Intraabdominal

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G. H. H. Mannaerts - One of the best experts on this subject based on the ideXlab platform.

Michael J Rosen - One of the best experts on this subject based on the ideXlab platform.

  • permissible Intraabdominal hypertension following complex abdominal wall reconstruction
    Plastic and Reconstructive Surgery, 2015
    Co-Authors: Clayton C Petro, Siavash Raigani, Mojtaba Fayezizadeh, James R Rowbottom, John C Klick, Ajita S Prabhu, Yuri W Novitsky, Michael J Rosen
    Abstract:

    Background:Repair of hernias with loss of domain can lead to elevated Intraabdominal pressure. The authors aimed to characterize the effects of elective hernia repair on Intraabdominal pressure, as well as its predictors and association with negative outcomes.Methods:Patients undergoing elective her

  • Permissible Intraabdominal Hypertension following Complex Abdominal Wall Reconstruction.
    Plastic and reconstructive surgery, 2015
    Co-Authors: Clayton C Petro, Siavash Raigani, Mojtaba Fayezizadeh, James R Rowbottom, John C Klick, Ajita S Prabhu, Yuri W Novitsky, Michael J Rosen
    Abstract:

    Repair of hernias with loss of domain can lead to elevated Intraabdominal pressure. The authors aimed to characterize the effects of elective hernia repair on Intraabdominal pressure, as well as its predictors and association with negative outcomes. Patients undergoing elective hernia repair requiring myofascial release had Intraabdominal and pulmonary plateau pressures measured preoperatively, postoperatively, and on the morning of the first postoperative day. Loss of domain was measured by preoperative computed tomography. Outcome measures included predictors of an increase in plateau pressure, respiratory complications, and acute kidney injury. Following 50 consecutive cases, diagnoses of Intraabdominal hypertension (92 percent), abdominal compartment syndrome (16 percent), and abdominal perfusion pressure less than 60 mmHg (24 percent) were determined. Changes in Intraabdominal pressure (preoperative, 12.7 ± 4.0 mmHg; postoperative, 18.2 ± 5.4 mmHg; postoperative day 1, 12.9 ± 5.2 mmHg) and abdominal perfusion pressure (preoperative, 74.7 ± 15.7; postoperative, 70.0 ± 14.4; postoperative day 1, 74.9 ± 11.6 mmHg) consistently resolved by postoperative day 1, and were not associated with respiratory complications or acute kidney injury. Patients who remained intubated postoperatively for an elevation in pulmonary plateau pressure (≥6 mmHg) all demonstrated an improvement in plateau pressure by postoperative day 1 (preoperative, 18.9 ± 4.5 mmHg; postoperative, 27.4 ± 4.0 mmHg; postoperative day 1, 20.1 ± 3.7 mmHg), and could be identified preoperatively as having a hernia volume of greater than 20 percent of the abdominal cavity (p < 0.001), but were still more likely to have postoperative respiratory events (p = 0.01). Elevated Intraabdominal pressure following elective hernia repair requiring myofascial releases is common but transient. Change in plateau pressure by 6 mmHg or more following repair can be expected with a loss of domain greater than 20 percent and is a more useful surrogate than Intraabdominal pressure measurements with regard to predicting postoperative pulmonary complications. The perception and management of elevated Intraabdominal pressure should be considered distinct and "permissible" in this context.

A. J. Runia - One of the best experts on this subject based on the ideXlab platform.

Nelson Jamel - One of the best experts on this subject based on the ideXlab platform.

  • Effects of different tidal volumes in pulmonary and extrapulmonary lung injury with or without Intraabdominal hypertension
    Intensive Care Medicine, 2012
    Co-Authors: Cíntia L. Santos, Lillian Moraes, Raquel S. Santos, Mariana G. Oliveira, Johnatas D. Silva, Tatiana Maron-gutierrez, Débora S. Ornellas, Marcelo M. Morales, Vera L. Capelozzi, Nelson Jamel
    Abstract:

    Purpose We hypothesized that: (1) Intraabdominal hypertension increases pulmonary inflammatory and fibrogenic responses in acute lung injury (ALI); (2) in the presence of Intraabdominal hypertension, higher tidal volume reduces lung damage in extrapulmonary ALI, but not in pulmonary ALI. Methods Wistar rats were randomly allocated to receive Escherichia coli lipopolysaccharide intratracheally (pulmonary ALI) or intraperitoneally (extrapulmonary ALI). After 24 h, animals were randomized into subgroups without or with Intraabdominal hypertension (15 mmHg) and ventilated with positive end expiratory pressure = 5 cmH_2O and tidal volume of 6 or 10 ml/kg during 1 h. Lung and chest wall mechanics, arterial blood gases, lung and distal organ histology, and interleukin (IL)-1β, IL-6, caspase-3 and type III procollagen (PCIII) mRNA expressions in lung tissue were analyzed. Results With Intraabdominal hypertension, (1) chest-wall static elastance increased, and PCIII, IL-1β, IL-6, and caspase-3 expressions were more pronounced than in animals with normal Intraabdominal pressure in both ALI groups; (2) in extrapulmonary ALI, higher tidal volume was associated with decreased atelectasis, and lower IL-6 and caspase-3 expressions; (3) in pulmonary ALI, higher tidal volume led to higher IL-6 expression; and (4) in pulmonary ALI, liver, kidney, and villi cell apoptosis was increased, but not affected by tidal volume. Conclusions Intraabdominal hypertension increased inflammation and fibrogenesis in the lung independent of ALI etiology. In extrapulmonary ALI associated with Intraabdominal hypertension, higher tidal volume improved lung morphometry with lower inflammation in lung tissue. Conversely, in pulmonary ALI associated with Intraabdominal hypertension, higher tidal volume increased IL-6 expression.

  • effects of different tidal volumes in pulmonary and extrapulmonary lung injury with or without Intraabdominal hypertension
    Intensive Care Medicine, 2012
    Co-Authors: Cíntia L. Santos, Lillian Moraes, Raquel S. Santos, Mariana G. Oliveira, Johnatas D. Silva, Débora S. Ornellas, Marcelo M. Morales, Vera L. Capelozzi, Tatiana Marongutierrez, Nelson Jamel
    Abstract:

    We hypothesized that: (1) Intraabdominal hypertension increases pulmonary inflammatory and fibrogenic responses in acute lung injury (ALI); (2) in the presence of Intraabdominal hypertension, higher tidal volume reduces lung damage in extrapulmonary ALI, but not in pulmonary ALI. Wistar rats were randomly allocated to receive Escherichia coli lipopolysaccharide intratracheally (pulmonary ALI) or intraperitoneally (extrapulmonary ALI). After 24 h, animals were randomized into subgroups without or with Intraabdominal hypertension (15 mmHg) and ventilated with positive end expiratory pressure = 5 cmH2O and tidal volume of 6 or 10 ml/kg during 1 h. Lung and chest wall mechanics, arterial blood gases, lung and distal organ histology, and interleukin (IL)-1β, IL-6, caspase-3 and type III procollagen (PCIII) mRNA expressions in lung tissue were analyzed. With Intraabdominal hypertension, (1) chest-wall static elastance increased, and PCIII, IL-1β, IL-6, and caspase-3 expressions were more pronounced than in animals with normal Intraabdominal pressure in both ALI groups; (2) in extrapulmonary ALI, higher tidal volume was associated with decreased atelectasis, and lower IL-6 and caspase-3 expressions; (3) in pulmonary ALI, higher tidal volume led to higher IL-6 expression; and (4) in pulmonary ALI, liver, kidney, and villi cell apoptosis was increased, but not affected by tidal volume. Intraabdominal hypertension increased inflammation and fibrogenesis in the lung independent of ALI etiology. In extrapulmonary ALI associated with Intraabdominal hypertension, higher tidal volume improved lung morphometry with lower inflammation in lung tissue. Conversely, in pulmonary ALI associated with Intraabdominal hypertension, higher tidal volume increased IL-6 expression.

  • effects of different tidal volumes in pulmonary and extrapulmonary lung injury with or without Intraabdominal hypertension
    Intensive Care Medicine, 2012
    Co-Authors: Cíntia L. Santos, Lillian Moraes, Raquel S. Santos, Mariana G. Oliveira, Johnatas D. Silva, Débora S. Ornellas, Marcelo M. Morales, Vera L. Capelozzi, Tatiana Marongutierrez, Nelson Jamel
    Abstract:

    Purpose We hypothesized that: (1) Intraabdominal hypertension increases pulmonary inflammatory and fibrogenic responses in acute lung injury (ALI); (2) in the presence of Intraabdominal hypertension, higher tidal volume reduces lung damage in extrapulmonary ALI, but not in pulmonary ALI.

Edward I. Miller - One of the best experts on this subject based on the ideXlab platform.

  • Seminomas complicating undescended Intraabdominal testes in patients with prior negative findings from surgical exploration
    AJR. American journal of roentgenology, 1999
    Co-Authors: Frank H. Miller, Winston S. Whitney, S W Fitzgerald, Edward I. Miller
    Abstract:

    OBJECTIVE: This paper describes the clinical and imaging characteristics of seminoma complicating Intraabdominal testes in three patients who had a history of negative findings from surgical exploration of the inguinal canal and scrotum. CONCLUSION: Because inguinal surgery for cryptorchidism may fail to disclose Intraabdominal testes, surgery that has negative findings should be followed up by a close examination of the abdomen. Seminomas arising from an Intraabdominal testis can be large and asymptomatic or may simulate other diseases. Torsion may occur and occasionally appear as acute abdomen. The imaging features can be nonspecific, and the history of cryptorchidism may not be provided. Awareness of the clinical and imaging features of neoplasms involving undescended Intraabdominal testes is important for appropriate diagnosis and therapy.