The Experts below are selected from a list of 20292 Experts worldwide ranked by ideXlab platform
Ming-heng Cai - One of the best experts on this subject based on the ideXlab platform.
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Effects of massage therapy for patients with thoracic facet Joint Disorders: A protocol for systematic review and meta-analysis.
Medicine, 2020Co-Authors: Ke-lin Zhou, Shuo Dong, Jing-yi Yang, Mei-ling Ren, Sheng Guo, Ming-heng CaiAbstract:Background Thoracic facet Joint Disorder is a common thoracic Disorder in clinic, inducing pain and discomfort at the dislocated thoracic vertebrae, radiating to pain of the neck and back. The incidence of thoracic facet Joint Disorder is higher than the facet Disorder of the cervical and lumbar vertebrae. Therefore, an ideal strategy to relieve thoracic facet Joint Disorder is urgently needed. In recent years, massage therapy has been increasingly accepted by thoracic facet Joint Disorder patients due to its lower costs, fewer unwanted side effects and safety for clinical use. In this systematic review, we aim to evaluate the effectiveness and safety of massage therapy for patients with thoracic facet Joint Disorder. Methods We will search the following electronic databases for randomized controlled trials to evaluate the effectiveness of massage therapy in treating thoracic facet Joint Disorder: Wanfang and PubMed Database, CNKI, CENTRAL, CINAHL and EMBASE. Each database will be searched from inception to October 2020. The entire process will include study selection, data extraction, risk of bias assessment and meta-analyses. Results This proposed study will evaluate the effectiveness of massage therapy for patients with thoracic facet Joint Disorder. Conclusions This proposed systematic review will evaluate the existing evidence on the effectiveness and safety of massage therapy for patients with thoracic facet Joint Disorder. Dissemination and ethics The results of this review will be disseminated through peer-reviewed publication. Because all of the data used in this systematic review and meta-analysis has been published, this review does not require ethical approval. Furthermore, all data will be analyzed anonymously during the review process. Osf registration number DOI 10.17605/OSF.IO/XMEJD.
Ke-lin Zhou - One of the best experts on this subject based on the ideXlab platform.
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Effects of massage therapy for patients with thoracic facet Joint Disorders: A protocol for systematic review and meta-analysis.
Medicine, 2020Co-Authors: Ke-lin Zhou, Shuo Dong, Jing-yi Yang, Mei-ling Ren, Sheng Guo, Ming-heng CaiAbstract:Background Thoracic facet Joint Disorder is a common thoracic Disorder in clinic, inducing pain and discomfort at the dislocated thoracic vertebrae, radiating to pain of the neck and back. The incidence of thoracic facet Joint Disorder is higher than the facet Disorder of the cervical and lumbar vertebrae. Therefore, an ideal strategy to relieve thoracic facet Joint Disorder is urgently needed. In recent years, massage therapy has been increasingly accepted by thoracic facet Joint Disorder patients due to its lower costs, fewer unwanted side effects and safety for clinical use. In this systematic review, we aim to evaluate the effectiveness and safety of massage therapy for patients with thoracic facet Joint Disorder. Methods We will search the following electronic databases for randomized controlled trials to evaluate the effectiveness of massage therapy in treating thoracic facet Joint Disorder: Wanfang and PubMed Database, CNKI, CENTRAL, CINAHL and EMBASE. Each database will be searched from inception to October 2020. The entire process will include study selection, data extraction, risk of bias assessment and meta-analyses. Results This proposed study will evaluate the effectiveness of massage therapy for patients with thoracic facet Joint Disorder. Conclusions This proposed systematic review will evaluate the existing evidence on the effectiveness and safety of massage therapy for patients with thoracic facet Joint Disorder. Dissemination and ethics The results of this review will be disseminated through peer-reviewed publication. Because all of the data used in this systematic review and meta-analysis has been published, this review does not require ethical approval. Furthermore, all data will be analyzed anonymously during the review process. Osf registration number DOI 10.17605/OSF.IO/XMEJD.
Jennifer A Haythornthwaite - One of the best experts on this subject based on the ideXlab platform.
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exploring the role of negative cognitions in the relationship between ethnicity sleep and pain in women with temporomandibular Joint Disorder
The Journal of Pain, 2018Co-Authors: Sheera F Lerman, Michael T Smith, C Campbell, L Buenaver, Mary Medak, Jane Phillips, Michelle Polley, Jennifer A HaythornthwaiteAbstract:Negative cognitions are central to the perpetuation of chronic pain and sleep disturbances. Patients with temporomandibular Joint Disorder (TMJD), a chronic pain condition characterized by pain and limitation in the jaw area, have a high comorbidity of sleep disturbances that possibly exacerbate their condition. Ethnic group differences are documented in pain, sleep, and coping, yet the mechanisms driving these differences are still unclear, especially in clinical pain populations. We recruited 156 women (79% white, 21% African American) diagnosed with TMJD as part of a randomized, controlled trial evaluating the effectiveness of interventions targeting sleep and pain catastrophizing on pain in TMJD. Analysis of baseline data demonstrated that, relative to white participants, African Americans exhibited higher levels of clinical pain, insomnia severity, and pain catastrophizing, yet there was no ethnic group difference in negative sleep-related cognitions. Mediation models revealed pain catastrophizing, but not sleep-related cognitions or insomnia severity, to be a significant single mediator of the relationship between ethnicity and clinical pain. Only the helplessness component of catastrophizing together with insomnia severity sequentially mediated the ethnicity-pain relationship. These findings identify pain catastrophizing as a potentially important link between ethnicity and clinical pain and suggest that interventions targeting pain-related helplessness could improve both sleep and pain, especially for African American patients. Perspective:Pain-related helplessness and insomnia severity contribute to ethnic differences found in clinical pain among woman with TMJD. Findings can potentially inform interventions that target insomnia and catastrophizing to assist in reducing ethnic disparities in clinical pain.
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sleep continuity and architecture associations with pain inhibitory processes in patients with temporomandibular Joint Disorder
European Journal of Pain, 2009Co-Authors: Robert R Edwards, Estherann Grace, Stephen Peterson, Brendan Klick, Jennifer A Haythornthwaite, Michael T SmithAbstract:Recent research suggests bi-directional interactions between the experience of pain and the process of sleep; pain interferes with the ability to obtain sleep, and disrupted sleep contributes to enhanced pain perception. Our group recently reported, in a controlled experimental study, that sleep fragmentation among healthy adults resulted in subsequent decrements in endogenous pain inhibition. The present report follows up that observation by extending this line of research to a sample of patients experiencing persistent pain. Patients with chronic temporomandibular Joint Disorder (TMD) pain were studied using polysomnography and psychophysical evaluation of pain responses. We assessed whether individual differences in sleep continuity and/or architecture were related to diffuse noxious inhibitory controls (DNIC), a measure of central nervous system pain inhibition. Among 53 TMD patients, higher sleep efficiency and longer total sleep time were positively associated with better functioning of DNIC (r = 0.42–0.44, p < 0.01; ps < 0.05 for the multivariate analyses). These results suggest the possibility that disrupted sleep may serve as a risk factor for inadequate pain-inhibitory processing and hint that aggressive efforts to treat sleep disturbance early in the course of a pain condition might be beneficial in reducing the severity or impact of clinical pain.
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sleep Disorders and their association with laboratory pain sensitivity in temporomandibular Joint Disorder
Sleep, 2009Co-Authors: Michael T Smith, Robert R Edwards, Stephen Peterson, Brendan Klick, L Buenaver, Emerson M Wickwire, Edward G Grace, Jennifer A HaythornthwaiteAbstract:TEMPOROMANDIBULAR Joint Disorder (TMD) HAS BEEN DESCRIBED AS A PROTOTYPIC IDIOPATHIC PAIN SYNDROME CHARACTERIZED BY POORLY understood, episodic, masticatory muscle and/or Joint pain. TMD affects an estimated 12% of the population.1 As in other idiopathic pain Disorders such as fibromyalgia and irritable bowel syndrome, patients often present with overlapping signs and symptoms including psychological distress, neuroendocrine abnormalities, and chronic insomnia.2,3 Recent theoretical perspectives have proposed that these “central sensitivity syndromes” share a common central nervous system substrate characterized by heightened processing of noxious input, which contributes to overlapping daytime sequelae among these Disorders.4 Several cross-sectional studies have demonstrated that compared to controls, TMD patients exhibit enhanced responsivity to a variety of painful stimuli measured both at facial and extracranial anatomic sites.5–7 Pain sensitivity at “unaffected” (i.e., non-jaw) sites suggests the involvement of central pain processing mechanisms, beyond peripheral contributions. Recent longitudinal work has reported that enhanced laboratory pain sensitivity in pain free individuals is linked to genetic polymorphisms that predict the development of new onset TMD.8 This suggests that central processes associated with pain amplification may be critical to understanding the etiopathophysiology of TMD. Clinical factors that contribute to pain amplification in TMD, however, are poorly understood. Our group has focused on the possibility that sleep disturbance is one such factor that may directly contribute to central sensitization and pain amplification.9 We recently reported, for example, that sleep onset insomnia symptoms predict the development of chronic pain following serious burn injury.10 While it is often assumed that insomnia or sleep loss occurring in the context of chronic pain occurs secondarily to the sleep interrupting effects of pain, we and others have demonstrated that insomnias associated with chronic pain are often phenotypically similar to primary insomnia.11 Shared features include high levels of pre-sleep cognitive rumination and evidence of maladaptive coping strategies that may exist prior to the development of pain and/or independently contribute to insomnia symptoms. It is unknown, however, whether primary insomnia is associated with alterations in laboratory pain sensitivity when it occurs either as a sole condition or as part of a chronic pain Disorder such as TMD. Only a handful of investigations have systematically sought to evaluate the sleep quality of TMD patients. These studies have consistently found that the majority ( > 50%) of TMD patients report poor sleep quality, and that subjective ratings of poor sleep are associated with increased clinical pain severity and psychological distress.12–14 Fundamental descriptive data using polysomnography and standard research diagnostic interviews to quantify the range of sleep Disorders in TMD and determine their possible associations with laboratory measures of pain sensitivity are lacking. The extant literature has largely focused on possible relationships between sleep bruxism and TMD.15,16 Sleep bruxism, however, has not been found to be associated with either poor sleep quality or polysomnographic measures of sleep continuity or architecture disturbances.17–19 The objective of this study was to address two critical gaps in the literature: (1) characterize the spectrum of sleep Disorders in a well-described sample of myofascial TMD patients, using polysomnography and state-of-the art structured diagnostic interviews; and (2) evaluate possible associations between observed sleep Disorder indices and laboratory measures of pain threshold. We hypothesized that rates of primary insomnia would be substantive in TMD and that primary insomnia would be associated with reductions in pain threshold at both masseter and extracranial sites.
Berthold Langguth - One of the best experts on this subject based on the ideXlab platform.
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temporomandibular Joint Disorder complaints in tinnitus further hints for a putative tinnitus subtype
PLOS ONE, 2012Co-Authors: Veronika Vielsmeier, Jurgen Strutz, Tobias Kleinjung, Martin Schecklmann, Peter M Kreuzer, Michael Landgrebe, Berthold LangguthAbstract:Objective Tinnitus is considered to be highly heterogeneous with respect to its etiology, its comorbidities and the response to specific interventions. Subtyping is recommended, but it remains to be determined which criteria are useful, since it has not yet been clearly demonstrated whether and to which extent etiologic factors, comorbid states and interventional response are related to each other and are thus applicable for subtyping tinnitus. Analyzing the Tinnitus Research Initiative Database we differentiated patients according to presence or absence of comorbid temporomandibular Joint (TMJ) Disorder complaints and compared the two groups with respect to etiologic factors. Methods 1204 Tinnitus patients from the Tinnitus Research Initiative (TRI) Database with and without subjective TMJ complaints were compared with respect to demographic, tinnitus and audiological characteristics, questionnaires, and numeric ratings. Data were analysed according to a predefined statistical analysis plan. Results Tinnitus patients with TMJ complaints (22% of the whole group) were significantly younger, had a lower age at tinnitus onset, and were more frequently female. They could modulate or mask their tinnitus more frequently by somatic maneuvers and by music or sound stimulation. Groups did not significantly differ for tinnitus duration, type of onset (gradual/abrupt), onset related events (whiplash etc.), character (pulsatile or not), hyperacusis, hearing impairment, tinnitus distress, depression, quality of life and subjective ratings (loudness etc.). Conclusion Replicating previous work in tinnitus patients with TMJ complaints, classical risk factors for tinnitus like older age and male gender are less relevant in tinnitus patients with TMJ complaints. By demonstrating group differences for modulation of tinnitus by movements and sounds our data further support the notion that tinnitus with TMJ complaints represents a subgroup of tinnitus with clinical features that are highly relevant for specific therapeutic management.
Michael T Smith - One of the best experts on this subject based on the ideXlab platform.
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exploring the role of negative cognitions in the relationship between ethnicity sleep and pain in women with temporomandibular Joint Disorder
The Journal of Pain, 2018Co-Authors: Sheera F Lerman, Michael T Smith, C Campbell, L Buenaver, Mary Medak, Jane Phillips, Michelle Polley, Jennifer A HaythornthwaiteAbstract:Negative cognitions are central to the perpetuation of chronic pain and sleep disturbances. Patients with temporomandibular Joint Disorder (TMJD), a chronic pain condition characterized by pain and limitation in the jaw area, have a high comorbidity of sleep disturbances that possibly exacerbate their condition. Ethnic group differences are documented in pain, sleep, and coping, yet the mechanisms driving these differences are still unclear, especially in clinical pain populations. We recruited 156 women (79% white, 21% African American) diagnosed with TMJD as part of a randomized, controlled trial evaluating the effectiveness of interventions targeting sleep and pain catastrophizing on pain in TMJD. Analysis of baseline data demonstrated that, relative to white participants, African Americans exhibited higher levels of clinical pain, insomnia severity, and pain catastrophizing, yet there was no ethnic group difference in negative sleep-related cognitions. Mediation models revealed pain catastrophizing, but not sleep-related cognitions or insomnia severity, to be a significant single mediator of the relationship between ethnicity and clinical pain. Only the helplessness component of catastrophizing together with insomnia severity sequentially mediated the ethnicity-pain relationship. These findings identify pain catastrophizing as a potentially important link between ethnicity and clinical pain and suggest that interventions targeting pain-related helplessness could improve both sleep and pain, especially for African American patients. Perspective:Pain-related helplessness and insomnia severity contribute to ethnic differences found in clinical pain among woman with TMJD. Findings can potentially inform interventions that target insomnia and catastrophizing to assist in reducing ethnic disparities in clinical pain.
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sleep continuity and architecture associations with pain inhibitory processes in patients with temporomandibular Joint Disorder
European Journal of Pain, 2009Co-Authors: Robert R Edwards, Estherann Grace, Stephen Peterson, Brendan Klick, Jennifer A Haythornthwaite, Michael T SmithAbstract:Recent research suggests bi-directional interactions between the experience of pain and the process of sleep; pain interferes with the ability to obtain sleep, and disrupted sleep contributes to enhanced pain perception. Our group recently reported, in a controlled experimental study, that sleep fragmentation among healthy adults resulted in subsequent decrements in endogenous pain inhibition. The present report follows up that observation by extending this line of research to a sample of patients experiencing persistent pain. Patients with chronic temporomandibular Joint Disorder (TMD) pain were studied using polysomnography and psychophysical evaluation of pain responses. We assessed whether individual differences in sleep continuity and/or architecture were related to diffuse noxious inhibitory controls (DNIC), a measure of central nervous system pain inhibition. Among 53 TMD patients, higher sleep efficiency and longer total sleep time were positively associated with better functioning of DNIC (r = 0.42–0.44, p < 0.01; ps < 0.05 for the multivariate analyses). These results suggest the possibility that disrupted sleep may serve as a risk factor for inadequate pain-inhibitory processing and hint that aggressive efforts to treat sleep disturbance early in the course of a pain condition might be beneficial in reducing the severity or impact of clinical pain.
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sleep Disorders and their association with laboratory pain sensitivity in temporomandibular Joint Disorder
Sleep, 2009Co-Authors: Michael T Smith, Robert R Edwards, Stephen Peterson, Brendan Klick, L Buenaver, Emerson M Wickwire, Edward G Grace, Jennifer A HaythornthwaiteAbstract:TEMPOROMANDIBULAR Joint Disorder (TMD) HAS BEEN DESCRIBED AS A PROTOTYPIC IDIOPATHIC PAIN SYNDROME CHARACTERIZED BY POORLY understood, episodic, masticatory muscle and/or Joint pain. TMD affects an estimated 12% of the population.1 As in other idiopathic pain Disorders such as fibromyalgia and irritable bowel syndrome, patients often present with overlapping signs and symptoms including psychological distress, neuroendocrine abnormalities, and chronic insomnia.2,3 Recent theoretical perspectives have proposed that these “central sensitivity syndromes” share a common central nervous system substrate characterized by heightened processing of noxious input, which contributes to overlapping daytime sequelae among these Disorders.4 Several cross-sectional studies have demonstrated that compared to controls, TMD patients exhibit enhanced responsivity to a variety of painful stimuli measured both at facial and extracranial anatomic sites.5–7 Pain sensitivity at “unaffected” (i.e., non-jaw) sites suggests the involvement of central pain processing mechanisms, beyond peripheral contributions. Recent longitudinal work has reported that enhanced laboratory pain sensitivity in pain free individuals is linked to genetic polymorphisms that predict the development of new onset TMD.8 This suggests that central processes associated with pain amplification may be critical to understanding the etiopathophysiology of TMD. Clinical factors that contribute to pain amplification in TMD, however, are poorly understood. Our group has focused on the possibility that sleep disturbance is one such factor that may directly contribute to central sensitization and pain amplification.9 We recently reported, for example, that sleep onset insomnia symptoms predict the development of chronic pain following serious burn injury.10 While it is often assumed that insomnia or sleep loss occurring in the context of chronic pain occurs secondarily to the sleep interrupting effects of pain, we and others have demonstrated that insomnias associated with chronic pain are often phenotypically similar to primary insomnia.11 Shared features include high levels of pre-sleep cognitive rumination and evidence of maladaptive coping strategies that may exist prior to the development of pain and/or independently contribute to insomnia symptoms. It is unknown, however, whether primary insomnia is associated with alterations in laboratory pain sensitivity when it occurs either as a sole condition or as part of a chronic pain Disorder such as TMD. Only a handful of investigations have systematically sought to evaluate the sleep quality of TMD patients. These studies have consistently found that the majority ( > 50%) of TMD patients report poor sleep quality, and that subjective ratings of poor sleep are associated with increased clinical pain severity and psychological distress.12–14 Fundamental descriptive data using polysomnography and standard research diagnostic interviews to quantify the range of sleep Disorders in TMD and determine their possible associations with laboratory measures of pain sensitivity are lacking. The extant literature has largely focused on possible relationships between sleep bruxism and TMD.15,16 Sleep bruxism, however, has not been found to be associated with either poor sleep quality or polysomnographic measures of sleep continuity or architecture disturbances.17–19 The objective of this study was to address two critical gaps in the literature: (1) characterize the spectrum of sleep Disorders in a well-described sample of myofascial TMD patients, using polysomnography and state-of-the art structured diagnostic interviews; and (2) evaluate possible associations between observed sleep Disorder indices and laboratory measures of pain threshold. We hypothesized that rates of primary insomnia would be substantive in TMD and that primary insomnia would be associated with reductions in pain threshold at both masseter and extracranial sites.
