The Experts below are selected from a list of 29553 Experts worldwide ranked by ideXlab platform
Johannes Siegrist - One of the best experts on this subject based on the ideXlab platform.
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measuring effort reward imbalance in school settings a novel approach and its association with self rated health
Journal of Epidemiology, 2010Co-Authors: Li Shang, Tao Wang, Johannes SiegristAbstract:Background: We attempted to apply the model of effort–reward imbalance (ERI) to school settings in order to measure students’ psychosocial stress and analyze its association with self-rated health in adolescents.Methods: A cross-sectional survey was conducted in Kunming, China among 1004 Chinese students (468 boys and 536 girls) in grades 7 through 12, using a 19-item effort–reward imbalance questionnaire.Results: Satisfactory internal consistencies for the scales for effort and reward were obtained; the value for the scale for overcommitment was acceptable. Factor analysis replicated the theoretical structure of the ERI construct in this sample of Chinese students. All 3 scales were associated with an elevated odds ratio for diminished self-rated health, and the effect was strongest for the effort–reward ratio, as predicted by the theory. Sex and grade differences were also observed.Conclusions: The ERI questionnaire is a valid instrument for identifying sources of Stressful Experience, in terms of effort–reward imbalance, among adolescents in school settings.
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work stress and health risk behavior
Scandinavian Journal of Work Environment & Health, 2006Co-Authors: Johannes Siegrist, Andreas RodelAbstract:This contribution discusses current knowledge of associations between psychosocial stress at work and health risk behavior, in particular cigarette smoking, alcohol consumption and overweight, by reviewing findings from major studies in the field published between 1989 and 2006. Psychosocial stress at work is measured by the demand-control model and the effort-reward imbalance model. Health risk behavior was analyzed in the broader context of a health-related Western lifestyle with socially and economically patterned practices of consumption. Overall, the review, based on 46 studies, only modestly supports the hypothesis of a consistent association between work stress and health risk behavior. The relatively strongest relationships have been found with regard to heavy alcohol consumption among men, overweight, and the co-manifestation of several risks. Suggestions for further research are given, and the need to reduce Stressful Experience in the framework of worksite health promotion programs is emphasized.
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reported nonreciprocity of social exchange and depressive symptoms extending the model of effort reward imbalance beyond work
Journal of Psychosomatic Research, 2003Co-Authors: Olaf Von Dem Knesebeck, Johannes SiegristAbstract:Objective: To study associations of Stressful Experience in close social relationships with depressive symptoms a measure of nonreciprocal social exchange in marital, parental and less specific civic roles was developed. This measure aims at extending the model of effort–reward imbalance beyond work. Methods: Data from two national surveys in Germany (n=682) and the United States (n=608) were collected, using probability samples of persons 60 years or older. Reported nonreciprocity was measured by a Likert scale. Psychometric properties are described. Depressive symptoms were measured by the CES-D Scale and relevant covariates were assessed. Results: Logistic regression analysis indicates that the risk of depressive symptoms was about twice as high in elderly men and women reporting nonreciprocity of social exchange compared to nonstressed subjects. Conclusion: Findings provide preliminary evidence of usefulness of a measure that extends the notion of nonreciprocal social exchange beyond working life.
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stress management in bus drivers a pilot study based on the model of effort reward imbalance
International Journal of Stress Management, 1997Co-Authors: Birgit Aust, Richard Peter, Johannes SiegristAbstract:A number of studies documented associations between work stress and elevated morbidity in professional drivers. The model of effort–reward imbalance (ERI) identifies distinct situational and personal characteristcs which elicit chronically Stressful Experience at work in terms of a mismatch between high costs and low gain. Fifty-four highly stressed male inner-city bus drivers (mean age 49,5 ± 5,3) were recruited to participate in a 12 weeks stress management program based on the ERI model (intervention group (IG) n = 26, control group (CG) n = 28). Intervention included relaxation, coping with anger and excessive work commitment (“high need for control”), management of conflicts with superiors, and recommendations for structural changes at work. After 12 weeks, mean level of “need for control,” a critical, health adverse style of coping with job demands, was significantly reduced in IG vs. CG, and this effect persisted after 3 months. In conclusion, a theory-based worksite stress management program in an occupational risk group is feasible and shows beneficial psychological effects.
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adverse health effects of high effort low reward conditions
Journal of Occupational Health Psychology, 1996Co-Authors: Johannes SiegristAbstract:In addition to the person-environment fit model (J. R. French, R. D. Caplan, & R. V. Harrison, 1982) and the demand-control model (R. A. Karasek & T. Theorell, 1990), a third theoretical concept is proposed to assess adverse health effects of Stressful Experience at work: the effort-reward imbalance model. The focus of this model is on reciprocity of exchange in occupational life where high-cost/low-gain conditions are considered particularly Stressful. Variables measuring low reward in terms of low status control (e.g., lack of promotion prospects, job insecurity) in association with high extrinsic (e.g., work pressure) or intrinsic (personal coping pattern, e.g., high need for control) effort independently predict new cardiovascular events in a prospective study on blue-collar men. Furthermore, these variables partly explain prevalence of cardiovascular risk factors (hypertension, atherogenic lipids) in 2 independent studies. Studying adverse health effects of high-effort/low-reward conditions seems well justified, especially in view of recent developments of the labor market.
Jaideep S Bains - One of the best experts on this subject based on the ideXlab platform.
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paraventricular nucleus crh neurons encode stress controllability and regulate defensive behavior selection
Nature Neuroscience, 2020Co-Authors: Nuria Daviu, Tamas Fuzesi, David Rosenegger, Neilen P Rasiah, Tonilee Sterley, Govind Peringod, Jaideep S BainsAbstract:In humans and rodents, the perception of control during Stressful events has lasting behavioral consequences. These consequences are apparent even in situations that are distinct from the stress context, but how the brain links prior Stressful Experience to subsequent behaviors remains poorly understood. By assessing innate defensive behavior in a looming-shadow task, we show that the initiation of an escape response is preceded by an increase in the activity of corticotropin-releasing hormone (CRH) neurons in the paraventricular nucleus (PVN) of the hypothalamus (CRHPVN neurons). This anticipatory increase is sensitive to Stressful stimuli that have high or low levels of outcome control. Specifically, experimental stress with high outcome control increases CRHPVN neuron anticipatory activity, which increases escape behavior in an unrelated context. By contrast, stress with no outcome control prevents the emergence of this anticipatory activity and decreases subsequent escape behavior. These observations indicate that CRHPVN neurons encode stress controllability and contribute to shifts between active and passive innate defensive strategies. Prior Stressful Experience affects subsequent behavior even in different situations. Daviu et al. demonstrate that CRHPVN neurons encode stress controllability and contribute to shifts between active and passive innate defensive strategies.
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paraventricular nucleus crh neurons encode stress controllability and regulate defensive behavior selection
Nature Neuroscience, 2020Co-Authors: Nuria Daviu, Tamas Fuzesi, David Rosenegger, Neilen P Rasiah, Tonilee Sterley, Govind Peringod, Jaideep S BainsAbstract:In humans and rodents, the perception of control during Stressful events has lasting behavioral consequences. These consequences are apparent even in situations that are distinct from the stress context, but how the brain links prior Stressful Experience to subsequent behaviors remains poorly understood. By assessing innate defensive behavior in a looming-shadow task, we show that the initiation of an escape response is preceded by an increase in the activity of corticotropin-releasing hormone (CRH) neurons in the paraventricular nucleus (PVN) of the hypothalamus (CRHPVN neurons). This anticipatory increase is sensitive to Stressful stimuli that have high or low levels of outcome control. Specifically, experimental stress with high outcome control increases CRHPVN neuron anticipatory activity, which increases escape behavior in an unrelated context. By contrast, stress with no outcome control prevents the emergence of this anticipatory activity and decreases subsequent escape behavior. These observations indicate that CRHPVN neurons encode stress controllability and contribute to shifts between active and passive innate defensive strategies.
Xiao-dong Wang - One of the best experts on this subject based on the ideXlab platform.
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Blockade of corticotropin-releasing hormone receptor 1 attenuates early-life stress-induced synaptic abnormalities in the neonatal hippocampus.
Hippocampus, 2014Co-Authors: Xue-mei Liao, Mathias V. Schmidt, Xiaodun Yang, Jiao Jia, Xiao-meng Xie, Xiao-dong WangAbstract:Adult individuals with early Stressful Experience exhibit impaired hippocampal neuronal morphology, synaptic plasticity and cognitive performance. While our knowledge on the persistent effects of early-life stress on hippocampal structure and function and the underlying mechanisms has advanced over the recent years, the molecular basis of the immediate postnatal stress effects on hippocampal development remains to be investigated. Here, we reported that repeated blockade of corticotropin-releasing hormone receptor 1 (CRHR1) ameliorated postnatal stress-induced hippocampal synaptic abnormalities in neonatal mice. Following the stress exposure, pups with fragmented maternal care showed retarded dendritic outgrowth and spine formation in CA3 pyramidal neurons and reduced hippocampal levels of synapse-related proteins. During the stress exposure, repeated blockade of glucocorticoid receptors (GRs) by daily administration of RU486 (100 µg g(-1) ) failed to attenuate postnatal stress-evoked synaptic impairments. Conversely, daily administration of the CRHR1 antagonist antalarmin hydrochloride (20 µg g(-1) ) in stressed pups normalized hippocampal protein levels of synaptophysin, postsynaptic density-95, nectin-1, and nectin-3, but not the N-methyl-d-aspartate receptor subunits NR1 and NR2A. Additionally, GR or CRHR1 antagonism attenuated postnatal stress-induced endocrine alterations but not body growth retardation. Our data indicate that the CRH-CRHR1 system modulates the deleterious effects of early-life stress on dendritic development, spinogenesis, and synapse formation, and that early interventions of this system may prevent stress-induced hippocampal maldevelopment.
Nuria Daviu - One of the best experts on this subject based on the ideXlab platform.
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paraventricular nucleus crh neurons encode stress controllability and regulate defensive behavior selection
Nature Neuroscience, 2020Co-Authors: Nuria Daviu, Tamas Fuzesi, David Rosenegger, Neilen P Rasiah, Tonilee Sterley, Govind Peringod, Jaideep S BainsAbstract:In humans and rodents, the perception of control during Stressful events has lasting behavioral consequences. These consequences are apparent even in situations that are distinct from the stress context, but how the brain links prior Stressful Experience to subsequent behaviors remains poorly understood. By assessing innate defensive behavior in a looming-shadow task, we show that the initiation of an escape response is preceded by an increase in the activity of corticotropin-releasing hormone (CRH) neurons in the paraventricular nucleus (PVN) of the hypothalamus (CRHPVN neurons). This anticipatory increase is sensitive to Stressful stimuli that have high or low levels of outcome control. Specifically, experimental stress with high outcome control increases CRHPVN neuron anticipatory activity, which increases escape behavior in an unrelated context. By contrast, stress with no outcome control prevents the emergence of this anticipatory activity and decreases subsequent escape behavior. These observations indicate that CRHPVN neurons encode stress controllability and contribute to shifts between active and passive innate defensive strategies. Prior Stressful Experience affects subsequent behavior even in different situations. Daviu et al. demonstrate that CRHPVN neurons encode stress controllability and contribute to shifts between active and passive innate defensive strategies.
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paraventricular nucleus crh neurons encode stress controllability and regulate defensive behavior selection
Nature Neuroscience, 2020Co-Authors: Nuria Daviu, Tamas Fuzesi, David Rosenegger, Neilen P Rasiah, Tonilee Sterley, Govind Peringod, Jaideep S BainsAbstract:In humans and rodents, the perception of control during Stressful events has lasting behavioral consequences. These consequences are apparent even in situations that are distinct from the stress context, but how the brain links prior Stressful Experience to subsequent behaviors remains poorly understood. By assessing innate defensive behavior in a looming-shadow task, we show that the initiation of an escape response is preceded by an increase in the activity of corticotropin-releasing hormone (CRH) neurons in the paraventricular nucleus (PVN) of the hypothalamus (CRHPVN neurons). This anticipatory increase is sensitive to Stressful stimuli that have high or low levels of outcome control. Specifically, experimental stress with high outcome control increases CRHPVN neuron anticipatory activity, which increases escape behavior in an unrelated context. By contrast, stress with no outcome control prevents the emergence of this anticipatory activity and decreases subsequent escape behavior. These observations indicate that CRHPVN neurons encode stress controllability and contribute to shifts between active and passive innate defensive strategies.
Xue-mei Liao - One of the best experts on this subject based on the ideXlab platform.
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Blockade of corticotropin-releasing hormone receptor 1 attenuates early-life stress-induced synaptic abnormalities in the neonatal hippocampus.
Hippocampus, 2014Co-Authors: Xue-mei Liao, Mathias V. Schmidt, Xiaodun Yang, Jiao Jia, Xiao-meng Xie, Xiao-dong WangAbstract:Adult individuals with early Stressful Experience exhibit impaired hippocampal neuronal morphology, synaptic plasticity and cognitive performance. While our knowledge on the persistent effects of early-life stress on hippocampal structure and function and the underlying mechanisms has advanced over the recent years, the molecular basis of the immediate postnatal stress effects on hippocampal development remains to be investigated. Here, we reported that repeated blockade of corticotropin-releasing hormone receptor 1 (CRHR1) ameliorated postnatal stress-induced hippocampal synaptic abnormalities in neonatal mice. Following the stress exposure, pups with fragmented maternal care showed retarded dendritic outgrowth and spine formation in CA3 pyramidal neurons and reduced hippocampal levels of synapse-related proteins. During the stress exposure, repeated blockade of glucocorticoid receptors (GRs) by daily administration of RU486 (100 µg g(-1) ) failed to attenuate postnatal stress-evoked synaptic impairments. Conversely, daily administration of the CRHR1 antagonist antalarmin hydrochloride (20 µg g(-1) ) in stressed pups normalized hippocampal protein levels of synaptophysin, postsynaptic density-95, nectin-1, and nectin-3, but not the N-methyl-d-aspartate receptor subunits NR1 and NR2A. Additionally, GR or CRHR1 antagonism attenuated postnatal stress-induced endocrine alterations but not body growth retardation. Our data indicate that the CRH-CRHR1 system modulates the deleterious effects of early-life stress on dendritic development, spinogenesis, and synapse formation, and that early interventions of this system may prevent stress-induced hippocampal maldevelopment.
