The Experts below are selected from a list of 273 Experts worldwide ranked by ideXlab platform
Qiu Shi - One of the best experts on this subject based on the ideXlab platform.
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The in vitro Growth Inhibition Effect of Taraxacum mongolicum Hand.-Mazz (TMHM) on Bacteria
Lishizhen Medicine and Materia Medica Research, 2002Co-Authors: Qiu ShiAbstract:Objective:To research on the in vitro Growth Inhibition effect of TMHM on bacteria. Methods:K B paper dispersion method was used and observed the Inhibition effect of circular filter paper pieces of TMHM on Staphylococcus. aureus, P. vulgaris , α hemolytic streptpccus , β hemolytic streptpccus . Results:It had obvious effect of Growth Inhibition on bacteria. Conclusion:TMHM has significant in vitro effect of Growth Inhibition on bacteria.
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The in vitro Growth Inhibition Effect of Coptis chinensis Franch (CCF) on Bacteria
Lishizhen Medicine and Materia Medica Research, 2002Co-Authors: Qiu ShiAbstract:Objective:To research on the in vitro Growth Inhibition effect of Coptis chinensis Franch (CCF) on bacteria. Methods:K B paper dispersion method was used and observed the Inhibition effect of circular filter paper pieces of CCF on Escherichia coli , Staphylococcus aureus, Staphylococcus albicans, P. vulgaris , B. anthracis .Results:It had obvious effect of Growth Inhibition on bacteria.Conclusion:CCF has significant in vitro effect of Growth Inhibition on bacteria.
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The in Vitro Growth Inhibition Effect of Cortex Cinnamomi on Bacteria
Lishizhen Medicine and Materia Medica Research, 2001Co-Authors: Qiu ShiAbstract:Objective: To research on the in vitro Growth Inhibition effect of Cortex Cinnamomi on bacteria. Methods: K B paper dispersion method was used, the circular filter paper pieces were presoaked with 100% Cortex Cinnamomi water extract and observed the effect of it on following bacteria: Escherichia Coli, Shigella, Salmonella typhi, staphylococcus aureus, staphylococcus albicans, candida albicans. Results: It had obvious effect of Growth Inhibition on bacteria. Conclusion: Cortex Cinnamomi has significant in vitro effect of Growth Inhibition on bacteria and anti fungal effect.
Yuan Xiao-chun - One of the best experts on this subject based on the ideXlab platform.
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In vitro Growth Inhibition effect of Taraxacum mongolicum Hand .-Mazz(TMHM) on Bacteria
Journal of Shaanxi Normal University, 2004Co-Authors: Yuan Xiao-chunAbstract:It is observed that the water extract and ethyl alcohol extract of whole herb of Taraxacum mongolicum, Compare antibiotic effect of the two kinds culture had significant the in vitro Growth Inhibition effect on Staphylococcus. aureus and Escherichia coli. The experiments had showed that the water extract had more powerful effect of Growth Inhibition on two species bacteria than ethyl alcohol extract.
Shuan Shian Huang - One of the best experts on this subject based on the ideXlab platform.
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Cellular Growth Inhibition by TGF-β1 involves IRS proteins ☆
FEBS Letters, 2004Co-Authors: Shuan Shian Huang, Sandra M. Leal, Chun-lin Chen, I-hua Liu, Jung San HuangAbstract:Abstract In Mv1Lu cells, insulin partially reverses transforming Growth factor-β1 (TGF-β1) Growth Inhibition in the presence of α5β1 integrin antagonists. TGF-β1 appears to induce phosphorylation of IRS-2 in these cells; this is inhibited by a TGF-β antagonist known to reverse TGF-β Growth Inhibition. Stable transfection of 32D myeloid cells (which lack endogenous IRS proteins and are insensitive to Growth Inhibition by TGF-β1) with IRS-1 or IRS-2 cDNA confers sensitivity to Growth Inhibition by TGF-β1; this IRS-mediated Growth Inhibition can be partially reversed by insulin in 32D cells stably expressing IRS-2 and the insulin receptor (IR). These results suggest that Growth Inhibition by TGF-β1 involves IRS proteins.
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Cellular Growth Inhibition by TGF-beta1 involves IRS proteins.
FEBS letters, 2004Co-Authors: Shuan Shian Huang, Sandra M. Leal, Chun-lin Chen, I-hua Liu, Jung San HuangAbstract:In Mv1Lu cells, insulin partially reverses transforming Growth factor-beta1 (TGF-beta1) Growth Inhibition in the presence of alpha5beta1 integrin antagonists. TGF-beta1 appears to induce phosphorylation of IRS-2 in these cells; this is inhibited by a TGF-beta antagonist known to reverse TGF-beta Growth Inhibition. Stable transfection of 32D myeloid cells (which lack endogenous IRS proteins and are insensitive to Growth Inhibition by TGF-beta1) with IRS-1 or IRS-2 cDNA confers sensitivity to Growth Inhibition by TGF-beta1; this IRS-mediated Growth Inhibition can be partially reversed by insulin in 32D cells stably expressing IRS-2 and the insulin receptor (IR). These results suggest that Growth Inhibition by TGF-beta1 involves IRS proteins.
Kotaro T. Yamamoto - One of the best experts on this subject based on the ideXlab platform.
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Phytochrome-mediated Growth Inhibition of seminal roots in rice seedlings
Physiologia plantarum, 2009Co-Authors: Hisayo Shimizu, Takanari Tanabata, Xianzhi Xie, Noritoshi Inagaki, Makoto Takano, Tomoko Shinomura, Kotaro T. YamamotoAbstract:In rice (Oryza sativa) seedlings, continuous white-light irradiation inhibited the Growth of seminal roots but promoted the Growth of crown roots. In this study, we examined the mechanisms of photoInhibition of seminal root Growth. PhotoInhibition occurred in the absence of nitrogen but increased with increasing nitrogen concentrations. In the presence of nitrogen, photoInhibition was correlated with coiling of the root tips. The seminal roots were most photosensitive 48-72 h after germination during the 7-day period after germination. White-light irradiation for at least 6 h was required for photoInhibition, and the Bunsen-Roscoe law of reciprocity was not observed. Experiments with phytochrome mutants showed that far-red light was perceived exclusively by phyA, red light was perceived by both phyA and phyB, and phyC had little or no role in Growth Inhibition or coiling of the seminal roots. These results also suggest that other blue-light photoreceptors are involved in Growth Inhibition of the seminal roots. Fluence-response curve analyses showed that phyA and phyB control very low-fluence response and low-fluence response, respectively, in the seminal roots. This was essentially the same as the Growth Inhibition previously observed at the late stage of coleoptile development (80 h after germination). The photoperceptive site for the root Growth Inhibition appeared to be the roots themselves. All three phytochrome species of rice were detected immunochemically in roots.
Jeffrey C. Suttle - One of the best experts on this subject based on the ideXlab platform.
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Auxin-induced sprout Growth Inhibition: Role of endogenous ethylene
American Journal of Potato Research, 2003Co-Authors: Jeffrey C. SuttleAbstract:El rol del etileno endógeno en la inhibición del crecimiento del brote mediado por auxinas fue determinado en minitubérculos de papa ( Solanum tuberosum L. cv. Russet Burbank). El tratamiento de tubérculos con auxinas biológicamente activas resultó en un incremento pasajero en la producción de etileno e inhibición de crecimiento del brote dependiente de la dosis. Análogos de enzimas biológicamente activas no produjeron ninguna respuesta. La contínua exposicion de etileno exógeno a una concentración > de 0.001 μL L^-1 inhibió el crecimiento de brotes de mariera dependiente de la dosis, con una inhibición completa con concentraciones de etileno > a 1μL L^-1. En estudios adicionales con la auxina activa α-naftaleno ácido acético (NAA), se determinó el rol del etileno en la inhibición de crecimiento del brote inducido por auxinas utilizando inhibidores de biosintesis y de accién del etileno. La aminoetoxivinilglicina, inhibidor de la biosíntesis de etileno, inducida por NAA redujo la biosíntesis de etileno en más del 80%, pero no tuvo efecto sobre la inhibición de crecimiento del brote. La acción no competitiva del tiosulfato de plata, inhibidor del etileno no tuvo efecto en la inhibición de crecimiento del brote inducida por NAA. De la misma mariera, tanto la acción competitiva del 2,5-nor-bordarieno, inhibidor del etileno, como la acción irreversible del inhibidor 1-metilcloropeno no fueron efectivos para reducir la inhibición de crecimiento del brote por mediación del NAA. Colectivamente estos resultados no apoyan la opinion de que la inhibición de crecimiento de los brotes de papa inducida por auxinas sea realizada por mediación del etileno endógeno. The role of endogenous ethylene in auxin-mediated tuber sprout Growth Inhibition was determined in potato ( Solanum tuberosum L. cv. Russet Burbank) minitubers. Treatment of tubers with biologically active auxins resulted in a transient, dose-dependent increase in ethylene production and Inhibition of sprout Growth. Biologically inactive auxin analogs elicited neither response. Continuous exposure to > 0.001 μL L^-1 exogenous ethylene inhibited sprout Growth in a dose-dependent manner with complete Inhibition occurring at ethylene concentrations ≥1 μL L^-1. In further studies with the active auxin α-naphthalene acetic acid (NAA), the role of ethylene in auxin-induced sprout Growth Inhibition was determined using ethylene biosynthesis and action inhibitors. The ethylene biosynthesis inhibitor aminoethoxyvinylglycine reduced NAA-induced ethylene biosynthesis by over 80% but had no effect on sprout Growth Inhibition. The non-competitive ethylene action inhibitor silver thiosulfate had no effect on NAA-induced sprout Growth Inhibition. Similarly, both the competitive ethylene action inhibitor 2,5-norbornadiene and the irreversible action inhibitor 1-methylcyclopropene were ineffective in reducing NAA-mediated sprout Growth Inhibition. Collectively, these results do not support the proposal that, in the case of potato tuber sprouts, auxininduced Growth Inhibition is mediated by endogenous ethylene action.
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Auxin-induced sprout Growth Inhibition: Role of endogenous ethylene
American Journal of Potato Research, 2003Co-Authors: Jeffrey C. SuttleAbstract:The role of endogenous ethylene in auxin-mediated tuber sprout Growth Inhibition was determined in potato (Solanum tuberosum L. cv. Russet Burbank) minitubers. Treatment of tubers with biologically active auxins resulted in a transient, dose-dependent increase in ethylene production and Inhibition of sprout Growth. Biologically inactive auxin analogs elicited neither response. Continuous exposure to > 0.001 μL L-1 exogenous ethylene inhibited sprout Growth in a dose-dependent manner with complete Inhibition occurring at ethylene concentrations ≥1 μL L-1. In further studies with the active auxin α-naphthalene acetic acid (NAA), the role of ethylene in auxin-induced sprout Growth Inhibition was determined using ethylene biosynthesis and action inhibitors. The ethylene biosynthesis inhibitor aminoethoxyvinylglycine reduced NAA-induced ethylene biosynthesis by over 80% but had no effect on sprout Growth Inhibition. The non-competitive ethylene action inhibitor silver thiosulfate had no effect on NAA-induced sprout Growth Inhibition. Similarly, both the competitive ethylene action inhibitor 2,5-norbornadiene and the irreversible action inhibitor 1-methylcyclopropene were ineffective in reducing NAA-mediated sprout Growth Inhibition. Collectively, these results do not support the proposal that, in the case of potato tuber sprouts, auxininduced Growth Inhibition is mediated by endogenous ethylene action.
