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Calcium Formate

The Experts below are selected from a list of 282 Experts worldwide ranked by ideXlab platform

M A R De Benyacar – 1st expert on this subject based on the ideXlab platform

  • thermal decomposition of copper ii Calcium ii Formate
    IEEE Journal of Solid-state Circuits, 1996
    Co-Authors: A G Leyva, G Polla, P K De Perazzo, H Lanza, M A R De Benyacar

    Abstract:

    Abstract The presence of different stages in the thermal decomposition process of CuCa(HCOO) 4 has been established by means of TGA at different heating rates, X-ray powder diffraction of quenched samples, and DSC methods. During the first stage, decomposition of one of the two copper Formate structural units contained in the unit cell takes place. The presence of CuCa 2 (HCOO) 6 has been detected. Calcium Formate structural units break down at higher temperatures; the last decomposition peak corresponds to the appearance of different Calcium–copper oxides.

Paolo Bosi – 2nd expert on this subject based on the ideXlab platform

  • the influence of fat protection of Calcium Formate on growth and intestinal defence in escherichia coli k88 challenged weanling pigs
    Animal Feed Science and Technology, 2007
    Co-Authors: Paolo Bosi, G Sarli, L Casini, S De Filippi, Paolo Trevisi, Maurizio Mazzoni, G Merialdi

    Abstract:

    Abstract An experiment was conducted to test whether free or fat-protected dietary Calcium Formate improves the growth and health of weanling pigs that may or may not be susceptible to intestinal adhesion of enterotoxigenic Escherichia coli (ETEC), when orally challenged with ETEC. Sixty pigs, weaned at 21 days of age (day 0), were divided into three groups (20 pigs each), balanced for number, litter and live weight, and fed the same base diet but three different 30.5 g/kg supplementations: a control (CO—with diCalcium phosphate, Calcium sulphate, lard and sepiolite); a free Calcium Formate (FF), with this additive, monosodium phosphate and lard; a fat-protected Calcium Formate (PF), with this additive and monosodium phosphate only. The final diets did not differ for total Calcium and phosphorus content. The pigs, individually penned, were orally challenged with 1.5 ml of a 10 10  CFU E. coli K88 suspension on day 2 and slaughtered on day 7 or 8. Data were analysed using analysis of variance with a three-factor design, including diet, block, sensitivity of intestinal villus to ETEC adhesion, and first level interactions. The factor diet never interacted with the others. Compared to the control diet, the Formate supplementations improved growth (P E. coli faecal excretion (P E. coli K88 faecal excretion, and increased villus height in the small intestine (P Fat-protected Calcium Formate did not improve the results obtained with free Formate supplementation. Calcium Formate has a growth-promoting effect in weanling pigs challenged with E. coli K88, independently of their susceptibility to the intestinal adhesion of this strain. Its action seems to be more related to a general control of the total E. coli rather than of E. coli K88. At the same dietary concentration, no particular advantage comes from using the protected Formate instead of the free additive.

  • a continuous dietary supply of free Calcium Formate negatively affects the parietal cell population and gastric rna expression for h k atpase in weaning pigs
    Journal of Nutrition, 2006
    Co-Authors: Paolo Bosi, L Casini, Paolo Trevisi, Maurizio Mazzoni, Sara De Filippi, G Petrosino, Giovanna Lalattacosterbosa

    Abstract:

    : Baby formula acidification can be used to reduce diarrhea. Calcium Formate is a dietary acidifier frequently used in animal weaning diets; it is also a source of available Calcium. Gastric acidification reduces gastrin release and hydrochloric acid (HCl) secretion. To study the medium-term effects on fundic gastric mucosa, we fed weaning pigs control diets or diets supplemented with free or fat-protected Calcium Formate. We evaluated the following: 1) the number of HCl-secreting parietal cells, by immunohistochemistry using an antibody against H(+)/K(+)-ATPase; 2) the number of enteroendocrine cells immunohistochemically stained with chromogranin A (CGA), somatostatin, and histamine (HIS); and 3) the expression of the H(+)/K(+)-ATPase gene, by real-time RT-PCR in the oxyntic mucosa. Cells co-staining for CGA and HIS were defined as enterochromaffin-like (ECL) cells. Pigs fed Calcium Formate had fewer parietal cells and a lower expression of the H(+)/K(+)-ATPase gene than the controls (P < 0.05). This reduction did not occur in pigs fed fat-protected Calcium Formate. Somatostatin immune-reactive cells were also more numerous in pigs fed free Calcium Formate than in controls (P < 0.05). The number of ECL cells was not affected. Using covariance analysis, the number of parietal cells explained part of the differences in the expression of H(+)/K(+)-ATPase gene (positive correlation, r = 0.385, P < 0.01), and excluded the statistical significance of the diet. In the future, the effects on the oxyntic mucosa should be checked when the diet supplemented with Calcium Formate is discontinued. Furthermore, a reduction in the number of parietal cells could impair the absorption of vitamin B-12 due to a reduced secretion of the intrinsic factor by these cells.

G Merialdi – 3rd expert on this subject based on the ideXlab platform

  • the influence of fat protection of Calcium Formate on growth and intestinal defence in escherichia coli k88 challenged weanling pigs
    Animal Feed Science and Technology, 2007
    Co-Authors: Paolo Bosi, G Sarli, L Casini, S De Filippi, Paolo Trevisi, Maurizio Mazzoni, G Merialdi

    Abstract:

    Abstract An experiment was conducted to test whether free or fat-protected dietary Calcium Formate improves the growth and health of weanling pigs that may or may not be susceptible to intestinal adhesion of enterotoxigenic Escherichia coli (ETEC), when orally challenged with ETEC. Sixty pigs, weaned at 21 days of age (day 0), were divided into three groups (20 pigs each), balanced for number, litter and live weight, and fed the same base diet but three different 30.5 g/kg supplementations: a control (CO—with diCalcium phosphate, Calcium sulphate, lard and sepiolite); a free Calcium Formate (FF), with this additive, monosodium phosphate and lard; a fat-protected Calcium Formate (PF), with this additive and monosodium phosphate only. The final diets did not differ for total Calcium and phosphorus content. The pigs, individually penned, were orally challenged with 1.5 ml of a 10 10  CFU E. coli K88 suspension on day 2 and slaughtered on day 7 or 8. Data were analysed using analysis of variance with a three-factor design, including diet, block, sensitivity of intestinal villus to ETEC adhesion, and first level interactions. The factor diet never interacted with the others. Compared to the control diet, the Formate supplementations improved growth (P E. coli faecal excretion (P E. coli K88 faecal excretion, and increased villus height in the small intestine (P Fat-protected Calcium Formate did not improve the results obtained with free Formate supplementation. Calcium Formate has a growth-promoting effect in weanling pigs challenged with E. coli K88, independently of their susceptibility to the intestinal adhesion of this strain. Its action seems to be more related to a general control of the total E. coli rather than of E. coli K88. At the same dietary concentration, no particular advantage comes from using the protected Formate instead of the free additive.