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R. Sandhir - One of the best experts on this subject based on the ideXlab platform.
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Biochemical and morphological perturbations in rat erythrocytes exposed to Ethion: protective effect of vitamin E
Cellular and molecular biology (Noisy-le-Grand France), 2011Co-Authors: Gurjit Kaur Bhatti, Jasvinder Singh Bhatti, R. Kiran, R. SandhirAbstract:Erythrocyte membranes are an excellent model system to study interaction of pro-oxidants with membranes. The aim of the present study is to examine the effect of vitamin E on Ethion-induced biochemical and morphological alterations in erythrocytes. Ethion was administered to the rats orally at a daily dose of 2.7 mg/kg body weight for a period of 7, 14, 21 and 28 days. The results from the present study show that administration of Ethion resulted in oxidative damage to erythrocyte membranes as evident by increased lipid peroxidation and decreased phospholipid content. This was accompanied by decrease in membrane cholesterol levels. In addition, Ethion exposure inhibited the activities of membrane bound enzymes; Na+ K+ ATPase and Mg2+ATPase. Scanning electron micrographs of erythrocytes from animals exposed to Ethion revealed morphological changes. Supplementation of vitamin E (50 mg/kg body weight) to Ethion exposed animals ameliorated the Ethion-induced oxidative stress, restored membrane lipid composition and activity of membrane bound enzymes along with erythrocyte shape. The results clearly demonstrate that Ethion-induced damage involves increase in oxidative stress that results in alterations in erythrocyte membrane structure and function. Furthermore, supplementation with vitamin E reversed Ethion induced alterations suggesting its beneficial role in individuals exposed to Ethion.
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Alterations in Ca2+ homeostasis and oxidative damage induced by Ethion in erythrocytes of Wistar rats: Ameliorative effect of vitamin E
Environmental toxicology and pharmacology, 2011Co-Authors: Gurjit Kaur Bhatti, Jasvinder Singh Bhatti, R. Kiran, R. SandhirAbstract:Organophosphate (OP) insecticides have been reported to induce oxidative stress due to lipid peroxidation and alteration in defense mechanisms. It is known that calcium content in erythrocytes plays a very important in normal physiology of cells. Erythrocytes are a very convenient model to understand the susceptibility of membrane to oxidative damage induced by various xenobiotic compounds. The aim of present study was to investigate the effects of Ethion induced oxidative damage, alterations in membrane bound enzymes and Ca(2+) homeostasis and a possible protective role of vitamin E. Adult male albino rats of Wistar strain were orally administered Ethion and vitamin E daily for 28 days. Animals were randomly divided into four groups: control; Ethion treated (2.7 mg/kgbw/day); vitamin E treated (50mg/kg of bw/day); Ethion+vitamin E treated. The animals were sacrificed after 7, 14, 21 and 28 days. Erythrocyte membranes were prepared and analyzed for protein, lipid peroxidation (LPO) and membrane bound ATPases. Furthermore, Ca(2+) homeostasis as function of time and concentration was evaluated in erythrocytes. The results from the present study show that in vivo administration of Ethion resulted in oxidative damage to erythrocyte membranes as evident by increased lipid peroxidation. The increased LPO following Ethion intoxication was accompanied by significant decrease in the activities of Na(+)/K(+)-ATPase, Mg(2+)-ATPase and Ca(2+)-ATPase and disturbed Ca(2+)homeostasis in erythrocytes. Furthermore, vitamin E treatment had a beneficial effect by decreasing lipid peroxidation; partially restoring activities of membrane bound ATPases and Ca(2+) homeostasis. The present study suggests that Ethion exerts its toxic effect by increasing LPO, altering the activity of membrane bound enzymes and disturbing Ca(2+) homeostasis. Vitamin E treatment ameliorated the toxic effects of Ethion suggesting its role as a potential antioxidant.
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Modulation of Ethion-induced hepatotoxicity and oxidative stress by vitamin E supplementation in male Wistar rats
Pesticide Biochemistry and Physiology, 2010Co-Authors: Gurjit Kaur Bhatti, R. Kiran, R. SandhirAbstract:Organophosphorus insecticides (OPIs) may induce oxidative stress leading to generation of free radicals and alteration in antioxidant system of animals. Many studies reported that enzymatic and non-enzymatic antioxidant may play protective role against OPIs induced toxicity in human and rats. The aim of present study was to investigate the possible protective role of vitamin E on Ethion-induced hepatotoxicity in rats using qualitative, quantitative and biochemical approaches. Adult male albino rats of Wistar strain were randomly divided into four groups; each group consists of six animals. Animals were treated for a period of 28 days. Group I (control group received corn oil); Group II [Ethion treated (2.7 mg/kg bw/day)]; Group III (vitamin E treated (50 mg/kg of bw/day)]; Group IV (Ethion + vitamin E treated). Animals were sacrificed after 7, 14, 21 and 28 days by decapitation and liver tissue was used for the measurement of proteins, lipid peroxidation (LPO), reduced glutathione (GSH) content and activities of antioxidant enzymes such as superoxide dismutase (SOD), catalase (CAT), glutathione peroxidase (GPx) glutathione reductase (GR) and glutathione-S-transferase (GST). Erythrocytes were analyzed for acetyl cholinesterase activity. The result of this study shows that in vivo administration of Ethion caused a significant induction of oxidative damage in liver tissue as evidenced by increased level of LPO and decreased GSH content. Ethion toxicity also led to a significant increase in the activities of SOD, CAT, GPx and GST in liver tissue. In addition, decrease in GR activity was observed in Ethion administered rats compared to control. Histopathological findings revealed that exposure to Ethion caused damage in liver tissue. However, simultaneous supplementation with vitamin E restored these parameters partially. In conclusion, the results of the current study revealed that Ethion-induced toxicity caused lipid peroxidation, alterations in the antioxidant enzymes and histopathological changes in liver. Supplementation of vitamin E exhibited protective effect by inhibiting Ethion-induced toxicity in liver and erythrocytes.
R. S. Sethi - One of the best experts on this subject based on the ideXlab platform.
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Study of Ethion and lipopolysaccharide interaction on lung in a mouse model.
Laboratory animal research, 2020Co-Authors: Geetika Verma, R. S. SethiAbstract:Ethion is an organophosphate used commonly in India despite being banned in many other countries. The present study was designed to study the interaction of Ethion and lipopolysaccharide (LPS) together on lung after single low dose Ethion exposure. Mice (n = 20) were alienated into control and treatment groups (n = 10 each). The treatment group was orally fed Ethion (8 mg/kg/animal/day) dissolved in corn oil. The animals (n = 5 each) from both the groups were challenged with 80 μg Escherichia coli lipopolysaccharide (LPS) intranasally and the remaining animals (n = 5 each) were administered normal saline solution after 24 h. Ethion along with LPS induced lung inflammation as indicated by increased neutrophils and total leukocyte count (TLC) in broncheoalveolar lavage fluid. Ethion induced histomorphological alterations in lung as shown by increased pulmonary inflammation score in histopathology. Real time PCR analysis showed that Ethion followed by LPS resulted significant (p
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study of Ethion and lipopolysaccharide interaction on lung in a mouse model
Laboratory Animal Research, 2020Co-Authors: Geetika Verma, R. S. SethiAbstract:Ethion is an organophosphate used commonly in India despite being banned in many other countries. The present study was designed to study the interaction of Ethion and lipopolysaccharide (LPS) together on lung after single low dose Ethion exposure. Mice (n = 20) were alienated into control and treatment groups (n = 10 each). The treatment group was orally fed Ethion (8 mg/kg/animal/day) dissolved in corn oil. The animals (n = 5 each) from both the groups were challenged with 80 μg Escherichia coli lipopolysaccharide (LPS) intranasally and the remaining animals (n = 5 each) were administered normal saline solution after 24 h. Ethion along with LPS induced lung inflammation as indicated by increased neutrophils and total leukocyte count (TLC) in broncheoalveolar lavage fluid. Ethion induced histomorphological alterations in lung as shown by increased pulmonary inflammation score in histopathology. Real time PCR analysis showed that Ethion followed by LPS resulted significant (p < 0.05) increase in pulmonary Toll-like receptor (TLR)-4 (48.53 fold), interleukin (IL)-1β (7.05 fold) and tumor necrosis factor (TNF)-α (5.74 fold) mRNA expression. LPS co-exposure suggested synergistic effect on TLR4 and TNF-α mRNA expression. Ethion alone or in combination with LPS resulted genotoxicity in blood cells as detected by comet assay. The data suggested single dietary Ethion exposure alone or in conjunction with LPS causes lung inflammation and genotoxicity in blood cells.
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long term exposures to Ethion and endotoxin cause lung inflammation and induce genotoxicity in mice
Cell and Tissue Research, 2019Co-Authors: Geetika Verma, C S Mukhopadhyay, Ramneek Verma, Baljit Singh, R. S. SethiAbstract:Ethion, an organophosphorus pesticide, is used worldwide and has potential for toxicity and inflammation. There are very limited data on the pulmonary and genotoxic effects of Ethion especially when the exposure is combined with lipopolysaccharide. Therefore, we used a mouse model to test the hypothesis that prolonged exposure to Ethion alone or in conjunction with lipopolysaccharide (LPS) will cause lung inflammation and genotoxicity in a mouse model. Swiss albino (n = 30) were divided into a control (n = 10) and two treatment groups (n = 10; each group). The treatment groups were orally administered Ethion (4 or 2 mg/kg/animal/day; n = 10 each) dissolved in corn oil for 90 days. After 90 days of exposure, five animals from each of the groups were challenged with 80 μg Escherichia coli lipopolysaccharide (LPS) intranasally and the remaining five animals with normal saline solution via the same route. Ethion at both dosages induced lung inflammation as indicated by increased (p < 0.05) perivascular and peribronchial accumulation of inflammatory cells along with thickening of the alveolar septal wall. Ethion at 4 mg/kg altered (p < 0.05) the mRNA and protein expression of TLR-9 and IL-1β in the lungs and induced genotoxicity in blood cells as determined by single cell gel electrophoresis (Comet assay). Further, both dosages of Ethion in combination with E. coli LPS caused genotoxicity and increased (p < 0.05) pulmonary expression of TLR-4, TLR-9 and IL-1β. The data taken together suggest Ethion induces lung inflammation and interaction between Ethion and LPS increases genotoxicity in blood cells.
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Long-term exposures to Ethion and endotoxin cause lung inflammation and induce genotoxicity in mice.
Cell and tissue research, 2018Co-Authors: Geetika Verma, Ramneek Verma, Baljit Singh, Chandra Sekhar Mukhopadhyay, R. S. SethiAbstract:Ethion, an organophosphorus pesticide, is used worldwide and has potential for toxicity and inflammation. There are very limited data on the pulmonary and genotoxic effects of Ethion especially when the exposure is combined with lipopolysaccharide. Therefore, we used a mouse model to test the hypothesis that prolonged exposure to Ethion alone or in conjunction with lipopolysaccharide (LPS) will cause lung inflammation and genotoxicity in a mouse model. Swiss albino (n = 30) were divided into a control (n = 10) and two treatment groups (n = 10; each group). The treatment groups were orally administered Ethion (4 or 2 mg/kg/animal/day; n = 10 each) dissolved in corn oil for 90 days. After 90 days of exposure, five animals from each of the groups were challenged with 80 μg Escherichia coli lipopolysaccharide (LPS) intranasally and the remaining five animals with normal saline solution via the same route. Ethion at both dosages induced lung inflammation as indicated by increased (p
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Acute Ethion Exposure Alters Expression of TLR 9 in Lungs of Mice
Indian Journal of Veterinary Anatomy, 2016Co-Authors: Geetika Verma, Ramneek Ramneek, Chandra Sekhar Mukhopadhyay, R. S. SethiAbstract:The present study was conducted to investigate the expression of Toll like receptor (TLR) 9 in lungs of mice following acute Ethion exposure with or without lipopolysaccharide (LPS). The animals were divided into two groups viz. treatment and control (n=10; each group). The treatment group-was administered Ethion @8 mg kg-1 body weight/day (1/5LD50) orally for 24 hours while control was fed corn oil. At the end of exposure, half animals from both groups were challenged with LPS @ 80μg/animal and other half with normal saline solution.The lung samples were collected in paraformaldehyde for immunohistochemistry and RNA later solution for quantitative real time PCR (qPCR) analysis. LPS resulted strong immunopositive TLR9 reaction in septal and airways epithelial cells. Further, acute exposure to Ethion alone or in combination with LPS showed intense immunopositive expression of TLR9.The qPCR analysis revealed that LPS caused 2.76 folds increase in the expression of TLR9 mRNA. Ethion exposure further increased the expression to 6.69 folds. However, Ethion followed by LPS resulted 9.52 folds increase in TLR9 mRNA expression. It is the first data on the expression TLR9 following acute exposure to Ethion suggesting that Ethion alone or in combination with LPS increases the expression of TLR9 mRNAat protein and mRNA levels.
M S Dhooria - One of the best experts on this subject based on the ideXlab platform.
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Relative efficacy of different pesticides against red spider mite, Tetranychus cinnabarinus (Boisd.) (Acari: Tetranychidae)
Journal of research, 2004Co-Authors: Kamal Deep, M S DhooriaAbstract:The relative efficacy of eight pesticides namely diafenthiuron, dicofol, dimethoate, Ethion, monocrotophos, oxydemeton methyl, propargite and sulphur studied against different developmental stages of red spider mite, Tetranychus cinnabarinus (Boisd.) through bioassays indicated that diafenthiuron, propargite, dicofol, monocrotophos, Ethion and oxydemeton methyl were found respectively 1402.38, 361.35, 198.32, 20.31, 5.21 and 2.13 times more toxic than dimethoate against eggs. Sulphur did not show any ovicidal action. These pesticides and sulphur were more toxic than dimethoate against both the larval and nymphal stages of the mite. Regarding adulticidal action-diafenthiuron, propargite, dicofol, monocrotophos, Ethion, oxydemeton methyl and sulphur were found respectively 516.43, 162.58, 60.45, 28.75, 6.76, 1.87 and 1.45 times more toxic than dimethoate. Diafenthiuron proved to be the most effective pesticide against different stages of the mite followed by propargite and dicofol. However, oxydemeton methyl and dimethoate were the least effective. Rest of the pesticides were moderate in their response.
Gurjit Kaur Bhatti - One of the best experts on this subject based on the ideXlab platform.
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Biochemical and morphological perturbations in rat erythrocytes exposed to Ethion: protective effect of vitamin E
Cellular and molecular biology (Noisy-le-Grand France), 2011Co-Authors: Gurjit Kaur Bhatti, Jasvinder Singh Bhatti, R. Kiran, R. SandhirAbstract:Erythrocyte membranes are an excellent model system to study interaction of pro-oxidants with membranes. The aim of the present study is to examine the effect of vitamin E on Ethion-induced biochemical and morphological alterations in erythrocytes. Ethion was administered to the rats orally at a daily dose of 2.7 mg/kg body weight for a period of 7, 14, 21 and 28 days. The results from the present study show that administration of Ethion resulted in oxidative damage to erythrocyte membranes as evident by increased lipid peroxidation and decreased phospholipid content. This was accompanied by decrease in membrane cholesterol levels. In addition, Ethion exposure inhibited the activities of membrane bound enzymes; Na+ K+ ATPase and Mg2+ATPase. Scanning electron micrographs of erythrocytes from animals exposed to Ethion revealed morphological changes. Supplementation of vitamin E (50 mg/kg body weight) to Ethion exposed animals ameliorated the Ethion-induced oxidative stress, restored membrane lipid composition and activity of membrane bound enzymes along with erythrocyte shape. The results clearly demonstrate that Ethion-induced damage involves increase in oxidative stress that results in alterations in erythrocyte membrane structure and function. Furthermore, supplementation with vitamin E reversed Ethion induced alterations suggesting its beneficial role in individuals exposed to Ethion.
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Alterations in Ca2+ homeostasis and oxidative damage induced by Ethion in erythrocytes of Wistar rats: Ameliorative effect of vitamin E
Environmental toxicology and pharmacology, 2011Co-Authors: Gurjit Kaur Bhatti, Jasvinder Singh Bhatti, R. Kiran, R. SandhirAbstract:Organophosphate (OP) insecticides have been reported to induce oxidative stress due to lipid peroxidation and alteration in defense mechanisms. It is known that calcium content in erythrocytes plays a very important in normal physiology of cells. Erythrocytes are a very convenient model to understand the susceptibility of membrane to oxidative damage induced by various xenobiotic compounds. The aim of present study was to investigate the effects of Ethion induced oxidative damage, alterations in membrane bound enzymes and Ca(2+) homeostasis and a possible protective role of vitamin E. Adult male albino rats of Wistar strain were orally administered Ethion and vitamin E daily for 28 days. Animals were randomly divided into four groups: control; Ethion treated (2.7 mg/kgbw/day); vitamin E treated (50mg/kg of bw/day); Ethion+vitamin E treated. The animals were sacrificed after 7, 14, 21 and 28 days. Erythrocyte membranes were prepared and analyzed for protein, lipid peroxidation (LPO) and membrane bound ATPases. Furthermore, Ca(2+) homeostasis as function of time and concentration was evaluated in erythrocytes. The results from the present study show that in vivo administration of Ethion resulted in oxidative damage to erythrocyte membranes as evident by increased lipid peroxidation. The increased LPO following Ethion intoxication was accompanied by significant decrease in the activities of Na(+)/K(+)-ATPase, Mg(2+)-ATPase and Ca(2+)-ATPase and disturbed Ca(2+)homeostasis in erythrocytes. Furthermore, vitamin E treatment had a beneficial effect by decreasing lipid peroxidation; partially restoring activities of membrane bound ATPases and Ca(2+) homeostasis. The present study suggests that Ethion exerts its toxic effect by increasing LPO, altering the activity of membrane bound enzymes and disturbing Ca(2+) homeostasis. Vitamin E treatment ameliorated the toxic effects of Ethion suggesting its role as a potential antioxidant.
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Modulation of Ethion-induced hepatotoxicity and oxidative stress by vitamin E supplementation in male Wistar rats
Pesticide Biochemistry and Physiology, 2010Co-Authors: Gurjit Kaur Bhatti, R. Kiran, R. SandhirAbstract:Organophosphorus insecticides (OPIs) may induce oxidative stress leading to generation of free radicals and alteration in antioxidant system of animals. Many studies reported that enzymatic and non-enzymatic antioxidant may play protective role against OPIs induced toxicity in human and rats. The aim of present study was to investigate the possible protective role of vitamin E on Ethion-induced hepatotoxicity in rats using qualitative, quantitative and biochemical approaches. Adult male albino rats of Wistar strain were randomly divided into four groups; each group consists of six animals. Animals were treated for a period of 28 days. Group I (control group received corn oil); Group II [Ethion treated (2.7 mg/kg bw/day)]; Group III (vitamin E treated (50 mg/kg of bw/day)]; Group IV (Ethion + vitamin E treated). Animals were sacrificed after 7, 14, 21 and 28 days by decapitation and liver tissue was used for the measurement of proteins, lipid peroxidation (LPO), reduced glutathione (GSH) content and activities of antioxidant enzymes such as superoxide dismutase (SOD), catalase (CAT), glutathione peroxidase (GPx) glutathione reductase (GR) and glutathione-S-transferase (GST). Erythrocytes were analyzed for acetyl cholinesterase activity. The result of this study shows that in vivo administration of Ethion caused a significant induction of oxidative damage in liver tissue as evidenced by increased level of LPO and decreased GSH content. Ethion toxicity also led to a significant increase in the activities of SOD, CAT, GPx and GST in liver tissue. In addition, decrease in GR activity was observed in Ethion administered rats compared to control. Histopathological findings revealed that exposure to Ethion caused damage in liver tissue. However, simultaneous supplementation with vitamin E restored these parameters partially. In conclusion, the results of the current study revealed that Ethion-induced toxicity caused lipid peroxidation, alterations in the antioxidant enzymes and histopathological changes in liver. Supplementation of vitamin E exhibited protective effect by inhibiting Ethion-induced toxicity in liver and erythrocytes.
Hassan Abdel-gawad - One of the best experts on this subject based on the ideXlab platform.
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Green process for adsorptive removal of Ethion (O,O,O′,O′-tetraethyl S,S′-methylene bis(phosphorodithioate) from agricultural wastewater using modified surface of orange peel and apricot kernel
The Egyptian Journal of Chemistry, 2020Co-Authors: Reda M. Abdelhameed, Hassan Abdel-gawad, Fathia Mahdy, Bahira HegaziAbstract:Smart absorber with engineered surface, low cost price becomes utmost importance for application in water treatment. Pesticide represents the fatal problem in water contamination due to large quantity misused by the farmers. Ethion is a common contaminated insecticide found in wastewater, so it is really important for removal of Ethion from contaminant water. Here, the adsorption characteristics of Ethion and its degradation products were studied using modified and functionalized low cost adsorbent. The modified agricultural wastes [orange peels (OP) and the outer crust of apricot kernel (AK)] were characterized and analyzed. Physical and chemical depositions of Ethion residues on the surface of absorbers (OP and AK) were utilized.Theoretical calculations/modelling on surfaces of OP and AK were studied using Langmuir and Freundlich isotherm model. The results show that the removal efficiency of Ethion increased when the pH of adsorption system increased then decreased at pH greater than 7. The using of different adsorbent doses found that the 95.5 % Ethion removal was achieved when 16.5 g/L of the outer crust of apricot kernel. The kinetic model shows that the Ethion uptake with the orange peels and apricot kernel adsorbents is a pseudo-second order adsorption.
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Kinetic and equilibrium studies on the removal of ^14C-Ethion residues from wastewater by copper-based metal–organic framework
International Journal of Environmental Science and Technology, 2018Co-Authors: Reda M. Abdelhameed, Hassan Abdel-gawad, C. M. Silva, J. Rocha, B. Hegazi, A. M. S. SilvaAbstract:There are compelling economic and environmental reasons to remove pesticides from wastewater because they are toxic and carcinogenic. The effectiveness of copper-based metal–organic framework (Cu-BTC) for adsorbing the insecticide ^14C-Ethion from wastewater has been studied as function of contact time, adsorbent dosage, temperature and pH. ^14C-Ethion/Cu-BTC isotherms exhibit two plateaus (BET type IV) and are reliably represented by Brunauer–Deming–Deming–Teller and Zhu–Gu models, with deviations of only 1.99 and 3.95%, respectively. The removal curve measured under batch operation is well represented by a pseudo-first-order equation, yielding results equivalent to the theoretical linear driving force model of Glueckauf. At pH 7, 75 mg L^−1 Ethion concentration, 150 min, 25 °C and 0.425 g L^−1 Cu-BTC dose, the sorbent capacity is ca. 122 mg g^−1. Moreover, Cu-BTC has a good stability after six adsorptions cycles. Finally, our results disclose the fundamental understanding of the adsorption mechanism: the Ethion molecule coordinates to two copper(II) atoms across the metal–organic framework channel via the phosphoryl (P–O) group.
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Kinetic and equilibrium studies on the removal of ^14C-Ethion residues from wastewater by copper-based metal–organic framework
International Journal of Environmental Science and Technology, 2018Co-Authors: Reda M. Abdelhameed, Hassan Abdel-gawad, C. M. Silva, J. Rocha, B. Hegazi, A. M. S. SilvaAbstract:There are compelling economic and environmental reasons to remove pesticides from wastewater because they are toxic and carcinogenic. The effectiveness of copper-based metal–organic framework (Cu-BTC) for adsorbing the insecticide ^14C-Ethion from wastewater has been studied as function of contact time, adsorbent dosage, temperature and pH. ^14C-Ethion/Cu-BTC isotherms exhibit two plateaus (BET type IV) and are reliably represented by Brunauer–Deming–Deming–Teller and Zhu–Gu models, with deviations of only 1.99 and 3.95%, respectively. The removal curve measured under batch operation is well represented by a pseudo-first-order equation, yielding results equivalent to the theoretical linear driving force model of Glueckauf. At pH 7, 75 mg L^−1 Ethion concentration, 150 min, 25 °C and 0.425 g L^−1 Cu-BTC dose, the sorbent capacity is ca. 122 mg g^−1. Moreover, Cu-BTC has a good stability after six adsorptions cycles. Finally, our results disclose the fundamental understanding of the adsorption mechanism: the Ethion molecule coordinates to two copper(II) atoms across the metal–organic framework channel via the phosphoryl (P–O) group.
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Kinetic and equilibrium studies on the removal of 14C-Ethion residues from wastewater by copper-based metal–organic framework
International Journal of Environmental Science and Technology, 2017Co-Authors: Reda M. Abdelhameed, Hassan Abdel-gawad, Bahira Hegazi, C. M. Silva, J. Rocha, A. M. S. SilvaAbstract:There are compelling economic and environmental reasons to remove pesticides from wastewater because they are toxic and carcinogenic. The effectiveness of copper-based metal–organic framework (Cu-BTC) for adsorbing the insecticide 14C-Ethion from wastewater has been studied as function of contact time, adsorbent dosage, temperature and pH. 14C-Ethion/Cu-BTC isotherms exhibit two plateaus (BET type IV) and are reliably represented by Brunauer–Deming–Deming–Teller and Zhu–Gu models, with deviations of only 1.99 and 3.95%, respectively. The removal curve measured under batch operation is well represented by a pseudo-first-order equation, yielding results equivalent to the theoretical linear driving force model of Glueckauf. At pH 7, 75 mg L−1 Ethion concentration, 150 min, 25 °C and 0.425 g L−1 Cu-BTC dose, the sorbent capacity is ca. 122 mg g−1. Moreover, Cu-BTC has a good stability after six adsorptions cycles. Finally, our results disclose the fundamental understanding of the adsorption mechanism: the Ethion molecule coordinates to two copper(II) atoms across the metal–organic framework channel via the phosphoryl (P–O) group.
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Cu–BTC@cotton composite: design and removal of Ethion insecticide from water
RSC Advances, 2016Co-Authors: Reda M. Abdelhameed, Hassan Abdel-gawad, Mahmoud El-shahat, Hossam E. EmamAbstract:Pesticide removal from wastewater is of significant general benefit to protect of humans from the effect of pollution. Current study offers excellent material for organophosphate insecticide pollution remediation based on metal–organic frameworks (MOFs). A facile method was used to functionalize cotton fabric by assembly with Cu–BTC MOFs. The so-prepared Cu–BTC@cotton composite was characterized by X-ray diffraction, infrared spectroscopy, elemental analysis, and electron microscope. Cu–BTC was successfully bonded with cotton fabrics through interaction between Cu and cellulose functional groups. Adsorption of Ethion as organophosphorus insecticide onto Cu–BTC@cotton composite was systematically studied. Binding sites of composite represented in cellulose functional groups and Cu of MOF were both linked with Ethion via sulfur. The equilibrium adsorption isotherm proved that the adsorption of Ethion insecticide was fitted well to the Langmuir model. The maximum sorption capacity of Cu–BTC@cotton composite reached 182 mg g−1 and the removal percent of Ethion exceeded 97%. Furthermore, Cu–BTC@cotton composite is very stable and can be easily recycled using a simple organic solvent. After recycling five times, the adsorption efficiency of Cu–BTC@cotton composite was still very good and surpassing 85%. Therefore, Cu–BTC@cotton is a perfect sorbent to remove insecticides from wastewater with excellent efficiency.