Interpersonal Stress

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Karen D Rudolph - One of the best experts on this subject based on the ideXlab platform.

  • Interpersonal Stress Severity Longitudinally Predicts Adolescent Girls’ Depressive Symptoms: the Moderating Role of Subjective and HPA Axis Stress Responses
    Journal of Abnormal Child Psychology, 2019
    Co-Authors: Sarah A. Owens, Paul D Hastings, Karen D Rudolph, Sarah W Helms, Matthew K Nock, Mitchell J Prinstein
    Abstract:

    In recent decades, Stress response models of adolescent depression have gained attention, but it remains unclear why only certain adolescents are vulnerable to the depressogenic effects of Stress while others are not. Building on evidence that affective and physiological responses to Stress moderate the impact of Stress exposure on depression, the current study examined whether the interaction between severity of Interpersonal Stress, subjective affective reactivity, and hypothalamic-pituitary-adrenal (HPA) axis reactivity to an acute, in-vivo psychosocial Stressor prospectively predicted depressive symptoms nine months later. Hypotheses were examined with a clinically-oversampled group of 182 adolescent girls (aged 12–16) to ensure an examination of the widest possible range of risk. Self-report measures of affect and salivary cortisol samples were collected before and after an in-vivo Stress task to assess affective reactivity and HPA axis reactivity, respectively. Severity of Interpersonal Stress between baseline and nine months was assessed using a semi-structured interview and was objectively coded for severity and content theme (i.e., Interpersonal vs. non-Interpersonal). Results indicate that experiences of severe Interpersonally-themed Stress predict elevated levels of depressive symptoms longitudinally only for adolescent girls with elevated affective reactivity to Stress, and suggest that these deleterious effects of Stress are most exacerbated for girls with elevated physiological responses to Stress. Findings suggest that it may be critical to examine both affective and physiological Stress responses when assessing risk for depression in adolescents.

  • preliminary associations among relational victimization targeted rejection and suicidality in adolescents a prospective study
    Journal of Clinical Child and Adolescent Psychology, 2019
    Co-Authors: Maya Massingschaffer, Paul D Hastings, Karen D Rudolph, George M Slavich, Sarah W Helms, Matteo Giletta, Matthew K Nock, Mitchell J Prinstein
    Abstract:

    This study examined associations between multiple types of Interpersonal and nonInterpersonal Stressors and the subsequent occurrence of suicide ideation and attempts among female adolescents. Adolescents ages 12 to 18 years old (n = 160) at elevated risk for suicidal thoughts and behaviors were followed for 18 months, divided into two 9-month epochs for data analysis (Periods 1 and 2). Exposure to acute relational victimization, targeted rejection, nonspecified Interpersonal, and nonInterpersonal life Stressors over the first 9-month epoch (Period 1) was assessed using semistructured interviews and an independent life Stress rating team. Participants also completed phone-based semistructured interviews of suicidal thoughts and behaviors. Preliminary analyses showed significant prospective associations between acute targeted rejection and nonspecified Interpersonal Stress during Period 1 and suicide ideation during Period 2, as well as relational victimization and nonInterpersonal Stress during Period 1 and suicide attempts during Period 2. However, in logistic regression analyses that adjusted for prior suicidality and depressive symptoms, relational victimization during Period 1 (but not targeted rejection, nonspecified Interpersonal or nonInterpersonal events) was associated with increased odds of suicide attempt during Period 2. Therefore, acute relational victimization exposure is associated with heightened risk for suicidal behaviors in female adolescents. Future studies should examine potential mediators and moderators of this association, and these Stressors should be considered for inclusion in clinical screening tools.

  • reciprocal associations between adolescent girls chronic Interpersonal Stress and nonsuicidal self injury a multi wave prospective investigation
    Journal of Adolescent Health, 2018
    Co-Authors: Adam Bryant Miller, Paul D Hastings, Karen D Rudolph, Katherine P Linthicum, Sarah W Helms, Matteo Giletta, Matthew K Nock, Mitchell J Prinstein
    Abstract:

    Abstract Purpose Nonsuicidal self-injury (NSSI) is associated with increased risk of suicide attempts. Theories of NSSI assert Interpersonal Stress as a common risk factor for, and perhaps consequence of, NSSI. Prior research has not examined reciprocal associations between chronic Interpersonal Stress and NSSI. This study used a multiwave, prospective design to address this gap in a sample of adolescent girls, a group with elevated risk for both chronic Interpersonal Stress and NSSI. Pubertal development was examined as a moderator of the reciprocal associations. Methods Adolescent girls (N = 220; ages 12–16, M age = 14.69 years) at heightened risk for NSSI completed a baseline assessment and follow-up assessments over 18 months, divided into two 9-month epochs (Time 1 and 2). Pubertal development was assessed via self- and parent-report. Chronic Interpersonal Stress was assessed using a semistructured interview at the end of each time period. NSSI was measured using a semistructured clinical interview every 3 months within both time periods to enhance accurate reporting. Results Path models revealed that chronic romantic Stress during Time 1, but not peer or parent–child Stress, predicted NSSI during Time 2 among girls with more advanced pubertal development. Moreover, NSSI during Time 1 predicted higher levels of chronic romantic and parent–child Stress during Time 2. Conclusions Results revealed a reciprocal relationship between chronic romantic Stress and engagement in NSSI. Further, this association may be best understood in the context of pubertal development.

  • responses to Interpersonal Stress normative changes across childhood and the impact of peer victimization
    Child Development, 2017
    Co-Authors: Wendy Troopgordon, Niwako Sugimura, Karen D Rudolph
    Abstract:

    This research examined the development of Stress responses across second to sixth grades and whether exposure to peer victimization alters Stress response trajectories. Youth (338 girls; 298 boys; Mage  = 7.97 years, SD = .37) reported on Stress responses; teachers and youth reported on peer victimization. Latent growth curve modeling revealed an increase in effortful engagement responses and a decrease in disengagement and involuntary engagement responses during this period. Peer victimization disrupted these normative trajectories, resulting in less effortful engagement and more effortful disengagement and involuntary Stress responses in early adolescence. These findings suggest that early peer victimization sensitizes youth to Stress by interfering with the development of effective coping and fostering maladaptive Stress responses.

  • Stress generation and adolescent depression contribution of Interpersonal Stress responses
    Journal of Abnormal Child Psychology, 2011
    Co-Authors: Megan Flynn, Karen D Rudolph
    Abstract:

    This research examined the proposal that ineffective responses to common Interpersonal problems disrupt youths’ relationships, which, in turn, contributes to depression during adolescence. Youth (86 girls, 81 boys; M age = 12.41, SD = 1.19) and their primary female caregivers participated in a three-wave longitudinal study. Youth completed a measure assessing Interpersonal Stress responses; youth and caregivers completed semi-structured interviews assessing youths’ life Stress and psychopathology. Consistent with the hypothesized model, ineffective Stress responses (low levels of effortful engagement, high levels of involuntary engagement and disengagement) predicted the generation of subsequent Interpersonal Stress, which partially accounted for the association between Stress responses and depression over time. Moreover, results revealed that self-generated Interpersonal, but not nonInterpersonal Stress, predicted depression, and that this explanatory model was specific to the prediction of depression but not anxiety. This research builds on Interpersonal Stress generation models of depression, and highlights the importance of implementing depression-focused intervention programs that promote effective Stress responses and adaptive Interpersonal relationships during adolescence.

Constance Hammen - One of the best experts on this subject based on the ideXlab platform.

  • additive genetic risk from five serotonin system polymorphisms interacts with Interpersonal Stress to predict depression
    Journal of Abnormal Psychology, 2015
    Co-Authors: Suzanne Vrshekschallhorn, Catherine B Stroud, Constance Hammen, Susan Mineka, Richard E Zinbarg, Eva E Redei, Emma K Adam, Michelle G Craske
    Abstract:

    Behavioral genetic research supports polygenic models of depression in which many genetic variations each contribute a small amount of risk, and prevailing diathesis-Stress models suggest gene-environment interactions (G×E). Multilocus profile scores of additive risk offer an approach that is consistent with polygenic models of depression risk. In a first demonstration of this approach in a G×E predicting depression, we created an additive multilocus profile score from 5 serotonin system polymorphisms (1 each in the genes HTR1A, HTR2A, HTR2C, and 2 in TPH2). Analyses focused on 2 forms of Interpersonal Stress as environmental risk factors. Using 5 years of longitudinal diagnostic and life Stress interviews from 387 emerging young adults in the Youth Emotion Project, survival analyses show that this multilocus profile score interacts with major Interpersonal Stressful life events to predict major depressive episode onsets (hazard ratio [HR] = 1.815, p = .007). Simultaneously, there was a significant protective effect of the profile score without a recent event (HR = 0.83, p = .030). The G×E effect with Interpersonal chronic Stress was not significant (HR = 1.15, p = .165). Finally, effect sizes for genetic factors examined ignoring Stress suggested such an approach could lead to overlooking or misinterpreting genetic effects. Both the G×E effect and the protective simple main effect were replicated in a sample of early adolescent girls (N = 105). We discuss potential benefits of the multilocus genetic profile score approach and caveats for future research.

  • chronic and episodic Interpersonal Stress as statistically unique predictors of depression in two samples of emerging adults
    Journal of Abnormal Psychology, 2015
    Co-Authors: Suzanne Vrshekschallhorn, Catherine B Stroud, Constance Hammen, Susan Mineka, Richard E Zinbarg, Kate Wolitzkytaylor, Michelle G Craske
    Abstract:

    Few studies comprehensively evaluate which types of life Stress are most strongly associated with depressive episode onsets, over and above other forms of Stress, and comparisons between acute and chronic Stress are particularly lacking. Past research implicates major (moderate to severe) Stressful life events (SLEs), and to a lesser extent, Interpersonal forms of Stress; research conflicts on whether dependent or independent SLEs are more potent, but theory favors dependent SLEs. The present study used 5 years of annual diagnostic and life Stress interviews of chronic Stress and SLEs from 2 separate samples (Sample 1 N = 432; Sample 2 N = 146) transitioning into emerging adulthood; 1 sample also collected early adversity interviews. Multivariate analyses simultaneously examined multiple forms of life Stress to test hypotheses that all major SLEs, then particularly Interpersonal forms of Stress, and then dependent SLEs would contribute unique variance to major depressive episode (MDE) onsets. Person-month survival analysis consistently implicated chronic Interpersonal Stress and major Interpersonal SLEs as statistically unique predictors of risk for MDE onset. In addition, follow-up analyses demonstrated temporal precedence for chronic Stress; tested differences by gender; showed that recent chronic Stress mediates the relationship between adolescent adversity and later MDE onsets; and revealed interactions of several forms of Stress with socioeconomic status (SES). Specifically, as SES declined, there was an increasing role for nonInterpersonal chronic Stress and nonInterpersonal major SLEs, coupled with a decreasing role for Interpersonal chronic Stress. Implications for future etiological research were discussed.

  • effects of chronic Interpersonal Stress exposure on depressive symptoms are moderated by genetic variation at il6 and il1β in youth
    Brain Behavior and Immunity, 2015
    Co-Authors: Margaret Tartter, Constance Hammen, Patricia A Brennan, Julienne E Bower, Steven W Cole
    Abstract:

    Aims Close to one third of patients with major depression show increases in pro-inflammatory cytokines, which are in turn associated with risk for inflammatory disease. Genetic variants that enhance immune reactivity may thus enhance inflammatory and depressive reactions to Stress. The aim of the present study was to investigate a trio of functional SNPs in the promoter regions of IL6 (-174G>C, rs1800795), IL1β (-511C>T, rs16944), and TNF (-308G>A, rs1800629) as moderators of the relationship between chronic Stress exposure and elevations in depressive symptoms.

  • refining the candidate environment Interpersonal Stress the serotonin transporter polymorphism and gene environment interactions in major depression
    Clinical psychological science : a journal of the Association for Psychological Science, 2014
    Co-Authors: Suzanne Vrshekschallhorn, Constance Hammen, James W Griffith, Susan Mineka, Richard E Zinbarg, Michelle G Craske, Eva E Redei, Kate Wolitzkytaylor, Jonathan M Sutton, Emma K Adam
    Abstract:

    Meta-analytic evidence supports a gene-environment (G×E) interaction between life Stress and the serotonin transporter polymorphism (5-HTTLPR) on depression, but few studies have examined factors that influence detection of this effect, despite years of inconsistent results. We propose that the "candidate environment" (akin to a candidate gene) is key. Theory and evidence implicate major Stressful life events (SLEs)-particularly major Interpersonal SLEs-as well as chronic family Stress. Participants (N = 400) from the Youth Emotion Project (which began with 627 high school juniors oversampled for high neuroticism) completed up to five annual diagnostic and life Stress interviews and provided DNA samples. A significant G×E effect for major SLEs and S-carrier genotype was accounted for significantly by major Interpersonal SLEs but not significantly by major non-Interpersonal SLEs. S-carrier genotype and chronic family Stress also significantly interacted. Identifying such candidate environments may facilitate future G×E research in depression and psychopathology more broadly.

  • differential exposure and reactivity to Interpersonal Stress predict sex differences in adolescent depression
    Journal of Clinical Child and Adolescent Psychology, 2006
    Co-Authors: Josephine H Shih, Nicole K Eberhart, Constance Hammen, Patricia A Brennan
    Abstract:

    This study tested the hypothesis that higher rates of depression in adolescent girls are explained by their greater exposure and reactivity to Stress in the Interpersonal domain in a large sample of 15-year-olds. Findings indicate that adolescent girls experienced higher levels of total and Interpersonal episodic Stress, whereas boys experienced higher levels of chronic Stress (academic and close friendship domains). Higher rates of depression in girls were explained by their greater exposure to total Stress, particularly Interpersonal episodic Stress. Adolescent girls were also more reactive (more likely to become depressed) to both total and Interpersonal episodic Stress. The findings suggest that girls experience higher levels of episodic Stress and are more reactive to these Stressors, increasing their likelihood of becoming depressed compared to boys. Results were discussed in terms of girls' greater Interpersonal focus and implications for understanding sex differences in depression.

Michelle G Craske - One of the best experts on this subject based on the ideXlab platform.

  • interaction of cd38 variant and chronic Interpersonal Stress prospectively predicts social anxiety and depression symptoms over 6 years
    Clinical psychological science, 2016
    Co-Authors: Benjamin A Tabak, Susan Mineka, Richard E Zinbarg, Suzanne Vrshekschallhorn, Jason M Prenoveau, Eva E Redei, Emma K Adam, Michelle G Craske
    Abstract:

    Variation in the CD38 gene, which regulates secretion of the neuropeptide oxytocin, has been associated with several social phenotypes. Specifically, rs3796863 A allele carriers have demonstrated increased social sensitivity. In 400 older adolescents, we used trait-state-occasion modeling to investigate how rs3796863 genotype, baseline ratings of chronic Interpersonal Stress, and their gene-environment (GxE) interaction predicted trait social anxiety and depression symptoms over six years. We found significant GxE effects for CD38 A-carrier genotypes and chronic Interpersonal Stress at baseline predicting greater social anxiety and depression symptoms. A significant GxE effect of smaller magnitude was also found for C/C genotype and chronic Interpersonal Stress predicting greater depression; however, this effect was small compared to the main effect of chronic Interpersonal Stress. Thus, in the context of chronic Interpersonal Stress, heightened social sensitivity associated with the rs3796863 A allele may prospectively predict risk for social anxiety and (to a lesser extent) depression.

  • additive genetic risk from five serotonin system polymorphisms interacts with Interpersonal Stress to predict depression
    Journal of Abnormal Psychology, 2015
    Co-Authors: Suzanne Vrshekschallhorn, Catherine B Stroud, Constance Hammen, Susan Mineka, Richard E Zinbarg, Eva E Redei, Emma K Adam, Michelle G Craske
    Abstract:

    Behavioral genetic research supports polygenic models of depression in which many genetic variations each contribute a small amount of risk, and prevailing diathesis-Stress models suggest gene-environment interactions (G×E). Multilocus profile scores of additive risk offer an approach that is consistent with polygenic models of depression risk. In a first demonstration of this approach in a G×E predicting depression, we created an additive multilocus profile score from 5 serotonin system polymorphisms (1 each in the genes HTR1A, HTR2A, HTR2C, and 2 in TPH2). Analyses focused on 2 forms of Interpersonal Stress as environmental risk factors. Using 5 years of longitudinal diagnostic and life Stress interviews from 387 emerging young adults in the Youth Emotion Project, survival analyses show that this multilocus profile score interacts with major Interpersonal Stressful life events to predict major depressive episode onsets (hazard ratio [HR] = 1.815, p = .007). Simultaneously, there was a significant protective effect of the profile score without a recent event (HR = 0.83, p = .030). The G×E effect with Interpersonal chronic Stress was not significant (HR = 1.15, p = .165). Finally, effect sizes for genetic factors examined ignoring Stress suggested such an approach could lead to overlooking or misinterpreting genetic effects. Both the G×E effect and the protective simple main effect were replicated in a sample of early adolescent girls (N = 105). We discuss potential benefits of the multilocus genetic profile score approach and caveats for future research.

  • chronic and episodic Interpersonal Stress as statistically unique predictors of depression in two samples of emerging adults
    Journal of Abnormal Psychology, 2015
    Co-Authors: Suzanne Vrshekschallhorn, Catherine B Stroud, Constance Hammen, Susan Mineka, Richard E Zinbarg, Kate Wolitzkytaylor, Michelle G Craske
    Abstract:

    Few studies comprehensively evaluate which types of life Stress are most strongly associated with depressive episode onsets, over and above other forms of Stress, and comparisons between acute and chronic Stress are particularly lacking. Past research implicates major (moderate to severe) Stressful life events (SLEs), and to a lesser extent, Interpersonal forms of Stress; research conflicts on whether dependent or independent SLEs are more potent, but theory favors dependent SLEs. The present study used 5 years of annual diagnostic and life Stress interviews of chronic Stress and SLEs from 2 separate samples (Sample 1 N = 432; Sample 2 N = 146) transitioning into emerging adulthood; 1 sample also collected early adversity interviews. Multivariate analyses simultaneously examined multiple forms of life Stress to test hypotheses that all major SLEs, then particularly Interpersonal forms of Stress, and then dependent SLEs would contribute unique variance to major depressive episode (MDE) onsets. Person-month survival analysis consistently implicated chronic Interpersonal Stress and major Interpersonal SLEs as statistically unique predictors of risk for MDE onset. In addition, follow-up analyses demonstrated temporal precedence for chronic Stress; tested differences by gender; showed that recent chronic Stress mediates the relationship between adolescent adversity and later MDE onsets; and revealed interactions of several forms of Stress with socioeconomic status (SES). Specifically, as SES declined, there was an increasing role for nonInterpersonal chronic Stress and nonInterpersonal major SLEs, coupled with a decreasing role for Interpersonal chronic Stress. Implications for future etiological research were discussed.

  • refining the candidate environment Interpersonal Stress the serotonin transporter polymorphism and gene environment interactions in major depression
    Clinical psychological science : a journal of the Association for Psychological Science, 2014
    Co-Authors: Suzanne Vrshekschallhorn, Constance Hammen, James W Griffith, Susan Mineka, Richard E Zinbarg, Michelle G Craske, Eva E Redei, Kate Wolitzkytaylor, Jonathan M Sutton, Emma K Adam
    Abstract:

    Meta-analytic evidence supports a gene-environment (G×E) interaction between life Stress and the serotonin transporter polymorphism (5-HTTLPR) on depression, but few studies have examined factors that influence detection of this effect, despite years of inconsistent results. We propose that the "candidate environment" (akin to a candidate gene) is key. Theory and evidence implicate major Stressful life events (SLEs)-particularly major Interpersonal SLEs-as well as chronic family Stress. Participants (N = 400) from the Youth Emotion Project (which began with 627 high school juniors oversampled for high neuroticism) completed up to five annual diagnostic and life Stress interviews and provided DNA samples. A significant G×E effect for major SLEs and S-carrier genotype was accounted for significantly by major Interpersonal SLEs but not significantly by major non-Interpersonal SLEs. S-carrier genotype and chronic family Stress also significantly interacted. Identifying such candidate environments may facilitate future G×E research in depression and psychopathology more broadly.

  • overgeneral autobiographical memory and chronic Interpersonal Stress as predictors of the course of depression in adolescents
    Cognition & Emotion, 2011
    Co-Authors: Jennifer A Sumner, James W Griffith, Susan Mineka, Kathleen Newcomb Rekart, Richard E Zinbarg, Michelle G Craske
    Abstract:

    This study investigated whether overgeneral autobiographical memory (OGM) predicts the course of depression in adolescents. As part of a larger longitudinal study of risk for emotional disorders, 55 adolescents with a past history of major depressive disorder or minor depressive disorder completed the Autobiographical Memory Test. Fewer specific memories predicted the subsequent onset of a major depressive episode (MDE) over a 16-month follow-up period, even when covarying baseline depressive symptoms. This main effect was qualified by an interaction between specific memories and chronic Interpersonal Stress: Fewer specific memories predicted greater risk of MDE onset over follow-up at high (but not low) levels of chronic Interpersonal Stress. Thus, our findings suggest that OGM, in interaction with chronic Interpersonal Stress, predicts the course of depression among adolescents, and highlight the importance of measuring Interpersonal Stress in OGM research.

Mitchell J Prinstein - One of the best experts on this subject based on the ideXlab platform.

  • Interpersonal Stress Severity Longitudinally Predicts Adolescent Girls’ Depressive Symptoms: the Moderating Role of Subjective and HPA Axis Stress Responses
    Journal of Abnormal Child Psychology, 2019
    Co-Authors: Sarah A. Owens, Paul D Hastings, Karen D Rudolph, Sarah W Helms, Matthew K Nock, Mitchell J Prinstein
    Abstract:

    In recent decades, Stress response models of adolescent depression have gained attention, but it remains unclear why only certain adolescents are vulnerable to the depressogenic effects of Stress while others are not. Building on evidence that affective and physiological responses to Stress moderate the impact of Stress exposure on depression, the current study examined whether the interaction between severity of Interpersonal Stress, subjective affective reactivity, and hypothalamic-pituitary-adrenal (HPA) axis reactivity to an acute, in-vivo psychosocial Stressor prospectively predicted depressive symptoms nine months later. Hypotheses were examined with a clinically-oversampled group of 182 adolescent girls (aged 12–16) to ensure an examination of the widest possible range of risk. Self-report measures of affect and salivary cortisol samples were collected before and after an in-vivo Stress task to assess affective reactivity and HPA axis reactivity, respectively. Severity of Interpersonal Stress between baseline and nine months was assessed using a semi-structured interview and was objectively coded for severity and content theme (i.e., Interpersonal vs. non-Interpersonal). Results indicate that experiences of severe Interpersonally-themed Stress predict elevated levels of depressive symptoms longitudinally only for adolescent girls with elevated affective reactivity to Stress, and suggest that these deleterious effects of Stress are most exacerbated for girls with elevated physiological responses to Stress. Findings suggest that it may be critical to examine both affective and physiological Stress responses when assessing risk for depression in adolescents.

  • preliminary associations among relational victimization targeted rejection and suicidality in adolescents a prospective study
    Journal of Clinical Child and Adolescent Psychology, 2019
    Co-Authors: Maya Massingschaffer, Paul D Hastings, Karen D Rudolph, George M Slavich, Sarah W Helms, Matteo Giletta, Matthew K Nock, Mitchell J Prinstein
    Abstract:

    This study examined associations between multiple types of Interpersonal and nonInterpersonal Stressors and the subsequent occurrence of suicide ideation and attempts among female adolescents. Adolescents ages 12 to 18 years old (n = 160) at elevated risk for suicidal thoughts and behaviors were followed for 18 months, divided into two 9-month epochs for data analysis (Periods 1 and 2). Exposure to acute relational victimization, targeted rejection, nonspecified Interpersonal, and nonInterpersonal life Stressors over the first 9-month epoch (Period 1) was assessed using semistructured interviews and an independent life Stress rating team. Participants also completed phone-based semistructured interviews of suicidal thoughts and behaviors. Preliminary analyses showed significant prospective associations between acute targeted rejection and nonspecified Interpersonal Stress during Period 1 and suicide ideation during Period 2, as well as relational victimization and nonInterpersonal Stress during Period 1 and suicide attempts during Period 2. However, in logistic regression analyses that adjusted for prior suicidality and depressive symptoms, relational victimization during Period 1 (but not targeted rejection, nonspecified Interpersonal or nonInterpersonal events) was associated with increased odds of suicide attempt during Period 2. Therefore, acute relational victimization exposure is associated with heightened risk for suicidal behaviors in female adolescents. Future studies should examine potential mediators and moderators of this association, and these Stressors should be considered for inclusion in clinical screening tools.

  • reciprocal associations between adolescent girls chronic Interpersonal Stress and nonsuicidal self injury a multi wave prospective investigation
    Journal of Adolescent Health, 2018
    Co-Authors: Adam Bryant Miller, Paul D Hastings, Karen D Rudolph, Katherine P Linthicum, Sarah W Helms, Matteo Giletta, Matthew K Nock, Mitchell J Prinstein
    Abstract:

    Abstract Purpose Nonsuicidal self-injury (NSSI) is associated with increased risk of suicide attempts. Theories of NSSI assert Interpersonal Stress as a common risk factor for, and perhaps consequence of, NSSI. Prior research has not examined reciprocal associations between chronic Interpersonal Stress and NSSI. This study used a multiwave, prospective design to address this gap in a sample of adolescent girls, a group with elevated risk for both chronic Interpersonal Stress and NSSI. Pubertal development was examined as a moderator of the reciprocal associations. Methods Adolescent girls (N = 220; ages 12–16, M age = 14.69 years) at heightened risk for NSSI completed a baseline assessment and follow-up assessments over 18 months, divided into two 9-month epochs (Time 1 and 2). Pubertal development was assessed via self- and parent-report. Chronic Interpersonal Stress was assessed using a semistructured interview at the end of each time period. NSSI was measured using a semistructured clinical interview every 3 months within both time periods to enhance accurate reporting. Results Path models revealed that chronic romantic Stress during Time 1, but not peer or parent–child Stress, predicted NSSI during Time 2 among girls with more advanced pubertal development. Moreover, NSSI during Time 1 predicted higher levels of chronic romantic and parent–child Stress during Time 2. Conclusions Results revealed a reciprocal relationship between chronic romantic Stress and engagement in NSSI. Further, this association may be best understood in the context of pubertal development.

Benjamin L. Hankin - One of the best experts on this subject based on the ideXlab platform.

  • the interplay of chronic Interpersonal Stress and rumination on nonsuicidal self injury in youth
    Research on Child and Adolescent Psychopathology, 2021
    Co-Authors: Tina H Schweizer, Jami F. Young, Benjamin L. Hankin
    Abstract:

    Nonsuicidal self-injury (NSSI) describes deliberate self-destructive behaviors without the intention to die. Little is known about what factors contribute to NSSI especially among youth. The current study tested two conceptual models for how chronic Interpersonal Stress and rumination may contribute to NSSI engagement across 18 months in a community sample of youth: (1) a mediation pathway based on the Emotional Cascade Model (i.e., Stress contributes to rumination and then to subsequent NSSI), and (2) a moderation model based on the cognitive vulnerability-Stress framework (i.e., rumination moderates the relation between Stress and NSSI). 516 youth aged 7–16 (Mage = 12.0; 56% female; 90% Non-Hispanic or Non-Latinx) reported on ongoing Interpersonal Stress occurring between T1-T2 (every 6 months from T1 to 12 months) via the Youth Life Stress Interview, rumination via the Children’s Response Styles Questionnaire (at T1 and 18 months later, T2), and NSSI engagement every six months from 18 to 36 months (T2-T3) via the Self-Injurious Thoughts and Behaviors Interview. Interpersonal Stress predicted later rumination (b = .43, p < .01), rumination forecasted later NSSI occurrence (OR = 1.06, p < .01), and mediation was supported via a significant indirect effect of Interpersonal Stress on NSSI through rumination (b = .03, 95% CI = .01, .07). Rumination did not significantly moderate the relation between Stress and NSSI. The prospective relation between chronic Interpersonal Stress and NSSI engagement was partly explained by rumination, aligning with the Emotional Cascade Model’s prediction that rumination contributes to NSSI. Youth may conduct NSSI to interrupt rumination elicited by chronic Interpersonal Stress.

  • Co-Occurring Trajectories of Depression and Social Anxiety in Childhood and Adolescence: Interactive Effects of Positive Emotionality and Domains of Chronic Interpersonal Stress
    Journal of Abnormal Child Psychology, 2020
    Co-Authors: Julianne M. Griffith, Erin E. Long, Jami F. Young, Benjamin L. Hankin
    Abstract:

    Deficits in positive emotionality (PE) have been implicated in the etiology of both social anxiety and depression; however, factors that contribute to divergent social anxiety and depression outcomes among youth low in PE remain unknown. Extant research suggests that parent-child Stress and peer Stress demonstrate differential patterns of associations with social anxiety and depression. Thus, the present study examined prospective interactive effects of PE and chronic parent-child and peer Stress on simultaneously developing trajectories of social anxiety and depression symptoms among 543 boys and girls (age 8–16 at baseline, M[SD] = 11.94[2.32] 55.6% female). Parents reported on youth PE at baseline. Domains of chronic Interpersonal (parent-child and peer) Stress occurring between baseline and 18-months were assessed via child-report by trained interviews using the Youth Life Stress Interview (Rudolph and Flynn Development and Psychopathology , 19 (2), 497–521, 2007 ). Youth completed self-report measures of depression and social anxiety every three months from 18- to 36- months (7 assessments). Conditional bivariate latent growth curve models indicated that main effects of parent-child Stress, but not peer Stress, predicted trajectories of depression in boys and girls. In girls, high levels of chronic Interpersonal Stress in both domains predicted stable, elevated trajectories of social anxiety symptoms regardless of PE. In boys, PE contributed to a pattern of differential susceptibility whereby boys high in PE were particularly susceptible to the effects of chronic Interpersonal Stress, for better or worse.

  • 5 httlpr Interpersonal Stress interaction and nonsuicidal self injury in general community sample of youth
    Psychiatry Research-neuroimaging, 2015
    Co-Authors: Benjamin L. Hankin, Jami F. Young, Andrea L Barrocas, Brett C Haberstick, Andrew Smolen
    Abstract:

    No research with youth has investigated whether measured genetic risk interacts with Stressful environment (G⁎E) to explain engagement in non-suicidal self-injury (NSSI). Two independent samples of youth were used to test the a priori hypothesis that the Transporter-Linked Polymorphic Region (5-HTTLPR) would interact with chronic Interpersonal Stress to predict NSSI. We tested this hypothesis with children and adolescents from United States public schools in two independent samples (N׳s=300 and 271) using identical procedures and methods. They were interviewed in person with the Self-Injurious Thoughts and Behaviors Interview to assess NSSI engagement and with the UCLA Chronic Stress Interview to assess Interpersonal Stress. Buccal cells were collected for genotyping of 5-HTTLPR. For both samples, ANOVAs revealed the hypothesized G⁎E. Specifically, short carriers who experienced severe Interpersonal Stress exhibited the highest level of NSSI engagement. Replicated across two independent samples, results provide the first demonstration that youth at high genetic susceptibility (5-HTTLPR) and high environmental exposure (chronic Interpersonal Stress) are at heightened risk for NSSI. Language: en

  • corumination Interpersonal Stress generation and internalizing symptoms accumulating effects and transactional influences in a multiwave study of adolescents
    Development and Psychopathology, 2010
    Co-Authors: Benjamin L. Hankin, Lindsey B Stone, Patricia Ann Wright
    Abstract:

    This multiwave longitudinal study investigated potential transactional and accumulating influences among corumination, Interpersonal Stressors, and internalizing symptoms among a sample of early and middle adolescents (N = 350; 6th-10th graders). Youth completed self-report measures of corumination at Times 1, 2, and 4, and negative life events, internalizing symptoms (general depressive, specific anhedonic depressive, anxious arousal, general internalizing), and externalizing problems at all four time points (5 weeks between each assessment across 4 months). Results supported hypotheses. First, baseline corumination predicted prospective trajectories of all forms of internalizing symptoms but not externalizing problems. Second, baseline corumination predicted generation of Interpersonal-dependent, but not Interpersonal-independent or nonInterpersonal Stressors. Third, Interpersonal-dependent events partially mediated the longitudinal association between baseline corumination and prospective internalizing symptoms. Fourth, a transactional, bidirectional set of associations was supported in that initial internalizing symptoms and Stressors predicted later elevations in corumination, and in turn, corumination predicted later symptoms through the mediating role of Interpersonal Stressors to complete both streams in the transactional chain of influence. Fifth, girls and older adolescents exhibited higher corumination, but neither age nor sex moderated any associations. These findings are discussed within a transactional, developmental cascade model.

  • adult attachment dimensions and specificity of emotional diStress symptoms prospective investigations of cognitive risk and Interpersonal Stress generation as mediating mechanisms
    Personality and Social Psychology Bulletin, 2005
    Co-Authors: Benjamin L. Hankin, Jon D Kassel, John R Z Abela
    Abstract:

    Three prospective studies examined the relation between adult attachment dimensions and symptoms of emotional diStress (anxiety and depression). Across all three studies, avoidant and anxious attachment prospectively predicted depressive symptoms, and anxious attachment was associated concurrently with anxiety symptoms. Study 2 tested a cognitive risk factors mediational model, and Study 3 tested an Interpersonal Stress generation mediational model. Both cognitive and Interpersonal mediating processes were supported. The cognitive risk factors pathway, including elevated dysfunctional attitudes and low self-esteem, specifically mediated the relation between insecure attachment and prospective elevations in depression but not anxiety. For the Interpersonal Stress generation model, experiencing additional Interpersonal, but not achievement, Stressors over time mediated the association between insecure attachment and prospective elevations in depressive and anxious symptoms. Results advance theory and empirical knowledge about why these Interpersonal and cognitive mechanisms explain how insecurely attached people become depressed and anxious.