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Rudolph L. Juliano - One of the best experts on this subject based on the ideXlab platform.

Ramon Eritja - One of the best experts on this subject based on the ideXlab platform.

Kyle Carver - One of the best experts on this subject based on the ideXlab platform.

Sudhir Agrawal - One of the best experts on this subject based on the ideXlab platform.

  • Antisense and/or immunostimulatory oligonucleotide therapeutics.
    Current Cancer Drug Targets, 2001
    Co-Authors: Sudhir Agrawal, Kandimalla Er
    Abstract:

    : Antisense technology, which is based on a simple and rational principle of Watson-Crick complementary base pairing of a short oligonucleotide with the targeted mRNA to downregulate the disease-causing gene product, has progressed tremendously in the last two decades. Antisense Oligonucleotides targeted to a number of cancer-causing genes are being evaluated in human clinical trials. While the first-generation phosphorothioate antisense Oligonucleotides are in clinical trials, a number of factors, including sequence motifs that could lead to unwanted mechanisms of action and side effects, have been identified. The severity of the side effects of first-generation antisense Oligonucleotides is mostly dependent on the presence of certain sequence motifs, such as CpG dinucleotides. A number of second-generation chemical modifications have been proposed to overcome the limitations of the first-generation antisense Oligonucleotides. The safety and efficacy of several second-generation mixed-backbone antisense Oligonucleotides are being evaluated in clinical trials. The immune stimulation affects observed with CpG-containing antisense Oligonucleotides are being exploited as a novel therapeutic modality, with several CpG Oligonucleotides being evaluated in clinical trials. A number of medicinal chemistry studies performed to date suggest that the immunomodulatory activity of CpG Oligonucleotides can be fine-tuned by site-specific incorporation of chemical modifications in order to design disease-specific oligonucleotide therapeutics.

  • Effect of aspirin on protein binding and tissue disposition of oligonucleotide phosphorothioate in rats
    Journal of Drug Targeting, 1998
    Co-Authors: Sudhir Agrawal, Xueshu Zhang, Qiuyin Cai, Ekambar R. Kandimalla, Alisa K. Manning, Zhiwie Jiang, T. Marcel, Ruiwen Zhang
    Abstract:

    AbstractPharmacokinetic studies of phosphorothioate oligodeoxynucleotides (PS-Oligonucleotides) in animals show that following intravenous administration, PS-oligonucleotide clears out rapidly from the plasma and is distributed to majority of the organs. PS-Oligonucleotides are bound to plasma proteins extensively. This study was aimed to determine the effect of aspirin, a commonly used drug, on pharmacokinetics of PS-Oligonucleotides. In the present study, PS-oligonucleotide was administered to rats that had received aspirin by gavage. Pharmacokinetic study shows that if PS-oligonucleotide was administered following aspirin administration in rats, a) plasma pharmacokinetic parameters (t1/2α?, t1/2β, AUC, etc.) had lower values, b) tissue disposition was different, and c) rate and route of elimination was affected in animals compared to rats receiving PS-oligonucleotide alone. This finding suggests that pharmacokinetics of PS-Oligonucleotides can be affected with certain class of drugs, which may have dir...

  • Effect of different chemically modified oligodeoxynucleotides on immune stimulation
    Biochemical Pharmacology, 1996
    Co-Authors: Qiuyan Zhao, Jamal Temsamani, Zhiwei Jiang, Patricia L. Iadarola, Sudhir Agrawal
    Abstract:

    Based on previous studies that certain Oligonucleotides can stimulate cell proliferation and immunoglobulin production, this study was carried out to establish the relationship between the stimulatory effect and the chemical modification of the oligonucleotide. First, the effects of oligonucleotide and analogs on immune stimulation were studied in vitro using murine splenic lymphocytes. Our results show that cell proliferation and immunoglobulin production (IgG and IgM) depend on the sequence and the chemical modification of the oligonucleotide. Phosphorothioate oligodeoxynucleotides displayed a greater stimulatory effect than partially modified phosphorothioate Oligonucleotides. Second, we studied the effects of these chemically modified olionucleotides after injection in mice. Massive splenomegaly and stimulation of cell proliferation were observed with some phosphorothioate Oligonucleotides. These effect were minimized markedly by chimeric and hybrid Oligonucleotides. We also demonstrate that in vitro the effects of Oligonucleotides on murine lymphocytes were unaffected by T cell depletion, suggesting that Oligonucleotides exert their effects mainly on the B cells.

  • Pharmacokinetics of Antisense Oligonucleotides
    Clinical Pharmacokinetics, 1995
    Co-Authors: Sudhir Agrawal, Jamal Temsamani, Wayne Galbraith, Jinyan Tang
    Abstract:

    Antisense Oligonucleotides are promising therapeutic agents for the treatment of life-threatening diseases. Intravenous injection of phosphodiester oligonucleotide analogue (P-oligonucleotide) in monkeys shows that the oligonucleotide is degraded rapidly in the plasma with a half-life of about 5 minutes. Administration of a single dose of the phosphorothioate (S-oligonucleotide) in animals by the intravenous route reveals biphasic plasma elimination. An initial short half-life (0.53 to 0.83 hours) represents distribution out of the plasma compartment and a second long half-life (35 to 50 hours) represents elimination from the body. This elimination half-life was similar when the oligonucleotide was administered subcutaneously. In contrast, methylphosphonate Oligonucleotides have an elimination half-life of 17 minutes in mice. S-Oligonucleotide was distributed into most of organs of rats and mice. Liver and kidney were the 2 organs with highest uptake of the oligonucleotide. The S-oligonucleotide was primarily excreted in urine. Up to 30% was excreted in the first 24 hours. Repeated daily intravenous injections of a 25-mer S-oligonucleotide into rats showed that the concentrations in the plasma are at steady-state during the 8 days’ administration. The data represented here support the potential utility of phosphorothioate and methylphosphonate Oligonucleotides as therapeutic agents in vivo .

  • Pharmacokinetics, biodistribution, and stability of capped oligodeoxynucleotide phosphorothioates in mice.
    Antisense Research and Development, 1993
    Co-Authors: Jamal Temsamani, Jinyan Tang, Abeysinghe Padmapriya, Michael Kubert, Sudhir Agrawal
    Abstract:

    Several end-modified oligodeoxynucleotide phosphorothioates (S-Oligonucleotides) were studied for their pharmacokinetics, biodistribution, excretion, and metabolic stability in vivo after intravenous administration in mice. The overall tissue distribution and excretion patterns of these S-Oligonucleotides were found to be independent of 5' or 3' end modification studied. However, the 3' end modification proved to be of considerable importance with respect to metabolic stability of the oligonucleotide. In the case of uncapped and 5'-capped S-oligonucleotide, only 50% of intact oligonucleotide was recovered out of the total bioavailable concentration in liver and kidney. In contrast, in the case of 3'-capped Oligonucleotides almost all bioavailable concentrations of 3'-capped oligonucleotide was found to be intact in kidney and liver at 24 hr after administration. These results demonstrate that superior pharmaceutical potential can be created by 3'-end modification of oligonucleotide phosphorothioates.

Yogesh S. Sanghvi - One of the best experts on this subject based on the ideXlab platform.