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Robert J. Schwartzman - One of the best experts on this subject based on the ideXlab platform.
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Printed in U.S.A. UROLOGICAL SYMPTOMATOLOGY IN PATIENTS WITH REFLEX Sympathetic Dystrophy
2014Co-Authors: Michael B Chancellor, Steven Mandel, Patrick J. Shenot, David A Rivas, Robert J. SchwartzmanAbstract:Purpose: We determined the effect of reflex Sympathetic Dystrophy on lower urinary tract function. Materials and Methods: A total of 20 consecutive patients (16 women and 4 men) with neurologically verified reflex Sympathetic Dystrophy was referred for voiding symp~~s, including urgency, frequency, incontinence and urinary retention. No patient had had VOidIng symptoms before the initial trauma that induced reflex Sympathetic Dystrophy. Evaluation included medical history, physical examination, video urodynamic testing and cystoscopy. Results: Mean patient age was 43.4::!: 10.2 years (range 28 to 58) and mean duration of urological symptoms was 4.9::!: 3.6 years (range 1 to 14). Urodynamic study demonstrated a mean cystometric bladder capacity of 417::!: 182 ml. (range 120 to 700). The urodynamic diagnoses included detrusor hyperreflexia in 8 patients, detrusor areflexia in 8, sensory urgency in 3 and detrusor hyperreflexia with detrusor-external sphincter dyssynergia in 1. In 4 women genuine stress urinary incontinence was also documented urodynamically. Conclusions: Reflex Sympathetic Dystrophy may have a profound effect on detrusor an
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new treatments for reflex Sympathetic Dystrophy
The New England Journal of Medicine, 2000Co-Authors: Robert J. SchwartzmanAbstract:Reflex Sympathetic Dystrophy was first described by Mitchell, in 1864.1 It has been difficult for clinicians to diagnose this disorder because it has many variations, often follows minor injury, and evolves and spreads over time.2,3 There are five main types of symptoms: pain, autonomic dysfunction, edema, a movement disorder, and Dystrophy and atrophy. A new classification of this disorder and a new name, the complex regional pain syndrome, have been proposed in an effort to describe its clinical features more accurately and avoid the implications of the name “reflex Sympathetic Dystrophy.” The role of the Sympathetic nervous system in . . .
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Urological Symptomatology in Patients with Reflex Sympathetic Dystrophy
The Journal of Urology, 1996Co-Authors: Michael B Chancellor, Steven Mandel, Patrick J. Shenot, David A Rivas, Robert J. SchwartzmanAbstract:AbstractPurpose: We determined the effect of reflex Sympathetic Dystrophy on lower urinary tract function.Materials and Methods: A total of 20 consecutive patients (16 women and 4 men) with neurologically verified reflex Sympathetic Dystrophy was referred for voiding symptoms, including urgency, frequency, incontinence and urinary retention. No patient had had voiding symptoms before the initial trauma that induced reflex Sympathetic Dystrophy. Evaluation included medical history, physical examination, video urodynamic testing and cystoscopy.Results: Mean patient age was 43.4 plus/minus 10.2 years (range 28 to 58) and mean duration of urological symptoms was 4.9 plus/minus 3.6 years (range 1 to 14). Urodynamic study demonstrated a mean cystometric bladder capacity of 417 plus/minus 182 ml. (range 120 to 700). The urodynamic diagnoses included detrusor hyperreflexia in 8 patients, detrusor areflexia in 8, sensory urgency in 3 and detrusor hyperreflexia with detrusor-external sphincter dyssynergia in 1. In ...
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Reflex Sympathetic Dystrophy.
Current opinion in neurology and neurosurgery, 1993Co-Authors: Robert J. SchwartzmanAbstract:: Reflex Sympathetic Dystrophy is a progressive illness most often initiated by trauma to a nerve, plexus, or soft tissue. Its five components are pain, edema, autonomic dysfunction, movement disorder, and trophic changes. The illness evolves in stages that progress insidiously over time. The length of time a patient remains in a specific stage is unknown. In any stage of reflex Sympathetic Dystrophy, the symptom complex may be dissociated. Reflex Sympathetic Dystrophy occurs in one part of the body that seems to sensitize a patient so that a succeeding injury may initiate the process in the newly traumatized area. The length of time this sensitization lasts is unknown. Pain is the most disabling and the most difficult aspect of the illness to treat. At least in early stages the pain is Sympathetically maintained, but with time becomes Sympathetically independent. The alpha 1 adrenoreceptor appears to be the peripheral link that, when activated, sensitizes directly or indirectly C-nociceptor fibers. Dynamic mechanoallodynia is mediated by A beta low threshold mechanoreceptors, whereas static primary mechanical hyperalgesia may be mediated by sensitized C-nociceptors. A peripheral afferent C-nociceptor input appears to be necessary to alter the dorsal horn central processing mechanisms to allow for the expression of dynamic mechanoallodynia. This nociceptive barrage could be driven by the Sympathetic efferent outflow or could be Sympathetically independent. The response of immediate early response genes may change the neuropeptide concentration of the dorsal horn. Central sensitization mediated by excitatory amino acids, neuropeptides, and the N-methyl-D-aspartate receptor may be responsible for the severe pain seen in the later stages of the illness.
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Reflex Sympathetic Dystrophy. Occurrence of inflammatory skin lesions in patients with stages II and III disease.
Archives of Dermatology, 1991Co-Authors: Guy F. Webster, Robert J. Schwartzman, Richard A. Jacoby, Robert L. Knobler, Jouni UittoAbstract:• Reflex Sympathetic Dystrophy is a poorly understood syndrome of posttraumatic pain and Sympathetic nervous aberration. We have observed previously unreported cutaneous manifestations of reflex Sympathetic Dystrophy. Seven patients with reflex Sympathetic Dystrophy were referred to our institution because of skin disorders. Three had recurrent ulcerating papules, and two had reticulate hyperpigmentation. Xerosis was common, and cutaneous atrophy was infrequent. Cutaneous ulceration and reticulate hyperpigmentation are previously unappreciated aspects of reflex Sympathetic Dystrophy. Further investigation regarding neural influences on the skin is warranted. ( Arch Dermatol. 1991;127:1541-1544)
Lawrence E. Holder - One of the best experts on this subject based on the ideXlab platform.
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Segmental reflex Sympathetic Dystrophy: Clinical and scintigraphic criteria☆
Journal of Hand Surgery (European Volume), 1993Co-Authors: Samuel C. Kline, Lawrence E. HolderAbstract:Abstract Clinical and scintigraphic criteria are proposed for the diagnosis of segmental reflex Sympathetic Dystrophy. Eight patients met previously described clinical criteria for reflex Sympathetic Dystrophy with involvement limited to only a portion of the hand. The delayed phase of the three-phase radionuclide bone scan was found to be highly sensitive (100%) for this small group of patients. Consecutive bone scans (n = 127) performed during a 6-month period for a variety of upper extremity problems were reviewed, and a segmentally diffuse pattern of tracer uptake was found to be highly specific (98%) for segmental reflex Sympathetic Dystrophy. Recognition and documentation of a more localized form of reflex Sympathetic Dystrophy will allow earlier recognition and treatment, which is an important factor in a successful outcome for managing pain dysfunction disorders.
F Kozin - One of the best experts on this subject based on the ideXlab platform.
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reflex Sympathetic Dystrophy syndrome a review
Clinical and Experimental Rheumatology, 1992Co-Authors: F KozinAbstract:: Reflex Sympathetic Dystrophy syndrome is a serious and potentially disabling condition. Early diagnosis and treatment are essential to control the disorder and restore the patient's quality of life. The cardinal clinical features, radiological changes, etiopathologic advances, and current approaches to proper diagnosis and treatment will be discussed.
Zyluk A - One of the best experts on this subject based on the ideXlab platform.
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Iatrogenic causes of post-traumatic reflex Sympathetic Dystrophy
Wiadomości lekarskie (Warsaw Poland), 1998Co-Authors: Zyluk AAbstract:: It is suspected that some faults in the course of the treatment of traumas such as: reduction of fractures without sufficient anaesthesia, repeated reductions, tightness of casts, immobilization of fingers, ignoring patients' complaints of the pain and swelling about or painful rehabilitation may have effect on the development of the post-traumatic reflex Sympathetic Dystrophy. These opinions have not been confirmed by clinical trials. The incidence of above mentioned factors was analysed retrospectively in 165 patients with post-traumatic reflex Sympathetic Dystrophy within upper extremity and in 86 patients following fracture of distal radius without features of the condition (control group). It was found that the patients with reflex Sympathetic Dystrophy significantly more frequently complained of pain and swelling in the course of the treatment of trauma in the cast and they were subjected more frequently to painful rehabilitation after removing the cast. These factors appeared to have an effect on developing the reflex Sympathetic Dystrophy (comparing with the control group). The role of remaining factors that are often considered as significant in the development of reflex Sympathetic Dystrophy--painful and repeated reductions of fractures as well as immobilization of fingers-was not confirmed.
Samuel C. Kline - One of the best experts on this subject based on the ideXlab platform.
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Segmental reflex Sympathetic Dystrophy: Clinical and scintigraphic criteria☆
Journal of Hand Surgery (European Volume), 1993Co-Authors: Samuel C. Kline, Lawrence E. HolderAbstract:Abstract Clinical and scintigraphic criteria are proposed for the diagnosis of segmental reflex Sympathetic Dystrophy. Eight patients met previously described clinical criteria for reflex Sympathetic Dystrophy with involvement limited to only a portion of the hand. The delayed phase of the three-phase radionuclide bone scan was found to be highly sensitive (100%) for this small group of patients. Consecutive bone scans (n = 127) performed during a 6-month period for a variety of upper extremity problems were reviewed, and a segmentally diffuse pattern of tracer uptake was found to be highly specific (98%) for segmental reflex Sympathetic Dystrophy. Recognition and documentation of a more localized form of reflex Sympathetic Dystrophy will allow earlier recognition and treatment, which is an important factor in a successful outcome for managing pain dysfunction disorders.
