The Experts below are selected from a list of 264 Experts worldwide ranked by ideXlab platform
Miso Sabovic - One of the best experts on this subject based on the ideXlab platform.
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an unusual case of Cortical Blindness associated with aortography a case report
Angiology, 2000Co-Authors: Barbara Bonac, Miso SabovicAbstract:Transient Cortical Blindness is a rare but well-recognized benign complication of angiog raphy that is due to neurotoxicity of the contrast agent. The Blindness completely resolves in a few days. This report describes a patient suffering an unusual course of Cortical Blindness following aortography resulting in permanent, partial Blindness. It was found that Blindness was the consequence of bilateral occipital embolisms. This case emphasizes that Cortical Blindness associatted with angiography does not always have a favorable outcome, since it might be, in rare cases, due to isolated occipital embolisms, which initially produce a clinical picture identical to that of the neurotoxic effect of contrast agents.
Rudiger J Seitz - One of the best experts on this subject based on the ideXlab platform.
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visual hallucinations in recovery from Cortical Blindness imaging correlates
JAMA Neurology, 2000Co-Authors: Gilbert Wunderlich, Boris Suchan, Jens Volkmann, Hans Herzog, Volker Homberg, Rudiger J SeitzAbstract:Objective To investigate the cerebral metabolic and functional patterns during recovery from Cortical Blindness. Design Follow-up study with serial clinical, metabolic, and functional imaging and visual evoked potentials. Case Presentation A 24-year-old woman suffered from Cortical Blindness after cardiac arrest and recovered over a 6-month period. During recovery, she experienced complex visual hallucinations that could be initiated by visual imagery. Results Initially, the regional cerebral metabolic rate of glucose was severely reduced in the visual and parieto-occipital cortex bilaterally but recovered almost completely. Visual hallucinations led to significant increases of the regional cerebral blood flow in the initially severely hypometabolic parieto-occipital and temporolateral cortex. Conclusions Recovery of vision was related to normalization of the postlesionally dysfunctional cortex. Visual hallucinations appeared as the clinical correlate of the electrophysiological hyperexcitability of the recovering partially damaged visual cortex.
R. S. Ackroyd - One of the best experts on this subject based on the ideXlab platform.
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Cortical Blindness following bacterial meningitis: a case report with reassessment of prognosis and aetiology.
Developmental medicine and child neurology, 2008Co-Authors: R. S. AckroydAbstract:A case of Cortical Blindness is presented which developed during H. influenzae type B meningitis. Cortical Blindness may appear late in the course of bacterial meningitis, during a period of clinical improvement, and recovery can be expected in 50 per cent of cases. In contrast, when the cause is ischaemic the onset of Blindness is immediate and usually recovery is complete. Radiological evidence is presented for the pathological process in meningitis.
M A Morgan - One of the best experts on this subject based on the ideXlab platform.
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Cortical Blindness and Anton syndrome in a patient with obstetric hemorrhage.
Obstetrics and gynecology, 1998Co-Authors: P A Argenta, M A MorganAbstract:Cortical Blindness is characterized by loss of vision in the presence of intact anterior visual pathways. Anton syndrome, a form of anosognosia, is a rare complication of Cortical Blindness involving compromise of the visual association centers, with resulting patient denial of Blindness. Both syndromes have been associated with computed tomography findings of localized Cortical ischemia. In most cases, both the clinical and radiologic features are reversible. A woman with hemorrhage from an incomplete abortion at 21 weeks experienced Cortical Blindness and visual anosognosia. Cortical Blindness and anosognosia are unusual manifestations of severe hemorrhage but should be considered in the differential diagnosis of a patient with atypical visual symptoms.
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Cortical Blindness and Anton syndrome in a patient with obstetric hemorrhage.
Obstetrics & Gynecology, 1998Co-Authors: P A Argenta, M A MorganAbstract:Abstract Background: Cortical Blindness is characterized by loss of vision in the presence of intact anterior visual pathways. Anton syndrome, a form of anosognosia, is a rare complication of Cortical Blindness involving compromise of the visual association centers, with resulting patient denial of Blindness. Both syndromes have been associated with computed tomography findings of localized Cortical ischemia. In most cases, both the clinical and radiologic features are reversible. Case: A woman with hemorrhage from an incomplete abortion at 21 weeks experienced Cortical Blindness and visual anosognosia. Conclusion: Cortical Blindness and anosognosia are unusual manifestations of severe hemorrhage but should be considered in the differential diagnosis of a patient with atypical visual symptoms.
Gilbert Wunderlich - One of the best experts on this subject based on the ideXlab platform.
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visual hallucinations in recovery from Cortical Blindness imaging correlates
JAMA Neurology, 2000Co-Authors: Gilbert Wunderlich, Boris Suchan, Jens Volkmann, Hans Herzog, Volker Homberg, Rudiger J SeitzAbstract:Objective To investigate the cerebral metabolic and functional patterns during recovery from Cortical Blindness. Design Follow-up study with serial clinical, metabolic, and functional imaging and visual evoked potentials. Case Presentation A 24-year-old woman suffered from Cortical Blindness after cardiac arrest and recovered over a 6-month period. During recovery, she experienced complex visual hallucinations that could be initiated by visual imagery. Results Initially, the regional cerebral metabolic rate of glucose was severely reduced in the visual and parieto-occipital cortex bilaterally but recovered almost completely. Visual hallucinations led to significant increases of the regional cerebral blood flow in the initially severely hypometabolic parieto-occipital and temporolateral cortex. Conclusions Recovery of vision was related to normalization of the postlesionally dysfunctional cortex. Visual hallucinations appeared as the clinical correlate of the electrophysiological hyperexcitability of the recovering partially damaged visual cortex.
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Visual Hallucinations in Recovery From Cortical Blindness
2000Co-Authors: Gilbert Wunderlich, Boris Suchan, Jens Volkmann, Hans HerzogAbstract:C ORTICAL Blindness refers to loss of vision caused by damage of the geniculocalcarine pathways. Cerebrovascular disease is the most common cause, followed by cardiac surgery and cerebral angiography. Cardiac arrest usually leads to global brain anoxia or to more focal neurological abnormalities, such as Cortical Blindness, resulting in a variable spectrum of diffuse or multifocal cerebral atrophy due to neuronal loss and Cortical and subCortical gliosis or even severe Cortical damage. Accordingly, positron emission tomography (PET) has shown that global ischemic anoxia results in diffuse hypometabolism with preferential localization of the metabolic alterations in the parieto-occipital cortex. Nevertheless, most patients recover from Cortical Blindness, but the mechanisms of this recovery are poorly understood. We describe a patient who experienced vivid visual hallucinations during recovery from prolonged Cortical Blindness. Using multitracer PET imaging, we demonstrated that the hallucinations were associated with activations of the parietooccipital cortex, which was severely metabolically compromised initially but which regained normal metabolic function as vision recovered.
