about this type of intoxication to veterinary toxicology

  • Estudo comparativo da intoxicação experimental por amitraz entre cães e gatos
    Universidade de São Paulo, 2008
    Co-Authors: Silvia Franco Andrade, Cecilia Braga Laposy, L T Rodrigues, J Marcicano, Carlos Vianna De Andrade, Thiago Luiz Appel
    Abstract:

    The objective of the present work was to study the characteristics in common and the differences observed in the intoxication by amitraz 1 mg/kg, IV, in dogs and cats. The main clinical signs observed in common were sedation, hypothermia, bradycardia, bradyarrhytmia, hypotension, Bradypnea, mydriasis and transitory hyperglycemia, however the intensity of these signs was different among the species. The hypothermia was more accentuated in cats than in dogs. The dogs were more sensitive to the cardiorespiratory alterations presenting more significant decrease in the heart and breathing frequency, besides occurrence of larger bradyarrhytmia number. The cats presented more prolong mydriasis than the dogs. It was observed hyperglycemia and transitory hypoinsulinemia and transitory decrease of the plasmatic levels of cortisol, however the cats presented a hyperglycemia pick more accentuated and more precocious than the dogs, and regarding the plasmatic levels of cortisol, the dogs presented a decrease more accentuated than the cats. In the medium time of return of the sedation smaller in cat. These results demonstrated that the amitraz intoxication between dogs and cats is very similar, however the cats showed more sensibility to the hypothermia and hyperglycemia induction, besides mydriasis and the medium time of return of sedation more prolong, while the dogs presented a decrease more accentuated in the cardiorespiratory parameters and of the plasmatic levels of cortisol than the cats

  • effects of experimental amitraz intoxication in cats
    Arquivo Brasileiro De Medicina Veterinaria E Zootecnia, 2007
    Co-Authors: Silvia Franco Andrade, Cecilia Braga Laposy, S F Valente, V M Bettanim, L T Rodrigues, Michiko Sakate, J Marcicano
    Abstract:

    This work studied the effects of experimental amitraz intoxication in cats. Sixteen cats were randomly divided equally into two groups: amitraz group - animals received 1.5% amitraz at 1mg/kg IV; and the control group - animals without amitraz. Physiological parameters from blood, cardiorespiratory system, and sedation indicators were quantified over time up to 360 minutes. Blood profile, urea, creatinine, alananine aminotransferase and aspartate aminotransferase were not affected by amitraz. Sedation, loss of reflexes, hypothermia, bradycardia, bradyarrhythmia, hypotension, Bradypnea, mydriasis, besides transitory hyperglycemia, hypoinsulinemia and decrease of cortisol levels were observed in cats experimentally exposed to amitraz. The a2-adrenergic effects induced by amitraz intoxication in cats are very similar to the same effects reported in others species, contributing with more information about this type of intoxication to veterinary toxicology.

Marcicano J. - One of the best experts on this subject based on the ideXlab platform.

  • Efeitos da intoxicação experimental por amitraz em gatos
    Universidade Federal de Minas Gerais (UFMG) Escola de Veterinária, 2007
    Co-Authors: Andrade S.f., Sakate M. [unesp], Laposy C.b., Valente S.f., Bettanim V.m., Rodrigues L.t., Marcicano J.
    Abstract:

    Este trabalho estudou os efeitos da intoxicação experimental por amitraz em 16 gatos, distribuídos, aleatoriamente, em dois grupos: grupo amitraz - animais receberam amitraz a 1,5% na dose de 1,0 mg/kg IV; e grupo controle - animais sem amitraz. Parâmetros fisiológicos sangüíneos, do sistema cardiorespiratório e de sedação foram aferidos até 360min. Perfil sangüíneo, uréia, creatinina, alanina aminotransferase e aspartato aminotransferase não foram afetados pelo amitraz. Sedação, perda de reflexos, hipotermia, bradicardia, bradiarritmias, hipotensão, bradipnéia, midríase, além de transitória hiperglicemia, hipoinsulinemia e diminuição dos níveis de cortisol, foram observados nos gatos experimentalmente expostos ao amitraz. Os efeitos alfa 2-adrenérgicos induzidos pela intoxicação por amitraz em gatos são muito similares aos mesmos efeitos relatados em outras espécies, contribuindo com mais informações dessa intoxicação para a toxicologia veterinária.This work studied the effects of experimental amitraz intoxication in cats. Sixteen cats were randomly divided equally into two groups: amitraz group - animals received 1.5% amitraz at 1mg/kg IV; and the control group - animals without amitraz. Physiological parameters from blood, cardiorespiratory system, and sedation indicators were quantified over time up to 360 minutes. Blood profile, urea, creatinine, alananine aminotransferase and aspartate aminotransferase were not affected by amitraz. Sedation, loss of reflexes, hypothermia, bradycardia, bradyarrhythmia, hypotension, Bradypnea, mydriasis, besides transitory hyperglycemia, hypoinsulinemia and decrease of cortisol levels were observed in cats experimentally exposed to amitraz. The alpha2-adrenergic effects induced by amitraz intoxication in cats are very similar to the same effects reported in others species, contributing with more information about this type of intoxication to veterinary toxicology