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Nouri Neamati - One of the best experts on this subject based on the ideXlab platform.

  • design and synthesis of novel pyrimidone analogues as hiv 1 Integrase Inhibitors
    Bioorganic & Medicinal Chemistry Letters, 2013
    Co-Authors: Shenghui Yu, Nouri Neamati, Tino W. Sanchez, Guisen Zhao
    Abstract:

    Abstract A series of novel pyrimidone analogues have been designed and synthesized as HIV-1 Integrase (IN) Inhibitors. This study demonstrated that introducing a substituent in the N1-position of the pyrimidone scaffold does not significantly influence IN inhibitory activity. Molecular docking studies showed these compounds could occupy the IN active site and form pi–pi interactions with viral DNA nucleotides DC16 and DA17 to displace reactive viral DNA 3′OH and block intasome activity.

  • Synthesis, biological evaluation and 3D-QSAR studies of 3-keto salicylic acid chalcones and related amides as novel HIV-1 Integrase Inhibitors.
    Bioorganic & medicinal chemistry, 2011
    Co-Authors: Horrick Sharma, Nouri Neamati, Shivaputra A. Patil, Tino W. Sanchez, Raymond F. Schinazi, John K. Buolamwini
    Abstract:

    Abstract HIV-1 Integrase is one of the three most important enzymes required for viral replication and is therefore an attractive target for anti retroviral therapy. We herein report the design and synthesis of 3-keto salicylic acid chalcone derivatives as novel HIV-1 Integrase Inhibitors. The most active compound, 5-bromo-2-hydroxy-3-[3-(2,3,6-trichlorophenyl)acryloyl]benzoic acid ( 25 ) was selectively active against Integrase strand transfer, with an IC 50 of 3.7 μM. While most of the compounds exhibited strand transfer selectivity, a few were nonselective, such as 5-bromo-3-[3-(4-bromophenyl)acryloyl]-2-hydroxybenzoic acid ( 15 ), which was active against both 3′-processing and strand transfer with IC 50 values of 11 ± 4 and 5 ± 2 μM, respectively. The compounds also inhibited HIV replication with potencies comparable with their Integrase inhibitory potencies. Thus, 5-bromo-2-hydroxy-3-[3-(2,3,6-trichlorophenyl)acryloyl]benzoic acid ( 25 ) and 5-bromo-3-[3-(4-bromophenyl)acryloyl]-2-hydroxybenzoic acid ( 15 ) inhibited HIV-1 replication with EC 50 values of 7.3 and 8.7 μM, respectively. A PHASE pharmacophore hypothesis was developed and validated by 3D-QSAR, which gave a predictive r 2 of 0.57 for an external test set of ten compounds. Phamacophore derived molecular alignments were used for CoMFA and CoMSIA 3D-QSAR modeling. CoMSIA afforded the best model with q 2 and r 2 values of 0.54 and 0.94, respectively. This model predicted all the ten compounds of the test set within 0.56 log units of the actual pIC 50 values; and can be used to guide the rational design of more potent novel 3-keto salicylic acid Integrase Inhibitors

  • hiv 1 Integrase Inhibitors 2007 2008 update
    ChemInform, 2011
    Co-Authors: Kavya Ramkumar, Erik Serrao, Srinivas Odde, Nouri Neamati
    Abstract:

    In recent years, HIV-1 Integrase (IN) has become an attractive target for designing antiretroviral agents. The first IN inhibitor approved for clinical use, raltegravir, has validated the pharmacological viability of IN Inhibitors and signals the advent of a new generation of antiretroviral drugs. The development of raltegravir and other successful lead IN Inhibitors has also influenced the IN inhibitor design strategy. This has led to the identification of several potent Inhibitors in these last two years. Further, an increased understanding of IN structural biology has opened up novel approaches to inhibiting IN, such as targeting its multimerization or interaction with cellular cofactors. This review covers recent developments in the field of IN inhibitor design from 2007 to 2008.   © 2010 Wiley Periodicals, Inc. Med Res Rev, 30, No. 6, 890–954, 2010

  • Raltegravir, elvitegravir, and metoogravir: the birth of "me-too" HIV-1 Integrase Inhibitors
    Retrovirology, 2009
    Co-Authors: Erik Serrao, Kavya Ramkumar, Srinivas Odde, Nouri Neamati
    Abstract:

    Merck's MK-0518, known as raltegravir, has recently become the first FDA-approved HIV-1 Integrase (IN) inhibitor and has since risen to blockbuster drug status. Much research has in turn been conducted over the last few years aimed at recreating but optimizing the compound's interactions with the protein. Resulting me-too drugs have shown favorable pharmacokinetic properties and appear drug-like but, as expected, most have a highly similar interaction with IN to that of raltegravir. We propose that, based upon conclusions drawn from our docking studies illustrated herein, most of these me-too MK-0518 analogues may experience a low success rate against raltegravir-resistant HIV strains. As HIV has a very high mutational competence, the development of drugs with new mechanisms of inhibitory action and/or new active substituents may be a more successful route to take in the development of second- and third-generation IN Inhibitors.

  • anti infectives clinical progress of hiv 1 Integrase Inhibitors
    Expert Opinion on Emerging Drugs, 2008
    Co-Authors: Laith Q Almawsawi, Rasha I Alsafi, Nouri Neamati
    Abstract:

    Background: HIV-1 Integrase (IN) represents a therapeutically advantageous viral target to treat HIV/AIDS in the clinic. Over a decade of progress in the field has resulted in IN inhibitor chemical classes that display specificity for strand transfer catalysis of the enzyme, thus blocking viral DNA integration into host cell nuclear DNA, an essential step for viral infectivity. Objective: In this manuscript we provide an update on recent HIV-1 IN Inhibitors that have been clinically evaluated, which include MK-0518, MK-2048, GS-9137, GS-9160, GS-9224, GSK-364735, and BMS-707035. The information presented here can aid in the IN drug developmental process. Methods: We have limited the scope of this review to information available on the clinical evaluation of promising strand transfer-specific IN Inhibitors and their potential drug–drug interaction profiles with other antiretroviral agents. Results/conclusion: The development of strand transfer-specific inhibitor classes is an important achievement for the ...

Yves Pommier - One of the best experts on this subject based on the ideXlab platform.

  • selectivity for strand transfer over 3 processing and susceptibility to clinical resistance of hiv 1 Integrase Inhibitors are driven by key enzyme dna interactions in the active site
    Nucleic Acids Research, 2016
    Co-Authors: Mathieu Metifiot, Christophe Marchand, Terrence R Burke, Xue Zhi Zhao, Barry C Johnson, Evgeny Kiselev, Laura Marler, Stephen H Hughes, Yves Pommier
    Abstract:

    : Integrase strand transfer Inhibitors (INSTIs) are highly effective against HIV infections. Co-crystal structures of the prototype foamy virus intasome have shown that all three FDA-approved drugs, raltegravir (RAL), elvitegravir and dolutegravir (DTG), act as interfacial Inhibitors during the strand transfer (ST) integration step. However, these structures give only a partial sense for the limited inhibition of the 3'-processing reaction by INSTIs and how INSTIs can be modified to overcome drug resistance, notably against the G140S-Q148H double mutation. Based on biochemical experiments with modified oligonucleotides, we demonstrate that both the viral DNA +1 and -1 bases, which flank the 3'-processing site, play a critical role for 3'-processing efficiency and inhibition by RAL and DTG. In addition, the G140S-Q148H (SH) mutant Integrase, which has a reduced 3'-processing activity, becomes more active and more resistant to inhibition of 3'-processing by RAL and DTG in the absence of the -1 and +1 bases. Molecular modeling of HIV-1 Integrase, together with biochemical data, indicate that the conserved residue Q146 in the flexible loop of HIV-1 Integrase is critical for productive viral DNA binding through specific contacts with the virus DNA ends in the 3'-processing and ST reactions. The potency of Integrase Inhibitors against 3'-processing and their ability to overcome resistance is discussed.

  • probing chelation motifs in hiv Integrase Inhibitors
    Proceedings of the National Academy of Sciences of the United States of America, 2012
    Co-Authors: Arpita Agrawal, Yves Pommier, Kasthuraiah Maddali, Jamie Desoto, Jessica L Fullagar, Shahrzad Rostami, Douglas D Richman, Seth M Cohen
    Abstract:

    A series of HIV Integrase (HIV-1 IN) Inhibitors were synthesized to evaluate the role of the metal-binding group (MBG) in this class of metalloenzyme Inhibitors. A total of 21 different raltegravir-chelator derivative (RCD) compounds were prepared that differed only in the nature of the MBG. These IN strand-transfer Inhibitors (INSTIs) were evaluated in vitro in cell-free enzyme activity assays, and the in vitro results were further validated in cell culture experiments. All of the active compounds showed selective inhibition of the strand-transfer reaction over 3′-processing, suggesting a common mode of action with raltegravir. The results of the in vitro activity suggest that the nature of the MBG donor atoms, the overall MBG structure, and the specific arrangement of the MBG donor atom triad are essential for obtaining maximal HIV-1 IN inhibition. At least two compounds (RCD-4, RCD-5) containing a hydroxypyrone MBG were found to display superior strand-transfer inhibition when compared to an abbreviated analogue of raltegravir (RCD-1). By isolating and examining the role of the MBG in a series of INSTIs, we have identified a scaffold (hydroxypyrones) that may provide access to a unique class of HIV-1 IN Inhibitors, and may help overcome rising raltegravir resistance.

  • evaluation of hiv 1 Integrase Inhibitors on human primary macrophages using a luciferase based single cycle phenotypic assay
    Journal of Virological Methods, 2010
    Co-Authors: Zuleika Michelini, Roberta Costi, Roberto Di Santo, Roberta Amici, Clementina Maria Galluzzo, Donatella R M Negri, Pasqualina Leone, Roberta Bona, Vincenzo Summa, Yves Pommier
    Abstract:

    Macrophages represent an important site for productive infection of HIV-1 and the evaluation of Integrase (IN) Inhibitors on this cell subset is of fundamental importance. In this report, preclinical evaluation of IN Inhibitors on primary human macrophages was attempted successfully using a 96-well microtiter phenotypic assay developed recently for the evaluation of IN Inhibitors in a cell-based system by taking advantage of HIV-derived lentiviral vectors expressing luciferase. IN Inhibitors were also tested using a lentiviral vector containing an IN with introduced T66I/S153Y mutations, known to affect the activity of azido-group-containing diketo acid (DKA) IN Inhibitors. Utilizing different classes of HIV Integrase Inhibitors against the wild-type IN and the mutant mentioned above, some of the IN Inhibitors used were also active on this particular mutant, suggesting that should HIV-1 develop additional or different mutations to become resistant to such anti-IN drugs, new drugs can be developed with a better resistance profile. This assay provides a standardized method for the preclinical evaluation of the efficacy of IN Inhibitors on wild-type and mutated IN that can be adapted easily for the evaluation of anti-IN activity on IN sequences derived from patients.

  • resistance to Integrase Inhibitors
    Viruses, 2010
    Co-Authors: Mathieu Metifiot, Christophe Marchand, Kasthuraiah Maddali, Yves Pommier
    Abstract:

    Integrase (IN) is a clinically validated target for the treatment of human immunodeficiency virus infections and raltegravir exhibits remarkable clinical activity. The next most advanced IN inhibitor is elvitegravir. However, mutant viruses lead to treatment failure and mutations within the IN coding sequence appear to confer cross-resistance. The characterization of those mutations is critical for the development of second generation IN Inhibitors to overcome resistance. This review focuses on IN resistance based on structural and biochemical data, and on the role of the IN flexible loop i.e., between residues G140-G149 in drug action and resistance.

  • diketoacid genre hiv 1 Integrase Inhibitors containing enantiomeric arylamide functionality
    Bioorganic & Medicinal Chemistry, 2009
    Co-Authors: Xue Zhi Zhao, Christophe Marchand, Yves Pommier, Kasthuraiah Maddali, Terrence R Burke
    Abstract:

    Using our recently disclosed 2,3-dihydro-6,7-dihydroxy-1H-isoindol-1-one and 4,5-dihydroxy-1H-isoindole-1,3(2H)-dione Integrase Inhibitors, we report differential effects on inhibitory potency induced by introduction of an α-chiral center into a key aryl substituent. We show that introduction of the chiral center is uniformly deleterious to binding, with the (R)-enantiomer being more deleterious than the (S)-enantiomer. A greater enantiomeric difference in potency is shown by Inhibitors that have restricted rotation of the aryl ring, with the larger difference being due to poorer potency of the (R)-enantiomer rather than higher potency of the (S)-enantiomer. The potency difference for enantiomers based on the isoindoline-1,3-dione ring system is less than for those derived from the isoindol-1-one ring system. Our findings provide useful information that should aid in understanding molecular binding interactions of DKA-derived IN Inhibitors.

Masahiro Fuji - One of the best experts on this subject based on the ideXlab platform.

  • Carbamoyl Pyridone HIV-1 Integrase Inhibitors 3. A Diastereomeric Approach to Chiral Nonracemic Tricyclic Ring Systems and the Discovery of Dolutegravir (S/GSK1349572) and (S/GSK1265744)
    Journal of medicinal chemistry, 2013
    Co-Authors: Brian A Johns, Takashi Kawasuji, Teruhiko Taishi, David Temelkoff, Hiroshi Yoshida, Akiyama Toshiyuki, Yoshiyuki Taoda, Hitoshi Murai, Ryuichi Kiyama, Masahiro Fuji
    Abstract:

    We report herein the discovery of the human immunodeficiency virus type-1 (HIV-1) Integrase Inhibitors dolutegravir (S/GSK1349572) (3) and S/GSK1265744 (4). These drugs stem from a series of carbamoyl pyridone analogues designed using a two-metal chelation model of the Integrase catalytic active site. Structure-activity studies evolved a tricyclic series of carbamoyl pyridines that demonstrated properties indicative of once-daily dosing and superior potency against resistant viral strains. An inherent hemiaminal ring fusion stereocenter within the tricyclic carbamoyl pyridone scaffold led to a critical substrate controlled diastereoselective synthetic strategy whereby chiral information from small readily available amino alcohols was employed to control relative and absolute stereochemistry of the final drug candidates. Modest to extremely high levels of stereochemical control were observed depending on ring size and position of the stereocenter. This approach resulted in the discovery of 3 and 4, which are currently in clinical development.

  • carbamoyl pyridone hiv 1 Integrase Inhibitors 3 a diastereomeric approach to chiral nonracemic tricyclic ring systems and the discovery of dolutegravir s gsk1349572 and s gsk1265744
    Journal of Medicinal Chemistry, 2013
    Co-Authors: Brian A Johns, Takashi Kawasuji, Teruhiko Taishi, David Temelkoff, Hiroshi Yoshida, Yoshiyuki Taoda, Hitoshi Murai, Ryuichi Kiyama, Toshiyuki Akiyama, Masahiro Fuji
    Abstract:

    We report herein the discovery of the human immunodeficiency virus type-1 (HIV-1) Integrase Inhibitors dolutegravir (S/GSK1349572) (3) and S/GSK1265744 (4). These drugs stem from a series of carbamoyl pyridone analogues designed using a two-metal chelation model of the Integrase catalytic active site. Structure–activity studies evolved a tricyclic series of carbamoyl pyridines that demonstrated properties indicative of once-daily dosing and superior potency against resistant viral strains. An inherent hemiaminal ring fusion stereocenter within the tricyclic carbamoyl pyridone scaffold led to a critical substrate controlled diastereoselective synthetic strategy whereby chiral information from small readily available amino alcohols was employed to control relative and absolute stereochemistry of the final drug candidates. Modest to extremely high levels of stereochemical control were observed depending on ring size and position of the stereocenter. This approach resulted in the discovery of 3 and 4, which a...

Brian A Johns - One of the best experts on this subject based on the ideXlab platform.

  • pyridopyrimidinone Inhibitors of hiv 1 rnase h
    European Journal of Medicinal Chemistry, 2014
    Co-Authors: Emile Johann Velthuisen, Brian A Johns, Peter Gerondelis, Yan Chen, Ke Mou, Wenwen Zhang, John W Seal, Kendra E Hightower, Sonia R Miranda, Kevin K Brown
    Abstract:

    Using a structure based pharmacophore design, a weak inhibitor of RNase H, identified from a small library of two metal binding HIV-1 Integrase Inhibitors, was optimized for potency and physicochemical properties. This manuscript describes the SAR and in vivo DMPK for the pyridopyrimidinone class of Inhibitors.

  • Carbamoyl Pyridone HIV-1 Integrase Inhibitors 3. A Diastereomeric Approach to Chiral Nonracemic Tricyclic Ring Systems and the Discovery of Dolutegravir (S/GSK1349572) and (S/GSK1265744)
    Journal of medicinal chemistry, 2013
    Co-Authors: Brian A Johns, Takashi Kawasuji, Teruhiko Taishi, David Temelkoff, Hiroshi Yoshida, Akiyama Toshiyuki, Yoshiyuki Taoda, Hitoshi Murai, Ryuichi Kiyama, Masahiro Fuji
    Abstract:

    We report herein the discovery of the human immunodeficiency virus type-1 (HIV-1) Integrase Inhibitors dolutegravir (S/GSK1349572) (3) and S/GSK1265744 (4). These drugs stem from a series of carbamoyl pyridone analogues designed using a two-metal chelation model of the Integrase catalytic active site. Structure-activity studies evolved a tricyclic series of carbamoyl pyridines that demonstrated properties indicative of once-daily dosing and superior potency against resistant viral strains. An inherent hemiaminal ring fusion stereocenter within the tricyclic carbamoyl pyridone scaffold led to a critical substrate controlled diastereoselective synthetic strategy whereby chiral information from small readily available amino alcohols was employed to control relative and absolute stereochemistry of the final drug candidates. Modest to extremely high levels of stereochemical control were observed depending on ring size and position of the stereocenter. This approach resulted in the discovery of 3 and 4, which are currently in clinical development.

  • carbamoyl pyridone hiv 1 Integrase Inhibitors 3 a diastereomeric approach to chiral nonracemic tricyclic ring systems and the discovery of dolutegravir s gsk1349572 and s gsk1265744
    Journal of Medicinal Chemistry, 2013
    Co-Authors: Brian A Johns, Takashi Kawasuji, Teruhiko Taishi, David Temelkoff, Hiroshi Yoshida, Yoshiyuki Taoda, Hitoshi Murai, Ryuichi Kiyama, Toshiyuki Akiyama, Masahiro Fuji
    Abstract:

    We report herein the discovery of the human immunodeficiency virus type-1 (HIV-1) Integrase Inhibitors dolutegravir (S/GSK1349572) (3) and S/GSK1265744 (4). These drugs stem from a series of carbamoyl pyridone analogues designed using a two-metal chelation model of the Integrase catalytic active site. Structure–activity studies evolved a tricyclic series of carbamoyl pyridines that demonstrated properties indicative of once-daily dosing and superior potency against resistant viral strains. An inherent hemiaminal ring fusion stereocenter within the tricyclic carbamoyl pyridone scaffold led to a critical substrate controlled diastereoselective synthetic strategy whereby chiral information from small readily available amino alcohols was employed to control relative and absolute stereochemistry of the final drug candidates. Modest to extremely high levels of stereochemical control were observed depending on ring size and position of the stereocenter. This approach resulted in the discovery of 3 and 4, which a...

Myriam Witvrouw - One of the best experts on this subject based on the ideXlab platform.