The Experts below are selected from a list of 27549 Experts worldwide ranked by ideXlab platform

Yahya M. Naguib - One of the best experts on this subject based on the ideXlab platform.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C
    Reproductive Biology and Endocrinology, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Purpose Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Methods Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Results Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Conclusion Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C.
    Reproductive biology and endocrinology : RB&E, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

Nermin I. Rizk - One of the best experts on this subject based on the ideXlab platform.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C
    Reproductive Biology and Endocrinology, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Purpose Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Methods Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Results Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Conclusion Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C.
    Reproductive biology and endocrinology : RB&E, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

Asmaa S. Mohamed - One of the best experts on this subject based on the ideXlab platform.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C
    Reproductive Biology and Endocrinology, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Purpose Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Methods Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Results Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Conclusion Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C.
    Reproductive biology and endocrinology : RB&E, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

Mohamed S. Rizk - One of the best experts on this subject based on the ideXlab platform.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C
    Reproductive Biology and Endocrinology, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Purpose Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Methods Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Results Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Conclusion Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

  • Attenuation of sleep deprivation dependent deterioration in male Fertility Parameters by vitamin C.
    Reproductive biology and endocrinology : RB&E, 2020
    Co-Authors: Nermin I. Rizk, Mohamed S. Rizk, Asmaa S. Mohamed, Yahya M. Naguib
    Abstract:

    Male Fertility is multifaceted and its integrity is as well multifactorial. Normal spermatogenesis is dependent on competent testicular function; namely normal anatomy, histology, physiology and hormonal regulation. Lifestyle stressors, including sleep interruption and even deprivation, have been shown to seriously impact male Fertility. We studied here both the effects and the possible underlying mechanisms of vitamin C on male Fertility in sleep deprived rats. Thirty male Wistar albino rats were used in the present study. Rats were divided (10/group) into: control (remained in their cages with free access to food and water), sleep deprivation (SD) group (subjected to paradoxical sleep deprivation for 5 consequent days, rats received intra-peritoneal injections of vehicle daily throughout the sleep deprivation), and sleep deprivation vitamin C-treated (SDC) group (subjected to sleep deprivation for 5 consequent days with concomitant intra-peritoneal injections of 100 mg/kg/day vitamin C). Sperm analysis, hormonal assay, and measurement of serum oxidative stress and inflammatory markers were performed. Testicular gene expression of Nrf2 and NF-κβ was assessed. Structural changes were evaluated by testicular histopathology, while PCNA immunostaining was conducted to assess spermatogenesis. Sleep deprivation had significantly altered sperm motility, viability, morphology and count. Serum levels of cortisol, corticosterone, IL-6, IL-17, MDA were increased, while testosterone and TAC levels were decreased. Testicular gene expression of Nrf2 was decreased, while NF-κβ was increased. Sleep deprivation caused structural changes in the testes, and PCNA immunostaining showed defective spermatogenesis. Administration of vitamin C significantly countered sleep deprivation induced deterioration in male Fertility Parameters. Treatment with vitamin C enhanced booth testicular structure and function in sleep deprived rats. Vitamin C could be a potential Fertility enhancer against lifestyle stressors.

Toshiyuki Wakatsuki - One of the best experts on this subject based on the ideXlab platform.

  • Geographical distribution of selected soil Fertility Parameters of inland valleys in West Africa
    Geoderma, 1997
    Co-Authors: Roland Nuhu Issaka, Fusako Ishida, Daisuke Kubota, Toshiyuki Wakatsuki
    Abstract:

    Abstract This study was part of an ongoing characterization of soil properties of inland valleys (IVs) of West Africa. Soil sampling was done during 1982 to 1989. Soil properties of the topsoil (0–15 cm) were used to evaluate the Fertility status of the various geographic regions. Large differences in soil Fertility Parameters were observed in the various geographic regions. Generally the drier regions [Sudan (SuS) and Sahel savannas (SS)] were relatively higher in ECEC [SuS, 7.7 cmol(+) kg−1 and SS, 8.2 cmol(+) kg−1, topsoil means] and exchangeable basic cations; but total C (SuS, 5.5 g kg−1 and SS, 6.2 g kg−1, topsoil means) and total N (SuS, 0.68 g kg−1 and SS, 0.66 g kg−1, topsoil means), available P (SuS, 2.6 mg kg− 1 and SS, 2.7 mg kg−1, topsoil mean) and exchangeable acidity [SuS, 0.14 cmol(+) kg−1 and SS, 0.46 cmol(+) kg−1, topsoil means] were relatively low. Soil Fertility Parameters were generally low in the Guinea savanna zone [ECEC, 2.5 cmol(+) kg−1; total C, 7.3 g kg−1; total N, 0.7 g kg−1 and available P, 2.9 mg kg−1, topsoil means]. In the Forest region, ECEC [5.7 cmol(+) kg−1], exchangeable Ca [2.3 cmol(+) kg−1], Mg [1.24 cmol(+) kg−1], acidity [1.67 cmol(+) kg−1], and total C (20.4 g kg− 1 ) and total N (1.66 g kg−1) were relatively high. Available P (5.3 g kg−1 ) and exchangeable K [0.27 cmol(+) kg−1] were relatively moderate. However, in the more wetter Equatorial Forest, available P and exchangeable K were low and acidity very high. Soil properties of these inland valleys were affected by both geology and climate. Geological formation of recent alluvial deposition, rejuvenation and the presence of basic rocks mostly resulted in relatively fertile soils while soils of very old geological origin were relatively less fertile. Climate had a dual effect on these soil properties: within the areas with tropudic and typic udic soil moisture regimes, serious leaching resulted in less exchangeable basic cations but high exchangeable acidity; however, rainfall enhanced high biomass production which resulted in relatively high organic matter content and hence high ECEC and total N. Annual burning, especially of the grass vegetation in savanna area brought about a low addition of plant residue into the soil. Total elemental oxides were almost the same for all the zones. However, values for the various sites were different due to differences in geological formation. The relative differences in retention (ECEC) and leaching of basic cations resulting from weathering also accounted for some of the differences in the soil Fertility properties observed.

  • soils of inland valleys of west africa general Fertility Parameters
    Soil Science and Plant Nutrition, 1996
    Co-Authors: Roland Nuhu Issaka, Tsugiyuki Masunaga, Takashi Kosaki, Toshiyuki Wakatsuki
    Abstract:

    Abstract In an effort to improve the critical food situation in West Africa, wetlands are currently receiving the necessary attention as the environment most suitable for rice production. Various scientific studies are required to effectively put these wetlands into sustainable crop production. Between 1983 and 1989 inland valleys (IVs) of West Africa were surveyed with the main objective of characterizing these soils; soil samples were therefore collected from these IVs throughout the various climatic zones. As inland valleys of West Africa have undergone intense leaching and weathering, the soil pH is generally low. Mean soil pH values (0–15 cm) are: West Africa, 5.3; Equatorial forest (EF), 5.3; Guinea savanna (GS), 5.3; Sudan savanna (SuS), 5.9; and Sahel savanna (SS), 6.0. Exchangeable cations for West Africa IVs are low; also low for IVs of EF; very low for GS IVs; and moderate for the drier zones (SuS and SS). Total carbon and nitrogen contents (12.8 g kg-1 C and 1.11 g kg-1 N) of these soils are l...